ライフサイエンスコーパス: vasogenic edema

1 tion of (131)I-mu81C6 mAb through regions of vasogenic edema.

2 Abnormalities ranged from cytotoxic edema to vasogenic edema.

3 presumed tumor-infiltrated edema from purely vasogenic edema.

4 reased blood-brain barrier permeability, and vasogenic edema.

5 ng lesion surrounded by a variable amount of vasogenic edema.

6 ously inhibiting angiogenesis and minimizing vasogenic edema.

7 longer treatment windows may be possible for vasogenic edema.

8 or 'paravasculitis' plays a critical role in vasogenic edema.

9 nt, yet distinct stage between cytotoxic and vasogenic edemas.

10 g was used to show that C57BL/6 mice develop vasogenic edema 4 to 5 weeks after infection with T. gon

11 MMP-9 contributes to the early formation of vasogenic edema after transient FCI.

12 y injury process reflects an extreme form of vasogenic edema and blood-brain barrier breakdown.

13 ropriate proinflammatory response leading to vasogenic edema and cerebral microhemorrhage.

14 rates a 1.6-cm right parietal mass with mild vasogenic edema and four additional brain metastases mea

15 the extracellular water differences between vasogenic edema and infiltrative tissue and training a c

16 (BBB) is associated with the development of vasogenic edema and intracranial hypertension in a numbe

17 ed inflammation (CAA-ri) is characterized by vasogenic edema and multiple cortical/subcortical microb

18 d other chronic retinal diseases, results in vasogenic edema and neural tissue damage, causing vision

19 tein and enzyme activity could contribute to vasogenic edema and the pathogenesis of neuronal dysfunc

20 creased vascular permeability contributes to vasogenic edema and tissue damage, with consequent adver

21 the lesions are likely caused by reversible vasogenic edema and transient breakdown of the blood-bra

22 en in the brain and bone, decreased cerebral vasogenic edema, and improved survival, despite increasi

23 d hematoma and edema volumes, T2 relaxation (vasogenic edema), apparent diffusion coefficient (ADC, c

24 with intracranial evidence of hydrocephalus, vasogenic edema, central venous thrombosis, and/or mass

25 in animals with focal injury associated with vasogenic edema (cryogenic injury).

26 se glibenclamide benefitted hematoma volume, vasogenic edema, cytotoxic edema, and BBB integrity afte

27 imaging abnormalities, interpreted as focal vasogenic edema, develop in some epileptic patients afte

28 These observations support the proposal that vasogenic edema due to cerebrovascular autoregulatory dy

29 Proteinase-3 might mediate vasogenic edema during diabetic ketoacidosis, and select

30 nt with this hypothesis, showing no signs of vasogenic edema, even in ApoE4 carriers.

31 This establishes that Pb(2+)-induced vasogenic edema formation develops via a Flk-1-dependent

32 Vasogenic edema had a minimal effect on MT ratio, and ra

33 e rapid growth of solid brain metastases and vasogenic edema in patients with advanced cancer, leadin

34 s can all result in the clinical syndrome of vasogenic edema in the central nervous system leading to

35 performed in the emergency department showed vasogenic edema in the right temporal parietal region, w

36 ry responses, in order to lower the risk for vasogenic edemas in the clinic.

37 th, pseudocyst-like or multiloculated shape, vasogenic edema, mass effect, and size greater than 3cm.

38 focus 5 mm or less in diameter surrounded by vasogenic edema that extended less than 7 mm in radius b

39 versely, high ADC values, indicative of mild vasogenic edema, were observed in both patients with SNC

40 lleled that of known resistance to spread of vasogenic edema, which suggested that anisotropy may be

41 he metabolic enzyme TYMS; and association of vasogenic edema with the oncogene FOXP1 and PIK3IP1, whi