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1 ose with esophageal, gastric, pancreatic, or uterine cancer.
2 ted at short-term follow-up for treatment of uterine cancer.
3 ncreased mortality risk in women with occult uterine cancer.
4 and most aggressive form of ovarian or extra-uterine cancer.
5 ey, liver, ovarian, pancreatic, melanoma, or uterine cancer.
6 al cancer subtypes, including PTEN-deficient uterine cancer.
7 vanced age was the strongest risk factor for uterine cancer.
8 t and progression of the most common type of uterine cancer.
9 omy for recurrence after surgical staging of uterine cancer.
10 formation for decision making for women with uterine cancer.
11 imaging to determine the anatomic origin of uterine cancer.
12 ogy and Obstetrics (FIGO) staging system for uterine cancer.
13 rotomy for comprehensive surgical staging of uterine cancer.
14 y the association between clomiphene use and uterine cancer.
15 equently inactivated in brain, prostate, and uterine cancer.
16 e involved in metastasis in both ovarian and uterine cancer.
17 s in prostate cancer, testicular cancer, and uterine cancer.
18 ing locoregional recurrence in patients with uterine cancer.
19 the initiation and metastasis of high-grade uterine cancer.
20 panic White (hereafter, White) patients with uterine cancer.
21 ching nor talc was associated with breast or uterine cancer.
22 NBS on three cancers: ovarian, bladder, and uterine cancer.
23 activating mutations in tamoxifen-associated uterine cancer.
24 te care product use and breast, ovarian, and uterine cancers.
25 and bladder, lung, pancreatic, prostate, and uterine cancers.
26 om most cancers, but are notably enriched in uterine cancers.
27 may be a useful marker for aggressive human uterine cancers.
28 eatly disproportionate amount of deaths from uterine cancers.
29 ms1, increases the chances of colorectal and uterine cancers.
31 endometrial cancer, 1 case of nonendometrial uterine cancer, 13 cases of cervical cancer, and 7 cases
33 7878 due to pancreatic cancer; 209314 due to uterine cancer; 421628 due to kidney cancer; 487518 due
35 and Relevance: Although the overall risk for uterine cancer after RRSO was not increased, the risk fo
36 ur exclusively within high-grade subtypes of uterine cancer and can drive tumorigenesis and metastasi
38 have less cardiovascular disease, breast and uterine cancer and menopausal symptoms than those eating
40 seful for determining the anatomic origin of uterine cancer and provides helpful information regardin
43 ts, unemployed women, women with ovarian and uterine cancers and choriocarcinoma, higher disease stag
45 ly nor in randomized trials for treatment of uterine cancers, and the associated toxic effects and pa
49 aneously arising estrogen-associated de novo uterine cancer, are significantly less frequent in tamox
50 with temporal and extratemporal features and uterine cancer as a prominent oncologic association.
51 leeding caused by endometrial hyperplasia or uterine cancer as a result of prolonged exposure to tumo
52 ovarian cancer mortality (OR = 1.5), and 3) uterine cancer as a risk factor for pancreatic cancer mo
54 tic, melanoma, kidney, bladder, gastric, and uterine cancer being treated in the first line were incl
55 ith a confirmed histopathologic diagnosis of uterine cancer between April 1, 2000, and March 31, 2009
56 ung cancer, endocrine therapy with increased uterine cancer (but reduced contralateral breast cancer)
57 as been identified as a potent suppressor of uterine cancer, but the biological modes of action of LK
62 g either HMGA1a or COX-2 in high-grade human uterine cancer cells blocks anchorage-independent cell g
63 inds directly to the COX-2 promoter in human uterine cancer cells in vivo and activates its expressio
64 Significance: Elevated IGFBP2 secretion by uterine cancer cells with heterozygous PPP2R1A mutations
66 mong Hispanic men and women and pancreas and uterine cancer deaths among Hispanic women increased fro
67 tage at diagnosis with racial disparities in uterine cancer deaths at the population-based level are
68 71.5%] White individuals), there were 16 797 uterine cancer deaths between 2010 and 2017, correspondi
69 spective cohort study included patients with uterine cancer diagnosed from 2000 to 2019, from 17 Surv
72 ely affect prognosis of patients with occult uterine cancer, empirical evidence has been limited and
73 d White patients who received a diagnosis of uterine cancer from January 1, 2004, to December 31, 201
76 he Black population, with the highest HR for uterine cancer (HR, 1.87; 95% CI, 1.63-2.15), and for 7
77 ons may explain in part the reduced rates of uterine cancer in Asian countries compared with those in
78 erms of detecting the presence or absence of uterine cancers in women with abnormal uterine bleeding.
81 ted that laparoscopic surgical management of uterine cancer is superior for short-term safety and len
84 tion between tamoxifen therapy and secondary uterine cancers is uncommon but well established; howeve
85 estrogen and progesterone receptors, but in uterine cancers, it is likely no longer under control of
87 association of subtype-specific trends with uterine cancer mortality and with the role of tumor subt
88 both men and women in all US census regions, uterine cancer mortality increased in all regions for wo
90 5RHH-siAXL treatment reduces metastasis in a uterine cancer mouse xenograft model, without causing an
91 BMI was associated with an increased risk of uterine cancer (OR 1.10, 95% CI 1.05-1.15; p < 0.001) an
92 to 65,499 single nuclei from samples of five uterine cancer patients and validate the clustering usin
94 efits of minimally invasive hysterectomy for uterine cancer, population-level data describing the pro
97 ved kappa s of 0.36 and 0.25 for ovarian and uterine cancers, respectively, exceeded chance expectati
98 results suggest that clomiphene may increase uterine cancer risk (rate ratio (RR) = 1.79, 95% confide
100 sitive associations between BMI and lung and uterine cancer risk as well as the inverse association b
105 cation of PhyloFrame to breast, thyroid, and uterine cancers shows marked improvements in predictive
108 sted associations of race and ethnicity with uterine cancer-specific survival (primary outcome) in th
109 Location-stratified analyses found worse uterine cancer-specific survival among Black patients co
113 equently inactivated in brain, prostate, and uterine cancers that acts as a phosphatase on phosphatid
114 echanism is specific to tamoxifen-associated uterine cancer, the concept of treatment-induced signali
116 ing that longer wait times from diagnosis of uterine cancer to definitive surgery have a negative imp
118 ciated with incontinence and falls; cervical/uterine cancer was associated with falls and osteoporosi
122 dian age 45.6 (IQR: 40.9 - 52.5), 8 incident uterine cancers were observed (4.3 expected; observed to
124 acial survival disparity among patients with uterine cancer, while insurance status represented the m
125 database to identify women with stage I-III uterine cancer who underwent hysterectomy from 2006 to 2
126 g cancer diagnosis by 2030, and melanoma and uterine cancer will become the fifth and sixth most comm
127 uates the overall survival for patients with uterine cancer with distant organ metastasis treated wit
130 ultimately developed bulky endometrioid-type uterine cancers with 100% mortality by 8 months of age.
131 s likely to have thromboembolic sequelae and uterine cancer), women without a uterus, and women at hi