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1 ster headache (galcanezumab and non-invasive vagus nerve stimulation).
2 ms of enhancing NTS neuronal activity during vagus nerve stimulation.
3 nd existing bioelectronic therapies, such as vagus nerve stimulation.
4 red for inhibition of cytokine production by vagus nerve stimulation.
5 r functional inhibition of TNF production by vagus nerve stimulation.
6 r requirements significantly decreased after vagus nerve stimulation.
7 of gut Lactobacillus species in response to vagus nerve stimulation.
10 ecause preclinical evidence shows electrical vagus nerve stimulation accelerates clotting to reduce h
12 , with the carrageenan air pouch model, both vagus nerve stimulation and cholinergic agonists signifi
14 eloped a method of transcutaneous mechanical vagus nerve stimulation and then investigated whether th
15 erve stimulation (SNS) and right and/or left vagus nerve stimulation and was compared with DOR during
16 receive electrical, transcutaneous, or sham vagus nerve stimulation and were followed for survival o
17 allosotomy), neuromodulation techniques (eg, vagus nerve stimulation), and dietary interventions repr
19 e association between temporal lobe surgery, vagus nerve stimulation, and other non-pharmacological t
21 animals, could help to explain the effect of vagus nerve stimulation as a treatment for headache diso
22 ne hemorrhage and thrombosis models, we show vagus nerve stimulation bypasses the factor VIII deficie
24 Our results demonstrate that noninvasive vagus nerve stimulation can significantly reduce insomni
25 te that bilateral autonomic activity (SNS or vagus nerve stimulation) cause reversible shifts of apex
26 Here, we use brief bursts of closed-loop vagus nerve stimulation (CL-VNS) delivered during rehabi
27 -focused training with real-time closed-loop vagus nerve stimulation (CLV) to enhance synaptic plasti
28 epeatedly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physio
30 on-invasive alternative to invasive cervical vagus nerve stimulation (cVNS) by activating vagal affer
31 ement anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively ac
32 veral different approaches in use, including vagus nerve stimulation, deep brain stimulation of the t
36 ntiinflammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically
37 mulation, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation
41 US Food and Drug Administration approval of vagus nerve stimulation for recurrent treatment-resistan
42 s: 1) sepsis group (eight pigs), 2) sepsis + vagus nerve stimulation group (nine pigs), and 3) contro
45 hese observations demonstrate that auricular vagus nerve stimulation holds promise for improving slee
47 hange in the direction of DOR, but bilateral vagus nerve stimulation increased and reversed DOR to ba
48 n of nicotine, an alpha7 agonist that mimics vagus nerve stimulation, increases proinflammatory cytok
55 We hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympatheti
56 tions for temporary neurotoxicity, low-level vagus nerve stimulation [LL-VNS], stellate ganglion bloc
63 vidence that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by
65 t may be exploited through either electrical vagus nerve stimulation or administration of alpha7 agon
74 ene-related peptide antibodies, non-invasive vagus nerve stimulation, sphenopalatine ganglion stimula
76 on celiac nerve abolishes TNF suppression by vagus nerve stimulation, suggesting that the cholinergic
83 lammatory effect of transcutaneous auricular vagus nerve stimulation (taVNS) and its ability to promo
84 The efficacy of transcutaneous auricular vagus nerve stimulation (taVNS) as a non-invasive method
85 tinuous noninvasive transcutaneous auricular vagus nerve stimulation (taVNS) during exposure to emoti
91 nightly, bilateral, transcutaneous auricular vagus nerve stimulation (taVNS) on insomnia and mental h
93 ared the effects of transcutaneous auricular vagus nerve stimulation (taVNS, Cerbomed Nemos) with sha
97 hase studies have supported potential use of vagus nerve stimulation to deliver autonomic regulation
98 cessful application of a phasic, noninvasive vagus nerve stimulation to improve cognitive control and
99 uromodulation by non-invasive transcutaneous vagus nerve stimulation (tVNS) can improve exercise capa
102 jection fraction (HFpEF) that transcutaneous vagus nerve stimulation (tVNS) reduced cardiac fibrosis
104 (iVNS) and transcutaneous auricular (taVNS) vagus nerve stimulation using intracranial neurophysiolo
106 to either rehabilitation paired with active vagus nerve stimulation (VNS group) or rehabilitation pa
107 Specifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic
109 ctive of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidat
110 erve fibers was implicated in several failed vagus nerve stimulation (VNS) clinical trials by effecti
111 ation for motor dysfunction, the delivery of vagus nerve stimulation (VNS) combined with tactile reha
112 controls multiple organs of the GI tract and vagus nerve stimulation (VNS) could provide a means for
123 NCE STATEMENT Recent studies have implicated vagus nerve stimulation (VNS) in enhanced learning and m
124 nvasive studies demonstrated that continuous vagus nerve stimulation (VNS) in rodents can activate th
125 ential benefits and underlying mechanisms of Vagus Nerve Stimulation (VNS) in the treatment of Chroni
132 e immunity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various in
138 sought to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CB
139 m effects of episodic inspiratory-inhibitory vagus nerve stimulation (VNS) on phrenic nerve activity.
140 gate this, mice were subjected to electrical vagus nerve stimulation (VNS) or sham surgery at the cer
144 he pilot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chro
145 loped a novel strategy that uses closed-loop vagus nerve stimulation (VNS) paired with tactile rehabi
147 eviously shown the safety and feasibility of vagus nerve stimulation (VNS) paired with upper-limb reh
148 nervation of multiple organs, and electrical vagus nerve stimulation (VNS) provides an adjunctive tre
150 evoked cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interac
155 own promise as a non-invasive alternative to vagus nerve stimulation (VNS) with implantable devices,
156 ow that combining brief bursts of electrical vagus nerve stimulation (VNS) with motor or tactile reha
157 ion Registry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in
159 t peripheral nerve stimulation, specifically vagus nerve stimulation (VNS), can alter neural activity
160 and approved in refractory focal epilepsies: vagus nerve stimulation (VNS), deep brain stimulation of
161 investigate the effect of vagotomy (VGX) and vagus nerve stimulation (VNS), on the development and se
168 nt large animal model of progressive sepsis, vagus nerve stimulation was associated with a number of
169 timulation frequency-dependent manner during vagus nerve stimulation, with comparable increases seen