1 into fetal and neonatal mice, using control
uninjected age-matched mice.
2 exhibited dysfunction similar to hearts from
uninjected animals (P = NS, each time point).
3 ent in cone ERG amplitudes in treated versus
uninjected animals.
4 blastomere at mitotic metaphase, whereas the
uninjected blastomere progressed through mid- to late cl
5 First,
uninjected blastomeres in whole-mounts reflect and scatt
6 owth in descendants of both the injected and
uninjected blastomeres.
7 it mitosis after approximately 12 min, while
uninjected cells remained in mitosis for at least 6 h, d
8 led to their engraftment in the injected and
uninjected contralateral and ipsilateral glands.
9 jected knee joint are able to traffic to the
uninjected contralateral knee joint.
10 k edema compared to mock antigen-injected or
uninjected control corneas.
11 No
uninjected control F-IX-deficient mice survived for >7 d
12 until 60 dpi, when they reached 75% of their
uninjected control number.
13 Uninjected control rats were exposed to light at the sam
14 A groups with respect to their contralateral
uninjected control sides.
15 ht eye only, with the left eye serving as an
uninjected control.
16 l (median: >164 to >175 days) as compared to
uninjected controls (median: 53 days).
17 m peripheral nerve graft twofold relative to
uninjected controls and threefold if injections were del
18 d's adjuvant (CFA) into the TMJ or served as
uninjected controls and were killed two days after CFA t
19 , stabilized at levels 2-fold higher than in
uninjected controls for both P5 and P22 injections, and
20 82.3 +/- 9.3% (P < 0.00001) when compared to
uninjected controls.
21 educed the level of muscle degeneration over
uninjected controls.
22 s injected with NP and naked DNA compared to
uninjected counterparts.
23 tially all injected tumors and a majority of
uninjected,
distant-site tumors.
24 response thresholds in injected ears than in
uninjected ears or ears injected with control complexes
25 The result that T cells are observed in the
uninjected eye as late as day 63 p.i. suggests that T ce
26 RNFL thinning in the injected eye versus the
uninjected eye were estimated using linear mixed models.
27 L thinning in the injected versus the fellow
uninjected eye.
28 iving intravitreal injections than in fellow
uninjected eyes among patients suspected of having or co
29 However, in the
uninjected eyes of KOSTNF-infected mice, TNF-alpha expre
30 The average rate of RNFL thinning in
uninjected eyes was -0.620 mum/year (P = .029).
31 The average rate of RNFL thinning in
uninjected eyes was -0.620 um/year (P = .029).
32 The
uninjected eyes were removed.
33 Some of the
uninjected eyes were sectioned and stained for CD4+ and
34 n these mice, and the titers of virus in the
uninjected eyes were significantly increased in KOSTNF-i
35 not significantly differ from that of fellow
uninjected eyes.
36 es of infiltrating inflammatory cells in the
uninjected eyes.
37 yes injected with AAV-CBA-PEDF compared with
uninjected eyes.
38 with levels in retinas isolated from normal,
uninjected eyes.
39 mparable visual outcomes in the injected and
uninjected eyes.
40 were unchanged in 8 of 9 injected and 9 of 9
uninjected eyes.
41 with AAV-FGF-2 were increased compared with
uninjected eyes; however, these amplitudes were not sign
42 injected into the contralateral, previously
uninjected,
eyes of 11 children and adults (aged 11-46 y
43 Pre-injection IOP was analyzed in study and
uninjected fellow eyes from baseline to week 96.
44 120
uninjected fellow eyes served as controls.
45 kness measurements between injected eyes and
uninjected fellow eyes was largely within the reported n
46 In
uninjected fellow eyes, only sustained IOP >21 mmHg even
47 ompared with the respective pressures of the
uninjected fellow eyes.
48 A second group of 10
uninjected female Lewis rats served as naive controls.
49 antly greater increase in temperature on the
uninjected foot was seen during the phentolamine infusio
50 The temperature of the
uninjected foot was used to monitor the degree of alpha-
51 Both control (water-injected and
uninjected)
groups exposed to constant light maintained
52 inferior olive, and thalamus relative to the
uninjected hemisphere.
53 typical neuroendocrine axis in the conscious
uninjected horse, sheep, and human (i) illustrates proba
54 axis when transplanted to the animal pole of
uninjected host embryos, indicating that nuclear beta-ca
55 Control eyes were
uninjected,
injected with PBS, or AAV-LacZ.
56 MRI of arthritic tibiotarsal joints of the
uninjected left hindpaws from AIA rats demonstrated 2 di
57 e rate, with durability in both injected and
uninjected lesions including visceral sites, together wi
58 leens, as compared with isotype controls and
uninjected mice (12% of CD4).
59 ualize the paravascular space (PVS) in naive
uninjected mice, we show that a single wave of cortical
60 equal to or marginally greater than that in
uninjected mice.
61 was significantly decreased when compared to
uninjected neurons.
62 Ca(2+) channel facilitation when compared to
uninjected neurons.
63 acilitation significantly when compared with
uninjected neurons.
64 Uninjected normal mice served as negative controls, and
65 a 27% increase in strength as compared with
uninjected old muscles.
66 the membrane currents to a similar extent in
uninjected oocytes and in oocytes expressing AQP4, indic
67 Crystals were not formed in
uninjected oocytes, but were formed in oocytes expressin
68 Proton-activated current was not observed in
uninjected oocytes.
69 essing SLC5A8 was severalfold higher than in
uninjected oocytes.
70 led to some stimulation of glucose uptake in
uninjected oocytes.
71 by extreme hyperpolarization (> -150 mV) of
uninjected oocytes.
72 ytes compared with native (water-injected or
uninjected)
oocytes (-1.0 +/- 0.2 nA); the Na+-dependent
73 11b(+) peritoneal cells compared with 18% in
uninjected or dextran-injected mice.
74 oocytes injected with TrkA cRNA, but not in
uninjected or mock injected oocytes.
75 injected with TrkA receptor cRNA, but not in
uninjected or mock-injected oocytes.
76 ia; (ii) expression of inflammatory genes in
uninjected periosteum and muscle is significantly higher
77 early identical to the level observed in the
uninjected periosteum and muscle of historical responder
78 ultaneous elimination of the tumor at second
uninjected sites.
79 zing dose of 2, 4-dinitro-1-fluorobenzene on
uninjected skin.
80 Although the
uninjected testis contains no detectable bacteria, it un
81 nabotulinumtoxinA-injected and onabotulinumA-
uninjected tissues.
82 ses were observed among injected compared to
uninjected Tmc1(Bth/+) mice.
83 were not found in either vehicle-injected or
uninjected transgenic mice or in any nontransgenic mice.
84 human prostatic tumor and also of a distant
uninjected tumor, without associated toxicity to the mic
85 o significant inhibition of growth of other,
uninjected tumors in the same animal.
86 ced antitumor responses in both injected and
uninjected tumors, thus improving survival and tumor reg
87 driving local tumour clearance while sparing
uninjected tumours in immunodeficient mice.
88 depleted vegetal masses by induction from an
uninjected vegetal mass.
89 ty and suppressed both injected and distant,
uninjected wild-type B16.F10 melanomas.
90 and studied in comparison with injected and
uninjected WT zebrafish and heterozygotes.