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1 ramyocardial traversal but did not cross the tricuspid valve.
2 of great arteries and Ebstein anomaly of the tricuspid valve.
3 be strongly influenced by the status of the tricuspid valve.
4 atrioventricular valve, and 97 had a single tricuspid valve.
5 that extends posteriorly to the plane of the tricuspid valve.
6 es, good operator technique, and a competent tricuspid valve.
7 by MRSA was evaluated for replacement of the tricuspid valve.
8 red with age- and size-matched controls with tricuspid valves.
9 many currently untreated patients with leaky tricuspid valves.
10 n and morphogenesis of the mature mitral and tricuspid valves.
11 n pulmonary valves, 22 mitral valves, and 21 tricuspid valves.
12 with bicuspid aortic valves than those with tricuspid valves.
13 ater gives rise to the septum and mitral and tricuspid valves.
14 ram abnormalities extended from perivalvular tricuspid valves (5 patients), pulmonic valves (6 patien
15 of tricuspid atresia that includes an absent tricuspid valve, a large ASD, a VSD, an elongated left v
16 almost exclusively into the right atrium and tricuspid valve; a small amount that was refluxed from t
18 lmonary atresia, six with severe obstructive tricuspid valve abnormalities, five with severe tetralog
19 gher clinical success rates according to the Tricuspid Valve Academic Research Consortium at 30 days
22 in the superior and inferior caval veins and tricuspid valve (adjusted r = 0.28-0.55; all P < .01), p
23 rmined by factors in the right ventricle and tricuspid valve and not the timing of or the type of sur
24 More flow dependence was associated with tricuspid valves and the morphologic features characteri
26 septal defects, Ebstein malformation of the tricuspid valve, and perimembranous and muscular ventric
27 cardiographic dataset at baseline revealed a tricuspid valve annular area of 14.1 cm(2), and effectiv
30 ly Feasibility of the Mitralign Percutaneous Tricuspid Valve Annuloplasty System (PTVAS) Also Known a
32 ve surgery, and discuss the emerging role of tricuspid valve annuloplasty with left ventricular assis
33 increasingly supports the use of corrective tricuspid valve annuloplasty, and the growing consensus
34 We aimed to examine the 3D geometry of the tricuspid valve annulus (TVA) in patients with functiona
35 95% confidence interval, 3.5-21.0; P<0.001), tricuspid valve annulus diameter z-score (odds ratio, 1.
36 ices of RV size and function, neo-aortic and tricuspid valve annulus dimensions and function, and aor
37 mined by atrial activation mapping along the tricuspid valve annulus during tachycardia and was furth
41 ns have been few, however, in the therapy of tricuspid valve anomalies, especially Ebstein's malforma
42 f correcting pathological alterations of the tricuspid valve apparatus may lead to more robust repair
43 munication, smaller aortic root size, larger tricuspid valve area z-score, and larger left ventricula
44 ve regurgitation (<5% at each time), indexed tricuspid valve area, and >/=moderate tricuspid valve re
45 he optimal ICD configuration in a paediatric tricuspid valve atresia patient; (3) establish whether t
46 onconduit positions such as in bioprosthetic tricuspid valves, branch pulmonary arteries, aortic and
47 ator leads on the incidence of bioprosthetic tricuspid valve (BTV) regurgitation compared with BTV pa
51 CAD), and those requiring concomitant mitral/tricuspid valve (CMTV) or concomitant ascending aorta re
53 affected by associated intracardiac defects, tricuspid valve competence, and systemic right ventricul
55 racterized by the congenital agenesis of the tricuspid valve connecting the right atrium to the right
56 e damage (Stage 2), pulmonary vasculature or tricuspid valve damage (Stage 3), or right ventricular d
57 ve damage; stage 3, pulmonary vasculature or tricuspid valve damage; and stage 4, right ventricular d
58 ter z score was lower (P<0.001) and the mean tricuspid valve diameter z score was higher in fetuses w
61 nscatheter interventions to treat mitral and tricuspid valve disease are becoming increasingly availa
64 tients who have mitral valve with or without tricuspid valve disease with a significant history of at
65 atients with evidence of moderate or greater tricuspid valve disease, left-sided valve disease, pulmo
66 ole of cross-sectional imaging in mitral and tricuspid valve disease, primarily valvular regurgitatio
70 mitral valve prolapse (OR 2.725; P < .0001), tricuspid valve disorders (OR 2.142; P < .0001), cardiac
71 LP therapy is associated with an increase in tricuspid valve dysfunction through 12 months of follow-
73 Fetuses diagnosed with Ebstein anomaly and tricuspid valve dysplasia from 2005 to 2011 were include
74 y series of fetuses with Ebstein anomaly and tricuspid valve dysplasia, perinatal mortality remained
76 ic data who underwent isolated transcatheter tricuspid valve edge-to-edge repair for significant tric
79 al in 4] and bicuspid in 458); 417 (45%) had tricuspid valves (either absent or minimal commissural f
80 ar-old woman who had received a diagnosis of tricuspid valve endocarditis caused by MRSA was evaluate
82 atrial pressure (RAP) score (comprising the tricuspid valve, foramen ovale, and ductus venosus Doppl
85 TR, comparing them with patients with normal tricuspid valve function and relating annular geometric
87 was not significantly different whether the tricuspid valve had been repaired (4 of 9 [44%]) or surg
89 st 1 A4C video with color Doppler across the tricuspid valve in 2410 of 2462 studies with a sensitivi
90 ients (aortic valve in 5, mitral valve in 9, tricuspid valve in 4, catheter in 2 and pacemaker in 2,
93 epatic shunts stent that migrated across the tricuspid valve in a patient with Child-Pugh category C
94 ion was achieved between 2 and 7 mm from the tricuspid valve in imaging planes containing the AV musc
96 ed normal intracardiac connections, with the tricuspid valve in the normal position and normal size o
98 ) category, with impairment of aortic and/or tricuspid valves in addition to mitral valve damage.
99 cular valve (SAVV) intervention (morphologic tricuspid valve) in congenitally corrected transposition
100 er aortic and pulmonary valve prostheses for tricuspid valve-in-valve implantation (TVIV) within dysf
101 tion of the LP was associated with increased tricuspid valve incompetence (odds ratio, 5.20; P=0.03).
102 5% CI, 30.2%-44.9%]) patients, predominantly tricuspid valve infection (43/177 [24.3%]), with associa
104 ffective, and safe in treating patients with tricuspid valve infective endocarditis refractory to med
107 of the systemic venous portion of the medial tricuspid valve/inferior vena cava isthmus was attempted
109 year later the patient developed mitral and tricuspid valve insufficiency and subsequently underwent
110 t as a result of chamber enlargement, severe tricuspid valve insufficiency resulted in a detectable r
111 se included intraventricular septum defects, tricuspid valve insufficiency, and diaphragm defects, wh
112 ages of the mitral valve (MV), aortic valve, tricuspid valve, interatrial septum, and left atrial app
113 sought to evaluate the impact of concomitant tricuspid valve intervention (TVI) on post-operative TR,
114 t selection in future trials to determine if tricuspid valve intervention improves outcomes in this h
115 7.6% versus 29.3%; P<0.0001), driven by more tricuspid valve intervention in control patients who cro
116 ause mortality, tricuspid valve surgery, and tricuspid valve intervention through 2 years was signifi
117 spid regurgitation and RVD/PH, transcatheter tricuspid valve intervention was associated with high pr
118 evice-based therapies, such as transcatheter tricuspid valve intervention, gene therapy in patients w
120 from any cause (14.8% vs. 12.5%), postindex tricuspid-valve intervention (3.2% vs. 0.6%), and improv
123 t is not known whether the properties of the tricuspid valve isthmus differ from those of the remaind
124 m and superior vena cava, crista terminalis, tricuspid valve isthmus, coronary sinus orifice, membran
126 to the ventricular phenotype, in A-STR, the tricuspid valve leaflet tethering is typically trivial.
127 urred in 4 patients, lead impingement of the tricuspid valve leaflets occurred in 16 patients, and le
128 there is an insufficient adaptive growth of tricuspid valve leaflets that become unable to cover the
132 lenge this paradigm and hypothesize that the tricuspid valve maladapts in those patients rendering th
134 ricuspid valve dysplasia are rare congenital tricuspid valve malformations associated with high perin
137 ricular (RV) pacing are well-known causes of tricuspid valve, mitral valve, and cardiac dysfunction.
138 efine echocardiographic views for evaluating tricuspid valve morphology and function, and discuss ima
142 lead in 7 patients, lead entanglement in the tricuspid valve occurred in 4 patients, lead impingement
143 tricuspid regurgitation (r=0.692; P=0.009), tricuspid valve offset (r=0.583; P=0.004), and tricuspid
145 wed the records of 41 patients who underwent tricuspid valve operation for severe tricuspid regurgita
146 gh this seemed to be predominantly driven by tricuspid valve or pulmonary artery vasculature damage (
147 itral valve or left atrial damage), Stage 3 (tricuspid valve or pulmonary artery vasculature damage),
149 multivalve surgery that did not include the tricuspid valve (OR, 2.1; 95% CI, 1.3 to 3.3), preoperat
150 .7) and multivalve surgery that included the tricuspid valve (OR, 3.7; 95% CI, 2.3 to 6.1) were the s
151 observed in TAD, apical displacement of the tricuspid valve, or other features compared with the gro
152 artery bypass grafting or aortic, mitral or tricuspid valve, or thoracic aorta surgery between Octob
153 ry artery bypass grafting or aortic, mitral, tricuspid valve, or thoracic aorta surgical procedures b
155 propose a new staging system for functional tricuspid valve pathology using 3 parameters that may mo
159 y pressure of more than 60 mm Hg, a previous tricuspid valve procedure, or a cardiovascular implantab
160 itral valve procedures (TMVP), transcatheter tricuspid valve procedures (TTVP), as well as procedural
161 ) as independent predictors while concurrent tricuspid valve procedures (TVP) were not predictors.
162 th safety and performance of a transcatheter tricuspid valve reconstruction system in the treatment o
166 AF) is considered a risk factor for isolated tricuspid valve regurgitation (TR) in the absence of oth
167 Late echocardiographic failure (3 to 4+ tricuspid valve regurgitation [TR]) after TV surgery was
168 lyzes the factors responsible for changes in tricuspid valve regurgitation after pulmonary endarterec
169 /59; 14%) were found to have a minimum of 2+ tricuspid valve regurgitation before hemi-Fontan or Font
170 We report a series of patients with severe tricuspid valve regurgitation due to a permanent pacemak
171 etermine the incidence of moderate to severe tricuspid valve regurgitation in children with hypoplast
174 this study was to explore the full range of tricuspid valve regurgitation velocity (TRV) at rest and
177 during hybrid palliation, moderate to severe tricuspid valve regurgitation, and smaller ascending aor
182 al surgical strategies to improve results of tricuspid valve repair and close surveillance after surg
183 after excluding the 3 patients who underwent tricuspid valve repair as part of their HF procedure.
186 the first-in-human successful transcatheter tricuspid valve repair for severe tricuspid regurgitatio
189 antable cardiac defibrillator, and mitral or tricuspid valve repair or replacement, can be (partially
190 s such as cardiac resynchronization therapy, tricuspid valve repair or replacement, pulmonary artery
191 nalysed in all patients who had an attempted tricuspid valve repair procedure upon femoral vein punct
192 gating safety and performance of the TriClip Tricuspid Valve Repair System in patients with moderate
193 scular Outcomes In Patients Treated With the Tricuspid Valve Repair System Pivotal [TRILUMINATE Pivot
194 scular Outcomes in Patients Treated With the Tricuspid Valve Repair System Pivotal) found that transc
195 scular Outcomes In Patients Treated With the Tricuspid Valve Repair System Pivotal) is an internation
196 scular Outcomes In Patients Treated With the Tricuspid Valve Repair System Pivotal) is the first rand
197 scular Outcomes in Patients Treated with the Tricuspid Valve Repair System Pivotal), tricuspid transc
198 scular Outcomes In Patients Treated With the Tricuspid Valve Repair System Pivotal; NCT03904147).
199 TriClip, a minimally invasive transcatheter tricuspid valve repair system, for reducing tricuspid re
203 al role in guiding the procedure (mitral and tricuspid valve repair, left atrial appendage closure, a
204 ciency and subsequently underwent mitral and tricuspid valve repair, pulmonary valve replacement, and
208 09 per 5 mg/dL increase; P<0.0001), previous tricuspid valve repair/replacement (ORs, 2.01-10.09; P<0
209 o inform a decision about whether to perform tricuspid-valve repair during mitral-valve surgery in pa
212 n (TR) treated with the EVOQUE transcatheter tricuspid valve replacement (TTVR) system plus medical t
213 m valve-related outcomes after transcatheter tricuspid valve replacement (TTVR), including valve func
216 Ninety-seven patients who underwent initial tricuspid valve replacement are included in the present
218 % men; age, 67.5+/-11.3 years) who underwent tricuspid valve replacement for severe tricuspid regurgi
220 002) was reviewed to evaluate the results of tricuspid valve replacement in children <6 years of age.
221 increased mortality and worse outcome after tricuspid valve replacement in patients with severe tric
223 6% bioprosthetic valve; P=0.01) CONCLUSIONS: Tricuspid valve replacement in young children is associa
225 re or greater TR 2:1 to TTVR with the EVOQUE tricuspid valve replacement system plus optimal medical
227 ent successful tricuspid valve operation (22 tricuspid valve replacement), with one perioperative dea
228 TT-VIVR as a viable alternative to surgical tricuspid valve replacement, especially in high-risk pat
230 tality risk associated with reoperations for tricuspid valve replacement, these data suggest favorabl
231 a 2:1 ratio to undergo either transcatheter tricuspid-valve replacement and medical therapy (valve-r
233 evere tricuspid regurgitation, transcatheter tricuspid-valve replacement was superior to medical ther
234 +/-0.4 versus 0.6+/-0.3, P<0.0001), a larger tricuspid valve ring diameter (P<0.0001), and prolonged
235 3 to 3.7]) increased risk of CAD, and to the tricuspid valve (RR, 5.5 [95% CI, 2.0 to 15.1]) and righ
236 tware aided delineation of the RV free wall, tricuspid valve, RV outflow tract, and apex on 3D echo v
238 ocardiographic indices of RV, neoaortic, and tricuspid valve size and function at 14 months, pre-Font
240 odel, KEi(EDV) E/A ratio and 4D flow derived tricuspid valve stroke volume demonstrated independent a
241 all-cause mortality (17.9% versus 17.1%) and tricuspid valve surgery (2.3% versus 4.3%) were similar
242 d conservatively, and 551 underwent isolated tricuspid valve surgery (200 repairs and 351 replacement
243 years; P < .001), were undergoing mitral or tricuspid valve surgery (51% vs 32%, P < .001), or had h
244 In the subset of patients who underwent tricuspid valve surgery (n = 344), a post-operative impr
245 ared between patients who underwent isolated tricuspid valve surgery (repair or replacement) and thos
247 ate of patients being referred for mitral or tricuspid valve surgery after previous cardiac surgery i
249 roperatively in 120 patients who underwent a tricuspid valve surgery and using TTE (A4C) in 66 health
250 ectomy for constrictive pericarditis without tricuspid valve surgery and with pre- and postoperative
252 ry bypass grafting, and/or aortic, mitral or tricuspid valve surgery in Ontario, Canada, between 2008
254 RECENT FINDINGS: The rationale for offering tricuspid valve surgery is based upon an understanding o
255 based on anatomic complexity and history of tricuspid valve surgery or of subpulmonic obstruction.
258 failure symptoms of 2.03 (1.14-3.60), while tricuspid valve surgery was borderline protective with 0
260 oracotomy approach for reoperative mitral or tricuspid valve surgery was used in 62 patients from Jan
261 t study, 371 Ebstein patients that underwent tricuspid valve surgery were divided into 3 groups: norm
262 Y (1217 conservatively managed, 551 isolated tricuspid valve surgery, and 645 transcatheter valve rep
263 s favoring a more aggressive approach toward tricuspid valve surgery, and discuss the emerging role o
265 with isolated severe TR, 171 (5%) underwent tricuspid valve surgery, including 143 (84%) repairs and
266 Finally, operative mortality for isolated tricuspid valve surgery, particularly re-operative surge
267 ion sequence, ejection fraction, concomitant tricuspid valve surgery, type of valve operation, concom
269 y artery bypass grafting; aortic, mitral, or tricuspid valve surgery; ascending aorta surgery without
270 now include echocardiographic parameters of tricuspid valve tenting area, and associated right ventr
271 orm the transcatheter bicuspidization of the tricuspid valve, the Mitralign system was used to place
273 clinical trials for transcatheter mitral or tricuspid valve therapies, trial hospitals took care of
274 rthermore, novel surgical and interventional tricuspid valve treatment options are increasingly appli
278 ntation (TVIV) within dysfunctional surgical tricuspid valve (TV) bioprostheses has been described in
279 assess the remodeling potential of a tubular tricuspid valve (TV) bioprosthesis made of SIS-ECM by ev
282 le electronic device leads to interfere with tricuspid valve (TV) function has gained increasing reco
283 itation (TR), early results of transcatheter tricuspid valve (TV) intervention studies have shown sig
285 egurgitation (TR) with a structurally normal tricuspid valve (TV) may occur secondary to chronic atri
288 cant tricuspid regurgitation (TR) or require tricuspid valve (TV) surgery in the medium term have det
292 clustering approach, we defined atrial TR as tricuspid valve (TV) tenting height <=10 mm, midventricu
293 uspid valves, and in all 12 AR patients with tricuspid valves unassociated with the Marfan syndrome.
294 ciated defects, such as abnormalities of the tricuspid valve, ventricular septal defect, and pulmonar
300 alves (slope 0.21 cm2/100 ml per s) than for tricuspid valves with <10% commissural fusion (slope 0.3