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1 fter receiving intravesical inoculation with uropathogenic Escherichia coli.
2 1 fimbriae (FimH) are positively selected in uropathogenic Escherichia coli.
3 in two different pilus biogenesis systems in uropathogenic Escherichia coli.
4 flammatory markers typically associated with uropathogenic Escherichia coli.
5 mice, but not humans, and known to recognize uropathogenic Escherichia coli.
6 ired for assembly and secretion of P pili in uropathogenic Escherichia coli.
7 of P pili by the chaperone/usher pathway in uropathogenic Escherichia coli.
8 apC usher required for P pilus biogenesis by uropathogenic Escherichia coli.
9 P pili are important virulence factors in uropathogenic Escherichia coli.
10 essential for the expression of Pap pili by uropathogenic Escherichia coli.
11 PapG adhesins mediate the binding of uropathogenic Escherichia coli.
12 plicated as a preferred binding receptor for uropathogenic Escherichia coli.
13 l-binding adhesin molecules of P fimbriae of uropathogenic Escherichia coli.
14 n ICs impacts antibacterial defenses against uropathogenic Escherichia coli.
15 immune cell composition upon infection with uropathogenic Escherichia coli.
16 protect human kidney and bladder cells from uropathogenic Escherichia coli.
17 fimbria-mediated haemagglutination assay of uropathogenic Escherichia coli.
18 prototypic self-associating AT protein from uropathogenic Escherichia coli.
19 ed significant antiadhesive activity against uropathogenic Escherichia coli (13.7 +/- 1.59 % of inhib
20 ed significant antiadhesive activity against uropathogenic Escherichia coli (13.7 1.59 % of inhibitio
23 e show that purified CdiA-CT(536) toxin from uropathogenic Escherichia coli 536 translocates into bac
24 in-containing protein C (TcpC) from virulent uropathogenic Escherichia coli, a common human pathogen.
26 ArdB encoded on a pathogenicity island from uropathogenic Escherichia coli and a KlcA from an IncP-1
27 o interfere with adhesive fiber formation in uropathogenic Escherichia coli and oligomerization of am
28 ow that the coupling of fimbrial adhesins of uropathogenic Escherichia coli and pathogenic Neisseria
29 antivirulence strategies aimed at targeting uropathogenic Escherichia coli and potentially other Qse
31 natonium), umami (monosodium glutamate), and uropathogenic Escherichia coli; and release acetylcholin
37 ytotoxic necrotizing factor type 1 (CNF1) of uropathogenic Escherichia coli belongs to a family of ba
38 e most common bacterium implicated in UTI is uropathogenic Escherichia coli, but diverse pathogens in
39 side (a ligand that promotes the adhesion of uropathogenic Escherichia coli by binding to the FimH pr
40 nt communication pathway by which strains of uropathogenic Escherichia coli can inhibit the growth of
42 ic necrotizing factor type 1 (CNF1)-positive uropathogenic Escherichia coli caused more inflammation-
43 ), but the distribution of Dr subtypes among uropathogenic Escherichia coli causing UTI among otherwi
46 ladder epithelial cells were challenged with uropathogenic Escherichia coli (CFT073) and microbial PA
48 iae are homopolymeric adhesive organelles of uropathogenic Escherichia coli composed of DraE subunits
50 ession and attenuates enterohaemorrhagic and uropathogenic Escherichia coli (EHEC and UPEC), Salmonel
51 ration of different pathogenicity islands in uropathogenic Escherichia coli, enterohaemorrhagic E. co
52 The secreted autotransporter toxin (Sat) of uropathogenic Escherichia coli exhibits cytopathic activ
54 he otherwise healthy host is the movement of uropathogenic Escherichia coli from the intestinal tract
55 expression occurred in clinical isolates of uropathogenic Escherichia coli grown in media with diffe
57 nized variants of PapG, the major adhesin of uropathogenic Escherichia coli, have been deduced in par
59 pathway analysis rank following exposure to uropathogenic Escherichia coli in two of the intercalate
62 enetic approach in female mice, we deposited uropathogenic Escherichia coli into the distal urethra t
64 (pyelonephritis- associated pilus) operon of uropathogenic Escherichia coli is a functional homolog o
66 n of pyelonephritis-associated pili (Pap) in uropathogenic Escherichia coli is epigenetically control
67 ephritis-associated pili (Pap) expression in uropathogenic Escherichia coli is regulated by a complex
71 nsporter toxin (Sat), found predominantly in uropathogenic Escherichia coli, is a member of the SPATE
72 t infections (UTIs), predominantly caused by uropathogenic Escherichia coli, is the adhesion of bacte
73 h this platform, we observed the growth of a uropathogenic Escherichia coli isolate, with an initial
78 he assembly of type 1 pili on the surface of uropathogenic Escherichia coli proceeds via the chaperon
80 ribosome-associated resistance mechanisms in uropathogenic Escherichia coli samples and generating a
83 1Cer) monoclonal antibody, and P fimbriae on uropathogenic Escherichia coli (specific for Galalpha1-4
86 s study was to determine whether OMVs from a uropathogenic Escherichia coli strain can induce cardiac
89 e gene encoding d-serine deaminase, dsdA, in uropathogenic Escherichia coli strain CFT073 results in
91 ntly, we identified a fimbrial usher gene in uropathogenic Escherichia coli strain CFT073 that is abs
93 K5(-) mutants from an O75(+) K5(+) wild-type uropathogenic Escherichia coli strain in phagocytosis as
94 vic pain behavior elicited by infection with uropathogenic Escherichia coli strain NU14 and ASB strai
95 ide triggers rugose biofilm formation by the uropathogenic Escherichia coli strain UTI89 and by enter
98 tions between the fimbrial adhesin FimH from uropathogenic Escherichia coli strains and its natural h
99 tal insights into the adhesion properties of uropathogenic Escherichia coli strains to their target r
103 ry tract infections are caused by strains of uropathogenic Escherichia coli that encode filamentous a
104 pe 1 pili are important virulence factors of uropathogenic Escherichia coli that mediate bacterial at
105 of fimbriae are the type 1 and P fimbriae of uropathogenic Escherichia coli, the major causative agen
109 the P pilus, a key virulence factor used by uropathogenic Escherichia coli to adhere to the host uri
110 onserved chaperone/usher pathway and used by uropathogenic Escherichia coli to attach to bladder cell
111 Pyelonephritis-associated pili (pap) allow uropathogenic Escherichia coli to bind to epithelial cel
114 f adhesion resulting from the interaction of uropathogenic Escherichia coli to molecularly well defin
115 on of virulence factors that are produced by uropathogenic Escherichia coli to promote pyelonephritis
116 mH, which would otherwise mediate binding of uropathogenic Escherichia coli to the host urothelium to
118 polymeric structures that mediate binding of uropathogenic Escherichia coli to the surface of the kid
119 p of the P pilus that mediates attachment of uropathogenic Escherichia coli to the uroepithelium of t
122 copolymer nanoparticles are conjugated with uropathogenic Escherichia coli type 1 pilus adhesin FimH
123 the FmlH adhesin, has been shown to provide uropathogenic Escherichia coli (UPEC) a fitness advantag
125 Urinary tract infections (UTIs) caused by uropathogenic Escherichia coli (UPEC) affect 150 million
126 analyses have detailed interactions between uropathogenic Escherichia coli (UPEC) and the bladder me
127 Urinary tract infections (UTIs) caused by uropathogenic Escherichia coli (UPEC) are a significant
133 mmon and in general are caused by intestinal uropathogenic Escherichia coli (UPEC) ascending via the
135 ction of the prostate by clinically relevant uropathogenic Escherichia coli (UPEC) can initiate and e
140 izing mice intravesically with antigens from uropathogenic Escherichia coli (UPEC) combined with a Th
142 Invasion of bladder epithelial cells by uropathogenic Escherichia coli (UPEC) contributes to ant
144 describe the whole bladder transcriptome of uropathogenic Escherichia coli (UPEC) cystitis in mice u
145 e insights into the transcriptional state of uropathogenic Escherichia coli (UPEC) during infection.
148 The metV genomic island in the chromosome of uropathogenic Escherichia coli (UPEC) encodes a putative
149 While in transit within and between hosts, uropathogenic Escherichia coli (UPEC) encounters multipl
150 Here, we show in a murine model of UTI that uropathogenic Escherichia coli (UPEC) established quiesc
151 elial cells (BECs) that expels intracellular uropathogenic Escherichia coli (UPEC) from their intrace
152 binding, invasion, and biofilm formation of uropathogenic Escherichia coli (UPEC) in the host urothe
153 s bladder epithelial binding and invasion by uropathogenic Escherichia coli (UPEC) in the initial sta
154 ity, we applied PANTOMIM to the detection of uropathogenic Escherichia coli (UPEC) in urine samples.
159 with a history of either resolved or chronic uropathogenic Escherichia coli (UPEC) infection, elucida
161 Urinary tract infection (UTI) caused by uropathogenic Escherichia coli (UPEC) is a substantial e
162 The pathogenesis of pyelonephritis caused by uropathogenic Escherichia coli (UPEC) is not well unders
176 m of initiating innate host defenses against uropathogenic Escherichia coli (UPEC) is the production
177 is isolate TOP52 was compared to that of the uropathogenic Escherichia coli (UPEC) isolate UTI89 in a
178 Here, we show that Hfq is critical for the uropathogenic Escherichia coli (UPEC) isolate UTI89 to e
180 , as a novel approach for identifying AMR in uropathogenic Escherichia coli (UPEC) isolates within on
183 urinary tract infections (UTI), cystitis by uropathogenic Escherichia coli (UPEC) occurs through an
185 rostatitis cases are caused by Gram-negative uropathogenic Escherichia coli (UPEC) or Gram-positive E
186 tandem mass spectrometry to characterize the uropathogenic Escherichia coli (UPEC) outer membrane sub
191 ated urinary tract infection (UTI) caused by uropathogenic Escherichia coli (UPEC) represents a preva
192 of 976 isolates representing 119 lineages of uropathogenic Escherichia coli (UPEC) sampled longitudin
194 ans and the human gene transgenic mouse upon uropathogenic Escherichia coli (UPEC) stimulus in vitro
196 ified two chromosomal open reading frames in uropathogenic Escherichia coli (UPEC) strain CFT073 whic
199 ed transurethrally with the cystitis-derived uropathogenic Escherichia coli (UPEC) strain UTI89.
204 re highly recurrent and frequently caused by Uropathogenic Escherichia coli (UPEC) strains that can b
209 own that 36% (5 of 14) of mice infected with uropathogenic Escherichia coli (UPEC) will have at least
210 usly shown to contribute to the virulence of uropathogenic Escherichia coli (UPEC) within the urinary
213 (UTIs), the majority of which are caused by uropathogenic Escherichia coli (UPEC), afflict nearly 60
214 tract infections (UTIs), primarily caused by uropathogenic Escherichia coli (UPEC), annually affect o
215 tract infections (UTI), primarily caused by uropathogenic Escherichia coli (UPEC), are one of the le
216 t infections (UTIs), predominantly caused by uropathogenic Escherichia coli (UPEC), belong to the mos
217 re fundamental for keeping kidneys free from uropathogenic Escherichia coli (UPEC), but we have shown
218 pe 1 fimbria is a proven virulence factor of uropathogenic Escherichia coli (UPEC), causing urinary t
219 (CNF1), a toxin produced by many strains of uropathogenic Escherichia coli (UPEC), constitutively ac
224 cal for colonization of the urinary tract by uropathogenic Escherichia coli (UPEC), mediate opposing
225 formation and remove established biofilms of uropathogenic Escherichia coli (UPEC), Pseudomonas aerug
226 st intracellular infection in the bladder by uropathogenic Escherichia coli (UPEC), the leading cause
227 nfections (UTIs) have complex dynamics, with uropathogenic Escherichia coli (UPEC), the major causati
228 estigated deployment of this pathway against uropathogenic Escherichia coli (UPEC), the major cause o
233 haracterize the adaptive immune responses to uropathogenic Escherichia coli (UPEC), the predominant u
237 ulator of stress resistance and virulence in uropathogenic Escherichia coli (UPEC), the principal cau
241 1 pili (T1P) are major virulence factors for uropathogenic Escherichia coli (UPEC), which cause both
242 pe 1 pili are important virulence factors in uropathogenic Escherichia coli (UPEC), which cause the m
244 a key event in the pathogenesis mediated by uropathogenic Escherichia coli (UPEC), yet the mechanism
260 ciation of 3 putative genes for virulence of uropathogenic Escherichia coli; uropathogenic specific p
262 ction and subsequent MALDI-MS/MS analysis of uropathogenic Escherichia coli utilizing trapped ion mob
263 Using the P and type 1 pilus systems of uropathogenic Escherichia coli, we show that a conserved
264 s from Hib pili and from P-pili expressed on uropathogenic Escherichia coli were used to predict the
265 cine to provide long-term protection against uropathogenic Escherichia coli, which cause 80% of uncom