ライフサイエンスコーパス: ulcer

1 of pressure in pathogenesis of diabetic foot ulcers".

2 L (category 1: erythema/erosion; category 2: ulcer).

3 ad adverse events in the extension (aphthous ulcer).

4 therapy for early, limited lesions of Buruli ulcer.

5 tients with a nursing unit-acquired pressure ulcer.

6 ese cases, so that 33.65% of whom had peptic ulcer.

7 l virulent factor in the skin disease Buruli ulcer.

8 cantly associated with gastritis and gastric ulcer.

9 esophagus, small or large bowel, and peptic ulcer.

10 and 5% decrease in the odds of developing an ulcer.

11 ortic rupture, and 10 had penetrating aortic ulcer.

12 ving any risk assessment before the pressure ulcer.

13 al acuity (VA), size and location of corneal ulcer.

14 junctivitis, scleritis, uveitis, and corneal ulcer.

15 neuroischaemic (NI) or neuropathic (NP) foot ulcers.

16 ffective treatment for chronic diabetic foot ulcers.

17 (4) ng/mL [5.6 x 10(4)-1.1 x 10(5)]) develop ulcers.

18 of inflammatory bowel disease, fistulae and ulcers.

19 reatment, in patients with actively bleeding ulcers.

20 ociated with gastritis, gastric and duodenal ulcers.

21 role in the treatment of filamentous fungal ulcers.

22 ed with a decrease in unit-acquired pressure ulcers.

23 T cell regulation in the aetiology of mouth ulcers.

24 participants in the placebo group developed ulcers.

25 ent of moderate to severe filamentous fungal ulcers.

26 ere may have been some effect among Fusarium ulcers.

27 into the care of diabetic patients with foot ulcers.

28 mina densa blistering, erosions, and chronic ulcers.

29 gainst HD strains from both genital and skin ulcers.

30 n patients with neuroischaemic diabetic foot ulcers.

31 s, and all but one study focused on pressure ulcers.

32 per patient with hospital-acquired pressure ulcers.

33 s an adjuvant therapy for infectious corneal ulcers.

34 hcare cost due to hospital-acquired pressure ulcers.

35 ondary bacterial skin infections and genital ulcers.

36 percentage of patients at risk for pressure ulcers.

37 Sc-associated Raynaud phenomenon and digital ulcers.

38 297 patients had PCR-confirmed Buruli ulcer; 151 (51%) were assigned to RS8 treatment, and 146

39 derate-or-severe PTS (Villalta scale >=10 or ulcer: 18% versus 28%; risk ratio, 0.65; 95% CI, 0.45-0.

40 ty classification: type 1: erythema; type 2: ulcers (2a: superficial ulcers; 2b: deep ulcers); type 3

41 1: erythema; type 2: ulcers (2a: superficial ulcers; 2b: deep ulcers); type 3: perforation (3a: perfo

42 osal healing (CDEIS <4) with absence of deep ulcers 48 weeks after randomisation.

43 the occurrence of PTS (Villalta scale >=5 or ulcer: 49% PCDT versus 51% No-PCDT; risk ratio, 0.95; 95

44 0.021) or severe PTS (Villalta scale >=15 or ulcer: 8.7% versus 15%; risk ratio, 0.57; 95% CI, 0.32-1

45 s of the microbiota colonizing diabetic foot ulcers, a common and costly complication of diabetes, an

46 umber of subjects with large erosions and/or ulcers after 14 days of exposure.

47 scatheter arterial embolization of high-risk ulcers after successful endoscopic therapy is not encour

48 ion (persistent epithelial defect or corneal ulcer) after 8 weeks of masked treatment.

49 cers identified when examined; three had two ulcers; all ulcers resolved after ring removal.

50 emergent laparotomy for a perforated peptic ulcer and died from sepsis.

51 i (Hp) infection is the main cause of peptic ulcer and gastric cancer.

52 ular, the most important of them i.e. peptic ulcer and gastric cancer.

53 There were two grade 4 toxicities: duodenal ulcer and hyponatremia.

54 eption to Aug 6, 2018, for records of Buruli ulcer and Mycobacterium ulcerans detection, with no limi

55 The prevalence rates of corneal ulcer and perforation were 6.2% and 4.0%, respectively,

56 o RS8 for treatment of early, limited Buruli ulcer and was associated with fewer adverse events.

57 eath data, all patients with lower extremity ulcers and a diagnosis of peripheral artery disease who

58 esions including fistula and deep esophageal ulcers and abscesses.

59 reatment of acute disorders, such as gastric ulcers and esophagitis, PPIs are now commonly used for p

60 re needs to be taken for patients with large ulcers and fungal infections, as well as elderly patient

61 astric pathologic conditions such as gastric ulcers and gastric adenocarcinomas.

62 sa by Helicobacter pylori can lead to peptic ulcers and gastric cancer.

63 the classification and assessment of digital ulcers and in understanding ulcer pathogenesis, and ther

64 h central and paracentral corneal infectious ulcers and initial visual acuity worse than 20/200.

65 The PPV of small flat ulcers and large flat ulcers were 40% to 50% and 29% to

66 dase deficiency characterized by severe skin ulcers and mental impairment.

67 ng their ICU stay, and ICU-acquired pressure ulcers and physical restraint use decreased (period 1 vs

68 tion of the GI tract is necessary to exclude ulcers and polyps or cancer as possible causes of GI ble

69 er the odds of developing non-specific mouth ulcers and replicate these in an independent cohort (n =

70 ite patients are more likely to present with ulcers and rest pain.

71 nts with culture-positive filamentous fungal ulcers and visual acuity of 20/40 to 20/400 reexamined 6

72 phagitis, Barrett's esophagus, or esophageal ulcer), and pH impedance testing on PPI therapy demonstr

73 . heilmannii (three gastritis, five duodenal ulcer, and five gastric ulcer samples), and 7 for H. biz

74 zzozeronii (zero gastric ulcer, two duodenal ulcer, and five gastritis samples).

75 for H. felis (one gastritis, three duodenal ulcer, and six gastric ulcer samples), 20 for H. salomon

76 ary treatment of moderate filamentous fungal ulcers, and it may result in decreased visual acuity.

77 n-Barr virus positive (EBV(+)) mucocutaneous ulcer are included as new provisional entities, and a ne

78 Pressure ulcers are a major problem for national healthcare syste

79 Digital ulcers are a painful and disabling visible manifestation

80 ion and assessment of SSc-associated digital ulcers are also covered, alongside an overview of manage

81 Enteropathy and small-intestinal ulcers are common adverse effects of nonsteroidal anti-i

82 Ulcers are deemed neuroischaemic if peripheral neuropath

83 st frequent pathogens that cause the corneal ulcers are P. aeruginosa and S. aureus.

84 Pressure ulcers are serious, avoidable, costly and common adverse

85 Mouth ulcers are the most common ulcerative condition and enco

86 orylated FAK and Ki-67 and measuring mucosal ulcer area, serum creatinine, ALT, and body weight at da

87 epatic enzyme increased, dizziness, aphthous ulcer, arthralgia, arthritis, increased appetite, increa

88 We defined the index ulcer as that present at the first (index) DFU admission

89 tified vancomycin tended to have more severe ulcers at presentation.

90 6 (95%CI: dominant to $144,525) per pressure ulcer avoided.

91 al examination of tissue sample from colonic ulcer biopsy revealed invasive intestinal mucormycosis.

92 Mortality in peptic ulcer bleeding remains high, especially in patients who

93 gests PPI therapy for patients with previous ulcer bleeding who require antiplatelet or anticoagulant

94 regarding prognosis after refractory peptic ulcer bleeding, and the shorter length of hospital stay

95 , obesity, type 2 diabetes, gastrointestinal ulcers/bleeds, fractures, and cataracts (odds, 1.21-1.44

96 with elevated TRV, microalbuminuria, and leg ulcers, but these vascular complications are not indepen

97 reating sight-threatening infectious corneal ulcers by promoting faster corneal epithelialization and

98 Buruli ulcer can cause disfigurement and long-term loss of func

99 volvement in decision-making around pressure ulcer care are important aspects from the patient perspe

100 eographical data on the occurrence of Buruli ulcer cases and M ulcerans detection from studies of any

101 marker of the noninvasive assessment of the ulcer-causing H. pylori gastric pathogen.

102 ggest that risk stratification from baseline ulcer characteristics can identify those at highest risk

103 To determine baseline patient and ulcer characteristics that predict a high risk of develo

104 ude radiation gastritis and gastrointestinal ulcers, cholecystitis, radiation pneumonitis, and radioe

105 y Network, and surveillance data from Buruli ulcer control programmes in the evidence consensus frame

106 (TP) are major causative agents of cutaneous ulcer (CU) in the tropics.

107 us ducreyi are causative agents of cutaneous ulcer (CU) in yaws-endemic regions in the tropics.

108 Differentiation of diabetic foot ulcer-derived induced pluripotent stem cells reveals dis

109 ion is an inflammatory disease in which skin ulcer development is associated with mononuclear cell in

110 e of pressure injury (also known as pressure ulcer) development among critical-care patients.

111 Diabetic foot ulcer (DFU) is a problem worldwide, and prevention is cr

112 ogical properties is vital for diabetic foot ulcer (DFU) treatment.

113 y of patients with neuropathic diabetic foot ulcers (DFU).

114 Diabetic foot ulcers (DFUs) are a leading cause of high morbidity amon

115 Diabetic foot ulcers (DFUs) are a life-threatening disease that often

116 Diabetic foot ulcers (DFUs) are a major complication of diabetes, and

117 Nonhealing diabetic foot ulcers (DFUs) are characterized by low-grade chronic inf

118 Diabetic foot ulcers (DFUs), a life-threatening complication of diabet

119 oor presenting visual acuity, larger corneal ulcer diameter, and causative organisms were not associa

120 ), intestinal obstruction 11 (6%) and peptic ulcer disease 9 (5%).

121 pic subgroup has been associated with peptic ulcer disease and an increased bleeding tendency.

122 n strains isolated from patients with peptic ulcer disease or gastric cancer, rather than asymptomati

123 her risk of gastric adenocarcinoma or peptic ulcer disease than cag PAI-negative strains.

124 better understand the pathogenesis of Buruli ulcer disease, aid in the development of diagnostics, an

125 t association observed in vacA s1 and peptic ulcer disease, as well as vacA s1/m2 and gastric erosion

126 stomach and induces acute gastritis, peptic ulcer disease, atrophic gastritis, and gastric adenocarc

127 reducing the prevalence of herpetic genital ulcer disease, but could also have an additional, indire

128 history (smoking, diabetes, bleeding, peptic ulcer disease, cancer, chronic liver disease, chronic pa

129 he PCSK9 T allele were also seen with peptic ulcer disease, depression, asthma, chronic kidney diseas

130 lop more adverse outcomes, including gastric ulcer disease, gastric adenocarcinoma, or gastric mucosa

131 ons for PPI use, including history of peptic ulcer disease, gastroesophageal reflux disease, or gastr

132 ory NVUGIB, the etiology of bleeding (peptic ulcer disease, unknown source, post surgical); patient f

133 the development of gastric cancer and peptic ulcer disease.

134 he pathogenesis of gastric cancer and peptic ulcer disease.

135 and s2m1 genotypes and development to peptic ulcer disease.

136 s a major cause of chronic gastritis, peptic ulcer diseases and cancer.

137 t the strongest available evidence on Buruli ulcer distribution so far and have many potential applic

138 to describe and evaluate evidence for Buruli ulcer distribution worldwide.

139 cute diverticulitis, cholecystitis, duodenal ulcer, duodenitis, enteritis, or adnexal or testis patho

140 n was increased in jejunal epithelium at the ulcer edge, and Ki-67 staining was unchanged in jejunal

141 to grade the strength of evidence for Buruli ulcer endemicity.

142 Of the 323 patients with smear-positive ulcers enrolled in MUTT-I, 299 (92.6%) were scraped and

143 samples), 20 for H. salomonis (four duodenal ulcer, five gastritis, and 11 gastric ulcer samples), 13

144 from immune infiltration, crypt dropout, and ulcers following administration of dextran sodium sulfat

145 complicated with ischemic colitis, stercoral ulcer formation and subsequent perforation.

146 verage, similar between fungal and bacterial ulcers, fungal ulcers were more likely to produce severe

147 II could not find a benefit for all corneal ulcers, Fusarium keratitis may benefit from the addition

148 ccompanying network in tissue infections and ulcers, future investigation into their role in EC is wa

149 a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC or IIC (as de

150 ns, Fondation Raoul Follereau France, Buruli ulcer Groningen Foundation, Sanofi-Pasteur, and BuruliVa

151 se than 20/400 was more common in the fungal ulcer group after spectacle correction (odds ratio [OR]

152 Consenting patients with skin ulcers >1 cm in diameter were eligible for this study an

153 In patients with gastric ulcer (GU) and IM, the GU may have a different healing r

154 esents with rapidly developing, painful skin ulcers hallmarked by undermined borders and peripheral e

155 Mooren ulcer has been considered as an idiopathic autoimmune ke

156 le, it seems that hospital-acquired pressure ulcers have significant economic implications for the he

157 essively incorporate treatment to accelerate ulcer healing and address MRSA into the care of diabetic

158 enhancing Wnt/beta-catenin signaling during ulcer healing in IBD.

159 which contributes to impaired diabetic foot ulcer healing.

160 dence intervals (CIs) adjusted for age, sex, ulcer history, comorbidity, and calendar period were con

161 symptomatic with vaginal discharge; four had ulcers identified when examined; three had two ulcers; a

162 wound image dataset consisting of 1109 foot ulcer images from 889 patients to train and test the dee

163 There was evidence of Buruli ulcer in 32 countries and consensus on presence in 12.

164 m2, s2m1 and s2m2) and development to peptic ulcer in Iranian population.

165 litis in two patients, and neuritis and skin ulcer in one patient.

166 pic finding of a longitudinal mid-esophageal ulcer in the presence of proximal stricture may be indic

167 endoscopy revealed only minor self-limiting ulcers in 5 patients.

168 chancroid and is a major cause of cutaneous ulcers in children.

169 rmal treatments for NVUGIB, particularly for ulcers in difficult locations or those with a rigid and

170 tive incidence of hospital-acquired pressure ulcers in neonates was 9.8% (95% CI: 2.9%-19.8%) and in

171 of persistent epithelial defects and corneal ulcers in patients with NK.

172 with elevated TRV, microalbuminuria, and leg ulcers in SS-Sbeta(0) adults, but these associations wer

173 minuria in the whole population and with leg ulcers in SS-Sbeta(0) adults.

174 s of khambir alleviated cold-induced gastric ulcers in the animal model as it exhibited histoprotecti

175 tivity Index (CDAI) of 220-450, with mucosal ulcers in the ileum or colon, or both, and a Crohn's Dis

176 ophago-pulmonary fistula and deep esophageal ulcers in the other animals.

177 105 of the 158 patients (66.4%) with peptic ulcers in the urgent-endoscopy group and in 76 of 159 (4

178 the microbial community structure of genital ulcers in women.

179 hese data suggest a diverse etiology of skin ulcers in yaws-endemic areas, which may help design more

180 od flow or among those with other causes for ulcers, including venous, neuropathic, or pressure chang

181 l of repurposing auranofin to treat pressure ulcers infected with MRSA.

182 Buruli ulcer is a neglected tropical disease caused by Mycobact

183 The global distribution of Buruli ulcer is uncertain and potentially wider than currently

184 is in patients with stage IV sacral pressure ulcers is controversial.

185 Treatment for chronic diabetic foot ulcers is limited by the inability to simultaneously add

186 th that of RS8 for treatment of early Buruli ulcer lesions.

187 h RS8 in patients with early, limited Buruli ulcer lesions.

188 ostablation endoscopy documenting esophageal ulcer may identify patients at higher risk for AEF.

189 pathologies including "conventional pressure ulcers", "medical device related pressure injuries", "pr

190 dical interventions with antibiotics or anti-ulcer medications, regulate phylogenetic variability and

191 Sample clustering derived from ulcer microbial composition did not show geographical pa

192 Diabetic foot ulcer microbiota were found to exist in one of four comm

193 In both mouse ulcer models, ZINC40099027accelerated mucosal wound heal

194 Pseudoachalasia, peptic ulcer, normal gastric emptying scintigraphy (GES) and pr

195 The AUC for ulcer number was significantly lower in the Bif195 group

196 sis indicate that hospital-acquired pressure ulcers occur frequently in pediatric populations with a

197 eetable abstract: Hospital-acquired pressure ulcers occur frequently in pediatric populations, prolon

198 No episodes of corneal melts or ulcers occurred.

199 nal failure, urinary tract calculus, chronic ulcer of skin, and back problems.

200 onsenting patients with atraumatic exudative ulcers of >1 cm diameter were enrolled.

201 ss or endovascular intervention for ischemic ulcers of the lower extremities.

202 Presentation with ulcer or gangrene, age >/=65 years, female sex, large ho

203 e stage, antiretroviral regimen, and genital ulcers or cervical tenderness.

204 rom 2012 to 2017, from 83 male patients with ulcers or skin lesions, and were examined using multiloc

205 some individuals while it causes gastritis, ulcers, or cancer in others.

206 Poor healing was associated with large ulcers (P < .001) and fungal cases (P < .001).

207 jor complications were associated with large ulcers (P < .006), fungal cases (P < .001), and comorbid

208 oor visual outcome was associated with large ulcers (P < .01) and age >= 60 years (P < .02).

209 tamycin over topical voriconazole for fungal ulcers, particularly among those caused by Fusarium.

210 sment of digital ulcers and in understanding ulcer pathogenesis, and there are a wide range of treatm

211 acterial communities may contribute to HSV-2 ulcer pathogenesis, severity, or prolonged healing.

212 Importantly, in both diabetic foot ulcer patients, metagenomic sequencing identified pathog

213 tion (OR 6.97, 95% CI 6.60-7.37), and peptic ulcer perforation (OR 3.67, 95% CI 3.40-3.96).

214 tion (OR 4.33, 95% CI 4.12-4.56), and peptic ulcer perforation (OR 4.63, 95% CI 4.27-5.02).

215 erone acetate plus prednisone group (gastric ulcer perforation, sudden death, and cerebrovascular acc

216 as good: healed/improving, or poor: enlarged ulcer, perforation or transplant/glue).

217 further investigated, 11 had attended a leg ulcer/podiatry clinic.

218 aroscopic surgery (LS) for perforated peptic ulcer (PPU) disease using a National dataset BACKGROUND:

219 tion or gangrene (PEH) and perforated peptic ulcer (PPU) was analyzed, independent of HV esophageal c

220 ux-en-Y reconstruction for nonhealing peptic ulcer presented to the emergency department and reported

221 ed to synthesise and evaluate data on Buruli ulcer prevalence and distribution.

222 We did a systematic review of Buruli ulcer prevalence and used an evidence consensus framewor

223 d population-based surveys presenting Buruli ulcer prevalence estimates, or data that allowed prevale

224 receiving either a patient-centred pressure ulcer prevention care bundle (n=799) or standard care (n

225 -effectiveness of a patient-centred pressure ulcer prevention care bundle compared to standard care.

226 ' ability and willingness to follow pressure ulcer prevention interventions.

227 veness and cost-benefit analyses of pressure ulcer prevention performed from the health system perspe

228 Hospitals aiming to improve pressure ulcer prevention should focus on organizational structur

229 pathy, including pulmonary hypertension, leg ulcers, priapism, chronic kidney disease, and large-arte

230 nt jet velocity [TRV], microalbuminuria, leg ulcers, priapism, stroke, and osteonecrosis) by clinical

231 nt of the neglected tropical disease, Buruli ulcer, produces a cytotoxic macrolide, mycolactone, whos

232 One out of 10 postablation esophageal ulcers progressed to perforation, and no patient without

233 are often prescribed for patients as stress ulcer prophylaxis drugs in the intensive care unit (ICU)

234 Two stress ulcer prophylaxis strategies were compared (preferential

235 mechanical ventilation, a strategy of stress ulcer prophylaxis with use of proton pump inhibitors vs

236 to evaluate the safety of withholding stress ulcer prophylaxis.

237 Pressure ulcers (PUs) frequently occur in individuals with limite

238 Fusarium ulcers randomized to oral voriconazole had a 0.43-fold d

239 Pressure ulcer rates are persistently high despite years of resea

240 associated with 17-18% and 5-6% decrease in ulcer rates in rural and urban units respectively.

241 care interventions on unit-acquired pressure ulcer rates over 4 years controlling for community, hosp

242 Ulcer rates were measured as percentage of patients with

243 ion in which within-unit changes in pressure ulcer rates were related to the within-unit changes in t

244 interventions, nurse outcomes, and pressure ulcer rates, using unit-level data from the National Dat

245 nd nursing unit characteristics and pressure ulcer rates.

246 l wall of the patient with perforated peptic ulcer, real time reverse transcriptase polymerase chain

247 s II H. ducreyi strains persist in cutaneous ulcer regions of endemicity following mass drug administ

248 mia, obesity, low hemoglobin, gastroduodenal ulcers, rehospitalization, critical illness, thrombocyto

249 Practice Advice 5: Patients at high risk for ulcer-related bleeding from NSAIDs should take a PPI if

250 Chronic wounds including pressure ulcers represent a significant burden to patients and he

251 Diabetic foot ulcers represent a significant source of morbidity in th

252 ied when examined; three had two ulcers; all ulcers resolved after ring removal.

253 ed prognostic models for predicting pressure ulcer risk and studies evaluating the clinical effects o

254 Whilst many prognostic models for predicting ulcer risk have been developed few have been validated.

255 erived prognostic models to predict pressure ulcer risk.

256 nal ulcer, three gastritis, and four gastric ulcer samples), 10 for H. felis (one gastritis, three du

257 odenal ulcer, five gastritis, and 11 gastric ulcer samples), 13 for H. heilmannii (three gastritis, f

258 ritis, three duodenal ulcer, and six gastric ulcer samples), 20 for H. salomonis (four duodenal ulcer

259 ritis, five duodenal ulcer, and five gastric ulcer samples), and 7 for H. bizzozeronii (zero gastric

260 stage appendiceal MAA presenting as inguinal ulcers, scrotal abscesses, and other nonspecific symptom

261 Esophageal ulcer seems to precede AEF development, and postablation

262 n, and no patient without esophageal thermal ulcers showed the occurrence of perforating esophageal c

263 of the esophagus showed a 9 cm longitudinal ulcer situated 12 cm from the esophago-gastric junction.

264 , appendicitis, perforated esophagus, peptic ulcer, small bowel or large bowel, and incarcerated or s

265 , appendicitis, perforated esophagus, peptic ulcer, small bowel or large bowel, and incarcerated or s

266 teral hypermetabolic adrenal masses, gastric ulcer, small hypermetabolic adenopathies, multiple focal

267 s available to both prevent and heal digital ulcers, some of which are also used in Raynaud phenomeno

268 robial treatment approaches to the cutaneous ulcer syndrome.

269 However, their levels are lower in chronic ulcers than in acute wounds at the proliferative phase.

270 f we included the participants with improved ulcers, the healing rates increased to 94.7%, 97.3%, and

271 H species was 10 for H. suis (three duodenal ulcer, three gastritis, and four gastric ulcer samples),

272 based mathematical model of an HSV-2 genital ulcer to integrate mechanistic observations of Trm cells

273 th a persistent epithelial defect or corneal ulcer, treated with topical rhNGF, and age-matched healt

274 The randomized controlled Mycotic Ulcer Treatment Trial (MUTT) I showed a benefit of topic

275 le, 1%, and after the results of the Mycotic Ulcer Treatment Trial (MUTT) II became available, topica

276 icrobiological cure at 6 days in the Mycotic Ulcer Treatment Trial I (MUTT-I).

277 This is a secondary analysis of Mycotic Ulcer Treatment Trial II, a multicenter, double-masked,

278 n for treating various diseases like cancer, ulcers, tumor, asthma and fever.

279 es), and 7 for H. bizzozeronii (zero gastric ulcer, two duodenal ulcer, and five gastritis samples).

280 2: ulcers (2a: superficial ulcers; 2b: deep ulcers); type 3: perforation (3a: perforation without co

281 orbit and eyelids, although marginal corneal ulcers, uveitis, and epibulbar masses have also been rep

282 t of gastric cancer and duodenal and gastric ulcers, was early associated with gastric disease, but i

283 symptoms of fatigue, fever, skin rashes, and ulcers, was measured using the Systemic Lupus Activity Q

284 The PPV of small flat ulcers and large flat ulcers were 40% to 50% and 29% to 33%, respectively.

285 Ulcers were detected a mean of 32 days after ring use (r

286 Murine small bowel ulcers were generated by topical serosal acetic acid or

287 In a logistic regression analysis, ulcers were identified to be a significant predictor for

288 mural edema, perienteric fat stranding, and ulcers were independently associated with CDEIS scores;

289 between fungal and bacterial ulcers, fungal ulcers were more likely to produce severe visual impairm

290 A total of 151 patients with smear-positive ulcers were screened and 70 were enrolled at Aravind Eye

291 Four of eight participants with ulcers were symptomatic with vaginal discharge; four had

292 l damage (Lewis score) and the AUC value for ulcers were the primary and first-ranked secondary end p

293 th smear-positive filamentous fungal corneal ulcers who enrolled between May 2010 and August 2015.

294 aged 5 years or older and had typical Buruli ulcer with no more than one lesion (caterories I and II)

295 A central neurotrophic ulcer with thinning to 50% and 360 degrees of conjunctiv

296 Ulcers with active bleeding or visible vessels were foun

297 group recommends that patients with bleeding ulcers with high-risk stigmata who have had successful e

298 scores of 2 or more detected severe lesions (ulcers) with 85% sensitivity and 92% specificity (area u

299 luminal aggressors, results in erosions and ulcers, with potential complications of bleeding, protei

300 s initially presented with an atypical large ulcer without associated lymphocutaneous spread, clinica