1 sociated, respectively, with lepromatous and tuberculoid leprosy.

2 crophages from patients with lepromatous and tuberculoid leprosy and from normal donors do not differ

3 ort describes elements resembling borderline tuberculoid leprosy in humans.

4 at T cell activation in situ by M. leprae in tuberculoid leprosy leads to local up-regulation of CD40

5  levels of IL-12, relative to macrophages in tuberculoid leprosy lesions.

6          Therefore, T cell responsiveness in tuberculoid leprosy may be mediated by the ability of HL

7 ripheral blood mononuclear cells (PBMC) from tuberculoid leprosy patients (n = 59) and healthy lepros

8 ge of responders) ranging from 19 to 47% for tuberculoid leprosy patients and 21 to 64% for healthy l

9                                 In contrast, tuberculoid leprosy patients had only minor signal trans

10                We found that the majority of tuberculoid leprosy patients responded to peptides corre

11 7-1, B7-2, and CD28 transcripts dominated in tuberculoid leprosy patients, who have potent T cell res

12 proteins induced good IFN-gamma responses in tuberculoid leprosy patients.

13 rosy (L-lep) transition toward self-limiting tuberculoid leprosy (T-lep), mounting effective antimicr

14  antigen (HLA) class II genes in 54 cases of tuberculoid leprosy (TL) and 44 controls has shown a pos

15 inical manifestations seen in patients, from tuberculoid leprosy with robust production of Th1-type c

16   Skin biopsy was consistent with borderline tuberculoid leprosy with type 1 lepra reaction.