1 - and 80-min poststressor, from oral mucosal transudate.

2  were significantly higher than those of the transudates (2.91+/-8.53), (p<0.001).

3 ral effusions were classified as exudates or transudates according to the Light's criteria.

4 lly significant difference was found between transudate and exudate patients in terms of pleural thic

5                            With oral mucosal transudate and serum samples from 101 human immunodefici

6 hree (26%) of the 128 pleural effusions were transudates and 95 (74%) were exudates.

7  fluids, infection or inflammation, cysts or transudates and nonthyroid tumors.

8  pathogenesis, we propose that the volume of transudate generated at the OD is the critical factor ra

9 his does not necessarily rule out a role for transudated IgA in lung secretions because of "mucosal l

10 ted for distinguishing pleural exudates from transudates included pleural nodules, pleural thickening

11                      In contrast, epicardial transudate levels of adenosine do not change in the pres

12 V testing was carried out using oral mucosal transudate (OMT) tests.

13 osine, as determined from epicardial surface transudates, returned to preinfusion levels within 10 mi

14 mproved abdominal discomfort and disclosed a transudate, suggestive of portal hypertension.