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1 phalomyelitis), Guillain-Barre syndrome, and transverse myelitis).
2 rized by attacks of acute optic neuritis and transverse myelitis.
3 e initially diagnosed as NMOSD or idiopathic transverse myelitis.
4  marker NMO-IgG and its application to acute transverse myelitis.
5 lase, may be useful prognostic indicators in transverse myelitis.
6 ibutes to an evolving understanding of acute transverse myelitis.
7 ers in the setting of a paraneoplastic acute transverse myelitis.
8 cent advances in inflammatory non-infectious transverse myelitis.
9 arre syndrome (GBS), meningoencephalitis, or transverse myelitis.
10 ephalomyelitis, Guillain-Barre syndrome, and transverse myelitis.
11 ephalomyelitis, Guillain-Barre syndrome, and transverse myelitis.
12 e ataxia (4), and other sub-acute syndromes (transverse myelitis (1), opsoclonus myoclonus (1)).
13 ible/confirmed encephalitis, 6 meningitis, 1 transverse myelitis, 1 nonhemorrhagic leukoencephalopath
14 be answered before we truly understand acute transverse myelitis: (1) What are the various triggers f
15  acute disseminated encephalomyelitis (12%), transverse myelitis (12%), and cerebellar ataxia (10%).
16 ), 72.2% had spinal cord lesions (46.3% long transverse myelitis, 36.1% short transverse myelitis), 3
17 (46.3% long transverse myelitis, 36.1% short transverse myelitis), 37.1% satisfied 2010 McDonald crit
18 ad worse outcomes than typical patients with transverse myelitis/acute disseminated encephalomyelitis
19 cond event: 4 of 9 (44%) developed recurrent transverse myelitis and 1 of 9 (11%) developed optic neu
20 ncreased frequency of longitudinal extensive transverse myelitis and a favourable clinical outcome in
21            She then presented with recurrent transverse myelitis and a vesicobullous rash over her ar
22                                 Classically, transverse myelitis and acute disseminated encephalomyel
23  for approaching how we classify subtypes of transverse myelitis and acute disseminated encephalomyel
24 ation from hysterical paraplegia, the second transverse myelitis and its consequences for bladder fun
25 emity paralysis, and was diagnosed as having transverse myelitis and membranous glomerulonephritis se
26 Two patients had initial CNS manifestations: transverse myelitis and meningoencephalitis.
27 entral nervous system (CNS) characterized by transverse myelitis and optic neuritis.
28 ome; 0.1 for anaphylaxis and death; 0.04 for transverse myelitis and pancreatitis; and 0.009 for moto
29 tential immunopathogenic mechanisms in acute transverse myelitis and related inflammatory disorders o
30 nty), 7 (18%) with encephalitis, 3 (8%) with transverse myelitis, and 1 (3%) with newly diagnosed chr
31 comitant acute neuromuscular disease), 2 had transverse myelitis, and 1 had chronic inflammatory demy
32 ons of smallpox, which include encephalitis, transverse myelitis, and acute disseminated encephalomye
33 luding acute disseminated encephalomyelitis, transverse myelitis, and aseptic meningoencephalitis.
34 inated encephalomyelitis, optic neuritis, or transverse myelitis, and is less commonly associated wit
35  absence of Dawson fingers, presence of long transverse myelitis, and presence of periependymal lesio
36  well as rare presentations of encephalitis, transverse myelitis, and seizures.
37 itial diagnoses included multiple sclerosis, transverse myelitis, and unspecified neurodegenerative m
38 isystem inflammatory syndrome, Bell's palsy, transverse myelitis, appendicitis, pulmonary embolism, a
39 -old woman who had relapsing optic neuritis, transverse myelitis, AQP4-IgG seropositivity, and recurr
40              Prognostic factors in pediatric transverse myelitis are reviewed.
41  of optic neuritis, longitudinally extensive transverse myelitis, area postrema attacks and less comm
42 um of disorders associated with inflammatory transverse myelitis, as well as a greater appreciation o
43                        For example, in acute transverse myelitis associated with systemic disease (i.
44 with increased risk of relapse compared with transverse myelitis at onset (hazard ratio [HR], 2.66; 9
45 re were no differences in the frequencies of transverse myelitis, brainstem involvement, cerebellar s
46 isorders (including longitudinally extensive transverse myelitis), contributes to an evolving underst
47  who had an initial longitudinally extensive transverse myelitis, delay to diagnosis/treatment was gr
48                            Optic neuritis or transverse myelitis developed after vomiting onset in 11
49                                        Short transverse myelitis does not exclude consideration of AQ
50 on of novel biomarkers associated with acute transverse myelitis has led to a better understanding of
51                                        Acute transverse myelitis in children, in contrast to adults,
52 ignificant neurologic toxicity consisting of transverse myelitis in five patients, CNS toxicity in th
53                                        Acute transverse myelitis is a group of disorders characterize
54                                        Acute transverse myelitis is a pathogenetically heterogeneous
55                                        Short transverse myelitis is not uncommon in NMOSD and, when i
56           However, nonlongitudinal extensive transverse myelitis lesions were also observed frequentl
57 ative patients with longitudinally extensive transverse myelitis (LETM) behave differently from those
58                     Longitudinally extensive transverse myelitis (LETM) is a frequently devastating c
59 ial presentation of longitudinally extensive transverse myelitis (LETM) predicts relapse of myelitis
60 litis optica (NMO), longitudinally extensive transverse myelitis (LETM), multiple sclerosis with a hi
61                                        Acute transverse myelitis may be an isolated entity or may occ
62 ected, included radiation recall (n = 2) and transverse myelitis (n = 1).
63 taxia (n = 4), and other subacute syndromes (transverse myelitis [n = 1], opsoclonus myoclonus [n = 1
64                                           No transverse myelitis or late toxicities of grade 4 or gre
65 ted encephalomyelitis (ADEM), one episode of transverse myelitis or optic neuritis, multiple sclerosi
66 patterns of weakness and sensory loss due to transverse myelitis or peripheral nerve disease.
67 yelinating event (optic neuritis, incomplete transverse myelitis, or brain-stem or cerebellar syndrom
68 of demyelination (optic neuritis, incomplete transverse myelitis, or brain-stem/cerebellar syndrome)
69  monofocal syndromes such as optic neuritis, transverse myelitis, or isolated brainstem syndromes in
70  characterised by a longitudinally extensive transverse myelitis (p=0.003), more often a complete res
71 on length, total lesion burden and number of transverse myelitis relapses did not correlate with pain
72 r month to 1.4 per month, and admissions for transverse myelitis remained constant at 0.6 per month.
73  optic neuritis and longitudinally extensive transverse myelitis [reported on at least 1 magnetic res
74 ocused on recurrent longitudinally extensive transverse myelitis (rLETM) are lacking.
75  of 20 initially presented with 2 attacks of transverse myelitis (rLETM-onset NMO).
76                                        Short transverse myelitis (STM; <3 vertebral segments) is cons
77 s to be elucidated about the causes of acute transverse myelitis, tantalizing clues as to the potenti
78                          In idiopathic acute transverse myelitis, there is an intraparenchymal or per
79 ephalomyelitis (ADEM), two with ON, one with transverse myelitis (TM) and two with clinically isolate
80 Abs and present with optic neuritis (ON) and transverse myelitis (TM) are diagnosed with NMO and thos
81                                              Transverse myelitis (TM) is an immune-mediated spinal co
82 rts have suggested that vaccines may trigger transverse myelitis (TM) or acute disseminated encephalo
83 y isolated syndromes (CIS), 27 patients with transverse myelitis (TM), 50 patients with human immunod
84 racterised by severe optic neuritis (ON) and transverse myelitis (TM).
85 l optic neuritis (ON); 20% bilateral ON; 15% transverse myelitis (TM); 15% simultaneous TM&ON; 10% Ac
86                                              Transverse myelitis was rare (<5 events in all vaccinate
87                                              Transverse myelitis was the initial diagnosis.
88                     Longitudinally extensive transverse myelitis was the predominant SC lesion patter
89 nts with an initial longitudinally extensive transverse myelitis were excluded (n = 151).