ライフサイエンスコーパス: ultraviolet B

1 initially focused on the more erythemogenic ultraviolet B.

2 t A, broadband ultraviolet B, and narrowband ultraviolet B.

3 e consisting of 98.8% ultraviolet A and 1.2% ultraviolet B.

4 ated tumor necrosis factor alpha response to ultraviolet B.

5 do not completely protect keratinocytes from ultraviolet B.

6 to undergo apoptosis after irradiation with ultraviolet B.

7 more readily inhibited at higher fluences of ultraviolet B.

8 50 1B1 in human skin, and their induction by ultraviolet-B.

9 ling from perches receiving higher levels of ultraviolet-B.

10 This study demonstrates that ultraviolet B (280-320 nM) irradiation with physiologic

11 sun protection factor 15) offering primarily ultraviolet B (280-320 nm) protection.

12 yses revealed that exposure of human skin to ultraviolet-B (4 minimal erythema doses) resulted in enh

13 Ultraviolet B (40 mJ per cm2) significantly inhibited cu

14 After low daily doses of ultraviolet B (6-4) photoproduct repair was most affecte

15 We found that relatively low doses of ultraviolet-B (62.5-500 mJ per cm2) caused dose-dependen

16 d showed that the sunscreen was primarily an ultraviolet B absorber, with relatively poor absorption

17 tive oxygen species generated in response to ultraviolet B also contribute to this phosphorylation.

18 Ultraviolet B also induces perinuclear cytoplasmic reloc

19 rradiated skin to an acute "sunburn dose" of ultraviolet-B also produced significant epidermal hyperp

20 moters, including a striking synergy between ultraviolet B and added interleukin-1alpha in the induct

21 technological advances including narrowband ultraviolet B and excimer laser.

22 s been used successfully in combination with ultraviolet B and psoralen plus ultraviolet A.

23 res of nor-mal keratinocytes were exposed to ultraviolet B and showed a dose-dependent increase in nu

24 lated to offer equivalent protection against ultraviolet B and ultraviolet A.

25 cm2 ultraviolet A, 100 mJ per cm2 broadband ultraviolet B, and 3.0 J per cm2 narrowband ultraviolet

26 plants were experimentally exposed to wind, ultraviolet B, and grown in large pots in growth chamber

27 ultraviolet B, and 3.0 J per cm2 narrowband ultraviolet B, and its stability was compared with sampl

28 e associations between solar radiation, then ultraviolet B, and learning disabilities, adjusting for

29 ed upon exposure to ultraviolet A, broadband ultraviolet B, and narrowband ultraviolet B.

30 oxidant N-acetyl cysteine partially inhibits ultraviolet B-and oxidant-induced 27 kDa heat shock prot

31 was observed following exposure to doses of ultraviolet-B as low as 10 mJ per cm2.

32 n skin but are primarily designed to prevent ultraviolet-B-associated burning and damage.

33 atients exposed to high levels of tar and/or ultraviolet B before a first squamous cell carcinoma wer

34 Interleukin-1alpha plus ultraviolet B caused a remarkable 300-fold increase in -

35 When irradiated with ultraviolet-B, cells synchronized in G1 with mimosine di

36 resent at 44.3 ng per ml (SEM 6.2) following ultraviolet B compared with 26.0 ng per ml (SEM 8.0) in

37 synthesis of NO is induced by lower doses of ultraviolet B compared with that of prostaglandin E2.

38 ir positions longer on perches receiving low ultraviolet-B compared to perches receiving even slightl

39 ta from cancer genomes clearly implicate the ultraviolet B component of sunlight in melanoma skin can

40 In response to ultraviolet B damage, keratinocytes undergo apoptosis to

41 nt-independent manner associated with direct ultraviolet B DNA damage.

42 when organotypic mixtures were exposed to an ultraviolet B dose of 50 mJ per cm2, intraepithelial tum

43 protected skin (buttock site) resulted in an ultraviolet-B dose-dependent (0-4 minimal erythema doses

44 ough degradation, was induced in vivo by low ultraviolet B doses but was suppressed at erythemal dose

45 oxo-dG in normal human skin keratinocytes at ultraviolet-B doses relevant to human skin exposure.

46 odes with narrow-band compared to broad-band ultraviolet B, even when treatments are based on predete

47 the basal cell layer of the epidermis in non-ultraviolet-B exposed skin, whereas cytochrome P450 1B1

48 OR 0.89; 95% CI: 0.86, 0.93; p < 0.001) and ultraviolet B exposure (highest quintile; adjusted OR 0.

49 e protected from wind, herbivore attack, and ultraviolet B exposure and grown in restricted rooting v

50 These findings demonstrate that ultraviolet B exposure can induce the intraepithelial ex

51 Northern Europe where residents have reduced ultraviolet-B exposure and tend to have light skin color

52 yclin D1 associated kinase activity, whereas ultraviolet-B exposure had no effect on cyclin-dependent

53 higher than customary are required if solar ultraviolet-B exposure is low.

54 We show that ultraviolet-B exposure of human epidermoid carcinoma A43

55 kin tumorigenesis, we assessed the effect of ultraviolet-B exposure on cell cycle progression of tran

56 Ultraviolet-B exposure to solar-ultraviolet-protected sk

57 rization was greatly increased after chronic ultraviolet-B exposure with a significant increase of bo

58 ty incidence was associated with female sex, Ultraviolet-B exposure, and a history of diabetes.

59 One formulation contained the established ultraviolet B filter octyl methoxycinnamate, whereas the

60 sunscreen containing both ultraviolet A and ultraviolet B filters totally blocked ultraviolet-induce

61 the highest quintile of cumulative midrange ultraviolet B flux exposure versus the lowest quintile w

62 Attributable risks did not vary by age, ultraviolet B flux or hours outdoors.

63 t A and other ultraviolet exposures, such as ultraviolet B for skin cancer development in psoralen +

64 Psoriasis may be treated with ultraviolet B from lamps that have a broad emission spec

65 hemal response to narrow-band and broad-band ultraviolet B in 12 patients with psoriasis.

66 rative response of II-4 cells was altered by ultraviolet B in organotypic cultures.

67 -ultraviolet B versus placebo and narrowband-ultraviolet B in patients with nonsegmental vitiligo.

68 environmental insult, our data suggest that ultraviolet B, in addition to initiating recognized cyto

69 f other carcinogenic exposures, particularly ultraviolet B, in the development of skin cancer.

70 cultured JB6 cells, C3G was able to scavenge ultraviolet B-induced *OH and O2-* radicals.

71 e is thus the major transduction pathway for ultraviolet B-induced 27 kDa heat shock protein phosphor

72 ure, and this coincides with the reversal of ultraviolet B-induced actin reorganization.

73 89 blocks MSK1 activity but does not inhibit ultraviolet B-induced activation of MAP kinases p70/85(S

74 oyltransferase, significantly attenuated the ultraviolet B-induced apoptosis in a caspase-3-independe

75 sponsible pathway for ceramide generation in ultraviolet B-induced apoptosis of cultured normal human

76 increased de novo ceramide synthesis signals ultraviolet B-induced apoptosis, by a pathway independen

77 -chloromethyl-ketone) further attenuated the ultraviolet B-induced apoptosis.

78 ix times more sensitive than normal cells to ultraviolet B-induced apoptosis.

79 cancer is also thought to be due in part to ultraviolet B-induced impairment of immune responses.

80 cid, and phosphorylcholine revealed that the ultraviolet B-induced increase in ceramide results prima

81 y carotenoids were efficient in reducing the ultraviolet B-induced increases in the percentage of pro

82 The ultraviolet B-induced phosphorylation is not affected by

83 The ultraviolet B-induced phosphorylation is reversible, ret

84 ow that both ERKs and p38 kinase may mediate ultraviolet B-induced phosphorylation of H3 at serine 10

85 p38 kinase inhibitor, efficiently inhibited ultraviolet B-induced phosphorylation of H3.

86 l or C-terminal mutant MSK1 strongly blocked ultraviolet B-induced phosphorylation of histone H3 at s

87 These data illustrate that MSK1 mediates ultraviolet B-induced phosphorylation of histone H3 at s

88 f the nucleosomal response, totally inhibits ultraviolet B-induced phosphorylation of histone H3 at s

89 protein kinases MAP kinases are involved in ultraviolet B-induced phosphorylation of histone H3 at s

90 Ultraviolet B-induced skin cancer is also thought to be

91 which was confirmed histologically, whereas ultraviolet B-induced tanning of light-skinned swine was

92 suggested that II-4 cells were resistant to ultraviolet-B-induced alterations, which allowed these i

93 ed survivin expression and protected against ultraviolet-B-induced apoptosis.

94 Taken together, our results suggest that ultraviolet-B-induced cell cycle arrest in A431 cells is

95 ther inhibition of angiogenesis may diminish ultraviolet-B-induced cutaneous damage, wild-type and tr

96 , it is not known whether PAF is involved in ultraviolet-B-induced epidermal cell cytokine production

97 udies examined the role of the PAF system in ultraviolet-B-induced epidermal cell interleukin-8 biosy

98 Ultraviolet-B-induced erythema (one, two, or four times

99 Objective measurements of ultraviolet-B-induced erythema were performed using refl

100 xide also made a significant contribution to ultraviolet-B-induced erythema.

101 ling, which triggers increased pigmentation, ultraviolet-B-induced G1-like cell cycle arrest and cont

102 An ultraviolet-B-induced increase in H2O2 was observed in n

103 DNA is a target for ultraviolet-B-induced inhibition of contact hypersensiti

104 sts inhibited both carbamoyl-PAF-induced and ultraviolet-B-induced interleukin-8 production in the PA

105 Ascorbic acid-6-palmitate strongly promoted ultraviolet-B-induced lipid peroxidation, c-Jun N-termin

106 ould make the human skin more susceptible to ultraviolet-B-induced skin cancers or allergic and irrit

107 f the cutaneous vascular system in mediating ultraviolet-B-induced skin damage and suggest inhibition

108 e of the vascular system in the mediation of ultraviolet-B-induced skin damage, we performed quantita

109 rder to identify potential novel targets for ultraviolet-B-induced skin tumorigenesis, we assessed th

110 Because tumor necrosis factor alpha mediates ultraviolet-B-induced suppression of contact hypersensit

111 the exogenous interleukin-12 totally blocked ultraviolet-B-induced tumor necrosis factor alpha produc

112 that anti-interleukin-12 antibodies enhanced ultraviolet-B-induced tumor necrosis factor alpha secret

113 Ultraviolet-B induction of both cytochrome P450 1A1 and

114 ysis of their negative regulators, being the ULTRAVIOLET-B-INSENSITIVE4 protein and the DP-E2F-Like1

115 ight exposure and resembled that observed in ultraviolet-B-irradiated mice.

116 during oxidative stress, its accumulation in ultraviolet-B-irradiated normal human epidermal keratino

117 Ultraviolet-B-irradiated thrombospondin-1 transgenic mic

118 Skin cancer arising from exposure to ultraviolet B irradiation (UVB) from the sun is a promin

119 Ultraviolet B irradiation and traditional Chinese medici

120 ein and zeaxanthin diminishes the effects of ultraviolet B irradiation by reducing acute inflammatory

121 ison ivy or poison oak, and chronic low-dose ultraviolet B irradiation can damage the skin.

122 These data indicate that 90 mJ per cm2 of ultraviolet B irradiation induces a DNA damage response,

123 Ultraviolet B irradiation is thought to enable skin canc

124 Ultraviolet B irradiation of PAF-receptor-expressing KB

125 complish this, we have studied the effect of ultraviolet B irradiation on organotypic cultures that w

126 f this study was to characterize the role of ultraviolet B irradiation on the intraepithelial expansi

127 ct inhibited AP-1 transactivation induced by ultraviolet B irradiation or TPA in JB6 cells and AP-1-l

128 Chronic ultraviolet B irradiation significantly reduced the init

129 synthase (i.e., 1.7-fold increase 8 h after ultraviolet B irradiation), whereas serine palmitoyltran

130 ame evident in a time-dependent manner after ultraviolet B irradiation, in parallel with activation o

131 Furthermore, guinea pigs were exposed to ultraviolet B irradiation, known to enhance transport of

132 th cdk-2 and cdk-4 at 3-24 h and 6-24 h post-ultraviolet B irradiation, respectively.

133 and zeaxanthin in the cutaneous response to ultraviolet B irradiation.

134 levels of reactive oxygen species following ultraviolet B irradiation.

135 Chronic ultraviolet-B irradiation of the skin results in epiderm

136 of topical application of pTpT with those of ultraviolet-B irradiation on C57BL/6 mice sensitized to

137 thrombospondin-1 were subjected to the same ultraviolet-B irradiation regimen.

138 Mice pretreated with pTpT or ultraviolet-B irradiation showed markedly suppressed ear

139 actor alpha promoter, we examined effects of ultraviolet-B irradiation versus intradermal injection o

140 ate membranes, our data indicate that, after ultraviolet-B irradiation, H2O2 migrates from the cytoso

141 rrest in transformed keratinocytes following ultraviolet-B irradiation.

142 taneous blood vessels of mice after 10 wk of ultraviolet-B irradiation.

143 in 2 studies, subjects had received solar or ultraviolet-B irradiation.

144 Transmission of ultraviolet B is reduced by 67%-87% through vehicle and

145 er frogs on perches receiving relatively low ultraviolet-B levels maintained their positions for long

146 ed to perches receiving even slightly higher ultraviolet-B levels.

147 neous changes occur from chronic exposure to ultraviolet B light (290 to 320 nm) associated with sunb

148 Ultraviolet B light (UV-B; 280-320 nm) perception and si

149 Ultraviolet B light (UVB) causes cutaneous inflammation

150 atosus (SLE) patients are photosensitive and ultraviolet B light (UVB) exposure worsens cutaneous dis

151 Ultraviolet B light (UVB) is a common cause of human ski

152 histone H3 was enhanced by stimulation with ultraviolet B light (UVB) or epidermal growth factor (EG

153 el exercise decreased body fat and inhibited ultraviolet B light (UVB)-induced carcinogenesis in the

154 Treatment of SKH-1 hairless mice with ultraviolet B light (UVB; 30 mJ/cm(2)) twice a week for

155 entration >80 nmol/L in persons with limited ultraviolet B light exposure.

156 Ultraviolet B light induces EXO70H4 transcription in a C

157 idual cumulative lifetime ocular exposure to ultraviolet B light on cortical cataract risk for each l

158 Surprisingly, ultraviolet B light or Benzo(a)pyrene exposure of skin s

159 for vitamin D3 precursors after exposure to ultraviolet B light we found that the conversion of 7-de

160 he form of methyl jasmonate, salicylic acid, ultraviolet B light, and wounding, chosen on the basis o

161 Persons with limited exposure to ultraviolet B light, including space travelers, may not

162 s acquired through diet and skin exposure to ultraviolet B light.

163 oduce vitamin D after repeated high doses of ultraviolet B light.

164 Here we show that ultraviolet-B light (UV-B) perceived by the photorecepto

165 1 pal2 double mutants were more sensitive to ultraviolet-B light but more tolerant to drought than wi

166 3) There is new awareness of the hazards of ultraviolet-B light exposure in childhood and the subseq

167 ivin by RNA interference sensitizes cells to ultraviolet B-mediated apoptosis and results in enhanced

168 tes with ascorbic acid-6-palmitate inhibited ultraviolet-B-mediated activation of epidermal growth fa

169 adiation in skin and thus may act to augment ultraviolet-B-mediated production of cytokines such as i

170 her pTpT mimics the suppressive influence of ultraviolet-B on contact hypersensitivity, we compared t

171 that applying a sunscreen that contained an ultraviolet B only filter had no protective effect, wher

172 Solar ultraviolet B photons are absorbed by 7-dehydrocholester

173 atment of psoriasis in children, narrow band ultraviolet B phototherapy for atopic dermatitis and pso

174 on, daily treatment with a higher fluence of ultraviolet-B produced extensive hyperplasia and conside

175 sun protection factors (mainly indicative of ultraviolet B protection) quoted by the manufacturers (2

176 In contrast, ultraviolet B radiation (280-320 nm) initiates melanoma

177 Subjects who use a tanning bed that emits ultraviolet B radiation (290-315 nm) are likely to have

178 responses triggered by low fluence rates of ultraviolet B radiation (UV-B; 280-315 nm) are mediated

179 Ultraviolet B radiation (UVB) is a pro-oxidative stresso

180 To examine the association of myopia with ultraviolet B radiation (UVB; directly associated with t

181 response relation between ocular exposure to ultraviolet B radiation and cortical cataract were deriv

182 es for US cataract incidence of increases in ultraviolet B radiation due to ozone depletion.

183 Both the estimated exposure to ultraviolet B radiation during late pregnancy and the ma

184 (OH)D >/=50 nmol/L during periods of minimal ultraviolet B radiation exposure.

185 kin type and a greater likelihood of reduced ultraviolet B radiation exposure.

186 ated 3x per week for 12 wk with 15 kJ per m2 ultraviolet B radiation followed by application of the p

187 Ultraviolet B radiation has been shown to generate cutan

188 F receptor may be a pharmacologic target for ultraviolet B radiation in skin and thus may act to augm

189 Exposure to ultraviolet B radiation is an important trigger of both

190 mmatory cytokines synthesized in response to ultraviolet B radiation is the potent chemoattractant in

191 r in Antarctica at the McMurdo Station, when ultraviolet B radiation levels are essentially zero.

192 Low maternal exposure to ultraviolet B radiation may play a role in the seasonal

193 was subsequently exposed to acute, low-dose ultraviolet B radiation partially prevented tolerance in

194 own that chronic exposure to low fluences of ultraviolet B radiation reduced DNA repair capacity in m

195 dermal cell line A-431 with carbamoyl-PAF or ultraviolet B radiation resulted in interleukin-8 produc

196 When both cell lines were irradiated with ultraviolet B radiation, caspase 3 activity increased to

197 pic and exposure factors, including midrange ultraviolet B radiation, in our study populations.

198 uations, we subjected chemically- as well as ultraviolet B radiation-induced squamous papillomas in S

199 iet for 2 wk were exposed to single doses of ultraviolet B radiation.

200 altered during epidermal proliferation after ultraviolet B radiation.

201 le in the local immune aberrations caused by ultraviolet B radiation.

202 pevine during development and in response to ultraviolet B radiation.

203 to the tolerance promoted by acute, low-dose ultraviolet B radiation.

204 to the tolerance induced by acute, low-dose ultraviolet B radiation.

205 rsensitivity induction after acute, low-dose ultraviolet B radiation.

206 uman epidermal keratinocytes were exposed to ultraviolet-B radiation (290-320 nm).

207 ither oral administration of vitamin D(3) or ultraviolet-B radiation across a broad range of inputs.

208 Surface ultraviolet-B radiation and atmospheric CO2 concentratio

209 Ambient ultraviolet-B radiation can harm amphibian eggs, larvae

210 immortalized mouse keratinocytes exposed to ultraviolet-B radiation, we now report the induction of

211 se skin after chronic and acute exposures to ultraviolet-B radiation.

212 , delay the onset of skin tumors, and reduce ultraviolet-B-radiation-induced skin wrinkling.

213 that frogs called from perches receiving low ultraviolet-B regardless of perch height, and that frogs

214 ere down-regulated in cml42, and impaired in ultraviolet B resistance.

215 oredoxins, major latex protein-like protein, ULTRAVIOLET-B RESISTANCE8 photoreceptor) that contribute

216 effector molecules, we asked if pTpT mimics ultraviolet-B's upregulatory influence on tumor necrosis

217 cular analyses demonstrate that WXR3 is ROOT ULTRAVIOLET B-SENSITIVE1 (RUS1), a member of the conserv

218 ild-type mice, indicating that 86-90% of the ultraviolet B signal for keratinocyte apoptosis involved

219 iption factors following irradiation with an ultraviolet B source and solar-simulated ultraviolet irr

220 ese agents also inhibited the forskolin- and ultraviolet B-stimulated promoter activities of these ge

221 whether frogs at higher perches receive more ultraviolet-B than those calling from lower perches.

222 of these cells types to apoptosis induced by ultraviolet B, the tyrosine analog 4-tert-butylphenol, a

223 Ultraviolet A and ultraviolet B transmission were measured through thin an

224 erleukin-8 mRNA and protein in comparison to ultraviolet-B-treated control KB cells.

225 this study, keratinocytes were treated with ultraviolet-B, ultraviolet-A, or sham irradiation, witho

226 s, and giving large doses of platelets while ultraviolet B (UV-B) or gamma irradiation decreased plat

227 f absorption by BrC in the UV and diminished ultraviolet B (UV-B) radiation reaching the surface.

228 nvolved in development and translation under ultraviolet B (UV-B) stress.

229 Ultraviolet-B (UV-B) light exposure further promotes the

230 ISTANCE LOCUS8 (UVR8) is a photoreceptor for ultraviolet-B (UV-B) light that initiates photomorphogen

231 ry changes in gene expression in response to ultraviolet-B (UV-B) light through an unknown mechanism.

232 Ultraviolet-B (UV-B) photons can cause substantial cellu

233 The ultraviolet-B (UV-B) photoreceptor UV RESISTANCE LOCUS 8

234 Ultraviolet-B (UV-B) radiation affects leaf growth in a

235 ough climatic changes, increased exposure to ultraviolet-B (UV-B) radiation and increased prevalence

236 Ultraviolet-B (UV-B) radiation can have a negative impac

237 nses are induced in the cells by exposure to ultraviolet-B (UV-B) radiation.

238 h the environment, including the response to ultraviolet-B (UV-B) stress.

239 anding the molecular mechanisms by which the ultraviolet-B (UV-B)-exposed cells are being protected a

240 Solar ultraviolet B (UVB) acts as both an initiator and promot

241 the keratinocyte machinery that responds to ultraviolet B (UVB) and functions critically to convert

242 comprising ~95% ultraviolet A (UVA) and ~5% ultraviolet B (UVB) at the Earth's surface, promotes mel

243 Epidemiological studies suggest ultraviolet B (UVB) component (290-320 nm) of sun light

244 Ultraviolet B (UVB) damage is recognized as the most imp

245 The effects of a single ultraviolet B (UVB) exposure on hyaluronan content and m

246 longed nearly complete desiccation, and high ultraviolet B (UVB) exposure.

247 rradiation of human or murine epidermis with ultraviolet B (UVB) induces clones of p53-mutant keratin

248 Ultraviolet B (UVB) induces phosphorylation of histone H

249 on of two models of inflammatory pain, using ultraviolet B (UVB) irradiation alone and UVB irradiatio

250 Whereas ultraviolet B (UVB) irradiation of neonates yielded high

251 ctor for BCC is sun exposure, the ability of ultraviolet B (UVB) irradiation to activate PKD in prima

252 In response to sublethal ultraviolet B (UVB) irradiation, human keratinocytes tra

253 en studied using pure ultraviolet A (UVA) or ultraviolet B (UVB) irradiation, the signaling pathways

254 two models of inflammatory pain that involve ultraviolet B (UVB) irradiation, which can employ periph

255 A repair in normal human keratinocytes after ultraviolet B (UVB) irradiation.

256 nogens and environmental stresses, including ultraviolet B (UVB) irradiation.

257 ed MIF(-/-) BALB/c mice to acute and chronic ultraviolet B (UVB) irradiation.

258 Apoptotic NIT-1 cells were prepared by ultraviolet B (UVB) irradiation.

259 Narrow-band ultraviolet B (UVB) is an effective treatment for psoria

260 Ultraviolet B (UVB) light is the principal aetiological

261 -quadruplex forming human telomeric DNA with ultraviolet B (UVB) light results in the formation of an

262 In this study, by applying ultraviolet B (UVB) light, a physiologically relevant st

263 When irradiated with ultraviolet B (UVB) light, K14/Bcl-2 mice developed abou

264 We assessed the effects of ultraviolet B (UVB) on the p38 MAPK pathway and determin

265 factor activator protein 1 (AP-1) induced by ultraviolet B (UVB) or 12-O-tetradecanoylphorbol-13-acet

266 The ultraviolet B (UVB) portion (280-320 nm) of the ultravio

267 Sunlight contains ultraviolet B (UVB) radiation (290-315 nm) that affects

268 min D3 is generated secondary to exposure to ultraviolet B (UVB) radiation (whether from the sun or f

269 igments provide efficient protection against ultraviolet B (UVB) radiation but DNA repair also plays

270 Ultraviolet B (UVB) radiation causes cutaneous inflammat

271 Exposure to ultraviolet B (UVB) radiation from the sun can result in

272 cer in the United States, where DNA-damaging ultraviolet B (UVB) radiation from the sun remains the m

273 She was then exposed to ultraviolet B (UVB) radiation in a tanning bed wearing a

274 Solar ultraviolet B (UVB) radiation induces cutaneous ornithin

275 Ultraviolet B (UVB) radiation is a natural nonchemical s

276 SIRT1 in skin cancer development induced by ultraviolet B (UVB) radiation is dependent on its gene d

277 ry processes induced in the skin by moderate ultraviolet B (UVB) radiation on central nervous system

278 fibroblasts with the pro-oxidative stressor ultraviolet B (UVB) radiation resulted in increased reac

279 elling response to midrange UVR (280-320 nm, ultraviolet B (UVB) radiation) and UVB suppression of co

280 In addition, we found that the proportion of ultraviolet B (UVB) radiation-induced C>T transitions di

281 D8+ T cells, using monoclonal antibodies, on ultraviolet B (UVB) radiation-induced inflammation and t

282 ared to wild-type mice following exposure to ultraviolet B (UVB) radiation.

283 and promoter-specific transactivation in the ultraviolet B (UVB) response.

284 Ultraviolet B (UVB) therapy, used for the treatment of p

285 SKH-1 hairless mice were irradiated with ultraviolet B (UVB) twice weekly for 20 weeks.

286 ) analysis of nuclear extracts prepared from ultraviolet B (UVB)- and N-methyl-N'-nitro-N-nitrosoguan

287 Ultraviolet B (UVB)-induced cyclooxygenase-2 (COX-2) exp

288 lyunsaturated fatty acids (n-3 PUFA) inhibit ultraviolet B (UVB)-induced inflammation and other infla

289 xpression is a likely contributing factor in ultraviolet B (UVB)-induced non-melanoma skin cancer (NM

290 ega-3 polyunsaturated fatty acids (PUFAs) on ultraviolet B (UVB)-induced skin inflammation and photoc

291 urally occurring flavonoid silibinin against ultraviolet B (UVB)-induced skin tumorigenesis.

292 ern European subjects with multiple doses of ultraviolet B (UVB).

293 on assessed under basal conditions, or after ultraviolet-B (UVB) irradiation, or phorbol ester stimul

294 propriate response of human keratinocytes to ultraviolet-B (UVB) is dependent on the activation statu

295 lectin-3 mRNA was transiently upregulated in ultraviolet-B (UVB)-irradiated wild-type keratinocytes.

296 ta are, however, conflicting on the roles of ultraviolet B [UVB; 280-320 nanometers (nm)] and ultravi

297 hosphodiesterase 4 inhibitor, and narrowband-ultraviolet B versus placebo and narrowband-ultraviolet

298 Both ultraviolet-A and ultraviolet-B were capable of inducing interleukin-12 pr

299 f the solar spectrum (320-400 nm), devoid of ultraviolet B, were equally effective in activating immu

300 Melanocytes were relatively resistant to ultraviolet B, whereas keratinocytes were unresponsive t