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1 ing the new entity of 'chronic cerebrospinal venous insufficiency'.
2 ontroversial theory of chronic cerebrospinal venous insufficiency.
3 0 patients subjected to sclerotherapy due to venous insufficiency (45 women and 15 men, mean age 51.2
4 features of venous disorders such as chronic venous insufficiency and varicose veins.
5 inflammation probably contributes to chronic venous insufficiency, and little effective treatment exi
6          There was no evidence that alcohol, venous insufficiency, arterial disease or antibodies to
7                        Chronic cerebrospinal venous insufficiency (CCSVI) has been implicated in the
8        The presence of chronic cerebrospinal venous insufficiency (CCSVI) was defined according to pr
9 nervous system, termed chronic cerebrospinal venous insufficiency (CCSVI), is claimed to be a patholo
10 literature relating to chronic cerebrospinal venous insufficiency (CCSVI).
11 dy was to document the prevalence of chronic venous insufficiency (CVI) and its associated factors in
12    How complications associated with chronic venous insufficiency (CVI) develop is not clear.
13  in venous disease-specific quality of life (Venous Insufficiency Epidemiological and Economic Study
14 sability from pulmonary embolism and chronic venous insufficiency, especially when the proximal iliof
15 ous thermal ablation (EVTA) to treat chronic venous insufficiency has increased rapidly in the US.
16                                      Chronic venous insufficiency is characterized by inadequate veno
17 cularly for vascular diseases (e.g., chronic venous insufficiency [odds ratio (OR) (95% confidence in
18 s available for the investigation of chronic venous insufficiency of the lower limbs (CVI), with an o
19          Symptoms of lower extremity chronic venous insufficiency or pelvic venous disease can develo
20 evices), venous stasis (such as from chronic venous insufficiency or prolonged immobility), and hyper
21 ned as persistent symptoms, signs of chronic venous insufficiency, or both, occurs in 25% to 50% of p
22 ch is needed to determine what role, if any, venous insufficiency plays in the course of MS and also
23 g can show distinctive features of choroidal venous insufficiency previously identified with UWF ICGA
24 zed debridement samples from lower-extremity venous insufficiency ulcers using the following: convent
25                         They include chronic venous insufficiency, varicose veins, lipodermatoscleros