ライフサイエンスコーパス: tumor-associated fibroblast

1 lating mesenchymal precursors as a source of tumor-associated fibroblasts.

2 e microenvironment with increased stroma and tumor-associated fibroblasts.

3 oblast growth factor in normal compared with tumor-associated fibroblasts.

4 xenograft tumors, suggesting the presence of tumor-associated fibroblasts.

5 tochemistry confirm that MSF is expressed by tumor-associated fibroblasts and additionally indicate t

6 on protein (FAP) is selectively expressed on tumor-associated fibroblasts and pericytes in epithelial

7 asts) and adjacent cervical cancer biopsies (tumor-associated fibroblasts) and from primary keratinoc

8 infection of primary human dendritic cells, tumor-associated fibroblasts, and colorectal carcinoma c

9 owth on three-dimensional matrix produced by tumor-associated fibroblasts, and in formation of tumors

10 Tumor-associated fibroblasts are key regulators of tumor

11 We have investigated the role of Tiam1 in tumor-associated fibroblasts as a modulator of tumor cel

12 o-like three-dimensional system derived from tumor-associated fibroblasts at diverse stages of tumor

13 resistance effect in cancer cells induced by tumor associated fibroblasts (CAF).

14 on from Th2 to Th1 type while remodeling the tumor-associated fibroblasts, collagen, and blood vessel

15 Tumor-associated fibroblast-conditioned media similarly

16 product which is preferentially expressed in tumor-associated fibroblasts, could function as a tumor

17 that cells of mesenchymal origin, including tumor-associated fibroblasts, degrade substantial amount

18 ncluding mesenchymal stem cells, adipocytes, tumor associated fibroblasts, endothelial cells, and imm

19 e hepatocytes influences the network for the tumor-associated fibroblast environment.

20 Moreover, tumor-associated fibroblasts from mouse PDA were also re

21 Traction force microscopy revealed that tumor-associated fibroblasts generate larger forces on s

22 lial cells to initiate a paracrine loop with tumor-associated fibroblasts involving TGFbeta and HGF,

23 mutated smooth muscle cells and the adjacent tumor-associated fibroblasts lay down excessive quantiti

24 e involving host blood vessels, lymphocytes, tumor-associated fibroblasts, macrophages, dendritic cel

25 timulatory effects of conditioned media from tumor-associated fibroblasts on keratinocyte penetration

26 Tumor-associated fibroblasts or carcinoma-associated fib

27 Through CD8+ T cell-mediated killing of tumor-associated fibroblasts, our vaccine successfully s

28 conclusion, our data indicate that EREG and tumor-associated fibroblasts play a crucial role in cont

29 Tumor-associated fibroblasts produce C-X-C motif chemoki

30 epsin L to be predominantly expressed by the tumor-associated fibroblasts surrounding the invading me

31 e matrix-modifying hormone relaxin increased tumor-associated fibroblast (TAF) interaction with colla

32 rophages resulting in decreased abundance of tumor-associated fibroblasts (TAF) and robustly reduced

33 The tumor-promoting fibrotic stroma rich in tumor-associated fibroblasts (TAF) is drawing increased

34 A subset of the tumor-associated fibroblasts (TAF) was found to express

35 umbers of SMC and non-SMC, which were mostly tumor-associated fibroblasts (TAF).

36 erapeutic NP to distribute into, and deplete tumor-associated fibroblasts (TAFs) for improved therape

37 es and Cisplatin nanoparticles (Combo NP) on tumor-associated fibroblasts (TAFs).

38 geted immunotherapy to antigens expressed in tumor-associated fibroblasts, the predominant component

39 tor/scatter factor or conditioned media from tumor-associated fibroblasts to the Matrigel resulted in

40 the contribution of hypoxic signaling within tumor-associated fibroblasts to tumorigenesis remains un

41 ion protein (FAP), a product up-regulated in tumor-associated fibroblasts, using dendritic cells tran

42 To specifically kill tumor-associated fibroblasts, we constructed an oral DNA

43 e the functional relevance of EREG-producing tumor-associated fibroblasts, we developed a novel syste

44 Furthermore, tumor-associated fibroblasts were identified as a major

45 eveals that p53 mutations sometimes occur in tumor-associated fibroblasts, which correlate with an in