ライフサイエンスコーパス: triton

1 nteractions are more abundant in 1% NP-40 or Triton.

2 oglia and DI-TNC1 astrocytes were exposed to triton (1%) or CORM-3 (10-100 iM) and cytotoxicity (lact

3 terior to that of the large, retrograde moon Triton(1).

4 With Triton added at an optimal level, close to critical mice

5 lf; they formed shortly after the capture of Triton and most of them have probably been fragmented mu

6 ing molecule in the atmospheres of Pluto and Triton and probably the main nitrogen reservoir from whi

7 aration of non-canonical histone variants by triton AU(TAU) and 2D TAU electrophoresis; and immunoblo

8 al, human pancreata were decellularized with Triton-based solution and thoroughly characterized.

9 3% p-formaldehyde and permeabilized with 1% Triton before immunocytochemical detection of ZO-1 and n

10 using the Cirrus 5000, RTVue XR Avanti, and Triton DRI OCT platforms with default layer segmentation

11 both the recombinant 27-kDa (r27) and native Triton-extracted 17-kDa (TX17) Cryptosporidium antigens.

12 We followed myosin classes V, VI, and X on triton-extracted actin cytoskeletons from Drosophila S2,

13 inhibitors decreased the level of cTnT-ND in Triton-extracted myofibrils.

14 TRITON group was included as a positive control for tiss

15 These aggregates were hyperphosphorylated, Triton insoluble, and thioflavin-S positive, either comi

16 T) and phosphorylated (Ser129) hSYN(A53T) in Triton-insoluble aggregates.

17 A Triton-insoluble band ("raft fraction") at the 5%/30% su

18 states by cycling between Triton-soluble and Triton-insoluble forms.

19 smoplakin but not desmocollin (Dsc) 3 in the Triton-insoluble fraction of cell lysates within 2 hours

20 ted in the mutant oocytes and found that the Triton-insoluble fraction of tubulin was significantly d

21 Lipid analysis showed that, the Triton-insoluble fraction was highly enriched in cholest

22 m of MLK3 is inactive and redistributes to a Triton-insoluble fraction.

23 also abrogated interaction of F protein with Triton-insoluble lipid rafts.

24 h this effect and that Triton-soluble versus Triton-insoluble tau may be independently targeted by ki

25 Triton is Neptune's principal satellite and is by far th

26 of intracellular contents, analogous to the triton model in eukaryotic flagella and gliding Mycoplas

27 compounds was done by using an acute model (Triton model), in which compounds 3f and 3l showed signi

28 ual configuration has led to the belief that Triton originally orbited the Sun before being captured

29 mia and bleeding risk, a large proportion of TRITON participants (42%) were predicted to experience n

30 paired myofilament calcium responsiveness in Triton-permeabilized cardiomyocytes.

31 Neelsen stain involving cytospin slides with Triton processing, and an ESAT-6 immunocytochemical stai

32 lly inward on the silicon surface exposed to Triton((R)) X-100 containing HF based etchant solution.

33 when dimethyl sulfoxide was used instead of Triton((R)) X-100 in the etchant solution, no such morph

34 To transfer Triton-resistant chromatin binding to green fluorescent

35 ok domain from full-length LEDGF/p75 reduced Triton-resistant chromatin binding, while deletion of bo

36 ](2+) -doped silica nanoparticles, named bio-Triton@RuNP and bio-Igepal@RuNP, obtained following the

37 n-propylamine (TPrA) as coreactant, with bio-Triton@RuNps being more efficient than bio-Igepal@RuNP.

38 Neptune is a far more likely explanation for Triton's capture.

39 Triton shows the pattern of a segregating surfactant in

40 rce and relaxation rate were investigated in Triton-skinned rat caudal arterial smooth muscle strips.

41 were isolated by mechanical homogenization, Triton-skinned, and attached to micropipettes that proje

42 M myosin filament assembly, as assessed by a Triton solubility assay and a proximity ligation assay a

43 amentous actin (sF-actin) fractions based on Triton solubility of cell lysates.

44 uorescence recovery after photobleaching and Triton solubility.

45 in interaction network that remains after 1% Triton solubilization.

46 n and deactivation states by cycling between Triton-soluble and Triton-insoluble forms.

47 may be associated with this effect and that Triton-soluble versus Triton-insoluble tau may be indepe

48 cular events as compared with clopidogrel in TRITON-TIMI 38 (Trial to Assess Improvement in Therapeut

49 aintenance doses of prasugrel on events in a TRITON-TIMI 38 (TRial to Assess Improvement in Therapeut

50 e potent antiplatelet agent prasugrel in the TRITON-TIMI 38 (Trial to Assess Improvement in Therapeut

51 In the TRITON-TIMI 38 (TRial to Assess Improvement in Therapeut

52 red, and the data combined with the existing TRITON-TIMI 38 database.

53 Researchers in the TRITON-TIMI 38 randomized 13,608 subjects with acute cor

54 A subset of the TRITON-TIMI 38 study (Trial to Assess Improvement in The

55 In the TRITON-TIMI 38 study (Trial to Assess Improvement in The

56 prasugrel or clopidogrel with stents in the TRITON-TIMI 38 study.

57 The TRITON-TIMI 38 trial has shown that prasugrel-a novel, p

58 In the TRITON-TIMI 38 trial, 13,608 patients with an acute coro

59 In the TRITON-TIMI 38 trial, the primary endpoint was the compo

60 dogrel (n=1471) or prasugrel (n=1461) in the TRITON-TIMI 38 trial.

61 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38) demonstrated that treatment with prasugr

62 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38) showed an overall reduction in ischemic

63 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38), we fit risk models for ischemic events

64 el-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38).

65 l--Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38).

66 ugrel-Thrombolysis in Myocardial Infarction (TRITON-TIMI 38).

67 e index procedure following randomisation in TRITON-TIMI 38, and were further subdivided by type of s

68 Among clopidogrel-treated subjects in TRITON-TIMI 38, carriers had a relative increase of 53%

69 In TRITON-TIMI 38, the safety and efficacy outcomes of pras

70 In TRITON-TIMI 38, we classified 12,674 patients into low-d

71 nts treated with clopidogrel or prasugrel in TRITON-TIMI 38.

72 the US healthcare system by using data from TRITON-TIMI 38.

73 l with clopidogrel among subjects with DM in TRITON-TIMI 38.

74 ugrel-Thrombolysis in Myocardial Infarction (TRITON-TIMI) 38.

75 sing a Topcon swept-source system (DRI OCT-1 Triton, Topcon, Tokyo, Japan).

76 Our model predicts that Triton was once a member of a binary with a range of pla

77 ized indole-based fibrates were evaluated in Triton WR-1339 and high fat diet (HFD)-induced hyperlipi

78 assessed using [(35)S]methionine labeling in Triton WR1339-treated mice, was not increased in fasting

79 The Triton X (TX)-series are alkylphenol polyethoxylates -ty

80 r of FANCI foci form and become resistant to Triton X extraction in response to mitomycin C treatment

81 All extractions using SDS/Triton X with or without sonication were inhibited.

82 eal-time injection of a non-ionic detergent, Triton X, induced biphasic solubilization kinetics of su

83 ase activity required Mg(2+) ions (8 mm) and Triton X-100 (0.25 mm) at pH 5.0.

84 Maximum PAP activity was dependent on Triton X-100 (20 mm), PA (2 mm), Mg(2+) (0.5 mm), and 2-

85 mple dilution with an OIS and the surfactant Triton X-100 (inorganic media) or ethanol (organic media

86 o]-1-propanesulfonate (CHAPS, zwitterionic), Triton X-100 (nonionic), sodium dodecyl sulfate (SDS, an

87 derivatives in aqueous solutions of reduced Triton X-100 (RTX-100) were determined by measurements o

88 I) and ABI-attenuation by additives, such as Triton X-100 (TX) and human serum albumin (HSA), are not

89 ort, we confirm that TRAF2 translocates to a Triton X-100 (TX)-insoluble compartment upon TNF-R2 enga

90 month-old mice by intranasal irrigation with Triton X-100 (TX).

91 s the tegument was resistant to removal with Triton X-100 (TX-100), whereas it was lost nearly comple

92 e demonstrated that Gs alpha migrates from a Triton X-100 (TX-100)-insoluble membrane domain (lipid r

93 otein expression by Western blot analysis of Triton X-100 (TX-100)-soluble and TX-100-insoluble cell

94 (erythro-GUVs) when exposed to the detergent Triton X-100 (TX-100).

95 ity, and viability caused by the presence of Triton X-100 (TX100), a nonionic surfactant, were studie

96 Triton X-100 also thickens Lo domains, but partitions to

97 of these observations, we used a mixture of Triton X-100 and 1-butanol and observed that water-solub

98 % or 1% sodium dodecyl sulfate (SDS) or 0.1% Triton X-100 and assayed for clinical chemistry and mala

99 The subsequent introduction of Triton X-100 and CoQ10 causes the MLs lysis and the cres

100 amprenavir, and the Envs were solubilized in Triton X-100 and isolated by sedimentation in a sucrose

101 Milk sample slurries were prepared using Triton X-100 and nitric acid for direct analysis of Pb u

102 The protein was solubilized with Triton X-100 and purified to near-homogeneity.

103 en perfused with PBS, followed by successive Triton X-100 and SDS solutions in saline buffer.

104 In Triton X-100 and sodium cholate solutions, the aggregate

105 SDS, Brij-35, Brij-58, Triton X-100 and Span-40 were explored for the extractio

106 s dependent on the presence of the detergent Triton X-100 and the methyldonor S-adenosylmethionine.

107 KCl solution containing nonionic surfactant Triton X-100 and their translocation was studied at diff

108 In aqueous solution of nonionic surfactants (Triton X-100 and Tween 20) arrays from the second series

109 gh concentrations of inorganic ions, using a Triton X-100 aqueous solution to dilute the sample durin

110 ap the interface of the Bax oligomer we used Triton X-100 as a membrane surrogate and performed site-

111 duction of micellar organised media by using Triton X-100 as an extraction solvent.

112 nd basolateral plasma membrane domains using Triton X-100 as detergent, and characterized their lipid

113 ondrial membranes contains up to 120 nmol of Triton X-100 bound per nanomole of the enzyme.

114 Removal of Triton X-100 bound to bovine cytochrome bc(1) was accomp

115 (pH 7.6) containing 0.1 M NaCl and 1% (v/v) Triton X-100 caused liberation of the Ru(bpy)32+ from th

116 e complexation and extraction (pH, DDTC, and Triton X-100 concentration, vortex agitation time and co

117 , these GPC microdomains are soluble in cold Triton X-100 detergent and are thus distinct from conven

118 WT beta2 subunits are resistant to live cell Triton X-100 detergent extraction from the hippocampal a

119 activity by approximately 70% and abolished Triton X-100 detergent inhibition of Ca-dependent nucleo

120 glycosylation status, expression level, and Triton X-100 detergent sensitivity.

121 ions containing BIG/TIR3 and partitions into Triton X-100 detergent-resistant membrane (DRM) fraction

122 We demonstrated that Triton X-100 dissociated the heptameric complex into thr

123 Treatment of blood samples with 0.1% SDS or Triton X-100 does not inactivate EBOV.

124 all TM layers, but not in tissues killed by Triton X-100 exposure.

125 Triton X-100 extracted approximately 10% of the cellular

126 phalloidin staining and Western blotting of Triton X-100 extracted cell lysate.

127 was found to be largely insoluble following Triton X-100 extraction and cofractionationed with bioch

128 this fraction was resistant to high salt and Triton X-100 extraction at pH 6.5.

129 Experiments using Triton X-100 extraction followed by OptiPrep density gra

130 is showed that full-length LEDGF/p75 resists Triton X-100 extraction from chromatin.

131 Triton X-100 extraction of cardiac muscle fibers promote

132 teins CD4, CXCR4, and CCR5, as determined by Triton X-100 extraction.

133 We observed that Triton X-100 extracts of N. gonorrhoeae strain F62 conta

134 rthermore, GPC was more readily extracted by Triton X-100 from adenosine triphosphate (ATP)-depleted

135 A mixture of phospholipids and Triton X-100 in a molar ratio of 5:1 forms well-aligned

136 by sodium dodecyl sulfate, Nonidet P-40, or Triton X-100 in the mass spectrometry analysis.

137 e neuroglian is resistant to extraction with Triton X-100 in the sorting zone and nerve layer, possib

138 tion of cardiac myocytes with saponin and/or Triton X-100 increased NAADP synthesis, indicating that

139 otidase activities while greatly attenuating Triton X-100 inhibition of Mg-dependent nucleotidase act

140 with the actin cytoskeleton, we isolated the Triton X-100 insoluble actin cytoskeleton from platelets

141 cking the N-terminal domain, was detected in Triton X-100 insoluble fractions in Western blot analysi

142 ibited by DP-S2849G-GFP in the cytoskeletal (Triton X-100 insoluble) fraction, and keratin filament r

143 Triton X-100 is employed at finely tuned concentrations

144 t interact in the hetero-complex formed in a Triton X-100 micelle as a membrane surrogate.

145 ha (>15-fold) with a K(a) of 2.4 mol % C-1-P/Triton X-100 micelle.

146 We also show that smaller Triton X-100 micelles give a read length of 103 bases in

147 zyme activity in a cell-free system using PA/Triton X-100 mixed micelles as substrate, analyzing it i

148 centrations at all surface concentrations in Triton X-100 mixed micelles.

149 hange in the lipin 1beta affinity for the PA/Triton X-100 mixed micelles.

150 d point temperature of non-ionic surfactant, Triton X-100 occurred and complex was entrapped in surfa

151 We have studied the solubilization by Triton X-100 of binary mixtures composed of egg sphingom

152 ticity because the micelle-forming detergent Triton X-100 only minimally affects TRPV1 properties.

153 Confocal microscopy of Triton X-100 permeabilized neutrophils showed that a sma

154 lin than did the non-EGF receptor-containing Triton X-100 rafts.

155 c(1) is fully active; however, protein-bound Triton X-100 significantly interferes with structural st

156 Triton X-100 solubility assays demonstrate that mutant D

157 First, we did cold Triton X-100 solubility assays.

158 ve compound, did not significantly alter the Triton X-100 solubility properties of the membrane.

159 -48 h) certain mixtures were not amenable to Triton X-100 solubilization at one or more temperatures.

160 quantitatively removed using injection of 5% Triton X-100 solution, generating a fresh surface for ea

161 ively removed using a cleaning method of 0.5%Triton X-100 sonication plus 1 N nitric acid sonication.

162 he DMPC lipid membrane (tuned by addition of Triton X-100 surfactant or by the increase of the soluti

163 ional channels, even without the presence of Triton X-100 that has been found necessary for in vitro-

164 Some tissues were exposed to Triton X-100 to establish dead tissue controls.

165 ndorff-perfused mouse hearts were treated by triton X-100 to produce endothelial dysfunction and subs

166 can be stimulated 35-fold by the addition of Triton X-100 to the reaction mixture.

167 function during reperfusion was impaired in triton X-100 treated hearts compared with vehicle-treate

168 st, synaptic tau was partially soluble after Triton X-100 treatment and most likely represents aggreg

169 Triton X-100 treatment of transgenic mouse cardiac myofi

170 exhibit differential functional stability in Triton X-100 versus dodecyl maltoside.

171 An average LC50 value of 138 microM for Triton X-100 was obtained for an incubation period of 7-

172 Combining the use of Triton X-100 with the newly introduced UPLC method, we w

173 ted as detergent concentrations (Tween 80 or Triton X-100) are increased up to their critical micelle

174 les (e.g., diheptanoylphosphatidylcholine or Triton X-100) or 0.1-0.2 M inorganic salts.

175 to nonionic micelles in the running buffer (Triton X-100), linking the tagged DNA to the micellar dr

176 pon the addition of nonionic detergent (0.1% Triton X-100).

177 of Jurkat cells in the presence of the agent Triton X-100).

178 r the homogenization of the oil samples with Triton X-100, 200 muL of methanol was added to facilitat

179 Conversely pre-extraction in Triton X-100, a treatment that promotes SNARE complexes

180 onorhamnolipid was compared to that of SDBS, Triton X-100, and ethanol.

181 ocked with cis-Golgi, were solubilized in 2% Triton X-100, and proteins were immunoprecipitated using

182 e are solubilized by lower concentrations of Triton X-100, at least within certain temperature ranges

183 s was more easily perturbed by the detergent Triton X-100, but not other non-ionic detergents.

184 ECM by detergent decellularization methods (Triton X-100, DOC and SDS are compared).

185 ta activity was abolished by the denaturants triton X-100, Gua-HCl, Gua-thiocyanate, SDS and urea in

186 ed IC50 values of three cytotoxic chemicals, Triton X-100, H2O2, and cadmium chloride, as model compo

187 mphiphiles, sodium dodecyl sulfate (SDS) and Triton X-100, in addition to a similar synthesized dendr

188 TcGP63 is soluble in cold Triton X-100, in contrast to Leishmania GP63, which is d

189 as resistant to solubilization with urea and Triton X-100, indicating the formation of larger tau agg

190 erythrocytes were more easily extractable by Triton X-100, indicating weaker association to the cytos

191 in a highly purified plant PSII preparation (Triton X-100, octylthioglucoside).

192 LpxE activity is dependent upon Triton X-100, optimal near pH 6.5, and Mg2+-independent.

193 When lipid vesicles were disrupted by Triton X-100, PrP aggregation was necessary to maintain

194 ation of transfected cells with digitonin or Triton X-100, respectively.

195 7-s incubation times were 290 and 250 microM Triton X-100, respectively.

196 was diluted and homogenised by n-hexane and Triton X-100, respectively.

197 e inner membrane fraction was solubilized by Triton X-100, suggesting that GerD is a lipoprotein, and

198 Interestingly, CHAPS and Triton X-100, thanks to similar surface binding preferen

199 When extracted with Triton X-100, the isolated Ndi1 did not contain Q.

200 l sulfate (SDS), sodium deoxycholate, Chaps, Triton X-100, Triton X-114, NP-40, Brij-35, octyl glucos

201 llows: n-octyl glucoside, dodecyl maltoside, Triton X-100, Tween 20, 3-[(3-cholamidopropyl)dimethylam

202 xperiments in which neutral additives (e.g., Triton X-100, Tween 20, poly(ethylene glycol)) are remov

203 from beta(2)m upon exposure to the detergent Triton X-100, whereas a mutant expressing only glycan 2

204 nol gave slightly better results than when a Triton X-100-ethanol solution was used for dilution.

205 O flipping in proteoliposomes generated from Triton X-100-extracted Saccharomyces cerevisiae microsom

206 The resulting Triton X-100-free cytochrome bc(1) retained nearly full

207 of USA300 was found to be more resistant to Triton X-100-induced autolysis and also to lysis by lyso

208 on of gcp expression can effectively inhibit Triton X-100-induced lysis, eliminate penicillin- and va

209 lated (activated) EGFR was found only in the Triton X-100-insoluble (lipid raft) fraction, whereas to

210 ither WTsyn or A53Tsyn led to a reduction in Triton X-100-insoluble aggregates and an increase in pro

211 localized in Triton X-100-soluble as well as Triton X-100-insoluble cell fractions.

212 Flightless-I targets caspase-11 to the Triton X-100-insoluble cytoskeleton fraction and the cel

213 tin and moesin and was found enriched in the Triton X-100-insoluble fraction along with p67(phox) and

214 searched for Dyrk1A binding proteins in the Triton X-100-insoluble fraction extracted with urea and

215 ome isoforms being enriched primarily in the Triton X-100-insoluble fraction in lens tissue.

216 The Triton X-100-insoluble fractions of M. penetrans and M.

217 c tail abrogated the association of BHA with Triton X-100-insoluble lipid rafts.

218 fts, Triton X-100-soluble fractions, and the Triton X-100-insoluble pellet following apical infection

219 post-translationally converted into a 20-kDa Triton X-100-insoluble proteasome substrate.

220 The amount of endogenous p130Cas in the Triton X-100-insoluble protein fraction, and fibronectin

221 redistribution of Kir2.1 and Kv2.1 from the Triton X-100-insoluble to the Triton X-100-soluble membr

222 radient analysis of plasma membrane-labeled, Triton X-100-lysed cells shows that proximity measured b

223 t with EGCG caused a marked reduction in the Triton X-100-resistant membrane fraction.

224 class V myosin isoforms are associated with Triton X-100-resistant membranes isolated from mouse for

225 uced using nonspecific alkaline phosphatase, Triton X-100-solubilized membranes from DGK1-overexpress

226 Cytochrome bc(1) isolated from Triton X-100-solubilized mitochondrial membranes contain

227 Using proteoliposomes reconstituted from Triton X-100-solubilized rat liver ER membrane proteins,

228 osomes prepared from phosphatidylcholine and Triton X-100-solubilized rat liver ER-membrane proteins.

229 TJ proteins were redistributed/localized in Triton X-100-soluble as well as Triton X-100-insoluble c

230 ereas total cellular EGFR was present in the Triton X-100-soluble fraction.

231 hyperphosphorylated occludin in lipid rafts, Triton X-100-soluble fractions, and the Triton X-100-ins

232 Claudin-4 was localized to Triton X-100-soluble gradient fractions of control or CP

233 Kv2.1 from the Triton X-100-insoluble to the Triton X-100-soluble membrane fraction.

234 Ubr1 function leads slGFP to accumulate in a Triton X-100-soluble state with slGFP degradation interm

235 y by affinity isolation using native but not Triton X-100-treated budded vesicles.

236 ed iron oxidation, and was also sensitive to Triton X-100.

237 ut are disrupted in harsh detergents such as Triton X-100.

238 teinase K only when exosomes were exposed to Triton X-100.

239 e removed effectively by nonionic surfactant Triton X-100.

240 acrylate combined with a nonionic surfactant Triton X-100.

241 +), O 2, alpha-ketoglutarate, ascorbate, and Triton X-100.

242 t is extracted from low density fractions by Triton X-100.

243 nel is reversibly inhibited by the detergent Triton X-100.

244 olubilized from frozen RBC by addition of 1% Triton X-100.

245 ective to facilitate CeO2-NPs transport than Triton X-100.

246 Analysis of surface dilution kinetics with Triton X-100/PA-mixed micelles yielded constants for sur

247 of 4.2, 4.5, and 4.3 mol %, respectively) in Triton X-100/PA-mixed micelles.

248 t when the apposed bilayers are dissolved in Triton X-100; it is also observed during fusion of isola

249 the extraction efficiency, such as amount of Triton X-114 (5%, v/v), effect of pH, amount of Na(2)SO(

250 Circular dichroism (CD) spectroscopy, Triton X-114 (TX-114) phase partitioning, and liposome i

251 Five CPE factors, surfactant (Triton X-114 (TX-114)) concentration, pH, ionic strength

252 arabinogalactan-peptidoglycan complex, and a Triton X-114 (Tx114)-solubilized protein pool were effec

253 ive assays were performed in AMTPS formed by Triton X-114 and sodium tartrate.

254 PrcB localizes to the detergent phase of Triton X-114 cell surface extracts and migrates as a 22-

255 teins because of their aberrant behaviour by Triton X-114 detergent fractionation.

256 oteins were also found to partition into the Triton X-114 detergent phase and were sensitive to prote

257 o reduction by extracellularly added DTT and Triton X-114 detergent phase partitioning.

258 Triton X-114 extraction of outer membrane vesicle prepar

259 TULP1 partitions to the aqueous phase during Triton X-114 extraction.

260 Here we compare the biological properties of Triton X-114 extracts derived from avirulent and virulen

261 Recently, Triton X-114 extracts of a virulent strain of M. arthrit

262 eared to be genuine lipoproteins as shown by Triton X-114 fractionation and sensitivity to globomycin

263 eed, experiments in this work show that upon Triton X-114 fractionation of thylakoid membranes, PsbQ

264 onstrated endogenous Rac1 in the nucleus and Triton X-114 partition revealed that this pool is prenyl

265 P66 was examined by both Triton X-114 phase partitioning and circular dichroism,

266 r dichroism, heat modifiability by SDS-PAGE, Triton X-114 phase partitioning and liposome incorporati

267 proteomic protocols: solvent extraction and Triton X-114 phase partitioning method.

268 r dichroism, heat modifiability by SDS-PAGE, Triton X-114 phase partitioning, and liposome incorporat

269 Triton X-114 phase partitioning, liposome floatation ass

270 Mycoplasma membrane fractionation and Triton X-114 phase separation showed that MG_186 was a m

271 etween the detergent and aqueous phases upon Triton X-114 phase separation, demonstrating an intrinsi

272 d on the basis of a combination of selective Triton X-114 solubilization, radiolabeling with [(3)H]pa

273 Such derivatization of the Triton X-114 surfactant following CPE was found to provi

274 Triton X-114 was chosen as the surfactant for the extrac

275 Cloud point extraction (CPE) using Triton X-114 was successfully applied as an extractive p

276 Triton X-114 was used for sample clean-up and as a fluor

277 polyethylene glycol tert-octylphenyl ether (Triton X-114) as a surfactant prior to its detection by

278 polyethylene glycol tert-octylphenyl ether (Triton X-114) at pH 5.0.

279 e was extracted using a non-ionic detergent (Triton X-114), followed by temperature-induced phase par

280 he surfactant octylphenoxypolyethoxyethanol (Triton X-114).

281 ), sodium deoxycholate, Chaps, Triton X-100, Triton X-114, NP-40, Brij-35, octyl glucoside, octyl thi

282 Imaging and Triton X-114-extraction confirm TFPI and ADTRP associati

283 h-salt treatment of mycoplasma membranes and Triton X-114-partitioned mycoplasma fractions confirmed

284 terol and simple sphingolipids, thus forming Triton X-114-resistant DRMs.

285 hexafluorophosphate phase in the presence of Triton X-114.

286 hment for hydrophobic membrane proteins with Triton X-114.

287 nto the surfactant-rich phase of 0.06% (w/v) Triton X-114.

288 er proportion of total GFAP was found in the Triton X-insoluble fraction of plectin-deficient fibrobl

289 three nonlipid amphiphiles, vitamin E (VE), Triton-X (TX)-100, and benzyl alcohol (BA).

290 We utilize purified Triton-X 100, a nonionic surfactant, to make soap bubble

291 H 7), aqueous, micellar solutions of reduced Triton-X 100.

292 larized by successive sodium dodecyl sulfate/Triton-X cycles.

293 nts to study spectrin aggregate formation by Triton-X extraction and immunocytochemistry followed by

294 nding molecules partition into a low density Triton X100 resistant phase suggesting their association

295 Trace amounts of Triton X100, a nonionic surfactant, release the trapped

296 Using Triton X100, insoluble raft membranes contained homomeri

297 ine carbonate or different concentrations of Triton X100, Nonidet P40 and Brij-58 nonionic detergents

298 sions generated using surfactants: Tween 80, Triton X100, Sodium Dodecyl Sulfate (SDS) and Quillaja S

299 eric (curry) extract curcumin, and detergent Triton X100.

300 es, but its effect is different from that of Triton X100.