1 triuresis, with significantly lower tmax for
urine sodium (
1.3 +/- 0.5 vs 3.1 +/- 2.3 hours, P < 0.02
2 molarity, and urine osmolarity corrected for
urine sodium,
also exhibited an exponential relationship
3 However, 6-hour
urine sodium and 6-hour urine volume were not different
4 Recent studies that used spot
urine sodium and associated estimating equations suggest
5 pon establishment of high grade proteinuria,
urine sodium and creatinine clearance were measured.
6 acranial pressure, blood pressure, serum and
urine sodium and osmolality, and urine output.
7 ctivity, plasma aldosterone, plasma and 24 h
urine sodium and potassium, plasma concentrations of TXB
8 ma flow, plasma renin activity, aldosterone,
urine sodium,
and baroreflex sensitivity in both groups.
9 trial with complete data on urine output and
urine sodium concentration (UNa) were analyzed.
10 The average spot
urine sodium concentration immediately prior to diuretic
11 luid intake, and increases urine osmolality,
urine sodium concentration, and plasma AVP levels.
12 mice had increased urine volume and reduced
urine sodium concentration, but regardless of the level
13 c insights into the correlation between spot
urine sodium concentrations (UNa(+)) and urine dilution.
14 only 4% of patients having low (<20 mmol/L)
urine sodium consistent with CPDSR.
15 Urine sodium:
creatinine ratio (UNa:Cr; mmol:mmol) was ca
16 assessing furosemidePK, and urine volume and
urine sodium excretion for PD analyses.
17 validation of equations to estimate 24-hour
urine sodium excretion from casual urine samples.
18 Urine volume and
urine sodium excretion increased significantly during hB
19 Twenty-four-hour
urine sodium excretion is recommended for monitoring pop
20 We estimated 24-hour
urine sodium excretion level at each time point using th
21 ur results, based on multiple assessments of
urine sodium excretion over 5 years and standardized cli
22 to 2003, and estimated a mean (+/- SE) 24-h
urine sodium excretion per person of 3526 +/- 75 mg Na.
23 de-type diuretics can more than double daily
urine sodium excretion to induce weight loss and edema r
24 Mean (SD) estimated 24-hour
urine sodium excretion was 3.01 (0.82) g per day, and 10
25 led that a 1-g increase in estimated 24-hour
urine sodium excretion was associated with increased odd
26 The decrease in
urine sodium excretion was similar in patients with POTS
27 rrelations of estimated and measured 24-hour
urine sodium excretion were modest.
28 years) from the UK Biobank examined 24-hour
urine sodium excretion, which was estimated using a sing
29 Collections of 24-h
urine sodium excretions are reliable markers for dietary
30 We estimated mean
urine sodium excretions over time for all studies and de
31 We examined temporal trends in 24-h
urine sodium excretions to estimate temporal trends in s
32 ite; ejection fraction, 26+/-8%) had 24-hour
urine sodium &
gt;=3000 mg and agreed to randomly receive ei
33 erentiated on the basis of urine osmolality,
urine sodium level, and volume status.
34 Average 24-hour
urine sodium levels were not associated with conversion
35 Casual
urine sodium measurements are relatively easy to perform
36 to perform, but their relationship to timed
urine sodium measurements is unclear.
37 Higher pre-diuretic
urine sodium to creatinine ratio (r = 0.37, P < 0.001) w
38 anaka, and Kawasaki equations] that use spot
urine sodium to estimate 24-h sodium excretion in patien
39 placement therapy, serum creatinine, and the
urine sodium-
to-creatinine ratio.
40 to-creatinine ratio; lower serum albumin and
urine sodium-
to-potassium ratio; slower rate of decline
41 ring sodium intake is based on multiple 24-h
urine sodium (
UNa) collections, which are logistically c
42 Spot morning
urine sodium was used in 4 estimating equations.