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1 n of lateral junction proteins (occludin and vascular endothelial cadherin).
2 p38 activation, leading to the induction of vascular endothelial cadherin.
3 plasmalemma vesicle-associated protein-1 and vascular endothelial cadherin.
4 t such as junctional adhesion molecule-1 and vascular endothelial cadherin.
5 n VEGFR2 was, at least in part, dependent on vascular endothelial cadherin.
6 specific genes including VEGFR-2, Tie-2, and vascular endothelial cadherin.
7 could be rescued by exogenous expression of vascular endothelial cadherin.
8 mRNA levels for endogenous Robo4, CD31, and vascular endothelial cadherin.
9 s of TrpRS by enhancing its interaction with vascular endothelial-cadherin.
10 g von Willebrand factor, CD31, occludin, and vascular endothelial-cadherin.
11 in the region that putatively interacts with vascular endothelial-cadherin.
12 dherin 12 (Pcdh12), an ortholog of the mouse vascular endothelial cadherin-2 gene; hFmi1 and hFmi2, h
14 h factor C-mediated endosomal trafficking of vascular endothelial cadherin and induced overlapping ce
15 migrating leukocyte, as well as movement of vascular endothelial cadherin and its associated catenin
18 at Gal-3 silencing resulted in a decrease of vascular endothelial-cadherin and interleukin-8 promoter
19 se-1 in C8161-c9 cells resulted in a loss of vascular endothelial-cadherin and interleukin-8 promoter
21 f proteins involved with adherens-junctions (vascular endothelial-cadherin) and tight-junctions (zona
23 tion of epithelial, placental, neuronal, and vascular endothelial cadherins, and complete loss of cel
24 atelet endothelial cell adhesion molecule 1, vascular endothelial-cadherin, and CD34) and an endothel
25 icry markers factor VIII-associated antigen, vascular endothelial-cadherin, and laminin 5 gamma 2 cha
26 ated Src and higher levels of phosphorylated vascular endothelial cadherin at adherens junctions comp
27 co-localized and co-immunoprecipitated with vascular endothelial cadherin-based complexes, including
28 (2) exposure resulted in the dissociation of vascular endothelial cadherin/beta-catenin complexes and
30 osphatase-2 plasma membrane localization and vascular endothelial cadherin binding because Src homolo
31 -like CD133(+) fraction includes a subset of vascular endothelial-cadherin (CD144)-expressing cells t
34 ), angiotensin receptor-like 1 (Agtrl1), and vascular endothelial-cadherin (Cdh5) were all identified
35 hocytic leukemia protein 1 rescued hemogenic vascular-endothelial cadherin(+) cells and hematopoietic
36 tease activity on endothelial cells, causing vascular endothelial-cadherin cleavage and concomitant l
37 ockout (LKB1(endo-/-)) mice by crossbreeding vascular endothelial-cadherin-Cre mice with LKB1(flox/fl
38 enetically traceable subpopulation of BMDCs (vascular endothelial-cadherin-Cre-enhanced yellow fluore
40 or-beta superfamily, attenuated VEGF-induced vascular endothelial-cadherin disorganization, pore form
42 an irregular morphology and a less organized vascular endothelial cadherin distribution at cell-cell
44 cate that HSCs emerge from cadherin 5 (Cdh5; vascular endothelial-cadherin)(+) endothelial precursors
45 thelial cells required cell-cell contact and vascular endothelial cadherin engagement to transduce st
47 hanced contractility which downregulates the vascular endothelial cadherin expression and destabilize
48 d higher endothelialization rates and higher vascular endothelial-cadherin expression at 7 and 14 day
51 lial cells, yet experimental dissociation of vascular endothelial-cadherin from endothelial junctiona
55 that this fragment induces redistribution of vascular endothelial-cadherin in a process that is inhib
56 r and activator of transcription 3 and Snail/vascular endothelial cadherin-independent decrease in EM
58 human microvascular endothelial cells caused vascular endothelial cadherin internalization, disruptio
59 function is impaired and the localization of vascular endothelial cadherin is altered as function of
60 /C3aR signaling and a functional mediator of vascular endothelial cadherin junction and barrier integ
61 , as evidenced by junctional accumulation of vascular endothelial-cadherin, junctional adhesion molec
62 ate), where their introduction disrupted the vascular endothelial cadherin junctions in a dose-depend
63 el permeability resulting from disruption of vascular endothelial-cadherin junctions between endothel
64 e in the antecubital vein followed by CD144 (vascular endothelial cadherin) magnetic bead isolation.
65 and increased cell stiffness, thus favoring vascular endothelial cadherin-mediated transmission of i
66 d by an endothelial lineage-specific, murine vascular endothelial cadherin (mVEcad) promoter for the
68 cell junctions, which then induces increased vascular endothelial cadherin phosphorylation both in vi
69 same condition, the decreased expression of vascular endothelial cadherin, platelet-derived growth f
70 conditional deletion that Runx1 activity in vascular-endothelial-cadherin-positive endothelial cells
72 elial-specific Nox4 overexpression using the vascular endothelial cadherin promoter (VECad-Nox4 mice)
73 e transfected with a construct composed of a vascular endothelial cadherin promoter driving enhanced
77 ent angiostatic factors that act through the vascular endothelial-cadherin receptor and Akt signaling
79 Consistent with elevated VEGFR2 activity, vascular endothelial cadherin showed reduced localizatio
81 er formation that involves a TXNIP-dependent vascular endothelial cadherin-Src homology phosphatase-2
84 complex in the plasma membrane consisting of vascular endothelial cadherin, the transmembrane protein
85 ial-specific adherens junction protein, VEC (vascular endothelial cadherin), upregulate genes with ke
86 insulin-secreting pancreatic-like CHIPS with vascular endothelial cadherin(+) vascular-like networks.
87 l p120-catenin (p120) maintains the level of vascular endothelial cadherin (VE-Cad) by inhibiting VE-
89 I ligand found within the C-terminal tail of vascular endothelial cadherin (VE-Cad) suggests a role i
90 obin, which form intracellular links between vascular endothelial cadherin (VE-cadherin) and actin-bi
91 ation of adherens junction proteins, such as vascular endothelial cadherin (VE-cadherin) and beta-cat
92 modelling of adherens contacts consisting of vascular endothelial cadherin (VE-cadherin) and beta-cat
93 ociated with changes in permeability such as vascular endothelial cadherin (VE-Cadherin) and neuropil
94 r, both buttons and zippers were composed of vascular endothelial cadherin (VE-cadherin) and tight ju
96 ial cells display pronounced accumulation of vascular endothelial cadherin (VE-cadherin) at cell-cell
97 tein kinase 3 (RIPK3) and destabilization of vascular endothelial cadherin (VE-cadherin) at EC juncti
98 growth factor-induced signals impinge on the vascular endothelial cadherin (VE-cadherin) complex, the
100 tions, binding of the FAK FERM domain to the vascular endothelial cadherin (VE-cadherin) cytoplasmic
102 ction and the proteolytic disorganization of vascular endothelial cadherin (VE-cadherin) in HUVECs in
103 -NDSK II bound) and a monoclonal antibody to vascular endothelial cadherin (VE-cadherin) inhibited bi
104 noclonal antibodies directed against CD31 or vascular endothelial cadherin (VE-cadherin) inhibited th
107 mponents of angiogenesis and inflammation, a vascular endothelial cadherin (VE-cadherin) mutant defec
109 of adherens junctions and the degradation of vascular endothelial cadherin (VE-cadherin) protein.
110 ely determined by the homophilic assembly of vascular endothelial cadherin (VE-cadherin) within adher
113 dothelial growth factor receptor 2 (VEGFR2), vascular endothelial cadherin (VE-cadherin), and AC133.
114 phorylation of the adherens junction protein vascular endothelial cadherin (VE-cadherin), and leukocy
115 s and levels of von Willebrand factor (vWf), vascular endothelial cadherin (VE-cadherin), and prolife
116 eased permeability, abnormal distribution of vascular endothelial cadherin (VE-cadherin), and reduced
117 lines expressing either SNAP-tagged Notch or vascular endothelial cadherin (VE-cadherin), we provide
118 the inhibition of phosphorylation of Src and vascular endothelial cadherin (VE-cadherin), which incre
119 endothelial cell (HCAEC) wound closure via a vascular endothelial cadherin (VE-cadherin)-dependent me
125 ) have rare CTC subpopulations co-expressing vascular endothelial-cadherin (VE-cadherin) and cytokera
126 and embryonic SP isolated 8.0 dpc expressed vascular endothelial-cadherin (VE-cadherin) and vascular
127 ound to the VE-cadherin promoter, increasing vascular endothelial-cadherin (VE-cadherin) expression l
128 ndothelial cell-specific junctional molecule vascular endothelial-cadherin (VE-cadherin) in vitro and
131 and integrin alpha9 and exhibited continuous vascular endothelial-cadherin (VE-cadherin) junctions an
133 red hLECs with CGRP peptide in vitro induced vascular endothelial-cadherin (VE-cadherin) rearrangemen
134 tribution patterns of cytoskeletal actin and vascular endothelial-cadherin (VE-cadherin), both of whi
135 e-1 (Flk-1)-, stem cell leukemia (Scl)-, and vascular endothelial-cadherin (VE-cadherin)-expressing c
136 This study demonstrates that a CD34(-), vascular endothelial cadherin(-) (VE-cadherin(-)), AC133
137 not express detectable levels of the typical vascular endothelial cadherin, VE-cadherin (CDH5) as det
140 ly expression of endothelial markers such as vascular endothelial cadherin(VECAD) and occludin but lo
141 protein kinase C (PKC)-alpha interacts with vascular-endothelial cadherins (VECs) at the tight junct
142 l cell-adhesion molecule-1, CD34, KDR/Flk-1, vascular endothelial cadherin, von Willebrand factor), i