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1 QP2 plasma membrane localization and reduced urinary output.
2 ll maintained the increased water intake and urinary output.
3  mode of iron excretion, favoring fecal over urinary output.
4 ne and lidocaine group had three-fold higher urinary output (2.1 mL//kg/hr [95% CI, 1.2-3.8] vs 0.7 m
5 idneys after HMP showed significantly higher urinary output (5.31 2.06 versus 2.44 1.19 mL/min; P = 0
6 d and was the only reliable predictor of the urinary output after furosemide (area under the receiver
7 c ex vivo reperfusion, perfusate flow rates, urinary output, and oxygen consumption rates were compar
8 nts had faster creatinine recovery, superior urinary output, and reduced urinary neutrophil gelatinas
9 been shown to improve glomerular filtration, urinary output, and renal histopathology in laboratory a
10                        We identified 24-hour urinary output as a strong and easily available predicto
11                                              Urinary output doubled in 20 patients (67%) following IV
12        The number of patients who met hourly urinary output goals was higher in the computer decision
13 olute clearance (serum creatinine levels and urinary output), has been challenged by the identificati
14                                          The urinary output improved immediately in all patients, ser
15                     Fluconazole also reduced urinary output in tolvaptan-treated mice.
16 rticularly compelling because measurement of urinary output is inexpensive and routinely performed in
17                    Implementation of 24-hour urinary output leads to a substantial improvement of est
18  venous blood flow, decreases intraoperative urinary output, lowers respiratory compliance, increases
19 e metabolic acidosis [pH <= 7.15], oliguria [urinary output &lt; 200 mL/12 hr], or anuria).
20 omes (KDIGO) stage 2 (>/=2 times baseline or urinary output &lt;0.5 mL/kg/h for >/=12 hours) and plasma
21 ined subgroup of 120 patients with oliguria (urinary output, &lt; 400 ml per day), dialysis-free surviva
22 th respect to oxidative biomarkers, the 24-h urinary output of F2-Iso and 8-OHdG had median values of
23               Discrepancy between intake and urinary output of these electrolytes (i.e, UAG) indicate
24                                              Urinary output of water and electrolytes are markedly de
25  +/- 0.3 mL/kg; p < 0.001), body weight, and urinary output remained stable under decision assist and
26 , and was the only reliable predictor of the urinary output response to furosemide in acute kidney in
27      This study assessed the determinants of urinary output response to furosemide in acute kidney in
28 nary sodium excretion were not predictive of urinary output response to furosemide.
29                     Most remarkably, 24-hour urinary output showed additional prognostic value beyond
30 educed while patients were maintained within urinary output targets a higher percentage of the time.
31 use KDIGO guidelines but fail to include the urinary output (UO) criterion in their definition of AKI
32  on baseline serum creatinine (Screa) and/or urinary output (UO) criterion.
33 erations in serum creatinine (sCr) level and urinary output (UO).
34 ut a correlation with urinary osmolality and urinary output was also found during these periods.
35                  No difference in cumulative urinary output was found between the two states.
36                             Twenty-four-hour urinary output was reduced by -943 mL (95% CI: -1473, -4
37                             Twenty-four-hour urinary output was the strongest predictor of outcome am
38 rinary furosemide concentrations, and hourly urinary output were used to assess furosemide pharmacoki
39 refore, we evaluated the predictive value of urinary output within 24 hours after extracorporeal memb