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1 and the hamsters that survived showed severe tubulointerstitial nephritis.
2 some forms of immunologically mediated human tubulointerstitial nephritis.
3 nt of the changes characteristically seen in tubulointerstitial nephritis.
4 k accuracy for diagnosing histological acute tubulointerstitial nephritis.
5 et to attenuate fibrosis after toxin-induced tubulointerstitial nephritis.
6                              BackgroundAcute tubulointerstitial nephritis (AIN) is one of the few cau
7                  Accurate diagnosis of acute tubulointerstitial nephritis (AIN) often requires a kidn
8  polyendocrine syndrome type 1 who developed tubulointerstitial nephritis and ESRD in association wit
9 ains, develop ESRD associated with prominent tubulointerstitial nephritis and fibrosis within 3 month
10 ic interstitial nephropathy characterized by tubulointerstitial nephritis and formation of enlarged n
11 ed glomerular filtration rate (GFR), chronic tubulointerstitial nephritis and ultrastructural changes
12                                              Tubulointerstitial nephritis and uveitis (TINU) syndrome
13                                              Tubulointerstitial nephritis and uveitis syndrome is und
14                                              Tubulointerstitial nephritis and uveitis syndrome should
15 yndrome, renal proximal tubular acidosis, or tubulointerstitial nephritis and uveitis syndrome.
16 80%) carried the diagnosis of FSGS, 2 (6.7%) tubulointerstitial nephritis, and 1 (3.3%) nephrolithias
17                                              Tubulointerstitial nephritis antigen (TIN-Ag) is a recen
18                                              Tubulointerstitial nephritis antigen (TIN-ag) is an extr
19  467 aa was 46% identical with that of human tubulointerstitial nephritis antigen (TIN-ag), and there
20                         Here, we studied the tubulointerstitial nephritis antigen (TINag), a tubular
21 therapeutic delivery of the secreted protein Tubulointerstitial nephritis antigen-like 1 (Tinagl1) su
22 he pathogenesis of leukocyte infiltration in tubulointerstitial nephritis associated with glomerular
23 s of monocyte/macrophage infiltration in the tubulointerstitial nephritis associated with PAN nephros
24  69% were concurrently receiving a potential tubulointerstitial nephritis-causing medication.
25 h worse renal prognosis, whereas concomitant tubulointerstitial nephritis-causing medications and tre
26 athy is characterized by rapidly progressive tubulointerstitial nephritis culminating in end-stage re
27                    Our findings suggest that tubulointerstitial nephritis developed in these patients
28                               Immune complex tubulointerstitial nephritis due to antibodies to brush
29 from infantile polycystic kidneys to chronic tubulointerstitial nephritis, fibrosis, and cortical mic
30                 This disease may manifest as tubulointerstitial nephritis (IgG4-TIN), but its clinico
31 s affected with isolated kidney failure (and tubulointerstitial nephritis in individuals with availab
32 ficant histological changes corresponding to tubulointerstitial nephritis including interstitial infl
33 trogen levels, more severe histologic GN and tubulointerstitial nephritis, increased glomerular cresc
34                                              Tubulointerstitial nephritis is a common cause of kidney
35 phronophthisis (NPH), an autosomal-recessive tubulointerstitial nephritis, is the most common cause o
36 l-transfer RNA synthetase 1 in toxin-induced tubulointerstitial nephritis mice.
37 ur clinical tests accurately predicted acute tubulointerstitial nephritis on biopsy in two separate p
38 y injury (AKI), glomerulonephritis (GN), and tubulointerstitial nephritis (TIN) and assessed dispropo
39                                              Tubulointerstitial nephritis was observed in one (1%) pa
40                                              Tubulointerstitial nephritis was the dominant lesion in
41 nterior chamber inflammation and evidence of tubulointerstitial nephritis with either (1) a positive
42  a 36-year-old man with AIDS showed a severe tubulointerstitial nephritis with intranuclear inclusion
43 for immune and genetic causes of microcystic tubulointerstitial nephritis with little attention to it
44 ted animals showed renal dysfunction and had tubulointerstitial nephritis with nuclear inclusions, ap
45 2, 21.1%), infectious uveitis (6/52, 11.5%), tubulointerstitial nephritis with uveitis (6/52, 11.5%),