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1 atrix gla protein was found predominantly in undercarboxylated form and was associated with the miner
2 ting vitamin K and inactive proteins such as undercarboxylated forms of factor II and osteocalcin to
3             Bone-derived osteocalcin, in its undercarboxylated, hormonal form, regulates fat depositi
4                        We notably found that undercarboxylated matrix Gla protein precisely colocaliz
5 on, and change in dp-ucMGP (dephosphorylated-undercarboxylated matrix Gla-protein).
6  resulted in decreased circulating levels of undercarboxylated OCN, impaired glucose tolerance, and r
7                            The percentage of undercarboxylated osteocalcin (%ucOC) was measured at ba
8 ce in plasma phylloquinone, percentage serum undercarboxylated osteocalcin (%ucOC), and urinary gamma
9 plasma phylloquinone and serum percentage of undercarboxylated osteocalcin (%ucOC)] and IL-6, osteopr
10 hylloquinone concentration and percentage of undercarboxylated osteocalcin (%ucOC)] was measured at b
11  between circulating osteoblast (OB)-derived undercarboxylated osteocalcin (Glu-OCN) and pancreatic b
12                                        Serum undercarboxylated osteocalcin (ucOC) and total osteocalc
13 t reduction in circulating concentrations of undercarboxylated osteocalcin (ucOC), in both males and
14 -factor II, PIVKA-II), and the percentage of undercarboxylated osteocalcin (ucOC), was determined in
15 es, serum osteocalcin [total osteocalcin and undercarboxylated osteocalcin (ucOC)], plasma phylloquin
16                 High serum concentrations of undercarboxylated osteocalcin and low serum concentratio
17 cking IR in osteoblasts have low circulating undercarboxylated osteocalcin and reduced bone acquisiti
18                            The percentage of undercarboxylated osteocalcin increased an average of 28
19 min K1-2,3-epoxide, PIVKA-II, and percentage undercarboxylated osteocalcin increased significantly be
20 e SPI-fed rats was associated with increased undercarboxylated osteocalcin secretion and altered JNK/
21  appear to result from chronic elevations in undercarboxylated osteocalcin that lead to downregulatio
22                              Osteocalcin and undercarboxylated osteocalcin were measured at baseline
23                                Percentage of undercarboxylated osteocalcin, a functional measure of v
24                      These results show that undercarboxylated osteocalcin, plasma phylloquinone, and
25 ast insulin signaling reduce serum levels of undercarboxylated osteocalcin, which in turn exacerbate
26 es in these mice are improved by infusion of undercarboxylated osteocalcin.
27 K absence or antagonists-II), and percentage undercarboxylated osteocalcin.
28 lycemia with increased levels of insulin and undercarboxylated osteocalcin.
29 rin led to the accumulation of intracellular undercarboxylated protein S forms that were rapidly secr
30 inary gamma-carboxyglutamic acid, and plasma undercarboxylated prothrombin (PIVKA-II) were measured.
31 ns of serum folate, 25-hydroxyvitamin D, and undercarboxylated prothrombin remained unchanged.