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1                This report demonstrates that urinary 2-MPC can be considered an A. lumbricoides-speci
2  there was a significant association between urinary 2-MPC levels and both worm counts (p = 0.023) an
3  outputs were melatonin amplitude (overnight urinary 6-sulfatoxymelatonin (aMT6s)) and timing (dim li
4                                              Urinary 6-sulphatoxymelatonin is not a reliable measure
5  reduction in fecundability among males with urinary acetaminophen concentrations in the highest quar
6 ck ascending limb (mTAL) plays a key role in urinary acid and NaCl excretion.
7 ly hyperuricemia with UA crystalluria due to urinary acidification caused tubular obstruction, inflam
8                        Baseline and incident urinary ACR >=30 mg/g were not associated with probable
9 d with probable dementia or MCI, nor did the urinary ACR modify the effect of intensive treatment on
10  emphasizes the difficulties in interpreting urinary albumin levels in early disease models.
11 cal Evaluation) trial in patients with T2DM, urinary albumin-creatinine ratio >300 mg/g, and estimate
12                                HbA1c levels, urinary albumin-creatinine ratio (p = 0.041), average ce
13 sortium provided GWAS summary data for eGFR, urinary albumin-creatinine ratio (UACR), BUN, and serum
14                                              Urinary albumin-to-creatinine ratio (ACR) is a marker of
15 imated glomerular filtration rate (eGFR) and urinary albumin-to-creatinine ratio (ACR), with cancer i
16                      Eligible patients had a urinary albumin-to-creatinine ratio (with albumin measur
17 rea, and diabetic retinopathy, or they had a urinary albumin-to-creatinine ratio of 300 to 5000 and a
18 lobin A1c levels, creatinine levels, and the urinary albumin-to-creatinine ratio were recorded.
19 e protein, sST2, galectin-3, cystatin-C, and urinary albumin-to-creatinine ratio.
20 aseline and longitudinal changes in eGFR and urinary albumin-to-creatinine ratio.
21 GFR 20-44 or 45-59 ml/min per 1.73 m(2) with urinary albumin/creatinine (ACR) >=50 mg/g and serum bic
22 e, known diabetes duration, waist/hip ratio, urinary albumin/creatinine ratio (ACR) and fasting C-pep
23 ney disease and no history of gout who had a urinary albumin:creatinine ratio of 265 or higher (with
24                                              Urinary aldosterone was measured in participants in high
25                      Mean adjusted levels of urinary aldosterone were 6.5 mug/24 h (95% CI, 5.2 to 7.
26 ivo application of secretin induced a marked urinary alkalization, an effect absent in mice lacking p
27                      The higher dose lowered urinary ammonium excretion and increased serum bicarbona
28                                         Mean urinary ammonium excretion was 25% lower and serum bicar
29 s with T2D and DR had smaller PIPR and lower urinary aMT6s than other groups (p < 0.001).
30 hagia, optic atrophy, dysarthria, as well as urinary and bowel incontinence.
31                            The ratio between urinary and fecal excretion was also altered in antibiot
32                                              Urinary and plasma d3- and d6-alpha-CEHC concentrations
33 t were guideline-concordant in uncomplicated urinary and upper and lower respiratory tract infections
34 , whereas diuretics did significantly reduce urinary angiotensinogen and beta2-microglobulin excretio
35 1; 95% confidence interval [CI], 1.18-1.23), urinary antibiotic use (RR, 1.33; 95% CI, 1.28-1.38), an
36                                    Speciated urinary arsenic (As) (inorganic + monomethyl + dimethyl
37 ction, 5 CpGs remained associated with total urinary As (pBonferroni < 0.05), located in SLC7A11, ANK
38 , methylation at 20 CpGs was associated with urinary As after false discovery rate (FDR) correction (
39 e investigated the association between total urinary As and locus-specific DNA methylation in the Str
40 ults with low-to-moderate As exposure [total urinary As, mean (+/-SD) mug/g creatinine: 11.7 (10.6)].
41           We have successfully validated our urinary biomarker panel, which was improved by substitut
42                       The performance of the urinary biomarker, tissue inhibitor of metalloproteinase
43                                     Emerging urinary biomarkers continue to show promise in evaluatin
44 ostic potential of radiological findings and urinary biomarkers derived from the urothelium of patien
45  cytokine in mouse interstitial fluid and of urinary biomarkers in patient samples.
46  (MURs) that are able to specifically detect urinary biomarkers including gamma-glutamyl transferase
47 xamined whether trimester-specific blood and urinary biomarkers of DBP were associated with small for
48 e examined longitudinal associations between urinary biomarkers of oxidant stress, 8-OH deoxyguanosin
49 f archival renal biopsies and discovered two urinary biomarkers that may be used for specific clinica
50 hthalates and compared them to corresponding urinary biomarkers.
51 ter correlation coefficients with respective urinary biomarkers.
52 tions between concentrations or detection of urinary bisphenols and morbidity outcomes and assessed h
53 uracy to within approximately 20% of in vivo urinary bladder radiotracer concentrations.
54 e users analyzed SPECT/CT images for in vivo urinary bladder radiotracer uptake using quantitative so
55 1.4 muSv/MBq for an adult male, with a 1.5-h urinary bladder voiding interval.
56 ans with the highest absorbed doses were the urinary bladder wall (0.38 mSv/MBq) and kidneys (0.054 m
57 ysiologic accumulation of radiotracer in the urinary bladder which may cause some lesions in its vici
58 nal gland resembles a kidney, connected to a urinary bladder with a nephropore (exit opening) and a c
59  1.3 cm) and a fistula between the appendix, urinary bladder, right scrotum, and right groin.
60  prognosis than urothelial carcinomas of the urinary bladder.
61 ft ventricle; liver; spleen; kidneys; bowel; urinary bladder; gluteus muscle; and malignant lesions.
62 We estimated differences in patient-reported urinary, bowel, sexual, and hormonal function-Expanded P
63           We hypothesized that posttreatment urinary BPA (uBPA) concentrations would be higher among
64 SGS lack DAF and stain positive for C3d, and urinary C3a positively correlates with the degree of pro
65 usted odds ratio, 0.13; 95% CI, 0.1-0.2) and urinary catheter (adjusted odds ratio, 0.28; 95% CI, 0.1
66 I/1000-catheter days by 63% (5.9 to 2.2) and urinary catheter days/patient by 37% (1.1 to 0.69, all P
67 ment, early ambulation, and early removal of urinary catheter) was implemented in five academic and c
68 n a third cohort, we observed an increase in urinary CCL7 levels in AKI, supporting the clinical impo
69                                              Urinary CD163 <370 ng/mmol at 6 months predicted complet
70                                              Urinary CD163 <370 ng/mmol or >370 ng/mmol perfectly agr
71                    In addition, we evaluated urinary CD163 agreement with histologic activity in 19 p
72                                Evaluation of urinary CD163 in patients with persistent proteinuria at
73                                              Urinary CD163 levels were significantly higher in patien
74                                              Urinary CD163 reflects histologic inflammation in lupus
75       We also examined relationships between urinary compounds and blood pressure (BP).
76 ormed genome-wide association studies of the urinary concentrations of 1,172 metabolites among 1,627
77                         We measured maternal urinary concentrations of 18 phthalate metabolites and 8
78 ere fit to estimate the associations between urinary concentrations of 6 chemical exposure measures (
79                                 For example, urinary concentrations of benzoic acid were higher in ca
80                                     Maternal urinary concentrations of bisphenols were not associated
81                                              Urinary concentrations of BPA, PA, and phthalate metabol
82                                We quantified urinary concentrations of eight phthalate metabolites us
83 lite groups) and clinical renal outcomes and urinary concentrations of KIM-1, NGAL, 8-OHdG, and F2-is
84                                       Higher urinary concentrations of phthalate metabolites during e
85  mice exhibited reduced plasma and increased urinary concentrations of the cationic amino acids.
86                                   Tissue and urinary (corrected for creatinine) arsenic content was h
87      Here, we examine age-related changes in urinary cortisol in a 20-y longitudinal study of wild ch
88 odality for this purpose [GFR measurement by urinary Creatinine Clearance (uCrCl) versus GFR estimati
89 ed standard and kinetic equations as well as urinary creatinine clearance.
90 locker use, BMI z-score for age and sex, and urinary creatinine.
91                       We also show that that urinary CTXII had the strongest and consistent associati
92                                 Accordingly, urinary CTXII may aid in early diagnosis of OA in sympto
93 s only 4 (serum Coll2-1 NO2, CS846, COMP and urinary CTXII) were consistently associated with establi
94                                   Cumulative urinary d3-alpha-CEHC excretion was not significantly ch
95                      Mean +/- SEM cumulative urinary d6-alpha-CEHC derived from the intravenous dose
96                                              Urinary damage markers and IL6 levels were similarly red
97                              Other blood and urinary DBP biomarkers examined were unrelated to SGA, L
98                                  Mixtures of urinary DEHP metabolites were inversely associated with
99 7.4 billion [95% CI, $75.0-$100.1 billion]), urinary diseases ($86.0 billion [95% CI, $76.3-$95.9 bil
100 p and two (1%) cases of reversible renal and urinary disorders (one case of each) occurred in the 197
101 gressive therapy with radical cystectomy and urinary diversion or trimodal therapy with maximal endos
102 n cystogene proteins are detectable in human urinary ELVs and that all three undergo post-translation
103 whereas GFR, plasma renin concentration, and urinary endothelin-1 excretion were similar between knoc
104 ers the advantages of (18)F labeling and low urinary excretion compared with (68)Ga-PSMA-11.
105 h' efficiency uptake kinetics and fractional urinary excretion of 0.025%, whereas albumin and alpha(1
106 ptake kinetics and 50-fold higher fractional urinary excretion of 1.15%.
107                                              Urinary excretion of all other analytes was unchanged be
108 glomeruli compared with normal controls, and urinary excretion of both cathepsins was significantly g
109                             Herein we report urinary excretion of the latter analytes and related fra
110 d that reduced PT length can account for the urinary excretion profile in LS.
111 mall molecules with fast blood clearance and urinary excretion.
112 umulation of [(11)C]13 and hepatobiliary and urinary excretions.
113  PKD1, PKD2 and PKHD1 genes, are abundant in urinary exosome-like vesicles (ELVs) where they form the
114 evel of glycan microheterogeneity within the urinary exosomes, finding on average 5.9 glycans per sit
115 ction, 39 cm(3) +/- 39 (39% +/- 29); maximum urinary flow rate, 6 mL/sec +/- 10 (155% +/- 293); and p
116 Score (IPSS), quality-of-life score, maximum urinary flow rate, postvoid residual volume, prostate-sp
117 nges were demonstrated (p <.05) in PSA, peak urinary flow, QoL (quality of life) questionnaire and th
118                                              Urinary function was assessed by residual urine volume,
119  identifies PIEZO2 as a key mechanosensor in urinary function.
120 , an affected individual had mildly elevated urinary galactitol, which has been linked to cataract de
121 actose challenge whereas values obtained for urinary galactose/creatinine were lower than the existin
122                           Plasma glucose and urinary galactose/creatinine were unreliable (AUC < 0.70
123                       Integrated analysis of urinary gene expression and metabolite signatures unveil
124  empagliflozin promotes osmotic diuresis via urinary glucose excretion and therefore, might offer a n
125 in reduced postprandial PG through increased urinary glucose excretion.
126             Metabolic adaptations to induced urinary glucose loss include reduced fat mass and more k
127 e sodium-glucose cotransport) further limits urinary glucose loss.
128 ite-specific changes in regard to the common urinary glycoprotein, uromodulin.
129 t first step in understanding the functional urinary glycoproteome.
130 uminuria, mesangial matrix accumulation, and urinary H(2)O(2) Administration of MTP-131 significantly
131 ficantly inhibited increases in albuminuria, urinary H(2)O(2), and mesangial matrix accumulation in d
132  association between longitudinally measured urinary hCG and early miscarriage.
133  fibrosis (CF) do not respond with increased urinary HCO(3) (-) excretion after stimulation with secr
134 lts define the mechanism of secretin-induced urinary HCO(3) (-) excretion, explain metabolic alkalosi
135 F mice showed a greatly attenuated or absent urinary HCO(3) (-)-excreting ability.
136                     Conventional methods for urinary HP analysis based on liquid chromatography with
137 MSI-CE-MS/MS as compared to total hydrolyzed urinary HP by GC-MS due to the low residual levels of fr
138 racy with a mean recovery of (93 +/- 3%) for urinary HP-G at three concentration levels with adequate
139 ias (mean bias = 15%, n = 55) when measuring urinary HP-G by MSI-CE-MS/MS as compared to total hydrol
140 dinally measured in all participants include urinary hydroxy-polycyclic aromatic hydrocarbons, volati
141  [33%-41%] to 15% [12%-20%], p < 0.001), and urinary incontinence (43% [39%-47%) to 29% [24%-35%], p
142 ), reoperation (4.1% vs. 3.0%, p = 0.758) or urinary incontinence (5.0% vs. 0%, p = 0.195).
143  (1) identify risk factors related to stress urinary incontinence (SUI) and postnatal depression (PD)
144                                       Stress urinary incontinence (SUI) is a common and bothersome co
145                                              Urinary incontinence (UI) is common among women and cont
146 mplications such as erectile dysfunction and urinary incontinence persist at high incidence rates.
147                                Self-reported urinary incontinence was worse at 1 year for those in th
148 ructive pulmonary disease (COPD), asthma, or urinary incontinence.
149 es, and emergence of bacterial resistance in urinary, intestinal, and nasal microbiota.
150 erate iodine deficiency as a baseline median urinary iodine concentration (UIC) of 50-149 ug/L.
151 sCRP) measurement, and proteomic markers and urinary isoprostanes for oxidative measures, together wi
152 after hypoglycemia (91.7 ng/mL) was seen for urinary isoprostanes.
153           Acute tubular necrosis, apoptosis, urinary kidney damage markers, neutrophil gelatinase-ass
154  metabolites were positively associated with urinary KIM-1, NGAL, 8-OHdG, and F2-isoprostane levels o
155                    In this study we evaluate urinary LAM in HIV negative, pediatric and adult, pulmon
156 ifferences were found for the sleep/fatigue, urinary (large effects) and miscellaneous NMSS domains (
157                                              Urinary levels of both T-AN and H-AN negatively correlat
158 io to undergo screening (Xpert MTB/RIF test, urinary lipoarabinomannan test, and chest radiography) t
159 ivariate model analysis showed that the only urinary markers significantly related to eGFR slope were
160  using actigraphy (motion-based) and morning urinary melatonin metabolite.
161 h as knee loading and step aerobics, altered urinary metabolism and downregulated WISP1.
162 rhythms (profiles of serum melatonin and its urinary metabolite 6-sulphatoxymelatonin).
163                               Differences in urinary metabolite levels in cases and controls at each
164                                              Urinary metabolite profiles revealed decreases in acetam
165 n the general US population by measuring its urinary metabolite, N-acetyl-S-(4-hydroxy-2-methyl-2-but
166  significantly associated with corresponding urinary metabolites (r(s) = 0.2-0.6, p < 0.05).
167 d late pregnancy differences in non-targeted urinary metabolites among FGR cases and non-FGR controls
168  16S ribosomal RNA gene sequencing and fecal/urinary metabolites by 1H-NMR spectroscopy was complemen
169 he assay robustly detects and quantifies 142 urinary metabolites including 28 amino acids and derivat
170 r results in one of the largest non-targeted urinary metabolomics study to date demonstrate the role
171                                              Urinary microbes, especially in aging populations, are a
172                                  We analysed urinary microbial ATP as a biomarker of urinary tract in
173 large-scale study exploring factors defining urinary microbiome composition in community-dwelling old
174                                          The urinary microbiome is a relatively unexplored niche that
175                                          The urinary microbiome is distinct from nearby sites and unr
176                             Several elevated urinary molecules are also expressed within the kidneys
177 r proteinuria (e.g. immunoglobulin G [IgG]), urinary nephrin excretion (podocyte injury) and serum le
178          suPAR correlated significantly with urinary nephrin, IgG and albumin levels, suggesting suPA
179                We aimed to determine whether urinary neutrophil gelatinase-associated lipocalin (uNGA
180 is demonstrated strongly increased serum and urinary NGAL levels.
181 kd1 knockout mice were associated with lower urinary nitrite/nitrate excretion and markedly increased
182 diabetes mellitus; 'K', the presence of high urinary or blood ketoacids; and 'A', a high anion gap me
183  no statistically significant differences in urinary or bowel function and quality of life.
184 he highest in RS patients with high level of urinary oxalate, which was positively correlated with ge
185  unattainable sensitivity for characterizing urinary pathogen-derived peptides.
186  predominance of Enterobacteriaceae (54%) as urinary pathogens in heart, lung, and kidney transplant
187 inine ratio: UPPod:CR) and a tubular marker (Urinary pellet aquaporin 2:creatinine ratio) were measur
188 in:nephrin mRNA ratio), podocyte detachment (Urinary pellet podocin mRNA:creatinine ratio: UPPod:CR)
189                             Podocyte stress (Urinary pellet podocin:nephrin mRNA ratio), podocyte det
190                                              Urinary pellet podocyte and tubular mRNA markers were in
191 ite/nitrate excretion and markedly increased urinary PGE(2) excretion, whereas GFR, plasma renin conc
192 ture podocytes) has a profound effect on the urinary phenotype due to its critical roles in both the
193                                   Inadequate urinary phosphate excretion can lead to severe hyperphos
194                       Persistently increased urinary phosphate excretion maintains serum phosphate le
195 ate, parathyroid hormone, FGF23, and 24-hour urinary phosphate.
196  intervention and dust control on children's urinary phthalate metabolite concentrations.
197 otho-hypomorphic mouse (kl/kl) with impaired urinary Pi excretion, low autophagy, and premature organ
198 at new branches and longer paths form on the urinary pole side.
199 yed decreased glomerular filtration rate and urinary potassium excretion associated with hyperkaliemi
200 ed biomarker of platelet COX-1 activity, and urinary prostacyclin metabolite (PGIM) excretion were me
201  glomerular filtration and tubular uptake to urinary protein excretion, we developed a mathematical m
202 T) using Michaelis-Menten kinetics and molar urinary protein measurements taken from human Dent1 dise
203 reased proteinuria over time, as measured by urinary protein:creatinine ratio.
204                                           In urinary proteins, knee loading in mice and step aerobics
205 eptide on the surface that is cleaved into a urinary reporter upon exposure to specific proteases ove
206 rts the development of activatable molecular urinary reporters (MURs) that are able to specifically d
207  in men with lower urinary tract symptoms or urinary retention secondary to benign prostatic obstruct
208 h bothersome lower urinary tract symptoms or urinary retention secondary to benign prostatic obstruct
209  potentially preventable conditions, such as urinary retention, infection, and pain.
210 s of attraction, ultrasonic vocalization and urinary scent marking, and also serves as a reinforcer i
211 onal studies have found associations between urinary sodium (UNa) with obesity, body shape and compos
212                  Study meals reduced 24-hour urinary sodium by 137+/-21 mmol (1500-mg arm) and 82+/-1
213                   Because both diets reduced urinary sodium without adverse safety or quality of life
214 rage compliance with meals was 52% (based on urinary sodium) and was not significantly different betw
215 m that can be associated with alterations in urinary solute composition including hypercalciuria.
216 icipants provided >=1 24-h urine sample, and urinary solute excretion was analyzed.
217 , i.e., kidney infection (pyelonephritis) or urinary-source bacteremia, from non-invasive UPEC, defin
218 e that increased deaths were attributable to urinary-source BSI.
219 various conditions, such as kidney disorder, urinary stone disease, urinary tract infection, and cyst
220         TURP and ThuVARP were equivalent for urinary symptom improvement (IPSS) 12-months post-surger
221 rsing home residents is complex, as specific urinary symptoms are often absent and asymptomatic bacte
222 tients were followed up by questionnaires on urinary symptoms, sexual function and impact on quality
223  also observed for symptoms/disorders of the urinary system (OR 1.36, 95% CI 1.19-1.56), asthma (OR 1
224 ures it was 93.1%, and for cultures from the urinary system it was 99.2%.
225 nt organs of the respiratory, digestive, and urinary systems and the CNS.
226    Elimination was via the hepatobiliary and urinary systems.
227                                              Urinary TCF21 concentration in human showed a positive c
228                                   Changes in urinary [TIMP-2]*[IGFBP7] following initial fluid resusc
229 easured within 12 hours after admission, and urinary tissue inhibitor of metalloproteinase-2 and insu
230                                              Urinary tissue inhibitor of metalloproteinase-2 x insuli
231                                              Urinary tissue inhibitor of metalloproteinase-2 x insuli
232       Congenital anomalies of the kidney and urinary tract (CAKUTs) represent the leading cause of ch
233                                    The lower urinary tract (LUT) and micturition reflexes are sexuall
234           Urothelial carcinomas of the upper urinary tract (UTUCs) are rare, with poorer stage-for-st
235 l roles for the urothelium in the developing urinary tract and in the genesis of CAKUTs.
236 ng) to the quantification of the severity of urinary tract and periprosthetic joint infections.
237               Endometriosis of the bowel and urinary tract are types of extragenital endometriosis th
238 owed a significant association with lung and urinary tract cancers.
239 e biomarkers and their performance in common urinary tract cancers.
240 mportant in health and ailments of the lower urinary tract cause high pathological burden.
241     Patients were excluded if they had upper urinary tract disease, urothelial carcinoma within the p
242  was consistent across infection subtypes of urinary tract infection (482 cases), cellulitis/osteomye
243 hat most commonly define catheter-associated urinary tract infection (CAUTI) and encourage further ex
244 en and frequent cause of catheter-associated urinary tract infection (CAUTI).
245  pyrexia (three [4%]), diarrhoea (two [3%]), urinary tract infection (two [3%]), and acute kidney inj
246 spitalized patients treated for pneumonia or urinary tract infection (UTI) and determine whether over
247                  A significant proportion of urinary tract infection (UTI) patients experience recurr
248     National guidelines for pneumonia (PNA), urinary tract infection (UTI), and acute bacterial skin
249 r respiratory tract infection (RTI), skin or urinary tract infection (UTI), and antibiotic prescripti
250 ys, including surgical site infection (SSI), urinary tract infection (UTI), and lower respiratory tra
251 ysed urinary microbial ATP as a biomarker of urinary tract infection (UTI), confirming the capability
252                                              Urinary tract infection (UTIs) was higher in CC group (4
253 li (ExPEC) is the leading cause in humans of urinary tract infection and bacteremia.
254 diabetes, and graft function, posttransplant urinary tract infection and rejection treatment were imp
255                                              Urinary tract infection and rejection treatment were not
256 ted intraabdominal infection, or complicated urinary tract infection caused by imipenem-nonsusceptibl
257        Antibiotic prescribing for a presumed urinary tract infection is often preceded by inappropria
258 ent) and precipitating factors (for example, urinary tract infection) for delirium have been describe
259 h as kidney disorder, urinary stone disease, urinary tract infection, and cystic fibrosis.
260     Escherichia coli is the leading cause of urinary tract infection, one of the most common bacteria
261 hospital discharge with a diagnosis code for urinary tract infection, pneumonia, cellulitis/osteomyel
262 c obstructive pulmonary disease, arrhythmia, urinary tract infection, septicemia, and stroke from 200
263 regulating an inflammatory response during a urinary tract infection.
264 n mouse models of peritonitis, pneumonia and urinary tract infection.
265  high rate of bacteriuria without documented urinary tract infection.
266 ia, in the absence of signs or symptoms of a urinary tract infection.
267  personnel and number of catheter-associated urinary tract infections (CAUTI) and central line-associ
268 ulting in false positive catheter-associated urinary tract infections (CAUTI).
269                                    Recurrent urinary tract infections (R-UTIs) are the main cause of
270 sociates with an increased risk of recurrent urinary tract infections (rUTIs) linked to uropathogenic
271                                   Diagnosing urinary tract infections (UTI) in nursing home residents
272 uropathogenic E. coli - the primary cause of urinary tract infections (UTIs) - can adhere to vaginal
273                                              Urinary tract infections (UTIs) are common, recurrent in
274                                              Urinary tract infections (UTIs) represent a major burden
275 he acute pyelonephritis that can result from urinary tract infections (UTIs), which commonly ascend f
276 range of diseases, including respiratory and urinary tract infections and bacteremia.
277 e diseases like sepsis, acute kidney injury, urinary tract infections, and HIV/AIDS.
278 a range of infections, including pneumonias, urinary tract infections, and septicemia, in otherwise h
279 t that contains bacterial species related to urinary tract infections.
280 nd capsule-deficient mutants associated with urinary tract infections.
281 ce, including Escherichia-Shigella linked to urinary tract infections.
282 olvement of the urothelium in patterning the urinary tract is supported by evidence that CAKUTs can a
283                    Moreover, the presence of urinary tract malformation was associated with the need
284 symptomatic bacteriuria commonly result from urinary tract malformations or bladder disturbances.
285 AP) and performed a comprehensive screen for urinary tract microbiota signatures, metabolite, and cyt
286 rating endometriosis involving the bowel and urinary tract on abdominal ultrasonography and shows the
287           Enterococcus spp (20%) occurred as urinary tract pathogens in kidney transplant recipients
288 tigate TURP versus ThuVARP in men with lower urinary tract symptoms or urinary retention secondary to
289  in seven UK hospitals with bothersome lower urinary tract symptoms or urinary retention secondary to
290 -menopausal bleeding, rectal bleeding, lower urinary tract symptoms, haematuria, change in bowel habi
291 plasia with good long-term results for lower urinary tract symptoms.
292 mechanosensitive ion channel PIEZO2 in lower urinary tract tissues, where it is required for low-thre
293 li are detected inside vaginal cells and the urinary tract, indicating that vaginal colonization can
294 nd/or congenital anomalies of the kidney and urinary tract.
295                                              Urinary TXM was reduced by 35% in both experimental arms
296 3) relating rs-fMRI measures to self-report (urinary urge) and physiologic measures (voided volume).
297 lated to the stimulus (volume) and response (urinary urge).
298 symptom complex characterised by symptoms of urinary urgency, increased frequency, nocturia, with or
299                                 Detection of urinary volatile organic compounds and ELISA assays show
300    The primary outcome was change in 24-hour urinary volume from baseline to week 6.

 
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