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1 Orientia spp., which cause a zoonosis, scrub typhus.
2 th MIS-C and 87 patients hospitalized due to typhus.
3 ed to fully understand this aspect of murine typhus.
4 influence morbidity and mortality from scrub typhus.
5 combination of both in treating severe scrub typhus.
6 sis, scarlet fever, infectious diarrhea, and typhus.
7 virus disease-19 infection can mimic endemic typhus.
8 CCL23 and the receptor CCR3 in severe scrub typhus.
9 ine as oral therapy for uncomplicated murine typhus.
10 tsutsugamushi, the causative agent of scrub typhus.
11 M IFA for the serological diagnosis of scrub typhus.
12 ect immunofluorescence assay (IFA) for scrub typhus.
13 causes the vector-borne human disease scrub typhus.
14 sponses and correlates of immunity for scrub typhus.
15 pic correlates of immune protection in scrub typhus.
16 cially R. prowazekii, the causative agent of typhus.
17 ering advances in the prevention of epidemic typhus.
18 sutsugamushi is the causative agent of scrub typhus.
19 of therapeutics against epidemic and murine typhus.
20 accine candidate for the prevention of scrub typhus.
21 meters into 2 main groups: spotted fever and typhus.
22 ause the emerging global health threat scrub typhus.
23 , we identified leptospirosis (10.6%), scrub typhus (4.1%), dengue (3.7%), and Kyasanur forest diseas
24 monly identified aetiologies were JEV, scrub typhus (645 [18.5%] of 3489), and dengue virus (161 [5.2
25 tsia typhi is the causative agent of endemic typhus, a disease with increasing incidence worldwide th
26 priate antibiotic treatment for severe scrub typhus, a neglected but widespread reemerging zoonotic i
28 atic amino acids and histidine, causes scrub typhus, a potentially deadly infection that threatens 1
32 one, and highlighted the importance of scrub typhus and dengue virus as important infectious aetiolog
34 cyclines, needed for the treatment of murine typhus and scrub typhus, are not routinely advised for e
36 o detect Rickettsia prowazekii (the agent of typhus) and Bartonella quintana (the cause of trench fev
38 ttsiaspecies belonging to the spotted fever, typhus, and ancestral groups and, in parallel, compared
40 tive agents of Rocky Mountain spotted fever, typhus, and other human diseases with high mortality and
43 for the treatment of murine typhus and scrub typhus, are not routinely advised for empirical treatmen
45 DT) was used extensively to control malaria, typhus, body lice, and bubonic plague worldwide, until c
46 ommon problem with actual incidence of scrub typhus cases in Northeast India that were reported durin
47 we report three autochthonous cases of scrub typhus caused by O. tsutsugamushi acquired on Chiloe Isl
48 GR), typhus group rickettsioses (TGR), scrub typhus (caused by Orientia tsutsugamushi), ehrlichiosis,
50 s (caused by Orientia tsutsugamushi), murine typhus (caused by Rickettsia typhi), and leptospirosis a
52 cimens were additionally evaluated for scrub typhus, dengue virus, and West Nile virus by serum IgM E
53 %) met the clinical case definition of scrub typhus (detection of IgM against O. tsutsugamushi on enz
60 Phylogenomics reveals extreme gene loss in typhus group (TG) rickettsiae relative to the levels for
61 The lipopolysaccharides (LPSs) isolated from typhus group (TG) rickettsiae Rickettsia typhi and Ricke
62 mental support for the hypothesis that these typhus group genes, like the more degenerate spotted fev
64 gy involves arthropod vectors: for instance, typhus group rickettsiae are principally vectored by ins
65 E. coli metK control, demonstrating that the typhus group rickettsiae have the capability of synthesi
66 verlay modifications for plaque formation of typhus group rickettsiae on the continuous fibroblast ce
68 rs (Arfs), is critical for Rickettsia typhi (typhus group rickettsiae) entry but pseudogenized or abs
69 hods for evaluating the infectious titers of typhus group rickettsiae, cloning single plaque isolates
71 din-stained infected Vero cells revealed the typhus group rickettsiae, Rickettsia prowazekii and Rick
76 Spotted fever group rickettsioses (SFGR), typhus group rickettsioses (TGR), scrub typhus (caused b
77 ), 4,571 ( 30.8%) ehrlichiosis, 1,362 (9.2%) typhus group rickettsiosis (TGR), and 1,193 (8.0%) other
78 ic ehrlichiosis; a novel cat flea-associated typhus group rickettsiosis; bartonelloses of immunocompe
81 kettsia typhi, the causative agent of murine typhus, grows directly within the host cell cytoplasm, a
83 prowazekii, the causative agent of epidemic typhus, has been responsible for millions of human death
84 kettsia typhi, the etiologic agent of murine typhus, homologs of the Sec-translocon-associated protei
85 ologic and clinical characteristics of scrub typhus in 37 villages in Tamil Nadu, India, where the di
87 ion and convalescent-phase samples for scrub typhus indirect immunofluorescence assay using Bayesian
94 rowazekii, the etiological agent of epidemic typhus, is an obligate intracellular bacterium and is ap
95 kettsia typhi, the causative agent of murine typhus, is an obligate intracellular bacterium with a li
96 prowazekii, the causative agent of epidemic typhus, is an obligate intracellular parasitic bacterium
97 phi, the causative agent of murine (endemic) typhus, is an obligate intracellular pathogen with a lif
98 prowazekii, the causative agent of epidemic typhus, is an obligate, intracellular, parasitic bacteri
99 prowazekii, the etiologic agent of epidemic typhus, is an obligate, intracytoplasmic, parasitic bact
100 prowazekii, the causative agent of epidemic typhus, is an obligately intracytoplasmic bacterium, a l
105 eaction (receptors) from patients with scrub typhus (n = 129), patients with similar febrile illness
106 .75), malaria (OR 2.00, CI 1.46-2.73), scrub typhus (OR 2.37, CI 1.41-3.96) and spotted fever group d
108 l 120 serum samples from tuberculosis, scrub typhus, or leptospirosis patients were evaluated in all
109 ired infections (LAIs) associated with scrub typhus (Orientia tsutsugamushi) and murine typhus (Ricke
110 lid species as vector and reservoir of scrub typhus outside the tsutsugamushi triangle, providing uni
111 A significant percentage (>20%) of scrub typhus patient sera reacted strongly with recombinant hH
112 munity is a concern, the reactivity of scrub typhus patient sera with purified recombinant 47-kDa and
113 d raised a troubling observation; some scrub typhus patients responded poorly to doxycycline, which i
115 ii, which appears to have occurred after the typhus (R. prowazekii and R. typhi) and spotted fever (R
117 ttsia typhi, the etiological agent of murine typhus, research on vertebrate host biology is facilitat
118 s of Rocky Mountain spotted fever and murine typhus, respectively, were cloned and characterized.
120 such as plague (Yersinia pestis) and murine typhus (Rickettsia typhi) caused significant numbers of
121 b typhus (Orientia tsutsugamushi) and murine typhus (Rickettsia typhi) research to provide an evidenc
122 nthesis in the etiological agent of epidemic typhus, Rickettsia prowazekii, a slowly growing obligate
123 tic element), previously identified in scrub typhus rickettsiae (Orientia tsutsugamushi) genomes, is
124 e 120-kDa surface protein antigens (SPAs) of typhus rickettsiae are highly immunogenic and have been
127 macaque (Macaca fascicularis) model of scrub typhus, the leading cause of treatable undifferentiated
132 of the immunogenicity of a recombinant scrub typhus vaccine candidate in a nonhuman primate model.
133 , leptospirosis, melioidosis, typhoid, scrub typhus), viral infections (hepatitis A, B, C, D, and E;
136 e 15 years of age or older with severe scrub typhus with at least one organ involvement were enrolled