ライフサイエンスコーパス: venous blood

1 t birth (cord blood) and in early childhood (venous blood).

2 Baseline anemia prevalence was 58% (venous blood).

3 , and their expression, in placenta and cord venous blood.

4 mparing their concentrations in arterial vs. venous blood.

5 , Th1, Th2, Th17, and Treg-memory cells from venous blood.

6 LAgP patients were isolated from peripheral venous blood.

7 s were vascularly isolated and perfused with venous blood.

8 and less H(+) release into the great cardiac venous blood.

9 tissues and cannot be sampled in superficial venous blood.

10 LAgP patients were isolated from peripheral venous blood.

11 consistently higher in the arterial than in venous blood.

12 on of radioactivity into superior mesenteric venous blood.

13 ormal subjects were isolated from peripheral venous blood.

14 venous blood flow and lungs without hepatic venous blood.

15 ased and lactate levels increased in patient venous blood.

16 centrations in arterial, portal, and hepatic venous blood.

17 ntrols were genotyped using genomic DNA from venous blood.

18 Venous blood (5 mL) was obtained from 104 subjects aged

19 entrations of glucose and oxygen relative to venous blood, a comprehensive biochemical characterizati

20 ge volume right lung activity, normalized to venous blood activity, is a good proxy for arterial bloo

21 1 that are specifically increased in hepatic venous blood after CBDL.

22 A receptor imaging were assessed by PET with venous blood analysis.

23 asma and four DBS cards from anti-coagulated venous blood and a fifth card from finger-prick blood we

24 rstly, platelets were isolated from systemic venous blood and activated with the GPVI specific agonis

25 Heparinized venous blood and bone marrow were collected from the pat

26 7%, P < 0.001) compared to DBS prepared from venous blood and handled similarly.

27 nm at basal conditions in both arterial and venous blood and is formed at a level of 0.5-2.5 mum upo

28 ucosal pH, and serum lactate levels of mixed venous blood and mesenteric venous blood were recorded a

29 Venous blood and nasopharyngeal/oropharyngeal swabs were

30 Venous blood and oral rinse samples were obtained from f

31 n chamber was perfused with unanticoagulated venous blood and PDT evaluated using computerized morpho

32 ions declined rapidly after storage of fresh venous blood and that hypoxic vasodilation by banked RBC

33 , they are recalled, values are confirmed in venous blood and thyroxine replacement therapy (TRT) is

34 Venous blood and urine samples were collected at enrollm

35 A) directly diverts the splanchnic and renal venous blood assuring a good portal inflow to the graft.

36 We sampled peripheral venous blood at 0, 3, 6, 12, 24, 48, 72 and 168 hours po

37 s were vascularly isolated and perfused with venous blood at pulsatile pressures designed to simulate

38 partial pressure of O(2) (PO(2)) in cubital venous blood at rest, during handgrip exercise, and duri

39 port rat delivers constant pressure systemic venous blood at stable physiologic parameters to the ex

40 d calcium values of intraosseous and central venous blood at the baseline and during 5 mins of CPR wi

41 f radiotracer to the brain than arterialized venous blood, at least in some patient populations.

42 We collected venous blood before and after the intervention to measur

43 e and insulin concentrations in arterialized venous blood before and during FSIGT were virtually iden

44 ody fat metabolism, as validated by parallel venous blood beta-hydroxybutyrate (BOHB) measurements.

45 Intraosseous and central venous blood biochemical and hemoglobin values were simi

46 The venous blood BK1-5:bradykinin ratio correlated with plas

47 13-ketooctadecadienoic acid (KODE)] into the venous blood both in vivo and during perfusion.

48 tion is a rare condition in which splanchnic venous blood bypasses the liver draining directly into s

49 , it appears that increases in the tissue or venous blood CO(2) concentration are neither sensitive n

50 upport the notion that changes in tissue and venous blood CO(2) concentration during dysoxia reflect

51 hypoxia (dysoxia) to increases in tissue and venous blood CO(2) concentration.

52 collected by finger-prick was noninferior to venous blood collected in PAXgene tubes.

53 Samples of venous blood, collected at various time points, were ana

54 utility for applications in the field where venous blood collection and timely shipment of labile bl

55 ith AD showed a significant narrowing of the venous blood column diameter (131.7 +/- 10.8 microm) com

56 correlated with the percentage decreases in venous blood column diameter (P = 0.031, R(2) = 0.51).

57 hat leads to it receiving all the mesenteric venous blood, combined with its unique micro anatomy, al

58 e primary outcome was within-person GLP-1 in venous blood (concentrations and area under the curve).

59 Use of venous blood data introduced a large bias in VT (r(2) =

60 cted genomic DNA (gDNA) as an alternative to venous blood-derived gDNA from premature neonates for mo

61 were unable to cooperate with fingerstick or venous blood draw.

62 Plasma was collected from infants if venous blood draws were ordered by pediatricians.

63 n which inspiration-induced downward flow of venous blood due to reduced intrathoracic pressure is co

64 e acid-base status of intraosseous and mixed venous blood during cardiopulmonary resuscitation; and c

65 x gene Prox1 is necessary and sufficient for venous blood endothelial cells (BECs) to acquire a lymph

66 the number of neutrophils was greater in the venous blood entering the lungs than in the arterial blo

67 evelop PAVM compared with lungs with hepatic venous blood flow (12/12 and 3/16 respectively, p < 0.01

68 arterial blood flow (F(a)), absolute portal venous blood flow (F(p)), absolute total liver blood flo

69 Enhancing the portal venous blood flow (PVBF) has been shown to reduce portal

70 a significant improvement in post-LPS portal venous blood flow (PVBF, 79% of baseline vs. 45% of base

71 acquisitions, one optimized for arterial and venous blood flow (velocity encoding range, +/-50 cm/sec

72 t echocardiograms between lungs with hepatic venous blood flow and lungs without hepatic venous blood

73 the portal vein, and disturbances in portal venous blood flow could contribute to the formation of b

74 Concomitant analysis of brain venous blood flow indicated that CSF and venous flux act

75 the differential assessment of arterial and venous blood flow patterns in the retina that may facili

76 crom, P = 0.01), and a significantly reduced venous blood flow rate (9.7 +/- 3.1 microL/min) compared

77 Change in portal venous blood flow rates did not have an effect on the si

78 Liver portal venous blood flow was assessed during perfusion, and at

79 Lungs with no hepatic venous blood flow were more likely to develop PAVM compa

80 venous stasis, reduces intraoperative portal venous blood flow, decreases intraoperative urinary outp

81 tic IRI model, adjunctive BV improved portal venous blood flow, increased bile production, and decrea

82 HO-1 (Ad-HO-1) significantly improved portal venous blood flow, increased bile production, and decrea

83 mitant analyses of CSF dynamics and cerebral venous blood flow, that is, in epidural veins at cervica

84 For venous blood flow, the ICC with Bayesian multipoint MR i

85 n lesions is unaffected by changes in portal venous blood flow.

86 t inverse relationship to the rate of portal venous blood flow.

87 ps showed significant restoration of retinal venous blood flow.

88 ath, there was a large resurgence of femoral venous blood flow.

89 rtial pressure of a given inert gas in mixed-venous blood flowing back to the lungs is calculated fro

90 Obtaining venous blood for serological screening, typically perfor

91 multibed scanning over 4 h with sampling of venous bloods for radioactivity and radioactive metaboli

92 Venous blood from 72 Schistosoma haematobium-exposed par

93 Venous blood from diabetic patients was exposed to air t

94 Venous blood from healthy volunteers was tonometered to

95 ve immunoselection from the PBMC fraction of venous blood from healthy volunteers, and monocyte-deriv

96 dGuo in the DNA of lymphocytes isolated from venous blood from healthy young male volunteers in sever

97 Venous blood from human volunteers was stimulated with L

98 It is feasible and safe to collect portal venous blood from patients undergoing EUS.

99 MPs were prepared from ECs and venous blood from patients with ACS (n=30) and from heal

100 Venous blood from seven of the subjects was sampled for

101 ormed and corresponding arterial and central venous blood gas and lactate measurements were made.

102 Hemodynamic variables, systemic and mixed venous blood gas tensions and oxygenation, arterial lact

103 Arterial and venous blood gas values, glucose, and cardiac output wer

104 th ITPR-CPR for 15 minutes, and arterial and venous blood gases were collected at baseline and minute

105 Cerebral venous blood gases were drawn from a jugular bulb venous

106 Arterial and deep venous blood gases were measured and oxygen consumption

107 Arterial and mixed venous blood gases were measured at baseline, 1 min afte

108 Radial artery and femoral venous blood gases were measured.

109 leg blood flow and radial artery and femoral venous blood gases were measured.

110 the first minute of CPR, arterial and mixed venous blood gases were superior in the 3 experimental g

111 Arterial and venous blood gases, hemodynamics, and pulmonary mechanic

112 ge-pressure monitoring, measurement of mixed venous blood gases, or monitoring of cardiac output by o

113 nted for measurement of arterial and central venous blood gases.

114 and blood flow were measured with pulmonary venous blood gases.

115 rdiac output by thermodilution, arterial and venous blood gases; electrolytes; lactate; base excess;

116 sions by comparing the CGM glucose values to venous blood glucose measurements taken every 15 min.

117 All 12 lungs with no perfusion of hepatic venous blood had positive contrast echocardiographic stu

118 As expected, ACZ led to lower arterial and venous blood [HCO3-], pH and lactate levels (P < 0.05),

119 lse-positive heel-stick screening but normal venous blood hormone levels; (b) hypoplasia-ectopia in 3

120 lood [PVB]) and outflow compartment (hepatic venous blood [HVB]) of a training (n = 55) and validatio

121 ual parasites in: i) finger prick blood, ii) venous blood, iii) skin biopsies, and in pools of mosqui

122 near normal because of sequestered cerebral venous blood in capillaries and venous capacitance vesse

123 n of circulating tumor cells from peripheral venous blood in clinical practice.

124 of circulating tumor cells using peripheral venous blood in clinical practice.

125 Arterial blood (arterialized venous blood in healthy subjects) was collected for the

126 In CAS patients, glutamate concentrations in venous blood increased immediately after the procedure (

127 vels of inflammatory cytokines in the portal venous blood, induced activation of resident liver dendr

128 be a nonspecific feature of impaired portal venous blood inflow, whatever the cause, or a feature of

129 Venous blood ionized calcium was used as an index of int

130 ral blood (PPB) and intraoperative pulmonary venous blood (IPVB) could predict poor long-term surviva

131 Leukocytes were separated from the patient's venous blood, labeled with (18)F-FDG, and reinjected int

132 Here we describe and evaluate the changes in venous blood lead level (VBLL) associated with DMSA trea

133 9% to 74.0%) for identifying children with a venous blood lead level greater than 10 mug/dL (5 studie

134 ation was increased by approximately 7.5% in venous blood leaving the CNS capillary bed compared to a

135 hen feeding directly on the skin compared to venous blood (odds ratio 2.01; 95% CI 1.21 - 3.33, p = 0

136 from a primary tumor can be detected in the venous blood of cancer patients.

137 Human venous blood of different concentrations, various fluids

138 ry in concentration between the arterial and venous blood of human patients.

139 RNA was extracted from fresh venous blood or paraffin-embedded myocardial tissue of t

140 , and in pools of mosquitoes that fed iv) on venous blood or, v) directly on skin.

141 The reduction in mixed venous blood oxygen saturation and the increase in blood

142 Post-occlusive transient changes in venous blood oxygenation might provide a new measure of

143 between samples collected from capillary or venous blood (p = 0.547).

144 differential equation to calculate the mixed-venous blood partial pressure may be in error.

145 ovolemic conditions, especially when central venous blood pressure is critically low.

146 ternative explanations, like measurements of venous blood pressure, did not show correlation with hyd

147 Their arterial and central venous blood pressures were continuously monitored by in

148 Arterial blood flow, arterial and venous blood pressures, total vascular resistance, tissu

149 in arterial, hepatic venous (HV) and portal venous blood (PV).

150 were detected in the hepatic inflow (portal venous blood [PVB]) and outflow compartment (hepatic ven

151 ion ratios and taking into account pulmonary venous blood remixing yielded our lung-scale model.

152 Right lung activity, corrected to venous blood, represents the whole arterial blood curve

153 field units) of the systemic blood and renal venous blood, respectively, and CT(PRE) is the precontra

154 inferior vena cava and allows uninterrupted venous blood return during the anhepatic phase.

155 dequate forward blood flow out of the heart, venous blood return must be drawn back into the heart.

156 tion with inflammatory markers measured in a venous blood sample at the time of cognitive testing.

157 urements of known serum radioactivity from a venous blood sample obtained at the time of PET/CT.

158 A venous blood sample was collected and blood pressure was

159 A venous blood sample was taken at baseline and at 6 and 1

160 A sample of unstimulated whole saliva and a venous blood sample were obtained from each subject, and

161 ent of B-type natriuretic peptide level from venous blood sample were performed in 150 consecutive pa

162 er laboratory indicators, were measured in a venous blood sample.

163 Venous blood samples (0 h predose through 28 h after dos

164 CMRGlu quantified with venous blood samples (n = 6) showed excellent agreement

165 ompare pH and Pco2 of intraosseous and mixed venous blood samples after sequential infusions of fluid

166 O2 max , direct Fick by femoral arterial and venous blood samples and Doppler ultrasound blood flow m

167 d at peak exercise from femoral arterial and venous blood samples and leg blood flow (by thermodiluti

168 Femoral arterial and venous blood samples and muscle biopsy samples were coll

169 Venous blood samples and urine were collected for pharma

170 etics were derived from femoral arterial and venous blood samples and vastus lateralis muscle biopsie

171 Venous blood samples and vastus lateralis muscle biopsy

172 In addition, IDIF results obtained with venous blood samples and with a transformed venous-to-ar

173 Peripheral venous blood samples at 24 and 72 hrs after the onset of

174 Venous blood samples can be used for absolute quantifica

175 le angina pectoris undergoing PCI had serial venous blood samples drawn before PCI, after PCI, and at

176 cs were determined from femoral arterial and venous blood samples during a primed-constant d5-phenyla

177 mples to quantify S. japonicum infection and venous blood samples for hemograms and measures of iron

178 provided stools to quantify reinfection and venous blood samples for hemograms and measures of iron

179 ood samples could serve as an alternative to venous blood samples for the diagnosis of EVD in resourc

180 vels were measured in adrenal and peripheral venous blood samples from 2 patients.

181 H and PB NK cells in paired liver biopsy and venous blood samples from 70 patients with chronic HCV i

182 Venous blood samples from healthy volunteers were expose

183 We obtained peripheral venous blood samples in 23 subjects with NNV-ARMD or tre

184 Venous blood samples obtained before scanning were expos

185 ably frozen mononuclear cells separated from venous blood samples obtained from 111 infants born to H

186 Venous blood samples of 20 healthy volunteers were expos

187 moglobin concentration (Hb) in capillary and venous blood samples of HIV-negative and HIV-positive su

188 corticotropin levels were higher in adrenal venous blood samples than in peripheral venous samples,

189 ance between measurements of fingerprick and venous blood samples using the standard hematology analy

190 Concurrent venous blood samples were acquired for blood metabolite

191 Peripheral venous blood samples were analyzed including assessments

192 Peripheral and portal venous blood samples were assayed for midazolam and [15N

193 Venous blood samples were collected and assayed to exami

194 oth protocols, arterial and internal jugular venous blood samples were collected at rest and coupled

195 Venous blood samples were collected at weeks 0, 4, and 8

196 Additionally, venous blood samples were collected before (18)F-FLT inj

197 Venous blood samples were collected before blue dye infu

198 Dynamic scans were acquired and venous blood samples were collected during the (18)F-FLT

199 Fasted venous blood samples were collected for iron isotopic an

200 Venous blood samples were collected from 44 patients wit

201 s on TLR expression and function, peripheral venous blood samples were collected from healthy volunte

202 y on TLR expression and function, peripheral venous blood samples were collected from healthy volunte

203 Venous blood samples were collected from healthy, premen

204 Venous blood samples were collected from the extracorpor

205 Venous blood samples were drawn for thrombelastograpy (T

206 Serial venous blood samples were drawn from 1 to 240 min after

207 Venous blood samples were drawn from healthy, exclusivel

208 Serial venous blood samples were drawn over an 11-h period (8:3

209 Serial venous blood samples were drawn to assess concentrations

210 nical range from fingerprick (capillary) and venous blood samples were measured and compared using a

211 rization before and after the procedure, and venous blood samples were obtained 24, 48, and 72 hours

212 Arterial and hepatic venous blood samples were obtained after an overnight fa

213 Venous blood samples were obtained at 8 time points over

214 Serial venous blood samples were obtained for radioactivity mea

215 Methods: Venous blood samples were obtained from 48 patients subj

216 Venous blood samples were obtained from all participants

217 Venous blood samples were obtained.

218 Venous blood samples were taken at 0, 15, 30, 60, and 12

219 Venous blood samples were taken at 55 and 85 min after i

220 Venous blood samples were taken immediately after each b

221 Anthropometric measures and venous blood samples were taken monthly.

222 Venous blood samples, taken from volunteers were culture

223 Using 2 mixed venous blood samples, the method accurately assesses the

224 ers of endothelial function were measured on venous blood samples.

225 inflammatory marker levels were measured in venous blood samples.

226 Hematology was analyzed in venous blood samples.

227 Patients and controls provided venous blood samples.

228 th imaging-derived input function (IDIF) and venous blood samples.

229 ntial muscle biopsies, and femoral arterial, venous blood sampling allowed determination of glucose a

230 0, and 240 min postingestion with additional venous blood sampling at 5, 10, 15, and 30 min postinges

231 ated these at the 6-year follow-up alongside venous blood sampling for measurement of glucocerebrosid

232 PET acquisitions (10-20 min) and venous blood sampling were performed every 30-60 min thr

233 SPECT acquisitions (30 min) and venous blood sampling were performed every 60 min throug

234 30-min dynamic (18)F-FDHT PET/CT scans with venous blood sampling were performed in 14 patients.

235 ontinuous arterial and discrete arterial and venous blood sampling were performed to determine a plas

236 Arterial and femoral venous blood sampling, thermodilution blood flow measure

237 es were obtained with concurrent arterial or venous blood sampling.

238 often used as a less invasive alternative to venous blood sampling.

239 ment with constant infusion of (18)F-FDG and venous blood sampling.

240 Thirteen of these patients also underwent venous blood sampling.

241 90- to 140-min interval after injection with venous blood sampling.

242 On day 20, a venous blood specimen tested negative for Ebola virus by

243 examination, structured clinical interview, venous blood specimens, and masked grading of seven stan

244 0.0004) 55.2% +/- 22.5% increase in retinal venous blood speed accompanied the decreases in diameter

245 h, nitrite levels are higher in arterial vs. venous blood (suggesting systemic nitrite consumption),

246 CC xenografts that receive both arterial and venous blood supplies.

247 with clearance of CNS-derived Abeta into the venous blood supply with no increase from a peripheral c

248 umferential purple discolouration indicating venous blood supply, with heaping up of the mucosa invol

249 ed cell NO with appreciably higher values in venous blood than arterial blood.

250 lood that delivers nutrients to tissues, and venous blood that removes the metabolic by-products.

251 Based on venous blood, the analysis used mean corpuscular volume

252 heoretical hepatotrophic molecules in portal venous blood (Theme I) and with the contemporaneous para

253 omic right-to-left shunts, allowing systemic venous blood to bypass gas exchange and pulmonary capill

254 us malformations (PAVMs) that allow systemic venous blood to bypass the pulmonary capillary bed throu

255 continually exposed, via gut-derived portal venous blood, to potential antigens and bacterial produc

256 Stasis of venous blood triggers deep vein thrombosis by activating

257 Clonogenic MSCs were detected in venous blood up to 1 hour after infusion in 13 of 21 pat

258 st 28 days post-treatment when fed patients' venous blood using membrane feeding assays.

259 Increasing perfusion using venous blood (VB) would be an attractive option because

260 of factors control the ratio of arterial and venous blood vessel types during angiogenesis.

261 d for the proper development of arterial and venous blood vessels, and that a major role of Notch sig

262 topographic relationship of overt damage to venous blood vessels.

263 femoral arterial and venous, and peripheral venous blood vessels.

264 he feasibility and safety of sampling portal venous blood via endoscopic ultrasound (EUS) to count po

265 A 3-mm skin snip biopsy (PKDL) or venous blood (VL) was processed by quantitative PCR.

266 ,O2) due to the influence of the intervening venous blood volume and the contribution of body O2 stor

267 LV(EV), distal pulmonary venous blood volume for vessels smaller than 5 mm(2) in

268 Venous blood was also collected and centrifuged to obtai

269 In addition, 2 mL venous blood was collected by venipuncture from all part

270 t baseline and at the end of NEFA elevation; venous blood was collected for measurement of lipids and

271 Venous blood was collected for measurements of plasma ca

272 Venous blood was collected from 13 patients with CP desp

273 g (month 0) and end (month 10) of the study, venous blood was collected from family members >18 years

274 Venous blood was collected from normal subjects and peri

275 Internal jugular venous blood was drawn from both left and right sides an

276 Venous blood was drawn to compare protein binding, paren

277 Venous blood was measured for hemoglobin (Hb).

278 Fresh venous blood was obtained and stained with monoclonal an

279 placebo or inhaled enoxaparin (2 mg/kg), and venous blood was obtained for analysis of plasma antifac

280 During coronary angioplasty, venous blood was obtained for flow cytometric detection

281 Venous blood was obtained from healthy male lowlanders (

282 Venous blood was obtained from the rabbits before and af

283 Venous blood was sampled before and after the exposure a

284 Arterialized venous blood was sampled for 2 h, and measured meal GI a

285 Arterial and hepatic venous blood was sampled in postabsorptive (n = 6; study

286 Arterialized venous blood was sampled throughout the 2-h postchalleng

287 At the end of each CWR test, venous blood was sampled to determine [Lac](blood).

288 Venous blood was sampled to measure N-terminal (NT)-proA

289 nously; arterial, portal venous, and hepatic venous blood was sampled; and liver and visceral fat wer

290 duration of labeled MSCs in the circulation, venous blood was serially drawn from five additional rat

291 Six ml of venous blood was taken for the measurement of serum uric

292 ision and accuracy of the method with use of venous blood were also tested.

293 ples and placental tissue and umbilical cord venous blood were collected and analyzed for choline and

294 blood activity measurement, small samples of venous blood were collected at various time points after

295 a quality of life (AQLQ) questionnaires, and venous blood were collected during both visits.

296 levels of mixed venous blood and mesenteric venous blood were recorded at baseline, after pericardia

297 Arterial and venous blood were sampled frequently using a peristaltic

298 rprick samples accurately reflect those from venous blood, which confirms the potential of capillary

299 relative change in volume for arteriole vs. venous blood within primary vibrissa cortex of awake, he

300 on of spillover between arterial and hepatic venous blood without portal venous data.