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1 lassic mechanisms (reentry, automaticity, or triggered activity).
2                  Overdrive pacing suppressed triggered activity.
3  agonist increased TRPML1-dependent cellular triggered activity.
4 ases sarcoplasmic reticular calcium leak and triggered activity.
5 in delayed-afterdepolarisations that induced triggered activity.
6 c arrhythmias linked to Ca(2+)-wave-mediated triggered activity.
7  by early afterdepolarization (EAD)-mediated triggered activity.
8 in cardiac tissue caused by calcium-mediated triggered activity.
9 tion potentials were prolonged and displayed triggered activity.
10 myocytes to delayed afterdepolarizations and triggered activity.
11 opic firing via delayed afterdepolarizations/triggered activity.
12 tent with intracellular calcium overload and triggered activity.
13 tration, thereby enhancing susceptibility to triggered activity.
14 entry inducibility (23%), without increasing triggered activity.
15 bination were used to induce EADs, DADs, and triggered activity.
16 tial prolongation, afterdepolarizations, and triggered activity.
17 xchanger activation, and DADs, which promote triggered activity.
18 calcium exchanger), providing mechanisms for triggered activity.
19  release, action potential prolongation, and triggered activity.
20  RV by an early afterdepolarization-mediated triggered activity.
21 lity and by an increase in the threshold for triggered activity.
22 that differs from AT due to automaticity and triggered activity.
23  increasing vulnerability to stress-provoked triggered activity.
24             The mechanism is consistent with triggered activity.
25 hereby, more likely to initiate DAD-mediated triggered activity.
26 est onset served as a focus for DAD-mediated triggered activity.
27 lum, delayed-afterdepolarizations (DAD), and triggered activity.
28 d head position, which can modulate visually triggered activity.
29 and are thought to result from cAMP-mediated triggered activity.
30 a (VT) is thought to be due to cAMP-mediated triggered activity.
31 d pacing promoted both re-entry and ectopic (triggered) activity.
32                                              Triggered activity (14% versus 0% in controls) and incre
33 h their activation can be protective against triggered activity, additional repolarizing force under
34                           The reemergence of triggered activity after cessation of pacing was both ti
35    TAC myocytes showed a higher incidence of triggered activities and spontaneous Ca(2+) waves.
36 c reticulum Ca(2)(+) leak, PLN-KO suppresses triggered activities and stress-induced VTs in a mouse m
37 (81%) of these ATs were classified as due to triggered activity and 6/32 (19%) as due to enhanced aut
38 constitutive [Na(+)]i excess of PCs promotes triggered activity and arrhythmogenesis at lower levels
39 s during beta-adrenergic stimulation besides triggered activity and illustrate the importance of I(Ks
40 n combination, the 2 drugs suppressed AF and triggered activity and prevented the induction of AF in
41 s in long QT 3 syndrome (LQT3) involves both triggered activity and re-entrant excitation arising fro
42 y exertion or stress and is characterized by triggered activity and sudden cardiac death.
43  the development of late phase 3 EAD-induced triggered activity and that this mechanism may be respon
44 ronized in the intact myocardium, leading to triggered activity and the resultant DCs in the settings
45 translated into a high risk for induction of triggered activity and ventricular dysrhythmia.
46 We investigate the impact of PLN-KO on SCWs, triggered activities, and stress-induced ventricular tac
47 horter due to increased I(to1) and I(K1) and triggered activity, and Ca(2+)-aftertransients were less
48 clinic bifurcation mechanism of EAD-mediated triggered activity, and raise the possibility that this
49  vulnerability to early afterdepolarization, triggered activity, and ventricular arrhythmias.
50 that reduce excitation threshold and promote triggered activity; and (7) early after depolarizations
51 prolongation, and, occasionally, spontaneous triggered activity (apparently induced by early afterdep
52 eliminated late phase 3 EAD- and DAD-induced triggered activity as well as isoproterenol-induced auto
53 nse trials, but on slow-response trials they triggered activity associated with working memory repres
54 or contained amorphous ice, which could have triggered activity at greater distances from the Sun.
55 enerated delayed afterdepolarization-induced triggered activity at lower frequencies and level of adr
56 neous Ca(2+) release preceded pacing-induced triggered activity at subendocardial PCs.
57 tential mechanisms for any given PVC include triggered activity, automaticity, and reentry.
58 um (Ca) cycling during Ca overload can cause triggered activity because spontaneous calcium release (
59 induce early afterdepolarizations (EADs) and triggered activity by impairing Na current (I(Na)) inact
60                          Local blocking also triggered activity changes to nonblocked fingers across
61 to why hypokalemia promotes calcium-mediated triggered activity, despite the resting potential lying
62 narrhythmogenic stabilizer cells to suppress triggered activity due to delayed or early afterdepolari
63                         Eventually, EADs and triggered activity ensued, giving rise to intermittent e
64 waves and Ca(2)(+) sparks and suppressed the triggered activities evoked by sarcoplasmic reticulum Ca
65  a nonreentrant mechanism that may be due to triggered activity from delayed afterdepolarizations.
66 action potential duration (APD) and generate triggered activity from early afterdepolarizations.
67 mmon arrhythmia in humans, is initiated when triggered activity from the pulmonary veins propagates i
68 showed increased incidence of arrhythmogenic-triggered activities in female ventricular myocytes and
69 ding revealed cell-wide propagating SCWs and triggered activities in RyR2-R4496C+/- ventricular myocy
70 andom RyR gating gives rise to probabilistic triggered activity in a one-dimensional myocyte tissue m
71 anisms and role of calcium-mediated cellular triggered activity in AF, and addresses some of the curr
72 n completely abolished isoproterenol-induced triggered activity in both dyssynchronous HF and CRT myo
73 romoting atrial delayed afterdepolarizations/triggered activity in cAF patients.
74 ), and generated afterdepolarizations and/or triggered activity in drug-treated cells (11/20 acutely
75                                              Triggered activity in fibers obtained from affected dogs
76 nol-induced delayed afterdepolarizations and triggered activity in infected cells, (2) reduced adrene
77 f reduced local phosphatase activity enhance triggered activity in LQT2 syndrome.
78 vents, causing delayed after-depolarizations/triggered activity in pAF.
79 hesis that early afterdepolarization-induced triggered activity in Purkinje fibers is responsible for
80 itiated by early afterdepolarization-induced triggered activity in Purkinje fibers.
81 monstrate that both of these factors promote triggered activity in single cells and tissues.
82 ed afterdepolarization (EAD and DAD)-induced triggered activity in SVC sleeves and compares SVC and P
83 luences promote spontaneous automaticity and triggered activity in SVC sleeves, thus generating extra
84 polarizations and also led to development of triggered activity in the CPVT-hiPSCs-CMs.
85 etermine whether these changes contribute to triggered activity in the form of early after depolariza
86         Whereas brief odor pulses repeatedly triggered activity in the same coding ensemble, the temp
87 eased intracellular Ca promotes DAD-mediated triggered activity in tissue predominantly by increasing
88 ut not the remote region provide a source of triggered activity in vivo and of repolarization instabi
89 ial cause of proarrhythmic cellular ectopic (triggered) activity in AF.
90 nts, broad and double-humped transients, and triggered activity) in the CPVT cardiomyocytes that wors
91 erved manifestations of Ca(2+)-wave-mediated triggered activity, including phase 2 and phase 3 early
92 l ATs demonstrate properties consistent with triggered activity, including termination by adenosine.
93 n contrast, flecainide effectively prevented triggered activity induced by isoproterenol.
94 ne whether early afterdepolarization-induced triggered activity is responsible for the initiation of
95                                        This "triggered activity" is known to cause arrhythmias, but h
96 ddition to reentry, abnormal automaticity or triggered activity may also occur during VF.
97  impaired luminal Ca2+ sensing underlies the triggered activity observed in mutant CSQN2-expressing m
98 tion potential duration, slow conduction and triggered activity occur due to electrical and structura
99 X) currents and delayed afterdepolarizations/triggered activity occurred more often and the sensitivi
100               Early afterdepolarizations and triggered activity occurred spontaneously in Purkinje fi
101 ells (visualized as Ca2+ sparks) and the ICa-triggered activity of RyRs that gives rise to [Ca2+]i tr
102 hift in the dependence of the probability of triggered activity on sarcoplasmic reticulum Ca(2+) load
103  known about the mechanisms relating SCR and triggered activity on the tissue scale.
104 c for identifying focal AT mechanisms due to triggered activity or automaticity, respectively.
105 ial tachycardia (AT) caused by microreentry, triggered activity, or enhanced automaticity.
106 en elevated intracellular calcium levels and triggered activity originating from a specific location
107 he post-POC amplitude IPSPs elicited the POC-triggered activity pattern in this projection neuron and
108 ak and related delayed after-depolarizations/triggered activity promote cellular arrhythmogenesis in
109 preting complex and varied manifestations of triggered activity relevant to cardiac arrhythmias.
110 ease, delayed afterdepolarization (DAD), and triggered activity (TA) from tissues with high sensitivi
111 s of initiation and termination of bursts of triggered activity that are maintained by a positive fee
112 ted early afterdepolarization (EAD)-mediated triggered activity that led to sustained VF in 8 out of
113 ty showed early afterdepolarization-mediated triggered activity that led to VF.
114 r DADs with higher probability of generating triggered activity, the latency period variance and SR C
115 ivity promote EADs and underlie the enhanced triggered activity through increased forward mode of Na(
116  SHR ventricular myocytes developed EADs and triggered activity to the same extent as NR ventricular
117 that adenosine terminates AT attributable to triggered activity, transiently suppresses automatic rhy
118                           In one experiment, triggered activity was followed by a long episode of vor
119                                              Triggered activity was not induced by phenylephrine or e
120                                     Frequent triggered activity was observed with and without pharmac

 
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