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1 ation by administration of the mitochondrial uncoupling agent 2,4-dinitrophenol (DNP) in a large anim
2 ere readily resolved without pharmacological uncoupling agents, allowing identification of 4 major un
3 ps that modulate bacterial susceptibility to uncoupling agents and certain antibiotics.
4 tion were detected with blunted responses to uncoupling agents and reduced membrane potentials.
5 dependent on respiration and is prevented by uncoupling agents, and the Vmax for K+ is 1.2-1.5 microm
6  development of liver-targeted mitochondrial uncoupling agents as a potential novel therapy for lipod
7              Treatment with 2 microM AGA, an uncoupling agent, blocked the spread of Lucifer yellow i
8 cal blockade of adrenal gap junctions by the uncoupling agent carbenoxolone reduces nerve stimulation
9 tes, as well as in those treated with TNF or uncoupling agent carbonyl cyanide m-chlorophenyl hydrazo
10              Continuous superfusion with the uncoupling agent carbonyl cyanide-p-trifluoromethoxy-phe
11 ontrast, uptake of Cu-Mb is inhibited by the uncoupling agents carbonyl cyanide m-chlorophenylhydrazo
12 ing of mitochondria being confirmed with the uncoupling agent carbonylcyanidep-trifluoromethoxyphenyl
13 by hyperosmotic stress and the mitochondrial uncoupling agent, dinitrophenol, both of which lead to i
14 , including the discovery that mitochondrial uncoupling agent FCCP acts as a covalent-reversible cyst
15     Oscillations were also eliminated by the uncoupling agents heptanol, halothane or acidification.
16  cells with suramin (100 microM), a receptor-uncoupling agent, inhibited LPA-stimulated proliferation
17  mitochondrial membrane by treatment with an uncoupling agent inhibits import of the tandem tRNA subs
18 that globally targeting mitochondria with an uncoupling agent is unlikely to provide therapeutic bene
19 channels indicating that the actions of such uncoupling agents, like voltage gating, are intrinsic he
20  importantly, treatment with a mitochondrial uncoupling agent or adenosine triphosphate synthesis inh
21 gonists and antagonists or by treatment with uncoupling agents such as pertussis toxin or N-ethyl mal
22  mapping in the absence of electromechanical uncoupling agents, the method relieves a long-standing l
23 e stimulation by introducing a mitochondrial uncoupling agent to the microchannel, enabling determina
24                           Rotenone, NMDA and uncoupling agents were added to the brain slice superfus
25 he hypothesis that oxidative phosphorylation uncoupling agents would antagonize the effect of rotenon