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1 which contributes to impaired diabetic foot ulcer healing.
2 antibiotic therapy with improved venous leg ulcer healing.
3 They received enteric-coated ASA after ulcer healing.
4 n, and proliferation-essential components of ulcer healing.
5 hance mucosal barrier function or accelerate ulcer healing.
6 tivation of SRF is an important component of ulcer healing.
7 oxygenase-2 (COX-2) selective, delay gastric ulcer healing.
8 nced angiogenesis and accelerated esophageal ulcer healing.
9 increased neovascularization and accelerated ulcer healing.
10 xpression, significantly accelerates gastric ulcer healing.
11 l role for VEGF and enhanced angiogenesis in ulcer healing.
12 broblast growth factor and a reduced rate of ulcer healing.
13 c, Grb2, and Sos during experimental gastric ulcer healing.
14 n to be essential for cell proliferation and ulcer healing.
15 oblasts to evaluate their role in venous leg ulcers healing.
17 essively incorporate treatment to accelerate ulcer healing and address MRSA into the care of diabetic
21 Overall, strength of evidence was low for ulcer healing and low or insufficient for time to comple
23 gnificantly accelerated experimental gastric ulcer healing and promoted re-epithelialization and musc
24 hostin A46 treatment significantly inhibited ulcer healing and reduced EGF-R levels, EGF-R phosphoryl
25 extran sodium sulfate colitis due to delayed ulcer healing and reduced epithelial beta-catenin activa
27 h celecoxib significantly delayed esophageal ulcer healing and suppressed ulceration-triggered increa
29 role of VEGF and angiogenesis in esophageal ulcer healing, and 3) demonstrate the feasibility of gen
31 elective inhibitor, celecoxib, on esophageal ulcer healing as well as on the cellular and molecular e
32 ficantly changed at week 12 in segments with ulcer healing, based on endoscopic examination (CDEIS: 2
33 blood vessels, is crucial for gastroduodenal ulcer healing because it enables delivery of oxygen and
34 asis of solid tumors, but also for wound and ulcer healing, because without the restoration of blood
36 rvicovaginal HSV-2 DNA and slightly improved ulcer healing, but it did not decrease genital and plasm
38 in gastric ulcer margin accelerates gastric ulcer healing by promoting cell re-epithelization, proli
39 gs indicate that celecoxib delays esophageal ulcer healing by reducing ulceration-induced esophageal
40 eutic strategy that expedites chronic venous ulcer healing by treating the underlying ulcer pathophys
41 60 seconds reduced pain levels and enhanced ulcers' healing by reducing ulcers' size and duration.
42 ial agents associated with better venous leg ulcer healing compared with usual care (dressings and ba
43 Tayside, Scotland, receiving both NSAIDs and ulcer-healing drugs appeared to experience a large rise
44 ) to determine the efficacy of one exposure (ulcer-healing drugs) in preventing the serious upper gas
45 regression modeling showed that complicated ulcer healing (hazard ratio, 3.812; 95% confidence inter
46 tain whether episodic acyclovir will enhance ulcer healing if delivered at primary health care settin
48 creased crypt cell proliferation and delayed ulcer healing in GM-CSF(-/-) mice were rescued by exogen
51 n HGF (hepatocyte growth factor), to promote ulcer healing in patients with chronic limb-threatening
52 FAK activator, promotes acetic acid-induced ulcer healing in rats and may be useful in treating gast
53 cilitating epithelial cell proliferation and ulcer healing in response to injury of the colon crypt e
54 findings indicate that experimental gastric ulcer healing involves activation of EGF-R-ERK signal tr
55 ndicate that Raf-1 activation during gastric ulcer healing is Ras mediated, involves Shc-Grb2-Sos, an
58 ran sodium sulfate colitis exhibited delayed ulcer healing, more mucosal inflammation, and impaired W
59 with bleeding and/or perforations) and delay ulcer healing, presumably by blocking prostaglandin synt
63 g HCO3- secretion, the role of mechanisms in ulcer healing, the interaction of trefoil factors with t
65 as candidate surrogate markers of venous leg ulcer healing using the Curative Health Services populat
68 in the bandage group, and the proportion of ulcers healing was much the same in the two groups (70.9
69 efficacy of VEGF gene therapy in esophageal ulcer healing, we studied whether a single local injecti