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1  state, resulting in neointima formation and vascular occlusion.
2 the key to hemostasis and also to pathologic vascular occlusion.
3  spectrum of IOI, retinal vasculitis, and/or vascular occlusion.
4 nt responses but can also lead to thrombotic vascular occlusion.
5 nate blood flow to preserve tissue following vascular occlusion.
6 M can be the only presenting sign of retinal vascular occlusion.
7 ciation between cataract surgery and retinal vascular occlusion.
8  exclusion of patients with proxy of retinal vascular occlusion.
9 hrombolysis in a preclinical animal model of vascular occlusion.
10 gnificantly increased in RVO and any retinal vascular occlusion.
11              The primary outcome was retinal vascular occlusion.
12 nformation like the risk level caused by any vascular occlusion.
13 apturing haemodynamic changes in response to vascular occlusion.
14 osure and secure hemostasis while preventing vascular occlusion.
15 odialysis who subsequently developed retinal vascular occlusion.
16 Rs also enhance thrombotic and nonthrombotic vascular occlusion.
17 ncy procedure in 4 patients due to prolonged vascular occlusion.
18 chemic inflammation initiates already during vascular occlusion.
19 tion, incompetent blood-retinal barrier, and vascular occlusion.
20  are ineligible for IV t-PA but have a large vascular occlusion.
21 toring blood flow to ischemic tissue after a vascular occlusion.
22 -threatening diseases that result from acute vascular occlusion.
23  were imaged before, during, and after renal vascular occlusion.
24 id adaptation of the lymphatic pump to local vascular occlusion.
25 silateral parietal cortex to ensure adequate vascular occlusion.
26 and the most common cause of acute pulmonary vascular occlusion.
27  the impact of psychological stress on acute vascular occlusion.
28  heparin may be useful in preventing painful vascular occlusion.
29 ndothelium is important to the generation of vascular occlusion.
30 rease collateral blood flow after mesenteric vascular occlusion.
31 crosis and possibly plaque rupture and acute vascular occlusion.
32 estinal viability after segmental mesenteric vascular occlusion.
33  no association between dementia and retinal vascular occlusions.
34  potentially resulting in vision-threatening vascular occlusions.
35 cellular phenotype as fibroblasts from CTEPH vascular occlusions.
36 lood-retinal barrier with or without retinal vascular occlusions.
37  inflammatory environment that culminates in vascular occlusions.
38 n of Diseases 10 codes to select for retinal vascular occlusions.
39 ed toxicities were chorioretinal atrophy and vascular occlusions.
40 M)beta(2) microdomains leads to acute lethal vascular occlusions.
41      Mice deficient in VN (n = 12) developed vascular occlusion 77 +/- 11 minutes after injury, inter
42 lial cells has been thought to contribute to vascular occlusion, a major cause of morbidity in sickle
43 s (adjusted IRR 6.11, P < 0.001) and retinal vascular occlusion (adjusted IRR 3.81, P = 0.023) were s
44   We found the hazard ratio (HR) for retinal vascular occlusion after cataract surgery was 1.23 (95%
45              Diagnosing catastrophic retinal vascular occlusion after intravitreal injections as a re
46 the association between dementia and retinal vascular occlusions after adjusting for covariates which
47 on incidence also was increased with retinal vascular occlusions (aHR, 10, 95% CI, 3.0-33), retinal v
48 OI, including retinal vasculitis and retinal vascular occlusion, also were higher, leading to study t
49 ge-related macular degeneration, and retinal vascular occlusions, among others.
50      There are 1463 individuals with retinal vascular occlusion and 500 670 individuals without.
51  larger absolute force in men causes greater vascular occlusion and accumulation of dilators, while b
52 f retinal vasculitis with or without retinal vascular occlusion and an associated risk of visual acui
53                                              Vascular occlusion and complex plexiform lesions are hal
54                  This patient highlights the vascular occlusion and endothelial cell injury in the me
55 coherence tomography (OCT), with progressive vascular occlusion and evidence of the ischemic cascade
56 es inflammatory lung pathologic findings and vascular occlusion and hemorrhage.
57 in preclinical models of Phx with or without vascular occlusion and in donor livers.
58 urs during surgery involving renal or aortic vascular occlusion and is one of the leading causes of p
59 e report that FH(R/R) mice exhibited retinal vascular occlusion and ischemia.
60 et activation during disease states leads to vascular occlusion and ischemic damage.
61 stologic examination of the same rete showed vascular occlusion and moderate intraluminal foreign bod
62 nts with a history of retinopathy or retinal vascular occlusion and other physical or mental conditio
63 ive pulmonary vascular remodeling, including vascular occlusion and plexiform-like lesions, resemblin
64 echanism of reversing established neointimal vascular occlusion and pulmonary hypertension.
65 ment dysregulation can contribute to retinal vascular occlusion and that an anti-C5 antibody might be
66 pholipid antibodies in patients with retinal vascular occlusion and the spectrum of ophthalmologic fi
67  no significant associations between retinal vascular occlusions and dementia.
68 indicate that the connection between retinal vascular occlusions and neurodegenerative diseases causi
69  investigate the association between retinal vascular occlusions and neurodegenerative disorders that
70  systemic conditions associated with retinal vascular occlusions and offer guidelines for appropriate
71                                  Any retinal vascular occlusions and RVO are associated with increase
72 hypofluorescent areas, widespread peripheral vascular occlusion, and ischemia.
73 tion of endothelial VWF decreased hemolysis, vascular occlusion, and organ damage caused by TNF-induc
74 ical examination shows no objective signs of vascular occlusion, and this can lead the clinician to s
75  and transiently immediately after pulmonary vascular occlusion, and this rise was blunted significan
76 have an increased risk of developing retinal vascular occlusions, and more specifically CRAO and BRAO
77 n microscopic analysis of tumor sections for vascular occlusion; and analysis of tumor cryosections f
78 myocardial infarction, stroke and peripheral vascular occlusion are the leading causes of morbidity a
79                                Acute retinal vascular occlusions are common causes of visual impairme
80                                              Vascular occlusions are common structural modifications
81  with needles or microcannulas, but unwanted vascular occlusions are possible.
82 lculated to compare the incidence of retinal vascular occlusions before and after COVID-19 diagnosis
83 inal artery occlusion (RAO), and any retinal vascular occlusion (both RVO and RAO).
84 is a possible co-factor in atherogenesis and vascular occlusion, but its ability to actively infect m
85 soluble TM, prevents platelet activation and vascular occlusion by clots.
86 s estimated incidence rate ratios of retinal vascular occlusion by duration, dosage, and type of horm
87                            AION with visible vascular occlusion can be a rare retinal cause of acute
88                                              Vascular occlusion can lead to increased pressure in the
89 thus be attributed to impaired perfusion and vascular occlusion caused by deposition of Abeta and fib
90         Sickle cell disease (SCD) results in vascular occlusions, chronic hemolytic anemia, and cumul
91 frequency (24+/-2%) and severity (9+/-4%) of vascular occlusion compared with wild-type controls (n=7
92 lls into Rag2-deficient mice greatly reduced vascular occlusion compared with WT cells, clearly demon
93 athophysiology depicted with imaging include vascular occlusion, compensatory collateral flow, result
94 d to determine measurement agreement between vascular occlusion conditions.
95                  In patients with persistent vascular occlusions despite intravenous tissue plasminog
96 ls in 28, umbrella devices in 6 and a Grifka vascular occlusion device in 1.
97 PEDs and resolution of PAMM, with no further vascular occlusion during corticosteroid taper.Conclusio
98 tched on age and smoking who did not develop vascular occlusion during the prospective follow-up peri
99 tified through prescription records; retinal vascular occlusion events were detected through hospital
100 ith inflamed endothelium, triggering painful vascular occlusion events.
101 he patient had no classical risk factors for vascular occlusions, except for the presence of Flammer
102 in vascular injury in sickle cell anemia, in vascular occlusion following the rupture of atherosclero
103 inal vascular occlusions, those with retinal vascular occlusions had a higher prevalence of dementia
104  gabapentin administration had no effects on vascular occlusion, haemodynamic changes nor survival of
105 otein precursor, A beta PP) in the organized vascular occlusions harvested from patients with this di
106 emodialysis and subsequently develop retinal vascular occlusion have not been examined.
107                     Individuals with retinal vascular occlusions have a higher prevalence of dementia
108  Fundus examination revealed diffuse retinal vascular occlusion, hemorrhages clustered along venules,
109        For the proposed definitions of total vascular occlusion, hepatic flow occlusion and inferior
110 mentia was seen in patients with any retinal vascular occlusion (HR, 1.52; confidence interval [CI],
111                     Eyes with recent retinal vascular occlusion illustrating paracentral acute middle
112 stem-driven coagulation, both variants block vascular occlusion in an in vivo model for arterial thro
113 and parenchymal destruction with more severe vascular occlusion in grafts from IL-10 -/- recipients.
114 mation (IOI), retinal vasculitis, or retinal vascular occlusion in patients with neovascular age-rela
115 oma, episcleritis and scleritis, and retinal vascular occlusion in patients with SLE deserves vigilan
116 helial proteins is believed to contribute to vascular occlusion in sickle cell disease.
117 of irreversible PEm to ask whether pulmonary vascular occlusion in the absence of reperfusion is itse
118               This was followed by temporary vascular occlusion in the beam path that was resolved by
119 trated noninfectious uveitis and evidence of vascular occlusion in the presence of retinal vascular i
120 oma, episcleritis and scleritis, and retinal vascular occlusion in these patients.
121 ere significantly diminished, and thrombotic vascular occlusion in vivo was significantly blunted in
122 D) is characterized by recurring episodes of vascular occlusion in which neutrophil activation plays
123                           Background Retinal vascular occlusion in young adults warrants systematic e
124 suring hemostasis and thrombosis by inducing vascular occlusion in zebrafish larvae.
125     The presence of patent neovessels within vascular occlusions in chronic thromboembolic pulmonary
126 or (G-CSF), each induced NET-mediated lethal vascular occlusions in mice with combined genetic defici
127 ce R rickettsii infection induces thrombotic vascular occlusions in patients with Rocky Mountain Spot
128 we concluded that the extensive formation of vascular occlusions in PD-susceptible grapevines does no
129 ear-resistant adhesions ex vivo and unstable vascular occlusions in vivo.
130  mutation may manifest with combined retinal vascular occlusion involving multiple sites in both eyes
131                                      Retinal vascular occlusion is considered a risk factor for cardi
132 blood probing during the hyperacute stage of vascular occlusion is crucial to assess the role of infl
133 liferative processes that ultimately lead to vascular occlusion is enhanced by the abnormal metabolic
134                                              Vascular occlusion is the major cause of morbidity and m
135 nfarction (MI), as a result of thrombosis or vascular occlusion, is the most prevalent cause of morbi
136 of the inner retina, indicating long-lasting vascular occlusion lesions.
137 strategies that target gpIIbIIIa may prevent vascular occlusion, maintain tissue viability, and provi
138 d strategies targeting gpIIbIIIa may prevent vascular occlusion, maintain tissue viability, and provi
139 get platelet adherence molecules may prevent vascular occlusion, maintain tissue viability, and reduc
140 This is a rare presentation of mixed retinal vascular occlusion manifesting as PAMM and incomplete re
141 bed with the following categories: bleeding, vascular occlusion (myocardial infarction, stroke and pu
142 were acute stroke (n = 3), aneurysm (n = 3), vascular occlusion (n = 3), and disk herniation (n = 2).
143 orrhage (n = 7), optic nerve damage (n = 4), vascular occlusions (n = 2), pain (n = 2), eye or head m
144 ) receptors, whereas, in contrast, (3) brief vascular occlusion not associated with appreciable adeno
145 vessel wall is believed to contribute to the vascular occlusion observed in patients with sickle call
146                             The intermittent vascular occlusion occurring in sickle cell disease (SCD
147                                   Thrombotic vascular occlusion occurs in disorders of diverse etiolo
148 n resistance training (BFRT) employs partial vascular occlusion of exercising muscles via inflation c
149                                              Vascular occlusions of the retina and choroid can cause
150 iuretic peptide, and percentage of pulmonary vascular occlusion on chest computed tomography.
151 ase, such as diabetic retinopathy or retinal vascular occlusion, one would refer to the lesion as PAM
152 n patient populations who experience partial vascular occlusion or altered neuromuscular reflexes.
153  or preventing vertical progression of blood vascular occlusion or endothelial cell death.
154 nce of substantial progression (P=0.008), or vascular occlusion (P=0.005) when compared with the mode
155             Thus, by showing that NETs drive vascular occlusion, pleomorphic necrosis and metastasis,
156 ted blood loss, duration of surgery, hepatic vascular occlusion (rate or duration), portal vein embol
157                                      Retinal vascular occlusion rates were 1.6% vs 0.5% (KESTREL) and
158 ical dynamics for oxygen, growth factors and vascular occlusions, replicate key clinical observations
159                                   Thrombotic vascular occlusion resulting in infarction occurs during
160  lead to pathological thrombus formation and vascular occlusion, resulting in stroke, myocardial infa
161 l toxoplasmosis, retinal vasculitis, retinal vascular occlusions, rhegmatogenous and serous retinal d
162 S) had higher risk of developing any retinal vascular occlusion (RR, 2.33; 95% CI, 1.82-3.00), CRAO (
163 d not have higher risk of developing retinal vascular occlusions (RR, 1.01; 95% CI, 0.81-1.26) than m
164 e cases of retinal vasculitis and/or retinal vascular occlusion (RV/RO) were reported, often during t
165 mation, including retinal vasculitis/retinal vascular occlusion (RV/RO).
166  serous retinal detachment (SRD) and retinal vascular occlusion (RVO) are not available.
167                            New-onset retinal vascular occlusion (RVO) occurring acutely after messeng
168 terial neointimal formation (quantified by a vascular occlusion score) was more severe in vehicle-tre
169 score, 1.98 +/- 0.02) than in Group PMS5-35 (vascular occlusion score, 0.59 +/- 0.46; p < 0.001).
170 lls was more severe in vehicle-treated rats (vascular occlusion score, 1.98 +/- 0.02) than in Group P
171                                      Retinal vascular occlusions share the same risk factors as cardi
172  parenchymal infiltration and less prominent vascular occlusion than day 2 murine CTLA4Ig-treated or
173 95% CI, 1.95-5.06) of developing any retinal vascular occlusion than matched controls without SCD.
174 oportion of diabetic retinopathy and retinal vascular occlusions than the group without postoperative
175 vascular endothelium could contribute to the vascular occlusion that characterizes the acute crisis o
176 terial hypertension (PAH), a rare disease of vascular occlusion that leads to high blood pressure in
177      After 7 days, all embolic agents caused vascular occlusion that led to necrosis and fibrosis.
178                             Patients develop vascular occlusions that cause cardiovascular, cerebrova
179                        This study focuses on vascular occlusions that form in stem secondary xylem of
180      Patients develop painful neuropathy and vascular occlusions that progressively lead to cardiovas
181         Compared to subjects without retinal vascular occlusions, those with retinal vascular occlusi
182                            Platelets mediate vascular occlusion through thrombotic functions but are
183  diurnal variation in the time to thrombotic vascular occlusion (TTVO) subsequent to a photochemical
184              Patients who develop thrombotic vascular occlusions usually have underlying vascular dis
185 ques that precipitate acute atherothrombotic vascular occlusion ("vulnerable plaques") are abundant i
186                                      Retinal vascular occlusion was introduced by rose Bengal and las
187                                              Vascular occlusion was not permanent, and even when embo
188                  However, no risk of retinal vascular occlusion was observed among patients who had r
189 essed vasodilatory response to SNP caused by vascular occlusion was reversed with training.
190                                              Vascular occlusion was substantially increased when BM-d
191 I), including retinal vasculitis and retinal vascular occlusion, was 11.5% (0.8% and 2.2%) for broluc
192 omocysteine concentration, a risk factor for vascular occlusion, was examined in a representative sam
193 OI, including retinal vasculitis and retinal vascular occlusion, was higher with brolucizumab versus
194             By adjusting the severity of the vascular occlusion, we were able to generate hypertensiv
195         Individuals with and without retinal vascular occlusions were identified by International Cla
196 I), including retinal vasculitis and retinal vascular occlusion, were 9.3% (0.8% and 2.0%) for broluc
197 mong patients with RVO, RAO, and any retinal vascular occlusion, whereas the prevalence of VD is sign
198 cluded patients without a history of retinal vascular occlusion who were diagnosed with COVID-19 infe
199 nically relevant model of stroke utilizing a vascular occlusion with a freshly formed clot, and evalu
200 may be possible to inhibit platelet-mediated vascular occlusion with a minimal effect on primary hemo
201 large areas of splenic necrosis secondary to vascular occlusion with hepatocytes, whereas direct sple
202 na spacing were recorded and correlated with vascular occlusion with logistic regression analysis.
203 involvement (2.0% with vasculitis, 1.4% with vascular occlusion with or without vasculitis).

 
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