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1 l trigeminovascular neurons (recorded in the trigeminal ganglion).
2 ker-sensitive primary sensory neurons in the trigeminal ganglion.
3 ensory neurons in the ophthalmic lobe of the trigeminal ganglion.
4 acellular recording from single cells in the trigeminal ganglion.
5 placode-derived neurons of the fused amniote trigeminal ganglion.
6 number in either the dorsal root ganglion or trigeminal ganglion.
7 s its activity in the neocortex, retina, and trigeminal ganglion.
8 tosis and increases in neuron numbers in the trigeminal ganglion.
9 ain contribute to the ciliary as well as the trigeminal ganglion.
10 but increased viral titers in the stroma and trigeminal ganglion.
11 a and certain cranial ganglia, including the trigeminal ganglion.
12 presence of both cell types in the embryonic trigeminal ganglion.
13 us sensory neurons, their normal fate in the trigeminal ganglion.
14 y, originating primarily from neurons in the trigeminal ganglion.
15 lly regulated and different from that of the trigeminal ganglion.
16 e ophthalmic and mandibular divisions of the trigeminal ganglion.
17 lly (2 days), HSV-1 was observed only in the trigeminal ganglion.
18 two distinct roles during development of the trigeminal ganglion.
19 h in the retina or for PCD in the retina and trigeminal ganglion.
20 n of BDNF- and NGF-responsive neurons in the trigeminal ganglion.
21 bryos there were normal levels of PCD in the trigeminal ganglion.
22 ensory neurons in the ophthalmic lobe of the trigeminal ganglion.
23 e in blood flow following stimulation of the trigeminal ganglion.
24  and cytokine response gene-2 in the eye and trigeminal ganglion.
25 437-545/animal) were seen in the ipsilateral trigeminal ganglion.
26 ng on the early immune events in the eye and trigeminal ganglion.
27 Brn-3.0 is abundantly expressed in the adult trigeminal ganglion.
28 cripts (LAT) in the latently infected rabbit trigeminal ganglion.
29 eins that directly activate receptors in the trigeminal ganglion.
30 and differentiation into structures like the trigeminal ganglion.
31  responses in hundreds of neurons across the trigeminal ganglion.
32 likely acquired from VZV reactivation in the trigeminal ganglion.
33 xpression of CGRP and its receptor in rhesus trigeminal ganglion.
34 t precocious neuronal differentiation of the trigeminal ganglion.
35 pulations of corneal afferent neurons in the trigeminal ganglion.
36 ed for neuronal nitric oxide synthase in the trigeminal ganglion.
37 ate entirely from the ophthalmic lobe of the trigeminal ganglion.
38 phthalmic and maxillomandibular lobes of the trigeminal ganglion.
39 al crest cells to the ophthalmic lobe of the trigeminal ganglion.
40                               Removal of one trigeminal ganglion allowed axons of the contralateral g
41 s in chick neural crest cells populating the trigeminal ganglion also reduced the frequency of neurog
42 f primary sensory neurons of the ipsilateral trigeminal ganglion also stained positive for the report
43 neurons also received a novel input from the trigeminal ganglion and an overlapping input from a late
44 wholemount explant cultures of embryonic rat trigeminal ganglion and brainstem or in dissociated cell
45  into diverse derivatives, including cornea, trigeminal ganglion and branchial arch cartilage.
46 on of calcitonin gene-related peptide in the trigeminal ganglion and c-Fos in the trigeminal nucleus
47 derived neural crest or placode cells during trigeminal ganglion and corneal development, and after a
48 estern immunoblot analyses were performed on trigeminal ganglion and corneal lysates to determine abu
49 uring the initial stages of advance from the trigeminal ganglion and do not have a net attractive inf
50  are expressed by sensory neurons of the rat trigeminal ganglion and in what combinations these gene
51     Throughout the E10.5-E13.5 interval, the trigeminal ganglion and its targets in the CNS do not ex
52 HSV-1 infection in an area innervated by the trigeminal ganglion and may explain why the presence of
53                          Thus, somatotopy of trigeminal ganglion and nerve organization is only parti
54 e neural correlates of vibrissa resonance in trigeminal ganglion and primary somatosensory cortex (SI
55 lted in loss of the ophthalmic branch of the trigeminal ganglion and reduced corneal innervation.
56 irus replication in the eye and in the local trigeminal ganglion and reduced herpetic blepharitis and
57 icantly reduced CXCL10 levels in the eye and trigeminal ganglion and reduced mononuclear cell infiltr
58  deficient in Magel2, a PWS gene, within the trigeminal ganglion and regions that are anatomically re
59 w responses to electrical stimulation of the trigeminal ganglion and superior sagittal sinus.
60 and nociceptive brain pathways involving the trigeminal ganglion and thalamus.
61              A neural connection between the trigeminal ganglion and the auditory brainstem was inves
62 the dorsoventral axis of the embryonic mouse trigeminal ganglion and thus can be considered trigemina
63 CGRP immunoreactivity (IR) within the feline trigeminal ganglion and trigeminal nucleus of normal adu
64 t contribute to the ophthalmic branch of the trigeminal ganglion and use it, in conjunction with DiI
65 an-amine (BDA) were made into the guinea pig trigeminal ganglion, and anterograde labeling was examin
66 on and activation, extralymphoid sites (eye, trigeminal ganglion, and brain) contained only activated
67 ed and uninjured nerves in the skin, soma in trigeminal ganglion, and central terminals in the spinal
68 the developing retina, olfactory epithelium, trigeminal ganglion, and hair follicles.
69 wed quail nuclei in the proximal part of the trigeminal ganglion, and quail nerves in the pericorneal
70                                          The trigeminal ganglion, another potential source, had no NP
71 als that the regulatory role of Brn3a in the trigeminal ganglion appears to be conserved in more post
72 ensory neurons of the ophthalmic lobe of the trigeminal ganglion are derived from two embryonic cell
73 sumption that primary sensory neurons of the trigeminal ganglion are sensitive to various combination
74 tages of neurogenesis in the embryonic mouse trigeminal ganglion are supported by BDNF whereas most n
75   The sensory neurons of the embryonic mouse trigeminal ganglion are supported in culture by differen
76 genous Wnt signaling activity in the forming trigeminal ganglion, as indicated by Wnt reporter expres
77 ctron microscopy studies of the axons of the trigeminal ganglion at E13.5 revealed that in the p75 mu
78 of naturally occurring neuronal death in the trigeminal ganglion at E14, trigeminal neurons from bcl-
79 ttern of cells that infiltrate the A/J mouse trigeminal ganglion at various times after HSV-1 corneal
80 h in vitro, but thereafter permit or attract trigeminal ganglion axon outgrowth.
81                                        Thus, trigeminal ganglion axons always project to rhombomere (
82 ing embryo, in which the spinal ganglion and trigeminal ganglion-bound iodinated inhibin A.
83 howed quail nuclei in the distal part of the trigeminal ganglion, but no quail nerves in the cornea o
84  a transient rise in IL-6 mRNA levels in the trigeminal ganglion, but not other cytokine transcripts
85 A-, TrkB- and TrkC-expressing neurons in the trigeminal ganglion by activating directly each of these
86 and reduces the latent HSV-1 genome load per trigeminal ganglion by approximately 200-fold.
87 s type 1 (HSV) was introduced into the mouse trigeminal ganglion by stereotaxic injection.
88 , we could detect immunoreactive profiles of trigeminal ganglion cell axons that contained many vesic
89 s are able to form in the virtual absence of trigeminal ganglion cell death and suggest that mechanis
90 ptosis and subsequent ventricle enlargement, trigeminal ganglion cell loss, and abnormal hindbrain ar
91 inal complex established that these branched trigeminal ganglion cells also extended an axon into the
92                                        These trigeminal ganglion cells are unique among primary senso
93        Additionally, we cultured dissociated trigeminal ganglion cells in the presence of NGF, NT-3,
94      However, following infection of primary trigeminal ganglion cells in vitro with a recombinant HS
95  labeling techniques in rats to identify the trigeminal ganglion cells innervating each of these terr
96  can elicit persistent neural adaptations in trigeminal ganglion cells innervating the dura, prominen
97 ations from (1) cross-sectional areas of AEN trigeminal ganglion cells labeled with WGA-HRP, and (2)
98 e used compartmental cultures of dissociated trigeminal ganglion cells to determine the effect of Sem
99             These results indicate that some trigeminal ganglion cells with sensory endings in the na
100 d human embryonic kidney (HEK) cells and rat trigeminal ganglion cells) on the silica surface of a pl
101 nusual projections of primary afferents from trigeminal ganglion cells, and, subsequently, increased
102                                The number of trigeminal ganglion cells, their size distribution under
103 ion to compartmental cultures of dissociated trigeminal ganglion cells.
104 nal nerve (CN V) differentiation and altered trigeminal ganglion (CNgV) cellular composition prefigur
105      The cornea is extensively innervated by trigeminal ganglion cold thermoreceptor neurons expressi
106 igher levels of IFN-alphabeta (533 U) in the trigeminal ganglion compared with nontransgenic mice (70
107 s serve as the site of action for miR-203 in trigeminal ganglion condensation.
108 Following a survival period of 3-7 days, the trigeminal ganglion contained double-labeled, small (11.
109 h neural crest and placode contribute to the trigeminal ganglion, corneal innervation is entirely neu
110                     We hypothesized that the trigeminal ganglion could be one possible site.
111  which resides in the sensory neurons of the trigeminal ganglion, could be stress reactivated to prod
112 iability of the labeled DPANs in dissociated trigeminal ganglion cultures using calcium microfluorome
113  Here, we show that treatment of primary rat trigeminal ganglion cultures with WIN led to dephosphory
114 k HSV-1 reactivation from latency in ex vivo trigeminal ganglion cultures.
115 ral crest or placodal origin, neurons of the trigeminal ganglion derive from both populations.
116 rest- and placode-derived neurons throughout trigeminal ganglion development.
117  neurons from the dorsomedial portion of the trigeminal ganglion (DM-TG), which are largely cutaneous
118 ptor Neuropilin-1 (Npn1) is expressed in the trigeminal ganglion during cornea development.
119 thin the brainstem trigeminal nuclei and the trigeminal ganglion during elongation and branching-arbo
120 e imaging (fMRI) to assess activation in the trigeminal ganglion during innocuous mechanical (brush)
121 V-1-infected cell polypeptide 27 mRNA in the trigeminal ganglion during the acute (day 6 postinfectio
122 nt role in limiting HSV-1 replication in the trigeminal ganglion during the acute stage of infection.
123                                              Trigeminal ganglion electrophysiologic single unit recor
124 nstem or in dissociated cell cultures of the trigeminal ganglion, exogenous supply of NGF leads to ax
125                       We have used the mouse trigeminal ganglion explant model and reverse transcript
126 ctivation phenotype was determined using the trigeminal ganglion explant model.
127 term dissociated cultures, we cultured early trigeminal ganglion explants with and without their targ
128 ex vivo neurite outgrowth and myelination of trigeminal ganglion explants.
129 L-6 expression and HSV-1 reactivation in the trigeminal ganglion following hyperthermic stress of mic
130 ttern of HSV-1 latent infection of the mouse trigeminal ganglion following ocular inoculation with vi
131 he development of neural crest cells and for trigeminal ganglion formation, however, migrating neural
132 tonomously on neural crest cells and promote trigeminal ganglion formation.
133     Single unit activity was recorded in the trigeminal ganglion from 40 mechanosensitive dural affer
134 citonin gene-related peptide (CGRP) from the trigeminal ganglion has been established as a key player
135 ite stage, by which time condensation of the trigeminal ganglion has begun.
136  levels of infectious virus were detected in trigeminal ganglion homogenates from calves infected wit
137 ill be valuable for measuring changes in the trigeminal ganglion in human models of neuropathic pain
138 ter unilateral electrical stimulation of the trigeminal ganglion in mice lacking serotonin 5-HT1B (5-
139 h near complete loss of the ophthalmic (OpV) trigeminal ganglion in the most severe cases after overe
140 med cell death (apoptosis) in neurons of the trigeminal ganglion in vivo and in tissue culture cells
141 resents VZV reactivation, most likely in the trigeminal ganglion, in the absence of clinical herpes z
142 nt to feeding behavior and innervated by the trigeminal ganglion including the lateral periodontium,
143 ensity of the inflammatory infiltrate in the trigeminal ganglion increased until days 12 to 21 p.i.,
144                HSV antigen expression in the trigeminal ganglion (indicative of the viral lytic cycle
145 s study provides the first evidence that the trigeminal ganglion innervates the cochlear nucleus and
146 High-intensity electrical stimulation of the trigeminal ganglion is accompanied by mast cell degranul
147 al pulp innervation by nerve fibers from the trigeminal ganglion is an excellent example of nerve-tar
148                             Among these, the trigeminal ganglion is missing 70% of the normal number
149              Furthermore, we examined if the trigeminal ganglion is protected by the blood-brain barr
150              We also show that Kir4.1 in the trigeminal ganglion is reduced after chronic constrictio
151                                          The trigeminal ganglion is reduced in size and lacks project
152 ensory neurons of the ophthalmic lobe of the trigeminal ganglion, is a useful model of sensory neuron
153                      The organization of the trigeminal ganglion, its somatotopic projections upon th
154 e in IL-6 mRNA and protein expression in the trigeminal ganglion latently infected with HSV-1.
155 st follow-up questionnaire, were higher if a trigeminal-ganglion lesion had been created with radiofr
156 eyed to the somatosensory cortex through the trigeminal ganglion, medullary dorsal horn, and thalamus
157 sumptions: (1) electrical stimulation of the trigeminal ganglion mimics the neurogenic inflammatory p
158                              However, in the trigeminal ganglion, naturally occurring neuronal death
159 a latently infected with VZV and one control trigeminal ganglion negative for VZV DNA that were remov
160 ) to protect and regenerate isolated primary trigeminal ganglion neuronal cells (TGNC).
161 of NGF/TrkA signalling in vivo, the death of trigeminal ganglion neurones occurs independently of BAX
162 o2 expression occurs in approximately 26% of trigeminal ganglion neurons and 30% of corneal afferent
163 er with earlier work demonstrating a loss of trigeminal ganglion neurons and retinal ganglion cells i
164 owever, the mechanoreceptive and nociceptive trigeminal ganglion neurons and the visual sensory retin
165 elated with Cavalpha2delta1 up-regulation in trigeminal ganglion neurons and Vc/C2.
166 del in which dissociated wild-type embryonic trigeminal ganglion neurons are cultured on longitudinal
167 ulture showed that 65% of DiIC18-labeled rat trigeminal ganglion neurons are excited by capsaicin.
168 ing CGRP expression in the migraine-relevant trigeminal ganglion neurons are unknown.
169 rons at early embryonic (E) stages (E6-E12), trigeminal ganglion neurons at midstages (E9-E16), and v
170 er transgene was differentially regulated in trigeminal ganglion neurons depending upon age.
171 he periphery, where dorsal root ganglion and trigeminal ganglion neurons feed pain information into t
172                                              Trigeminal ganglion neurons from adult female Sprague Da
173  and show that this mutation renders DRG and trigeminal ganglion neurons hyperexcitable.
174 1632E renders dorsal root ganglion (DRG) and trigeminal ganglion neurons hyperexcitable.
175 VEGF enhanced neurite elongation in isolated trigeminal ganglion neurons in a dose-dependent manner.
176  made from dissociated guinea-pig nodose and trigeminal ganglion neurons in culture to study second m
177 (HMGA1), was readily detected in a subset of trigeminal ganglion neurons in latently infected calves
178 as no significant change in proliferation of trigeminal ganglion neurons in the noggin transgenic ani
179 hat activation of CGRP receptors on cultured trigeminal ganglion neurons increased endogenous CGRP mR
180              Recordings from primary sensory trigeminal ganglion neurons show that these neurons exhi
181           TRPA1 recorded from cell bodies of trigeminal ganglion neurons showed similar behavior with
182 ive-cycle genes in greater numbers of murine trigeminal ganglion neurons than LAT+ HSV type 1 at earl
183       Capsaicin evokes a membrane current in trigeminal ganglion neurons that is increased substantia
184 excitability and a sustained Na(+) influx in trigeminal ganglion neurons that may underlie pain in th
185          Here we recorded responses in mouse trigeminal ganglion neurons to investigate interactions
186 es have demonstrated that tonic responses of trigeminal ganglion neurons to maintained whisker deflec
187 e expression of key inflammatory proteins in trigeminal ganglion neurons under basal and inflammatory
188                     Compartmental culture of trigeminal ganglion neurons was performed in Campenot de
189                     Compartmental culture of trigeminal ganglion neurons was performed in Campenot de
190       Furthermore, no HSV strain 17-infected trigeminal ganglion neurons were apoptotic, but 0.087% o
191 Rohon-Beard, commissural, primary motor, and trigeminal ganglion neurons were distinctly stained in t
192                                              Trigeminal ganglion neurons were positive for beta-galac
193  selectively expressed in spinal sensory and trigeminal ganglion neurons within the peripheral nervou
194                              In cultured rat trigeminal ganglion neurons, knockdown of either USF1 or
195 unctional activities of the TRPA1 channel in trigeminal ganglion neurons.
196 acts with these repeats in latently infected trigeminal ganglion neurons.
197 s, which in turn modulate gene expression in trigeminal ganglion neurons.
198 l, by whole-cell patch clamping cultured rat trigeminal ganglion neurons.
199 mp recordings were performed on cultured rat trigeminal ganglion neurons.
200 motes export of endogenous deltaR in primary trigeminal ganglion neurons.
201  cocoa can prevent inflammatory responses in trigeminal ganglion neurons.
202 etected by polymodal and pure mechanosensory trigeminal ganglion neurons.
203 rotein kinase regulation of the CGRP gene in trigeminal ganglion neurons.
204                                  Neither the trigeminal ganglion nor the ophthalmic branch of the tri
205 esonate, conferring frequency specificity to trigeminal ganglion (NV) and primary somatosensory corte
206 ial dura, using single-unit recording in the trigeminal ganglion of anesthetized male rats.
207 There was also a significant increase in the trigeminal ganglion of cells expressing the gamma delta
208 nd IFN-alpha mRNA levels were reduced in the trigeminal ganglion of GFAP-IFN-alpha mice compared with
209 CD8+, and DX5+ (NK cell marker) cells in the trigeminal ganglion of latent HSV-1-infected mice 24 h a
210                                   Within the trigeminal ganglion of normal subjects, cell bodies and
211           Single-unit recordings made in the trigeminal ganglion of rats were used to test changes in
212 s, higher levels of infectious virus in eye, trigeminal ganglion, or brain, and virtually complete de
213 g chicken embryo, nerves from the ophthalmic trigeminal ganglion (OTG) innervate the cornea in a seri
214                                       In the trigeminal ganglion, P2X(3) receptors are often co-expre
215 el of chronic orofacial pain; in this model, trigeminal ganglion Panx1 expression and function are ma
216 ressed in the surface ectoderm overlying the trigeminal ganglion, play a role in promoting the assemb
217 f phenotypic, target, and sex differences in trigeminal ganglion primary afferent neurons.
218          Electrical stimulation of the right trigeminal ganglion produced a unilateral expression of
219                                          The trigeminal ganglion provides the somatosensory innervati
220  the cornea to the periocular tissue via the trigeminal ganglion rather than by direct spread from co
221 ed that in mouse, presumptive targets of the trigeminal ganglion, rather than intermediate structures
222      The largest of the cranial ganglia, the trigeminal ganglion, relays cutaneous sensations of the
223 arly striking was restoration of near-normal trigeminal ganglion replication and neurovirulence of an
224                           Stimulation of the trigeminal ganglion resulted in a frequency-dependent re
225        We established a co-culture system of trigeminal ganglion sensory neurons and vascular endothe
226 ents or whisker stimulation, because neither trigeminal ganglion sensory-evoked responses nor EMG act
227 at CD8(+) T cell control is expressed in the trigeminal ganglion, serving to curtail a source of viru
228                     Orthodromic latencies to trigeminal ganglion shocks and antidromic activation fro
229  central root that was still attached to the trigeminal ganglion showed an increase in CGRP-IR within
230                 In the largest of these, the trigeminal ganglion, Slit1-Robo2 signaling is essential
231 essed in skin, tongue, dorsal root ganglion, trigeminal ganglion, spinal cord and brain.
232                        After 2-6 days, their trigeminal ganglions, spinal cords and brainstems were c
233 avasation within dura mater after electrical trigeminal ganglion stimulation in both wild-type and kn
234  the neurogenic dural inflammation following trigeminal ganglion stimulation in the guinea pig after
235 achykinin (the substance P precursor) in the trigeminal ganglion, suggesting that increased NGF was d
236 y, transgenic overexpression of Brn3a in the trigeminal ganglion suppresses the expression of the end
237                   Nociceptors located in the trigeminal ganglion (TG) and DRG are the primary sensors
238  infection, CD8(+) T cells accumulate in the trigeminal ganglion (TG) and participate in the maintena
239                                 Both sensory trigeminal ganglion (TG) and sympathetic superior cervic
240 thy1-YFP mouse and determine if they promote trigeminal ganglion (TG) cell neurite growth.
241                          Primary cultures of trigeminal ganglion (TG) cells from herpes simplex virus
242 -1) in primary cultures of latently infected trigeminal ganglion (TG) cells.
243 (HSV-1) reactivation from latency in ex vivo trigeminal ganglion (TG) cultures without destroying the
244 characterize this mutant in murine embryonic trigeminal ganglion (TG) cultures.
245                       Axonal branches of the trigeminal ganglion (TG) display characteristic growth a
246 dding during reactivation from latency using trigeminal ganglion (TG) explants from Swiss Webster mic
247  virus 1 (HSV-1) infection in the tree shrew trigeminal ganglion (TG) following ocular inoculation.
248  KLF15 were frequently expressed in the same trigeminal ganglion (TG) neuron during reactivation and
249              Ectopic discharges from injured trigeminal ganglion (TG) neurons and thalamocortical reo
250 latency-associated transcript (LAT)-positive trigeminal ganglion (TG) neurons coexpressed SSEA3, 71%
251 solated mouse dorsal root ganglion (DRG) and trigeminal ganglion (TG) neurons expressing the cold-sen
252 expression of TRPV1 in anatomically relevant trigeminal ganglion (TG) neurons in both the xenograft a
253                       We report that ~50% of trigeminal ganglion (TG) neurons retrogradely labeled fr
254 CD8(+) T cells provide immunosurveillance of trigeminal ganglion (TG) neurons that harbor latent HSV-
255 in nonneuronal cells (MRC5) and adult murine trigeminal ganglion (TG) neurons using the Illumina plat
256 ordings were performed on cultured adult rat trigeminal ganglion (TG) neurons voltage-clamped near th
257    The number of latent sites established in trigeminal ganglion (TG) neurons was determined using a
258 f neurons, cultured dissociated adult murine trigeminal ganglion (TG) neurons were assessed for relat
259        The ability to genetically manipulate trigeminal ganglion (TG) neurons would be useful in the
260  virus type 1 (HSV-1) establishes latency in trigeminal ganglion (TG) neurons.
261 nt TRESK subunits in HEK293T cells and mouse trigeminal ganglion (TG) neurons.
262 gf8 as a novel modifier of BMP4 signaling in trigeminal ganglion (TG) neurons.
263 r biology.SIGNIFICANCE STATEMENT The DRG and trigeminal ganglion (TG) provide sensory information fro
264 ts a specific transcriptome signature in the trigeminal ganglion (TG) that includes Rictor, the rapam
265 fter reactivation of latent infection in the trigeminal ganglion (TG) was examined in the mouse using
266 f herpes simplex virus type 1 (HSV-1) in the trigeminal ganglion (TG) was induced by UV irradiation o
267 ger RNA (mRNA) in the sensory neurons of the trigeminal ganglion (TG) which innervate the nasal epith
268  cornea, the virus enters latency within the trigeminal ganglion (TG), from which it can reactivate t
269 nscriptomes from dorsal root ganglion (DRG), trigeminal ganglion (TG), medulla (MED), cortex, and spi
270 ades sensory neurons with cell bodies in the trigeminal ganglion (TG), replicates briefly, and then e
271  dental pulpal afferent (DPA) neurons of the trigeminal ganglion (TG).
272 ent protein kinase R (PKR) in the cornea and trigeminal ganglion (TG).
273 demonstrate receptor binding sites in rhesus trigeminal ganglion (TG).
274 ction in viral Ag expression in the eyes and trigeminal ganglion that correlated with a reduction in
275 ivity of primary afferent neurons in the rat trigeminal ganglion that innervate the dural venous sinu
276   Here, we show that proper formation of the trigeminal ganglion, the largest of the cranial ganglia,
277  the present study, we show that, in the rat trigeminal ganglion, the location of the primary sensory
278 e transport from the application site to the trigeminal ganglion, the numbers of stained DPANs, and t
279 somatosensory neurons of the dorsal root and trigeminal ganglion, the transient receptor potential me
280 ing analysis indicated that in the adult rat trigeminal ganglion there exist at least two nicotinic r
281                                              Trigeminal ganglion tissue sections were processed for s
282  (5-HT1D-IR) peripheral afferents within the trigeminal ganglion (TRG) and lumbar dorsal root ganglio
283 egulatory targets of Brn3a in the developing trigeminal ganglion using microarray analysis of Brn3a m
284  information, primary sensory neurons in the trigeminal ganglion (Vg) have often been described as en
285 n via afferents whose cell bodies lie in the trigeminal ganglion (Vg).
286           HSV was introduced into the murine trigeminal ganglion via stereotaxic guided injection.
287 noketone treatment on IL-6 expression in the trigeminal ganglion was also measured.
288                                 Briefly, the trigeminal ganglion was electrically stimulated for 30 s
289 l crest to form somatosensory neurons in the trigeminal ganglion was significantly reduced compared w
290  no change in the viral load from the eye or trigeminal ganglion when comparing the AED-treated with
291  the cornea, the virus is transmitted to the trigeminal ganglion, where a brief period of virus repli
292 e ophthalmic branch of the latently infected trigeminal ganglion, where they acquire and maintain an
293                                          The trigeminal ganglion, which arises lateral to the midbrai
294                             In contrast, the trigeminal ganglion, which innervates papillae but not t
295                        They originate in the trigeminal ganglion, which is derived from two embryonic
296                        Here we show that the trigeminal ganglion, which provides sensory innervation
297                                          The trigeminal ganglion, which provides sensory input to the
298 e limiting for CGRP receptor activity in the trigeminal ganglion, which raises the possibility that e
299  A 3-dimensional reconstruction of an entire trigeminal ganglion with 2-photon laser scanning fluores
300 for the identification of cell bodies in the trigeminal ganglion with axons in the IAN.

 
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