ライフサイエンスコーパス: traumatic injury

1 spiratory illness, nor was there a preceding traumatic injury.

2 iratory distress syndrome early after severe traumatic injury.

3 t are able to regenerate any body part after traumatic injury.

4 y of neurological disorders, including acute traumatic injury.

5 tection and prognostication in patients with traumatic injury.

6 nts for the treatment of tissue ischemia and traumatic injury.

7 of 20.5 million yielded 7080 cases of severe traumatic injury.

8 ne cataract surgery, and 10.4% had sustained traumatic injury.

9 d with chronic health conditions following a traumatic injury.

10 ne is to stimulate tissue regeneration after traumatic injury.

11 severe critical illnesses such as sepsis and traumatic injury.

12 tality in patients who suffered severe blunt traumatic injury.

13 educed CSPG to enhance CNS axon growth after traumatic injury.

14 ronal turnover and neuronal replacement upon traumatic injury.

15 n outgrowth, severely limiting recovery from traumatic injury.

16 ratory abdominal laparotomy was negative for traumatic injury.

17 ariety of growth related functions following traumatic injury.

18 tary casualties could be applied to civilian traumatic injury.

19 o develop organ dysfunction and sepsis after traumatic injury.

20 prouting'-in the human spinal cord following traumatic injury.

21 em cells into a rat spinal cord 9 days after traumatic injury.

22 es are activated in response to sepsis after traumatic injury.

23 , secondary to meningitis, encephalitis, and traumatic injury.

24 rom rat spinal cord at early times following traumatic injury.

25 bacterial cell wall/membrane components, or traumatic injury.

26 severity of the injury in patients suffering traumatic injury.

27 mon cause of death in each year, followed by traumatic injury.

28 Black adults who had recently experienced a traumatic injury.

29 ntributor to increased risk of unintentional traumatic injury.

30 spitalized for an acute medical emergency or traumatic injury.

31 spitalization for acute medical emergency or traumatic injury.

32 % CI, 1.06-1.21) increased odds of pediatric traumatic injury.

33 munocompromised patients or individuals with traumatic injury.

34 nts suffering from hyperfibrinolysis after a traumatic injury.

35 ndrome and delirium tremens in patients with traumatic injury.

36 ies of cancer or dementia, and admission for traumatic injury.

37 ing novel, noninvasive treatment options for traumatic injury.

38 t from neurodegenerative disease, stroke, or traumatic injury.

39 ons that range from acute coronary events to traumatic injury.

40 ths remain a major source of mortality after traumatic injury.

41 inflammatory conditions, including sepsis or traumatic injury.

42 tions, influence network structure following traumatic injury.

43 uromuscular pathology that was made worse by traumatic injury.

44 candidate biomarkers for survival following traumatic injury.

45 nt changes in the use of resuscitation after traumatic injury.

46 counts for approximately 20% of deaths after traumatic injury.

47 gh death rates and mass casualties with many traumatic injuries.

48 ic stroke and possibly for other ischemic or traumatic injuries.

49 in various neurological disorders, including traumatic injuries.

50 g old RBCs in patients with life-threatening traumatic injuries.

51 e ability of adult CNS axons to extend after traumatic injuries.

52 re, and understanding the pathophysiology of traumatic injuries.

53 al course in a group of patients with severe traumatic injuries.

54 l pain, staging in cancer, and evaluation of traumatic injuries.

55 ts required mechanical ventilation following traumatic injuries.

56 reated for acute medical emergencies but not traumatic injuries.

57 sed interventions to regenerate large scale, traumatic injuries.

58 conditions to neurodegenerative diseases and traumatic injuries.

59 litation is essential for many patients with traumatic injuries.

60 damage and can be reversible in ischemic and traumatic injuries.

61 juries and contusion or occult fracture) for traumatic injuries.

62 transections, spinal cord injuries and brain traumatic injuries.

63 ause of death on battlefield and in civilian traumatic injuries.

64 3 655 older adult patients who experienced a traumatic injury; 1 522 656 (62.1%) were female; 124 396

65 ligible deaths-83 percent from unintentional traumatic injuries, 14 percent from homicide, and the re

66 0, from patients aged 16 years or older with traumatic injury, admitted to the intensive care unit of

67 [SD, 16.2]; men, 197 [61%]; had experienced traumatic injury after a fall, 223 [69%]), 258 completed

68 All adult patients with traumatic injury aged 18 years or older who were admitte

69 ta (IL-1beta) that is up-regulated following traumatic injuries also promotes BBB dysfunction and hyp

70 angioedema and is involved in septic shock, traumatic injury, Alzheimer's disease (AD), and stroke,

71 mmon mechanism of axonal self-destruction in traumatic injuries and degenerative diseases.

72 uld lead to novel regenerative therapies for traumatic injuries and drug targets for chronic degenera

73 It is thought to be present in 10% of all traumatic injuries and in almost 40% of patients who sus

74 this study was to estimate the prevalence of traumatic injuries and injury-related deaths in low-reso

75 Both traumatic injuries and medical illnesses were treated.

76 These can be divided into two categories: traumatic injuries and overuse injuries.

77 copolymers, protects non-neuronal cells from traumatic injuries and rescues hippocampal neurons from

78 ertrophic scars (HTS), frequently seen after traumatic injuries and surgery, remain a major clinical

79 Twelve of these patients had suffered traumatic injuries and underwent a surgical procedure th

80 cluded adult patients who were admitted with traumatic injuries and underwent laparotomy with follow-

81 the prehospital allocation of patients with traumatic injuries and was incorporated in an applicatio

82 o 99 years who had experienced painful acute traumatic injuries and were treated and transported by a

83 btained from 27 patients with major burns or traumatic injury and 18 healthy persons and were studied

84 entral nervous system axons, associated with traumatic injury and demyelinating diseases such as mult

85 ally induced coma for brain protection after traumatic injury and during status epilepticus.

86 e pathologic lesions likely resulting from a traumatic injury and followed by secondary infection at

87 in many neurodegenerative diseases and after traumatic injury and is a self-destructive program indep

88 the nervous system begins within hours after traumatic injury and is characterized primarily by react

89 presence of tissue hypoperfusion and severe traumatic injury and is mediated by activation of the PC

90 inical research on cellular therapeutics for traumatic injury and its sequelae and discuss prospects

91 ased mortality in patients with sepsis after traumatic injury and may represent a novel marker of ris

92 versial for unconscious patients after blunt traumatic injury and negative findings on computed tomog

93 A total of 772 patients who presented with a traumatic injury and required an emergency surgical proc

94 l hemodynamically stable adult patients with traumatic injury and suspected moderate to severe TBI we

95 system is dysregulated in sepsis, shock, and traumatic injury and that interruption or termination of

96 as associated with increased mortality after traumatic injury and this association was primarily obse

97 no treatment or placebo among patients with traumatic injury and traumatic brain injury who were 15

98 ts who presented with a blunt or penetrating traumatic injury and underwent a laparotomy within 5 day

99 he spinal cord of squirrel monkeys following traumatic injuries, and their relationships to function

100 ears: 1 normal middle ear, 1 ear affected by traumatic injury, and 1 ear with cholesteatoma.

101 se in specific subsets of neurologic injury, traumatic injury, and cardiovascular surgery.

102 ty of insults including metabolic disorders, traumatic injury, and exposure to neurotoxins such as vi

103 les in neuronal development and maintenance, traumatic injury, and neurodegeneration.

104 operation to mitigate the effects of stroke, traumatic injury, and other neurological events.

105 dical services (EMS) personnel, did not have traumatic injury, and received attempts at external defi

106 sease; cause disabling symptoms, faints, and traumatic injuries; and substantially reduce quality of

107 Painful traumatic injuries are a common reason for 911 activatio

108 Severe traumatic injuries are a widespread and challenging clin

109 Many survivors of traumatic injuries are affected by mental disorders, whi

110 Pediatric patients with traumatic injuries are common at deployed U.S. military

111 missions after discharge from acute care for traumatic injury are frequent, persist beyond 30 days, a

112 cute respiratory distress syndrome following traumatic injury are substantially higher than previousl

113 intraoperatively from 10 patients with acute traumatic injuries as a result of motor vehicle collisio

114 provides baseline data on the mechanisms of traumatic injuries as well as the sociodemographic facto

115 Using traumatic injury as a model, we systematically investiga

116 to discharge for patients sustaining severe traumatic injury assessed by EMS.

117 years or younger who were admitted following traumatic injury between January 1, 2008, and December 3

118 immediate, early, and late responses to this traumatic injury by characterizing several histologic fe

119 Muscle loss and impairment resulting from traumatic injury can be alleviated by therapies using mu

120 Severe traumatic injury can lead to immune dysfunction that ren

121 acidification in inflammation, ischemia, or traumatic injury can sensitize VR1 to eicosanoids and tr

122 ing pursued for the biomedical management of traumatic injury, cancer, and degeneration.

123 d mortality in the pediatric population, and traumatic injury causes > 50% of all childhood deaths.

124 and local hypoxia that occurs as a result of traumatic injury, cell transplantation, or tumor growth,

125 ust literature that states that the CML is a traumatic injury commonly encountered in physically abus

126 rse gliosis-dependent central nervous system traumatic injury conditions and diseases, and for orphan

127 Traumatic injuries continue to be a significant cause of

128 After surgery or traumatic injury, corneal wound healing can cause a scar

129 al interventions can now be applied to treat traumatic injury, David J Lockey calls for research to d

130 incidence and outcome associated with severe traumatic injury differs across geographic regions of No

131 There were 320,450 (SE = 3,642) traumatic injury discharges from U.S. hospitals with sep

132 ntial for treatment of neurodegeneration and traumatic injuries (e.g., stroke) because this strategy

133 nternally validated on a data set of 372 573 traumatic injury encounters (mean [SD] age = 68.7 [19.3]

134 Widely different traumatic injuries evoke surprisingly similar gene expre

135 Traumatic injury evokes two characteristic forms of foca

136 ransplantation strategies in a wide range of traumatic injuries for which therapeutic intervention is

137 ords of patients aged 65 years or older with traumatic injury from 2018 to 2020 was conducted at a si

138 ites (6 US and 3 Canadian) sustaining severe traumatic injury from April 1, 2006 to March 31, 2007 fo

139 N. animaloris infections had occurred due to traumatic injury from grey seals.

140 r disease, cardiovascular disease, diabetes, traumatic injuries, gastrointestinal diseases, cancers,

141 In the immediate aftermath of traumatic injury, glucocorticoids stimulate excess gluta

142 In this study, severe traumatic injury had a significant association with empl

143 In traumatic injury, haemorrhage is responsible for almost

144 Traumatic injury has a profound immunomodulatory effect

145 bolism influence the normal host response to traumatic injury has not been extensively studied.

146 Racial disparities in survival after traumatic injury have been described.

147 egulatory mechanisms for tissue repair after traumatic injury have developed under strong evolutionar

148 (age >=18 years) with a primary diagnosis of traumatic injury hospitalized more than 2 days.

149 y injury (AKI) is a frequent complication of traumatic injury; however, long-term outcomes such as mo

150 aged 18 years or older who were admitted for traumatic injury, identified using International Statist

151 death caused by demyelinating, ischemic, and traumatic injuries, implying its involvement in a wide s

152 travenous injection of CR2-Crry 1 hour after traumatic injury improved functional outcome and patholo

153 years or older with inpatient admission for traumatic injury in 2014 to 2015.

154 employment and were hospitalized with severe traumatic injury in Canada between January 2008 and Dece

155 tory distress syndrome development following traumatic injury in children is associated with signific

156 n laboratory animals and naturally occurring traumatic injury in humans.

157 of 1,520,599 patients hospitalized following traumatic injury in Massachusetts or New York during the

158 CAP examinations rarely if ever reveal acute traumatic injury in patients who have experienced low-ve

159 findings suggest that, for axons undergoing traumatic injury in response to applied mechanical loads

160 d adverse medical event or overdose or (2) a traumatic injury in the 30 days after surgery.

161 Scar tissue at sites of traumatic injury in the adult central nervous system pre

162 ssion of other trophic factors in vivo after traumatic injury in the adult CNS.

163 of IL-6 in tissue repair processes following traumatic injury in the CNS.

164 The frequency of acute traumatic injury in the CT CAP examinations was also det

165 ty-two respondents (12%) reported at least 1 traumatic injury in the preceding year.

166 n a rat model of brachial plexus avulsion, a traumatic injury in which nerve roots are torn from the

167 experience a unique inflammatory response to traumatic injury in which the ability of alveolar effect

168 r for increased morbidity and mortality with traumatic injuries, in part through inhibition of bone f

169 the visual functional defects observed after traumatic injury, in degenerative diseases such as glauc

170 and important regional differences in severe traumatic injury, incidence, and outcome.

171 Acute painful traumatic injuries including burns.

172 factors, childhood behavioral problems, and traumatic injuries, including experiencing violence, did

173 Expectedly, traumatic injury induced dramatic changes in apparent ge

174 Here, we found that traumatic injury induced recruitment of myeloid progenit

175 Traumatic injury initiates a complex inflammatory respon

176 milar manner to typical penetrating or blunt traumatic injuries, injuries caused by the blast pressur

177 ansplantation strategies for a wide range of traumatic injuries is the determination of a suitable ti

178 Because traumatic injury is a common acute condition with import

179 ability to restore lost body parts following traumatic injury is a fascinating area of biology that c

180 Serious traumatic injury is a leading cause of death and disabil

181 Globally, traumatic injury is a leading cause of suffering and dea

182 Traumatic injury is a major human health problem, with m

183 The development of PTSD symptoms after traumatic injury is associated with a more fragmented pa

184 Traumatic injury is generally considered to have a suppr

185 Mortality and sepsis after a traumatic injury is greater in the elderly than in young

186 t effective at reducing alcohol misuse after traumatic injury is not known.

187 The loss of organ function following traumatic injury is often irreversible and the demand fo

188 Traumatic injury is the leading cause of death in childr

189 A small traumatic injury itself does not lead to recruitment of

190 Severe traumatic injury leads to marked systemic inflammation a

191 long-term pain outcome from musculoskeletal traumatic injuries may be estimated by measures recorded

192 These data suggest that traumatic injury may modify stem cell characteristics th

193 Following traumatic injury, mice deficient in C3 had a significant

194 Using an in vitro traumatic injury model, we examined the effects of mecha

195 is and decreased bleeding times in vivo in a traumatic injury model.

196 rated appendicitis, other perforated viscus, traumatic injuries more than 4 hours old, or intraabdomi

197 (n = 64) or chronic (n = 36) dissection, or traumatic injury (n = 39).

198 aphy (CT) scan showing no acute intracranial traumatic injury (negative head CT scan), yet the short-

199 ed axons are associated with conditions like traumatic injury, neurodegenerative disease, and seizure

200 tuberculosis-related death, one death due to traumatic injury); no adverse events, including deaths,

201 rvations using the Report of Early Childhood Traumatic Injury Observations & Symptoms inventory.

202 Traumatic injuries occur in > 20 million children each y

203 Recovery of neurological function after traumatic injury of the adult mammalian central nervous

204 ell transplantation to promote recovery from traumatic injury of the CNS, focusing on axonal regenera

205 otential of POSTN as a therapeutic agent for traumatic injury of the CNS.

206 nter experience of endograft repair of blunt traumatic injury of the thoracic aorta.

207 d ethnicities [27.7%]) were hospitalized for traumatic injuries, of whom 1339 (1.8%) died.

208 , we examined the effects of stretch-induced traumatic injury on the AMPA subtype of ionotropic gluta

209 dramatic change in the CNS environment after traumatic injury or disease is hemorrhage because of vas

210 ability to restore visual function following traumatic injury or disease-induced degeneration.

211 ul strategy to restore muscle function after traumatic injury or disease.

212 h as those that may be encountered following traumatic injury or during delayed reconstruction/regene

213 mage to the central nervous system caused by traumatic injury or neurological disorders can lead to p

214 ruption of the lymphatic system secondary to traumatic injury or obstruction.

215 the previous 30 days; (2) were admitted for traumatic injury or surgery; (3) had hypercoagulability

216 tions, which may influence outcome following traumatic injury or they have not been fully validated f

217 All patients 18 years suffered a traumatic injury or underwent a tumor resection of the l

218 children of other races and ethnicities with traumatic injuries (OR 1.01; 95% CI, 0.89-1.15).

219 Adult traumatic injury patients (N = 696) at a major urban Lev

220 postoperative cardiac surgery patients, post-traumatic injury patients, and neurologic injury patient

221 and depression screening are recommended for traumatic injury patients, routine screening is still un

222 e study of patients treated by EMS for acute traumatic injuries, patients from racial or ethnic minor

223 The median incidence of EMS-assessed severe traumatic injury per 100,000 population across sites was

224 In murine and porcine models of traumatic injury, PLPs reduced bleeding and facilitated

225 ry severity score, and the presence of blunt traumatic injury, pulmonary contusion, massive transfusi

226 Many patients with moderate-to-severe traumatic injuries receive care at nontrauma hospitals,

227 Information on traumatic injuries recorded between January 1, 2002, and

228 variation in incidence and outcome of severe traumatic injury remain to be analyzed.

229 Burns and other traumatic injuries represent a substantial biomedical bu

230 casualties with severe burns and associated traumatic injuries requires a coordinated interaction of

231 HO) after blast-related extremity injury and traumatic injuries, respectively.

232 addresses the origin and the role of DAM in traumatic injury response.

233 Mechanistically, we found that traumatic injury resulted in increased uncorrelated netw

234 US of the clenched fist in two patients with traumatic injury revealed dislocated but grossly intact

235 atients with tissue hypoperfusion and severe traumatic injury showed a strong activation of the PC wh

236 e CNS at both cognate antigen-containing and traumatic injury sites after intracerebral antigen deliv

237 ion of activated antigen-specific T-cells at traumatic injury sites, in addition to antigen-containin

238 the CNS, including autoimmune inflammation, traumatic injury, stroke, and neurodegenerative diseases

239 We prospectively recorded 92 traumatic injuries sustained by 73 mineclearers working

240 48-hr and 30-day survival for patients with traumatic injuries that require massive transfusion.

241 ts aged 66 years and older who experienced a traumatic injury that resulted in an emergency departmen

242 eposition in the mouse spinal cord following traumatic injury, the role of complement in the developm

243 and their concomitant heightened exposure to traumatic injury, the trauma burden among this patient p

244 taneously or after provoking events, such as traumatic injuries to the pelvis, upper and lower exterm

245 Traumatic injuries to the teeth are among the most serio

246 mple, and effective adjunct to the repair of traumatic injuries to the thoracic aorta.

247 emiology of pediatric patients admitted with traumatic injuries to U.S. combat support hospitals and

248 nrolled 1138 patients recently admitted with traumatic injury to 1 of 4 major trauma hospitals across

249 Traumatic injury to CNS fiber tracts is accompanied by f

250 Traumatic injury to mixed neuronal/glial cultures induce

251 Traumatic injury to one bird's retina was also successfu

252 mical and molecular changes that occur after traumatic injury to reproduce the pathological events as

253 fy the contribution of antigen deposition or traumatic injury to the accumulation of T-cells in the b

254 This review focuses on traumatic injury to the brain and spinal cord, and highl

255 Traumatic injury to the brain is a problem as old as hum

256 Traumatic injury to the brain often manifests itself sym

257 Emerging data indicate that traumatic injury to the brain or spinal cord activates B

258 Traumatic injury to the brain or spinal cord is one of t

259 ically relevant to most patients living with traumatic injury to the brain or spinal cord, very littl

260 as very recently been seen after ischemic or traumatic injury to the central nervous system (CNS), su

261 unction and cell loss following ischemic and traumatic injury to the central nervous system (CNS).

262 Traumatic injury to the central nervous system results i

263 a wide range of pathophysiologies including traumatic injury to the central nervous system, neurodeg

264 a for this study were found to have an acute traumatic injury to the chest, abdomen, or pelvis.

265 tudy of the Magnesium-Ibogaine: the Stanford Traumatic Injury to the CNS protocol (MISTIC), provided

266 Traumatic injury to the CNS results in chronic partial d

267 el species of Lophotrichus in a patient with traumatic injury to the cornea from a dog paw.

268 ndophthalmitis (in 1.3 percent of patients), traumatic injury to the lens (in 0.7 percent), and retin

269 Traumatic injury to the mammalian spinal cord activates

270 , we first demonstrate that a mouse model of traumatic injury to the pediatric brain reproduces many

271 The primary traumatic injury to the spinal cord triggers a cascade o

272 rapeutic potential in the treatment of acute traumatic injury to the spinal cord.

273 We show that traumatic injury to the ventral midline of the embryonic

274 evel decision that affects how patients with traumatic injury (trauma patients) interact with locoreg

275 8 patients (53%) had a traumatic injury (traumatic surgery group) and 7 patient

276 in LDEM to increase astronauts' autonomy in traumatic injury treatment and lower skill competency re

277 lated diseases and acute trauma (e.g., brain traumatic injury) via the implementation of pre- and pos

278 compare hospitalizations for and spending on traumatic injury vs heart failure, pneumonia, stroke, an

279 In this study, a traumatic injury was administered to a mouse model and h

280 The moderate and mild traumatic injury was inflicted by focal laser lesion and

281 patient sample, salivary CRP measured after traumatic injury was related to greater PTSD symptom sev

282 g mouse optic nerve crush as a model for CNS traumatic injury, we performed a detailed analysis of AI

283 The most common traumatic injuries were burns (n=4348, 18.3%), lower lim

284 Hospitalization for traumatic injuries were identified using International C

285 A total of 379 890 US adults with traumatic injury were included; 190 264 patients were in

286 Patients admitted for traumatic injury were significantly more likely to recei

287 leukocyte subpopulations, and the effect of traumatic injury, were also explored.

288 ncluded adult patients (age >=16 years) with traumatic injuries who were transported by ambulance bet

289 caregivers for adults 65 years or older with traumatic injury who were discharged from 1 of 2 level I

290 A scoring tool to identify which adults with traumatic injury will require early laparotomy could hel

291 and validated to identify which adults with traumatic injury will require laparotomy within 2 hours

292 n, Clinical Modification diagnosis codes for traumatic injuries with 1 or more concurrent open abdomi

293 providers to patients with life-threatening traumatic injuries with hemorrhage.

294 mortality when comparing Black children with traumatic injuries with Hispanic children, White childre

295 ine clearance in obtunded adults after blunt traumatic injury with negative results from a well-inter

296 osition of pediatric patients admitted after traumatic injury with SCA vs those without SCA.

297 included 4 781 396 EMS activations for acute traumatic injury, with a median (IQR) patient age of 59

298 Burn injury can be a devastating traumatic injury, with long-term personal and social imp

299 AIS] grades 3-5) undergoing laparotomy after traumatic injury within 6 hours of admission.

300 o 61 years who were hospitalized with severe traumatic injury, working in the 2 years prior to injury