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1 ER2 (apolipoprotein E receptor 2) and VLDLR (very-low-density lipoprotein receptor).
2 (d), approximately 30 nM) but not to soluble very low density lipoprotein receptor.
3 e gene expression for lipoprotein lipase and very-low-density lipoprotein receptor.
4 rs, apolipoprotein E receptor 2 (Apoer2) and very-low-density lipoprotein receptor.
5 s cortical layering by signaling through the very low density lipoprotein receptor and apolipoprotein
6 orylated form in mice lacking Reelin or both very low density lipoprotein receptor and apolipoprotein
7 ons for two LDL receptor family members, the very low density lipoprotein receptor and the apoE recep
8 eted extracellular protein that binds to the very low density lipoprotein receptor and the apolipopro
9 gnaling pathway that requires its receptors, very low-density lipoprotein receptor and apolipoprotein
10 by binding to the two lipoprotein receptors, very-low-density lipoprotein receptor and apolipoprotein
11 Reelin that is not mediated by Disabled1 or very-low-density lipoprotein receptors and apolipoprotei
12 y, including the receptors ApoER2 and VLDLR (very low density lipoprotein receptor) and the adapter p
14 ily, the apolipoprotein E receptor 2 and the very low density lipoprotein receptor, and regulates neu
15 eceptors for Reelin, including integrins and very-low-density lipoprotein receptor/apolipoprotein E2
16 ransfer experiments, we demonstrate that the very low density lipoprotein receptor can also bind and
17 r domains of apolipoprotein E receptor 2 and very low density lipoprotein receptor, Dab1 is preferent
18 l mouse models of retinal blood leakage, the very-low-density-lipoprotein receptor deficient mouse (V
19 l blood vessels invading photoreceptors: the very low-density lipoprotein receptor-deficient (Vldlr(-
21 the likelihood that this polymorphism in the very-low-density lipoprotein receptor gene is strongly a
22 rgets proteins involved in lipid metabolism (very low-density lipoprotein receptor), immunity (major
23 We determined the allele frequencies of the very-low-density lipoprotein receptor in 3 white populat
24 region of an apolipoprotein E receptor, the very-low-density lipoprotein receptor, is genetically as
25 ation and vascular leakage in the eyecups of very low-density lipoprotein receptor knockout mice, a m
26 creases low-density lipoprotein receptor and very low-density lipoprotein receptor levels via an extr
28 a with plasminogen activator inhibitor 1 and very low-density lipoprotein receptor on HUVEC or HAEC s
29 ells via apolipoprotein E receptor 2 and the very low density lipoprotein receptor, resulting in the
30 ptor proteins, apolipoprotein receptor 2 and very low density lipoprotein receptor, results in a Reln
32 an interaction with the RELN receptor VLDLR (very low-density lipoprotein receptor); this was confirm
33 ails of the lipoprotein receptors ApoER2 and very low density lipoprotein receptor through an amino-t
34 runcated TFPI molecule, is recognized by the very low density lipoprotein receptor (VLDL receptor) in
35 the LDL receptor-related protein (LRP), the very low density lipoprotein receptor (VLDL), and apoE r
37 rototype of this family, which also contains very-low-density lipoprotein receptors (VLDL-R), apolipo
39 Reelin signals via the lipoprotein receptors very low density lipoprotein receptor (VLDLR) and apolip
40 ther proteins, cell surface receptors termed very low density lipoprotein receptor (VLDLR) and apolip
41 to apolipoprotein E receptor 2 (ApoER2) and very low density lipoprotein receptor (VLDLR) and is int
44 lves two neuronal cell surface proteins, the very low density lipoprotein receptor (VLDLR) and the ap
47 ransfer to study the in vivo function of the very low density lipoprotein receptor (VLDLR) in low den
50 include the LDLR-related protein (LRP), the very low density lipoprotein receptor (VLDLR), the apoli
51 migrating cortical neurons by binding to the very low density lipoprotein receptor (VLDLR), the apoli
52 induced choroidal neovascularization and the very low density lipoprotein receptor (Vldlr)-knockout m
54 le seven-module cluster, vertebrate VgRs and very low density lipoprotein receptors (VLDLR) which hav
55 tly to lipoprotein receptors, preferably the very low-density lipoprotein receptor (VLDLR) and apolip
56 ion to binding human PCDH10, could also bind very low-density lipoprotein receptor (VLDLR) and apolip
57 nt in Reelin, Disabled 1 (Dab1), or both the very low-density lipoprotein receptor (VLDLR) and the ap
61 le pathways including through the receptors, Very low-density lipoprotein receptor (Vldlr), Apolipopr
62 Mice deficient in another Reelin receptor, very low-density lipoprotein receptor (VLDLR), had norma
63 r class A domain-containing 3 (LDLRAD3), and very low-density lipoprotein receptor (VLDLR), which fac
67 engagement for two of their entry receptors: very-low-density lipoprotein receptor (VLDLR) and protoc
68 ng embryonic neurons requires binding to the very-low-density lipoprotein receptor (VLDLR) and the ap
74 orted to be critical for binding EEEV to the very-low-density lipoprotein receptor (VLDLR), an EEEV r
76 retinopathy and another angiogenic model of very-low-density lipoprotein receptor (Vldlr)-deficient
78 the brain involves the binding of Reelin to very-low-density lipoprotein receptors (VLDLR) and apoli
79 scular entothelial growth factor [VEGF], and very low density lipoprotein receptor [VLDLR]) were test
80 induced CNV or mice with a deficiency in the very low-density lipoprotein receptor), we found that de
81 uncation disrupts an interaction with VLDLR (very low-density lipoprotein receptor), while the APOER2