ライフサイエンスコーパス: tubular reabsorption

1 rier, albuminuria, and the oxygen demand for tubular reabsorption.

2 asma concentrations as a result of saturable tubular reabsorption.

3 at changes in glomerular permselectivity and tubular reabsorption account, at least in part, for the

4 megalin and cubilin, mediates renal proximal-tubular reabsorption and is decreased in Dent disease be

5 dly increase renal excretion or reduce renal tubular reabsorption and thus blunt large increases in t

6 ntact by glomerular filtration, with partial tubular reabsorption and transient translocation into th

7 with chemicals that affected either proximal tubular reabsorption (cisplatin, gentamicin) or glomerul

8 ar to albumin, indicating decreased proximal tubular reabsorption following nephrectomy.

9 lation of megalin, a protein involved in the tubular reabsorption of albumin and lipid-bound proteins

10 sease indicators were the best predictors of tubular reabsorption of beta2-microglobulin (R2 = 0.53)

11 n of beta2-microglobulin (R2 = 0.53) whereas tubular reabsorption of beta2-microglobulin and RBP were

12 Sodium restriction can enhance the renal tubular reabsorption of drugs such as lithium, leading t

13 Renal net acid excretion requires tubular reabsorption of filtered bicarbonate, followed b

14 Tubular reabsorption of filtered sodium is tightly contr

15 Renal tubular reabsorption of glucose is substantially increas

16 ogous to recently defined kinetics for renal tubular reabsorption of glucose.

17 n with more severe disease also had impaired tubular reabsorption of low-molecular-weight proteins be

18 Impaired tubular reabsorption of low-molecular-weight proteins, s

19 e exhibited physiologic functions, including tubular reabsorption of macromolecules, that gained acce

20 ed, as does the relationship between TZR and tubular reabsorption of Mg.

21 Angiotensin II (ANG II) stimulates renal tubular reabsorption of NaCl by targeting Na(+)/H(+) exc

22 l [95% CI], 0.59 to 0.96; P=0.02), decreased tubular reabsorption of phosphate (OR, 0.41; 95% CI, 0.2

23 intermediate with decreased serum phosphate, tubular reabsorption of phosphate (TRP (%)), fibroblast

24 ffect of vitamin D (VITD) supplementation on tubular reabsorption of phosphate (TRP), parathyroid hor

25 actor 23 (FGF23) axis, creatinine, and renal tubular reabsorption of phosphate (TRP).

26 ls mineral metabolism by directly modulating tubular reabsorption of phosphate and calcium and by act

27 r 23, a hormone that inhibits proximal renal tubular reabsorption of phosphate and down-regulates ren

28 c cystinosis had significantly lower percent tubular reabsorption of phosphate and fibroblast growth

29 tration, plasma phosphate concentration, and tubular reabsorption of phosphate increased during the p

30 evaluating the fractional sodium excretion, tubular reabsorption of phosphate, and gamma-glutamyltra

31 tamin B(12)-intrinsic factor complex and the tubular reabsorption of protein in the proximal tubule.

32 n II (AngII) helps to regulate overall renal tubular reabsorption of salt and water, yet its effects

33 ion were associated with decreases in distal tubular reabsorption of sodium despite a mild decrease i

34 droxycortisol by CYP3A may mediate increased tubular reabsorption of sodium, leading to a state of mi

35 e studies examine the role of NHERF-1 in the tubular reabsorption of uric acid and regulation of mous

36 F-1 exerts a significant effect on the renal tubular reabsorption of uric acid in the mouse by modula

37 ransporter that is responsible for the renal tubular reabsorption of uric acid.

38 to the treatment of hyponatremia by blocking tubular reabsorption of water by binding to V2 receptors

39 The multiple effects of AngII to increase tubular reabsorption provide a powerful mechanism to pro

40 contributes importantly to regulation of the tubular reabsorption rate.

41 sis toward higher BP primarily by increasing tubular reabsorption rather than decreasing GFR.

42 Renal tubular reabsorption (RTR) following renal excretion is

43 Conceptually, modest inhibition of renal tubular reabsorption should provide effective relief for

44 two significant loci possibly related to its tubular reabsorption, SLC6A19, and its production, ERO1A