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1 ds to duplicate the efficacy of clearance by tubular secretion.
2 xic acid is at least partially eliminated by tubular secretion.
3 ower urine-to-plasma ratios reflecting worse tubular secretion.
4 tein binding and was found to mainly undergo tubular secretion.
5 the kidneys such as glomerular filtration or tubular secretion.
6 ous solutes that are primarily eliminated by tubular secretion.
7 h protein-binding affinity and dependence on tubular secretion.
8 e the glomerular filtration, tubular uptake, tubular secretion and extrusion of nanoparticles, includ
9 distribution of loop diuretics, reduce their tubular secretion, and enhance the inactivation of furos
10 , negligible metabolites in vivo, negligible tubular secretion, and excellent chemical and photo stab
11      It is unknown whether novel measures of tubular secretion are associated with adverse events (AE
12                                    Measuring tubular secretion as an independent marker of kidney fun
13 dofovir was also eliminated via the urine by tubular secretion as well as glomerular filtration.
14             This indicates the importance of tubular secretion, as well as the potential role of anio
15 microbial metabolite with high dependence on tubular secretion but low protein-binding affinity.
16 on-GFR factors (e.g., creatinine production, tubular secretion) conservatively biased associations wi
17             Therefore, estimates of proximal tubular secretion function correlate with glomerular fil
18                                        Renal tubular secretion is an active efflux pathway for the ki
19                                       Kidney tubular secretion is an essential mechanism for clearing
20 organic solutes that are normally removed by tubular secretion is hypothesized to contribute to cogni
21                            Although proximal tubular secretion is the primary mechanism of kidney dru
22          The clearance of solutes removed by tubular secretion may be altered out of proportion to th
23 ine-to-plasma ratios of ten novel endogenous tubular secretion measures, with lower urine-to-plasma r
24 ur results suggest that mIBG undergoes renal tubular secretion mediated by hOCT2 and hMATE1/2-K, and
25                            Maximal values of tubular secretion of creatinine (TScr) and tubular secre
26 f tubular secretion of creatinine (TScr) and tubular secretion of furosemide were obtained in the fir
27 in correlated with the increment of TScr and tubular secretion of furosemide.
28 rter (hOAT1) is a key component in the renal tubular secretion of negatively charged molecules includ
29                                              Tubular secretion of solutes is more efficient than glom
30 d 2j remained unchanged upon blockage of the tubular secretion pathway with probenecid, a necessary c
31                                              Tubular secretion rate modestly correlated with eGFR and
32 intrinsic kidney functions, such as proximal tubular secretion, typically are not quantified.
33 by the kidneys via glomerular filtration and tubular secretion using organic anion transporters.
34    Among SPRINT participants with CKD, worse tubular secretion was associated with greater risk of AE
35    Tc-99m MAG3 is cleared from the kidney by tubular secretion, whereas Tc-99m DTPA is cleared by glo
36 ce rates of four solutes normally cleared by tubular secretion with the plasma concentrations and cle