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1 ly a depressive episode and looks similar to unipolar depression.
2 chotic and nonpsychotic bipolar disorder and unipolar depression.
3 nts with schizophrenia, bipolar disorder, or unipolar depression.
4 network nodes was higher in bipolar than in unipolar depression.
5 o reward system function in both bipolar and unipolar depression.
6 d to depression severity in both bipolar and unipolar depression.
7 an even greater challenge than screening for unipolar depression.
8 ol subjects and suicide cases with confirmed unipolar depression.
9 center study of sibling pairs with recurrent unipolar depression.
10 d behavior were more common in patients with unipolar depression.
11 as 4.7 times less frequent, in bipolar as in unipolar depression.
12 ive episodes within individual patients with unipolar depression.
13 esponses were compared in DSM-IV bipolar and unipolar depression.
14 the treatment of patients 75 and older with unipolar depression.
15 living among elderly patients with recurrent unipolar depression.
16 vulnerable, given their history of recurrent unipolar depression.
17 ngulate of subjects with familial bipolar or unipolar depression.
18 ) was about 60% lower in patients with major unipolar depression.
19 associated with the symptomatology of major unipolar depression.
20 xplain the familial aggregation of recurrent unipolar depression.
21 rioration of functioning among patients with unipolar depression.
22 coding of loss events, which was present for unipolar depression.
25 ith mania, 10 controls and six subjects with unipolar depression (an affective patient control group)
27 patients with treatment-resistant recurrent unipolar depression and bipolar depression, in long-term
28 rkers in individuals 18 years and older with unipolar depression and healthy control individuals were
29 adults (ages 18-60) with treatment-resistant unipolar depression and lower-than-normative explicit se
30 ression and short REM latency, probands with unipolar depression and normal REM latency, and normal c
31 ry for parents and siblings of probands with unipolar depression and short REM latency, probands with
33 se impact of anxiety on treatment outcome in unipolar depression and the paucity of data on the role
34 Two types are treatment-resistant recurrent unipolar depression and treatment-resistant bipolar depr
35 Overall, both treatment-resistant recurrent unipolar depression and treatment-resistant bipolar depr
36 hizophrenia, 25 nonpsychotic inpatients with unipolar depression, and 25 nonpatient comparison subjec
37 ncluding 27 with bipolar depression, 25 with unipolar depression, and 37 healthy comparison subjects.
38 mbers of patients matched for schizophrenia, unipolar depression, and bipolar disorder with nonpsychi
40 d only if they were diagnosed with recurrent unipolar depression; and (2) "broad," in which relatives
41 various disorders (schizophrenia, bipolar or unipolar depression, anxiety disorders, and substance us
42 and common psychiatric disorders, including unipolar depression, anxiety disorders, bipolar disorder
43 sorders (including schizophrenia, bipolar or unipolar depression, anxiety, and substance use) to matc
44 the AC7 gene with major depressive illness (unipolar depression) based on Diagnostic and Statistical
45 focus on immune alterations associated with unipolar depression, bipolar depression, and anxiety dis
46 rimary affective disorder without psychosis (unipolar depression, bipolar disorder without psychosis)
47 cidence of schizophrenia spectrum disorders, unipolar depression, bipolar disorder, and organic menta
48 ple with schizophrenia, bipolar disorder, or unipolar depression born in Denmark in 1955 or later fro
49 singly prescribed to pediatric patients with unipolar depression, but little is known about the risk
50 (2+)] was higher in bipolar disorder than in unipolar depression, but not significantly different fro
51 ers that differentiate bipolar disorder from unipolar depression, but the problem in detection of a c
53 ses that differ between bipolar disorder and unipolar depression can both inform bipolar disorder dia
54 o examined the polymorphism in our recurrent unipolar depression cases (n=1159) and control (n=2592)
55 1.57; I2 = 0%) were higher in patients with unipolar depression compared with healthy control indivi
57 ly, twin, and adoption studies indicate that unipolar depression has both genetic and environmental c
59 der was associated with an increased risk of unipolar depression (HR, 1.84; 95% CI, 1.78-1.90), psych
60 ion (HR, 1.84; 95% CI, 1.78-1.90), psychotic unipolar depression (HR, 1.97; 95% CI, 1.73-2.25), and n
61 trials comparing CBT and pharmacotherapy for unipolar depression in 1,700 patients provided individua
63 of recurrence in individuals diagnosed with unipolar depression in hospital-based settings and estim
64 iating transient mood changes are present in unipolar depression independent of clinical illness stat
68 pared with those of a group of patients with unipolar depression (n= 24) and with those of a group of
69 e and unmedicated patients with nonpsychotic unipolar depression (N=14), antipsychotic-naive patients
70 e patients with a first episode of psychotic unipolar depression (N=20), antipsychotic-naive and unme
71 iagnosis were recruited (psychosis, n = 129; unipolar depression, n = 92; post-traumatic stress disor
74 lative to that of patients with nonpsychotic unipolar depression, patients with schizophrenia, and he
75 626 patients with schizophrenia, bipolar or unipolar depression, posttraumatic stress disorder, anxi
76 ferentiation of this disorder from recurrent unipolar depression (recurrent depressive episodes) in d
77 elative HR, 1.48; 95% CI, 1.21-1.81) but not unipolar depression (relative HR, 1.08; 95% CI, 0.92-1.2
79 patients with bipolar depression and 37 with unipolar depression, similar in age and sex distribution
80 al HDAC inhibitors to treat schizophrenia or unipolar depression, there are a number of key issues th
82 s in the brain that accurately differentiate unipolar depression (UD) and bipolar depression (BD) rem
83 ty of the prefrontal cortex in patients with unipolar depression (UD) and bipolar depression (BD) usi
84 tiating bipolar disorder (BD) from recurrent unipolar depression (UD) is a major clinical challenge.
85 such as bipolar disorder type-II (BD-II) and unipolar depression (UD) is challenging due to overlappi
87 ing 1) episode onset in patients with DSM-IV unipolar depression versus community comparison subjects
89 , noninferiority trial involving adults with unipolar depression, we randomly assigned patients to re
91 gn, 113 patients with incident and prevalent unipolar depression were followed for 12 months while th
93 adults (ages 18-60) with treatment-resistant unipolar depression were randomized in a double-blind, p
94 233 women 20-60 years of age with recurrent unipolar depression were treated in an outpatient resear
95 ured in saliva samples from 39 patients with unipolar depression who had been medication free for at
96 ctors at baseline, and sampled patients with unipolar depression who sought treatment for depression
97 emission tomography in hospitalized men with unipolar depression who were administered placebo as par
98 series for a major depressive episode (77.1% unipolar depression), who had a registered treatment out
99 t by the beginning of the next century major unipolar depression will be one of the most important ca
100 was used to compare 10 unmedicated men with unipolar depression with 12 normal men during the first
101 econdary care, were older than 18 years, had unipolar depression (with a current major depressive epi
102 sorder with psychosis, but not in those with unipolar depression without psychosis when compared with
103 features that could distinguish bipolar from unipolar depression would facilitate more appropriate tr
104 ion can be clinically indistinguishable from unipolar depression yet require different treatments, it
105 e the first-line treatment for patients with unipolar depression, yet there is little guidance on whi