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1 sulphated HS remained predominant within the tubular basement membrane.
2 talloproteinase-2 that specifically degraded tubular basement membrane.
3 sence of positively stained cells within the tubular basement membrane.
4 to cleavage of V1 isoform of versican in the tubular basement membrane.
5 an extracellular matrix protein expressed in tubular basement membranes.
6 matrix protein and is expressed in the renal tubular basement membranes.
7 and/or heavy chains along the glomerular and tubular basement membranes.
8 y showed deposits in the glomeruli and along tubular basement membranes.
9 les, peritubular capillaries, glomeruli, and tubular basement membranes.
11 , glomerular basement membranes (GBM) (42%), tubular basement membranes (85%), and vessel walls (96%)
12 MMP-2 leads to structural alterations in the tubular basement membrane, a process that triggers tubul
13 isoform of versican, which localized to the tubular basement membrane and adjacent interstitial mono
14 that epithelial cells migrate outside of the tubular basement membrane and differentiate into interst
15 iated with the ability of E. coli to bind to tubular basement membranes and Bowman's capsule and to b
16 matrix and thickening of the glomerular and tubular basement membranes and the number of dividing ce
17 n and actin reorganization, 3) disruption of tubular basement membrane, and 4) enhanced cell migratio
18 damage, IgG-positive immune deposits in the tubular basement membrane, and circulating antibodies re
19 direct epithelial injury, accumulate in the tubular basement membrane, and elicit an interstitial in
20 ies stained positive for C5b-9 in glomeruli, tubular basement membranes, and vessel walls, albeit at
21 and actin reorganization; (3) disruption of tubular basement membrane; and (4) enhanced cell migrati
22 s including beta1 chain in the mesangium and tubular basement membranes at 1, 2, 3, and 4 mo of diabe
23 mal kidney, HS was largely restricted to the tubular basement membrane; chondroitin-4-sulphate and ch
24 All pretreatment biopsies had glomerular and tubular basement membrane deposits that stained exclusiv
25 th diffuse linear staining of glomerular and tubular basement membranes for one light chain isotype (
28 IgG and C3 deposits were conspicuous in the tubular basement membrane of proximal tubules, correspon
29 the dra region, affecting E. coli binding to tubular basement membranes, prevented renal interstitial
30 ulointerstitial nephritis antigen (TINag), a tubular basement membrane protein, in the UUO model of t
31 ase in tubular cell numbers, suggesting that tubular basement membrane remodeling is important for cy
33 lar frequency in cases and controls, whereas tubular basement membrane staining was less frequently o
34 stitial space, tubular epithelial cells, the tubular basement membrane (TBM), and vascular structures
36 d quantitative changes in the composition of tubular basement membranes (TBMs) and interstitial matri
38 human NPH, including renal cyst development, tubular basement membrane thickening, retinal degenerati
39 n alpha5 chain content of the glomerular and tubular basement membranes was increased, with marked ex
41 tein-positive cells were confined within the tubular basement membrane, were not found in the renal i
42 estricted distribution to the glomerular and tubular basement membranes with scant expression in the