ライフサイエンスコーパス: vagina

1 clinically without further treatment of the vagina.

2 alic acid-containing mucus components in the vagina.

3 ay be more likely to have involvement of the vagina.

4 ents of the human gastrointestinal tract and vagina.

5 herniation of the uterus into or through the vagina.

6 five swabs from the lateral wall of the mid-vagina.

7 sity as physician collected swabs of the mid-vagina.

8 ivity on bone, plasma lipids, hot flush, and vagina.

9 precursor lesions in both the cervix and the vagina.

10 red the titers of the challenge virus in the vagina.

11 nized monkeys was minimal and limited to the vagina.

12 monads is preparatory to colonization of the vagina.

13 ated in milk and C. pecorum dominated in the vagina.

14 eficiency virus (SHIV) infection through the vagina.

15 appeared to be nonspecific in the uterus and vagina.

16 primordia of the oviducts, uterus and upper vagina.

17 ell as epithelia of the oviduct, cervix, and vagina.

18 for sustained long-term colonization of the vagina.

19 primary role in creating the acidity of the vagina.

20 ut did not entirely prevent infection of the vagina.

21 spp. and is therefore similar to that of the vagina.

22 els after controlled topical delivery to the vagina.

23 lective homing of T and B lymphocytes to the vagina.

24 approximately 1% of the concentration in the vagina.

25 in mucosal fluids of the eye as well as the vagina.

26 unction was not entirely eliminated from the vagina.

27 ns were not detected in either the uterus or vagina.

28 elia such as the oral cavity, esophagus, and vagina.

29 intermediate state between normal uterus and vagina.

30 did not increase plasma cell numbers in the vagina.

31 and specific IgG in both the cervix and the vagina.

32 lis to the mucous membrane of the urethra or vagina.

33 actors that influence drug movement into the vagina.

34 4) known bacteria formerly isolated from the vagina.

35 ucts and sinovaginal bulbs, give rise to the vagina.

36 eutrophil transepithelial migration into the vagina.

37 eral human niches such as the mouth, gut and vagina.

38 e from semen, 69 from urine, and 21 from the vagina.

39 factors and secreted proteins in the macaque vagina.

40 ations: caries lesions, the stomach, and the vagina.

41 ations through natural orifices, such as the vagina.

42 phonuclear neutrophil (PMN) migration to the vagina.

43 enuated N. gonorrhoeae colonization of mouse vaginas.

44 attenuates gonococcal colonization of mouse vaginas.

45 persistent interdigital webs and imperforate vaginas.

46 he endocervix and by patients from their own vaginas.

47 ethra/urine (83%, kappa=0.87) and endocervix/vagina (100%, kappa 1.0) (p<0.005 for all comparisons).

48 ty, nares, skin, gastrointestinal tract, and vagina (15 specimens from men and 18 from women).

49 cted in lymph nodes (3/3), uterus (2/3), and vagina (2/3) in PR isolate-infected dams.

50 ce receiving recombinant IFN-alphaA (5-500 U/vagina) 24 h PI showed no significant survival in compar

51 highest in the skin (61.3%), followed by the vagina (41.5%), mouth (30%), and gut (17.3%).

52 were dominated by bacteria from the mother's vagina (46%), ambient air (31%), and the sheep pen floor

53 ess common for cancers of the cervix (3.3%), vagina (8.3%), vulva (1.5%), and penis (8.3%).

54 docervix, 65 and 40%; urine, 72 and 24%; and vagina, 81 and 72%.

55 , cervix plus urine, 86 and 49%; cervix plus vagina, 91 and 93%; and vagina plus urine, 94 and 79%.

56 ory mice showed high in vivo CTL activity in vagina, a strong recall response, and robust protection

57 fic CD8(+) T cells in any tissues except the vagina after challenge.

58 54 cases of patients with artificial vagina after laparoscopic peritoneal vaginoplasty were i

59 Lymphocyte recruitment to the vagina after virus challenge appeared to involve memory

60 the intestine, oral cavity, nasopharynx, and vagina all have associated commensal flora.

61 The distal vagina also showed increased vascularization and perivas

62 nsenii, are common and abundant in the human vagina and absent from other habitats.

63 of nectin-1 in epithelial cells of the mouse vagina and also in neuronal cells of the central nervous

64 main supportive structures of the uterus and vagina and are often attenuated in women with POP.

65 re part of the normal bacterial flora of the vagina and are typically considered contaminants when cu

66 to what was reported for studies of HSV-2 in vagina and brain and suggests that receptor requirements

67 EM is dispensable for HSV-2 infection of the vagina and brain, but is required for WT pathogenesis of

68 imary entry receptors for HSV-2 in the mouse vagina and brain.

69 ) and clear cell adenocarcinoma (CCA) of the vagina and cervix is well known, yet there has been no s

70 me animals) of the SIV-infected cells in the vagina and cervix were Langerhans' cells.

71 uctive age, the microbiota of the lower FRT (vagina and cervix) microenvironment is dominated by Lact

72 ere preferentially found in the lower tract (vagina and cervix), whereas APCs and innate lymphoid cel

73 ased the number of viral target cells in the vagina and cervix, suggesting that the ratio of target c

74 was decreased in the laminae propriae of the vagina and cervix.

75 message was invariantly expressed by normal vagina and ectocervix and inflamed fallopian tube, but v

76 lls and silence gene expression in the mouse vagina and ectocervix for at least nine days.

77 of the stratified squamous epithelium of the vagina and ectocervix, with the intensity of cellular st

78 e of Lactobacillus strains isolated from the vagina and enhances the color production of H(2)O(2)-pro

79 echanism that coordinates development of the vagina and feminization of the urethra, which may accoun

80 Two sites, the vagina and gastrointestinal tract, are highlighted to ex

81 that inhabit key body niches, including the vagina and gastrointestinal tract.

82 were the least stable, whereas those in the vagina and gut were the most stable.

83 aginal development, including an imperforate vagina and hydrometrocolpos.

84 it is unknown whether ZIKV replicates in the vagina and impacts the unborn fetus.

85 y reduced C. muridarum survival in the mouse vagina and increased C. muridarum susceptibility to vagi

86 reased the number of IgG plasma cells in the vagina and increased vaginal secretion/serum titer ratio

87 l septum in adolescents with Obstructed hemi-vagina and ipsilateral renal agenesis (OHVIRA) syndrome.

88 ell types in the oviduct, uterus, cervix and vagina and is dependent upon specific mesenchymal-epithe

89 oundary structure between the uterus and the vagina and is key for the maintenance of pregnancy and t

90 de (H(2)O(2))-producing species found in the vagina and is under development as a probiotic for the t

91 meters both into the posterior fornix of the vagina and on the skin at the beam entrance site.

92 These may be adapted to the vagina and possess characteristics enabling them to thri

93 tion defined as reduced bacterial numbers in vagina and prevention of pathological changes in the upp

94 Upon infection, pDCs are recruited to the vagina and produce large amounts of type I IFNs in a TLR

95 suggest that the mucosal environments of the vagina and rectum both impose a strong selection for the

96 Cocolonization of the vagina and rectum by H(2)O(2)-producing lactobacilli was

97 ctions of challenge virus replication in the vagina and reduced latent viral loads in dorsal root gan

98 of innervation of epithelium of the proximal vagina and reduced proportions of VIP, CGRP, and SP cont

99 that IgG viral antibody was produced in the vagina and released into vaginal secretions at 6 weeks a

100 including stratified squamous epithelia from vagina and skin, as well as cuboidal epithelium from lun

101 s, and can colonize the periurethral area or vagina and subsequently ascend through the urethra to th

102 essing high levels of CCR5 reside within the vagina and that these cells are preferentially targeted

103 acterised by an abnormal opening between the vagina and the bladder or rectum, which results in conti

104 he squamous epithelium of the ectocervix and vagina and the columnar epithelium of the endocervical c

105 tion of immune effector function between the vagina and the periphery.

106 hat telomerase activity, particularly in rat vagina and uterus, appears to be associated with a cell

107 avity and esophagus, gastrointestinal tract, vagina and vascular system of humans.

108 The small-cell carcinomas of the vagina and vulva need to be distinguished from Merkel ce

109 ecific concordance suggests that the cervix (vagina) and anus may serve as reservoirs for HPV infecti

110 cus, and groin) and their mothers (mouth and vagina) and were obtained from infants weekly until they

111 usly isolated from body sites other than the vagina, and (4) known bacteria formerly isolated from th

112 fered from those of the oral cavity, rectum, vagina, and background DNA controls, but not of the cerv

113 erforate anus with confluence of the rectum, vagina, and bladder in a urogenital sinus.

114 sentative UTI isolate present in the rectum, vagina, and bladder of a woman with UTI was chosen as th

115 Following analysis of primary cervix, vagina, and first-void female urine specimens for Chlamy

116 tices (cleaning with the fingers, wiping the vagina, and inserting traditional substances) are associ

117 aea have been isolated from the human colon, vagina, and oral cavity, but have not been established a

118 that can harmlessly colonize the human gut, vagina, and rectum but can also cause pneumonia, sepsis,

119 ains from the three body sites (oral cavity, vagina, and rectum), regardless of host conditions.

120 ntrations of bacteria present in the rectum, vagina, and small intestine.

121 cells of primitive neurons, the cervix, the vagina, and the endometrium in 5- to 400-fold higher num

122 cit functional and structural effects on the vagina, and therefore E4 appears promising as a therapeu

123 al tumor at all sites except paratesticular, vagina, and uterus, or with alveolar RMS were randomly a

124 ell carcinomas of the cervix, ovary, uterus, vagina, and vulva.

125 ium, ovary, penis, prostate, rectum, testis, vagina, and vulva.

126 ary lymphoid follicles in the ectocervix and vagina; and 3) concentrated on the path of virus entry b

127 carcinomas and premalignancies of the vulva, vagina, anus, and oropharynx.

128 e 16) can cause cancer of the cervix, vulva, vagina, anus, penis, and oropharynx.

129 gulate development of the female urethra and vagina are largely unknown.

130 we demonstrated that these E4 effects on the vagina are mediated by nuclear ERalpha activation.

131 agement of cancers of the cervix, vulva, and vagina are reviewed.

132 Mucosal surfaces of the vagina are the portals for heterosexual transmission of

133 ns of peptide-containing nerves in the mouse vagina are unknown.

134 uding the oviducts, uterus, cervix and upper vagina, are derived from the Mullerian ducts, a pair of

135 innervate the distal urethra and the distal vagina, as well as the clitoris and perigenital skin and

136 f immunoglobulin G (IgG) plasma cells in the vagina at 6 weeks and 10 months after immunization, wher

137 hat vaginal lactobacilli could reacidify the vagina at the rate observed postcoitally following neutr

138 n serum and detectable levels of MAbs in the vagina at the time of infection, there was only modest p

139 rest in the vaginal stroma; as a result, the vagina becomes more vulnerable to pathogens.

140 HPV can be detected in the vagina before first sexual intercourse, highlighting the

141 have documented the detection of HPV in the vagina before first vaginal intercourse.

142 xynol 9 or a placebo film, inserted into the vagina before intercourse.

143 kin origin (e.g. that of cornea, oesophagus, vagina, bladder, prostate) that express the transcriptio

144 clear p63 protein was localized to the skin, vagina, bladder, urethra, and basal columnar cells of th

145 ation (> or =10(5) cfu/mL) of E. coli in the vagina (both, P<.001) and urine (all <10(5) cfu/mL; P=.0

146 duced a strong IgG response in the serum and vagina but was inefficient in generating a mucosal IgA r

147 rkedly reduced T cells in blood, spleen, and vagina, but major histocompatibility complex class II an

148 t implications regarding colonization of the vagina by G. vaginalis and may suggest an explanation fo

149 g sustained local drug concentrations in the vagina can be challenging, due to the high permeability

150 rgent epithelial cell differentiation in the vagina, cervix, and urinary tract.

151 lls were detected in the blood, lymph nodes, vagina, cervix, uterus, and fallopian tubes.

152 llenge with herpes simplex virus in skin and vagina challenge models, and were clearly superior to th

153 resulted in an inflammatory response in the vagina characterized by neutrophils and infiltrating sub

154 s significantly higher on CD4 cells from the vagina, compared with those from blood.

155 o weeks after birth support that the porcine vagina continues to develop postnatal.

156 an initial elevation in myeloid cells in the vagina (day 3) and uterine horns (day 7), followed by a

157 as varied individually in shape and that the vaginas demonstrated both significant directional and fl

158 Development of the vagina depends on sexual differentiation of the urogenit

159 Twenty-two patients had no discernible vagina (dimple or less).

160 estation and monthly after delivery from the vagina, distal gut, saliva, and tooth/gum.

161 yed bimodal (e.g., gut) or multimodal (e.g., vagina) distributions of sample abundances, but not all

162 r titers in the corneas, trigeminal ganglia, vaginas, dorsal root ganglia, spinal cords, and brains o

163 th contributing to the overall health of the vagina due to its direct and indirect effects on pathoge

164 a strong IL-17-related gene signature in the vagina during estrogen-dependent murine VVC.

165 applying antiseptic washes to the cervix and vagina during labor, are in progress.

166 es in elastic fiber homeostasis in the mouse vagina during pregnancy and parturition were determined.

167 In the adult vagina, E(2) induced expression of involucrin, a CCAAT/e

168 Epithelial cells derived from normal human vagina, ectocervix, and endocervix expressed mRNA for TL

169 the kidney and the epithelial layers of the vagina, ectocervix, endocervix, uterus, and fallopian tu

170 -related changes in innervation of the mouse vagina, effecting the distribution of neuropeptides with

171 lassical theory of Mullerian origin of upper vagina fails to explain complex urogenital malformations

172 ed body sites (gut, skin, oral, airways, and vagina), focusing on interpersonal and intrapersonal var

173 w outlines the anatomy and physiology of the vagina, focussing on areas relevant to drug delivery.

174 ting the ability of E. coli to reside in the vagina following UTI.

175 administer pharmaceutical drugs to the human vagina for periods typically ranging from three weeks to

176 f K5-expressing tissues, including the skin, vagina, forestomach, and odontogenic epithelium.

177 omized rats (very low proliferation) than in vagina from ovariectomized and estradiol-treated rats (h

178 The high telomerase levels observed in vagina from ovariectomized rats indicates that the same

179 re compared from the lymph nodes, blood, and vagina from uninfected and simian immunodeficiency virus

180 ward nanoparticle-based drug delivery in the vagina has been focused on HIV prevention, strategies fo

181 anti-candida host defence mechanisms in the vagina has developed slowly and, despite a growing list

182 Epithelial delivery of medication across the vagina has proven successful for administration of small

183 Normal vaginas have stratified epithelium and normal uteri have

184 ccurs when semen temporarily neutralizes the vagina, HIV maintained its native surface charge and dif

185 odes but not the ovaries, uterus, cervix, or vagina in FP isolate-infected dams.

186 se to the prostate in the male and the sinus vagina in the embryonic female.

187 We compared cervical and vulvar areas of the vagina in young nullipara and older multipara C57Bl/6 mi

188 le lower reproductive tracts (ie, cervix and vagina) in the human papillomavirus transgenic mouse mod

189 oral cavity, amniotic fluid, endometrium, or vagina (including women with bacterial vaginosis), were

190 mmunolabeling of ganglia associated with the vagina indicated the likely origin of some peptidergic f

191 Entry of bacteria from the vagina into the uterus raises the question of uterine ep

192 areas in intravaginal drug delivery, but the vagina is a challenging route of administration, due to

193 In women, a healthy, patent vagina is important for the maintenance of a good qualit

194 The vagina is innervated by a complex arrangement of sensory

195 that a similar acute PMN migration into the vagina is mediated by chemotactic S100A8 and S100A9 alar

196 ermine the prevalence of each species in the vagina, its association with BV, and the utility of PCR

197 oliferation and indicators of cellularity in vagina, mammary gland, and uterus from virgin, pregnant,

198 The most common tumor sites were vagina (n = 7), pelvis/retroperitoneum (n = 6), and blad

199 he pharynx (n=93), rectum (n=88), endocervix/vagina (n=89), and urethra/urine (46).

200 sitive at the mouth, n = 26; positive at the vagina, n = 18; positive at both sites, n = 10) at the t

201 tivity was significantly higher (P=0.003) in vagina obtained from ovariectomized rats (very low proli

202 atients (4.6%), of which mesh erosion to the vagina occurred in 7 patients (1.3%).

203 re simultaneous maternal colonization of the vagina (odds ratio [OR], 4.50; 95% confidence interval [

204 t to establish a productive infection in the vagina of 75% +/- 17% and in the spinal cord of 71% +/-

205 s) is a commensal of the human intestine and vagina of adult women but is the leading cause of invasi

206 the controlled release of lactic acid in the vagina of BV-infected women.

207 f herpesvirus entry mediator nectin-1 in the vagina of human and mouse at different stages of their h

208 ological and functional impacts of E4 on the vagina of ovariectomized mice, and we determined the mol

209 Swab samples were obtained from the lower vagina of participants at admission for delivery and ino

210 the oviduct, uterus and upper region of the vagina of the female reproductive tract.

211 oduced no pregnancies because the uterus and vagina of the MOER female mice were extremely atrophic.

212 S. pneumoniae was recovered from the vagina of the mother on a swab culture collected prior t

213 quences were also found within the colon and vagina of the same animal, and within the peripheral blo

214 entical to the E. coli found in the urine or vagina of their sex partner.

215 gens isolated from the rectum, urine, and/or vagina of women with UTIs and 54 E. coli isolates from t

216 h EVs released by lactobacilli isolated from vaginas of healthy women.

217 hydrogenase-C4 antibodies were placed in the vaginas of mice.

218 , activated mononuclear blood cells into the vaginas of mice; four hours later, numerous double-stain

219 ols used a pressure device placed within the vagina or anal canal, or electromyographic (EMG) sensors

220 ty of virus transmission when applied to the vagina or rectum of a person at risk of sexually acquiri

221 bserved in non-skin tissues (e.g. in cornea, vagina or thymus).

222 he vaginal apex below the upper third of the vagina, or anterior or posterior vaginal wall prolapse b

223 s in at least one of three body sites (nose, vagina, or anus), with approximately 9% colonized vagina

224 omplex, connective-tissue attachments of the vagina, or both, resulting in prolapse.

225 on the cervix, and distention of the uterus, vagina, or colon.

226 sites of keratinized epithelium, such as the vagina, or for adherence of these bacteria to damaged ep

227 r concentrations of BVAB in the mouth, anus, vagina, or labia prior to BV predict risk of incident BV

228 cally and/or mucosally into the nose, mouth, vagina, or rectum in a 4-dose schedule, followed by 2 do

229 risk types to be first detected in the vulva/vagina (P = .03).

230 arynx, rectum, urethra/urine, and endocervix/vagina paired specimens, respectively.

231 oof that Langerhans cells (LCs) in the human vagina participate in dissemination of infectious human

232 letion of CD4(+) T cells was observed in the vagina, particularly among the CCR5(+)CD4(+) subset.

233 ions obtained through the middle part of the vagina, perineal body, and anal canal.

234 ge seen in the Lactobacillus-dominated human vagina (pH 3.6 to 4.5 in most women).

235 and 49%; cervix plus vagina, 91 and 93%; and vagina plus urine, 94 and 79%.

236 ) were present at high concentrations in the vagina prior to treatment.

237 tious organisms (3 log(10)) from the cervico-vagina, produced a minimal inflammatory response in urog

238 ption factor for BMP4 signaling, was high in vagina-projecting sensory neurons after ovariectomy and

239 ry responses to infection in both cervix and vagina, recruits CD4(+) T cells to fuel this obligate ex

240 and compared them with those of the cervix, vagina, rectum, oral cavity, and controls for background

241 vical stroma, parametria and/or adnexae, and vagina, respectively, were 72%, 69%, 74%, and 85% for re

242 Specifically, HA depletion in the cervix and vagina resulted in inappropriate differentiation of epit

243 pening between the bladder and/or rectum and vagina resulting in continuous leakage of urine or stool

244 opening forms between a woman's bladder and vagina, resulting in urinary incontinence.

245 g single specimens, with the combined cervix-vagina results identifying the highest number of positiv

246 in sectioned murine lymph nodes draining the vagina revealed a profound cellular reorganization with

247 mmunocompartments matching blood, colon, and vagina samples from rhesus macaques were investigated.

248 or alpha, CXCL1, CCL2, CCL3, and CCL5 in the vagina, spinal cord, and/or brain stem than did wild-typ

249 lastin protein, and desmosine content in the vagina suggest that a burst of elastic fiber assembly an

250 e of the total number of lactobacilli in the vagina, suggest that vaginal lactobacilli could reacidif

251 quently identified in the cervix than in the vagina, suggesting that the expression levels of corecep

252 s and detectable levels of hemoglobin in the vagina, suggesting that the presence of hemoglobin in in

253 se (seeing/feeling a bulge in or outside the vagina) symptoms were assessed.

254 nsins (human neutrophil peptides 1-3) in the vagina than did uninfected women.

255 were more likely to be detected in the vulva/vagina than in the cervix (odds ratio, 4.38 [95% confide

256 significantly more T regulatory cells in the vagina than the unimmunized RM.

257 bition of challenge virus replication in the vagina than when the virus was used to vaccinate mice in

258 her E. coli to the urethra, periurethra, and vagina, the authors reasoned that uropathogenic E. coli

259 Of the 44 patients with a vagina, the mean length was 2.0 cm (range, 1.0-6.5 cm).

260 highly expressed in the epithelium of human vagina throughout the menstrual cycle, whereas the mouse

261 thmic anastomosis enabling continuity of the vagina to be preserved following insertion of an isthmic

262 emselves and/or by vaginal flora, causes the vagina to become acidic (pH approximately 4).

263 rom normal human endocervix, ectocervix, and vagina to characterize gonococcal epithelial interaction

264 sport of microbicides or immunogens from the vagina to local lymph organs is feasible.

265 pregnancy, ascending GBS infection from the vagina to the intrauterine space is associated with pret

266 Ascending infection from the colonized vagina to the normally sterile intrauterine cavity is a

267 osa explant model (endocervix and ectocervix/vagina) to mimic genital herpes infections caused by her

268 generally taught that LS does not affect the vagina, unlike lichen planus.

269 by-site, for primary melanomas of the vulva, vagina, urethra, ovary, and the uterine cervix.

270 nt mucosal antibody responses in the rectum, vagina, urine, seminal fluid, and blood.

271 report here the construction of a functional vagina using autologous cells expanded from a small vagi

272 nically significant anaerobes from the human vagina using conventional approaches with systematic mol

273 large bowel, resulting in protrusion of the vagina, uterus, or both.

274 rinary tract with adjacent organs namely the vagina, uterus, rectum and colon.

275 We found that the vaginas varied individually in shape and that the vagina

276 strated tissue selectivity toward uterus and vagina versus breasts in a rodent model after oral admin

277 nd not seen in HPV-related precursors of the vagina, vulva, and penis further support the notion that

278 ral and nasal cavities, small intestine, and vagina was carried out in female rhesus macaques to dete

279 Fungal CFU in each vagina was determined to assess the severity of infectio

280 activity in the keratinized (differentiated) vagina was probably due to dilution of the number of tel

281 The posterior vagina was swabbed using a cytology brush on PND 0, 2 an

282 ound in human clitoral corpus cavernosum and vagina, we hypothesized that human arginase II is simila

283 cluding stool, oral gingiva, nares, skin and vagina were collected for each maternal-infant dyad.

284 s 14 days postinfection, both the uterus and vagina were found to be positive compared to those of sa

285 dermal LCs, the DCs in the epithelium of the vagina were found to be repopulated mainly by nonmonocyt

286 Also, lymphocyte numbers in the vagina were markedly but similarly increased by vaginal

287 s, the numbers of T and B lymphocytes in the vagina were similar in vaginally and parenterally immuni

288 Quantum dots instilled into the mouse vagina were transported across the vaginal mucosa into d

289 lamina propria of the cervical region of the vagina, where a higher number of fibers containing immun

290 ure downregulated RUNX1 in the fornix of the vagina, where DES-associated adenosis is frequently foun

291 iking homeotic transformation towards cervix/vagina, where Hoxa13 is normally expressed.

292 were pure NOTES, through a SILS port in the vagina, whereas TV cholecystectomies were hybrid procedu

293 p into the oviduct, uterus, cervix and upper vagina, whereas Wolffian ducts regress.

294 the Acien's hypothesis of Wolffian origin of vagina which explains the development of OHVIRA syndrome

295 s induced adenosis in the cranial portion of vagina, which mimicked the effect of developmental DES-e

296 bulin A chlamydia-specific antibodies in the vagina, while mice immunized with the detergent-extracte

297 igh levels of effector memory CD8 T cells in vagina, while the pool of memory T cells in spleen assum

298 and cytobrushes and from the ectocervix and vagina with cervicovaginal lavage.

299 he immune response was reoriented toward the vagina with strikingly higher CD8 T cell responses in th

300 communities in oral, nasal, stool, skin, and vagina, with Proteobacteria as the dominant phylum (60 %