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4 ion of Thz led to a significant reduction of urine calcium and phosphorus excretion, whereas urine ox
10 m calcium in the normal range with decreased urine calcium excretion (P<.05 at 2 weeks and P<.Ol at 1
11 ncreased serum 1,25-dihydroxy vitamin D, and urine calcium excretion causing idiopathic hypercalciuri
12 of hypoparathyroidism with PTH 1-34 reduces urine calcium excretion compared with treatment with cal
16 azide diuretic chlorthalidone would decrease urine calcium excretion, supersaturation, and perhaps st
17 in addition to an acidosis-induced increased urine calcium excretion, without a corresponding increas
19 hate, oxalate, and pH levels were higher and urine calcium level was lower in rats fed potassium citr
21 ively raises urine citrate levels and lowers urine calcium levels; however, the increases in urine pH
22 ce (OR 0.4, 95% CI 0.2-0.8), lack of 24-hour urine calcium measurement (OR 0.5, 95% CI 0.3-0.8), Char
23 ude 25-hydroxyvitamin D measurement, 24-hour urine calcium measurement, dual-energy x-ray absorptiome
24 Thus, the effect of potassium citrate on urine calcium oxalate and calcium phosphate supersaturat
25 ate, alone or with chlorthalidone, increased urine calcium phosphate supersaturation, but chlorthalid