ライフサイエンスコーパス: vesicle

1 vesicle front and can be engaged on the same vesicle.

2 (i.e., lipid packing) in the synthetic lipid vesicles.

3 such as RNA and protein are loaded in these vesicles.

4 ased the overall numbers of axonal transport vesicles.

5 uncoating during endocytosis of presynaptic vesicles.

6 ng allowed for online analysis of individual vesicles.

7 0 msec) and synchronous fusion of the docked vesicles.

8 significantly reduced trafficking of CD63(+) vesicles.

9 as a signal for cargo loading into secretory vesicles.

10 y increased number of mitochondria and small vesicles.

11 sing the readily releasable pool of synaptic vesicles.

12 compartment to another is via intracellular vesicles.

13 virus progenies within autophagosome-derived vesicles.

14 olecules in individual nanometer transmitter vesicles.

15 t LPL adopts a filament-like distribution in vesicles.

16 RNA, miRNA, lincRNA, tRNA and piRNA in these vesicles.

17 r action-potential evoked fusion of synaptic vesicles.

18 ransmitter release from differentially-sized vesicles.

19 to take up droplets of medium into internal vesicles.

20 oligomers and microfilaments bound to lipid vesicles.

21 myloid polypeptide (hIAPP), with giant lipid vesicles.

22 differences in the surface phenotypes of the vesicles.

23 id rafts and the AP2A1/2 complex in clathrin vesicles.

24 , in contrast to synthetic giant unilamellar vesicles.

25 was also studied in right-side-out membrane vesicles.

26 virions en bloc via infectious extracellular vesicles, 100~1000 nm in diameter, secreted from host ce

27 lation resulted in the capture of many small vesicles (30 nm diameter) at the mitochondrial surface.

28 hat chemotherapy-induced small extracellular vesicles accelerate breast cancer metastasis, and target

29 Synaptic vesicles accumulate neurotransmitters, enabling the quan

30 es results from a gradient of actin-positive vesicle activity and is essential for developmental succ

31 RNAs (sRNA), which were protected within the vesicles against RNase treatment.

32 f genes related to microtubules, presynaptic vesicle alteration, and behavioral alterations.

33 ano-roughness, and relative abundance of non-vesicles among different isolation methods.

34 icles (EVs), and their subpopulations (micro-vesicles and exosomes), and microRNAs (miRNA-21-3p, miRN

35 re of tissue iron which co-localizes with DA vesicles and is necessary for DA synthesis, was assessed

36 mall molecules across the membranes of lipid vesicles and living cells.

37 bule V of CB (1) -KO contained less synaptic vesicles and lower vesicle density; by contrast, vesicle

38 DOPC and PGPC separate into vesicles and micelles.

39 equent interactions with other intracellular vesicles and organelles generate the final content of th

40 cated and accumulates in Lewy bodies rich in vesicles and other lipid membranes.

41 ered by ligation of NKp30 or NKG2D, included vesicles and SMAPs which contained TSP-1, perforin, and

42 by directing it to Rab 5-positive (Rab 5(+)) vesicles and targeting it for sequestration within the t

43 Proteomic studies of plasma extracellular vesicles and VSM from PTH1R-VKO mice identified C1r (com

44 neurotransmitters and protons from synaptic vesicles, and is supported by direct data from sensory r

45 se from photoactivatable polymers, micelles, vesicles, and photoswitches, along with the related emer

46 tunneling nanotubes (TNTs) or shed membrane vesicles, and this changes the phenotype of recipient ce

47 tion, involving gap junctions, extracellular vesicles, and tunneling nanotubes, some of which have be

48 Conditioned media containing these vesicles appeared to increase fibroblast motility.

49 Within 14 ms, new vesicles are recruited and fully replenish the docked po

50 We performed an extracellular vesicle array to find proteins on plasma sEVs that are d

51 g glycoproteins, microRNAs and extracellular vesicles as potential biomarkers of disease.

52 attachment protein receptor (SNARE) molecule vesicle-associated membrane protein 4 (VAMP4) as a key c

53 teins from the vesicle membrane (R-SNAREs or vesicle-associated membrane proteins [VAMPs]) and the ta

54 y involved in the biogenesis of intralumenal vesicles at endosomes (the source of exosomes) revealed

55 shown previously to capture endosome-derived vesicles at the TGN, were individually relocalised to mi

56 play in the formation of the double-membrane vesicle autophagosome, which is the functional unit of a

57 lles and proteins, in which double-membraned vesicles (autophagosomes) sequester cytoplasmic cargos,

58 the microtubule-mediated movement of insulin vesicles away from the plasma membrane limits insulin se

59 nt study unravels a new molecular system for vesicle-based axonal transport of proteins in male and f

60 n potentials and regulate the trafficking of vesicles between somatodendritic and axonal compartments

61 an be tuned by varying the relative rates of vesicle budding, fusion and biochemical conversion.

62 in which the formation of a double-membrane vesicle called the autophagosome is a key event in the t

63 , TIRF microscopy, and cell membrane-derived vesicles called blebs, Ward et al. visualize intermediat

64 Small extracellular vesicles called exosomes affect multiple autocrine and p

65 evidence that the RNA cargo of extracellular vesicles can alter recipient cell gene expression and fu

66 The membrane of the vesicles can be transiently switched between an impermea

67 During synchronous release, multiple vesicles can fuse at a single active zone.

68 D4(+) T cells, both TNF-alpha(+) and IL-2(+) vesicles can tether with endocytic organelles (lysosomes

69 DCs -containing vesicles can uniquely activate T cells, whereas DCs lack

70 ensation on the surface of giant unilamellar vesicles capable of undergoing lipid phase separations.

71 chanical stress and leading to near-complete vesicle cargo release in sub-seconds.

72 gate complex (GBS67-CpGODN+L) shared similar vesicle characteristics (size and charge) compared to fr

73 uption of the spherical shapes, reduction of vesicle circularity, lobe formation, and modulation of v

74 nisms both to prevent un-initiated fusion of vesicles (clamping) and to trigger fusion following Ca(2

75 cytoplasmic organelles-called intracellular vesicle clusters (IVCs)-serve as sites for the synthesis

76 atypical mitochondrion near the presynaptic vesicle clusters at the synapse.

77 es for subunits of the coat protein 1 (COPI)-vesicle coatomer, which regulates retrograde trafficking

78 rcularity, lobe formation, and modulation of vesicle compactness.

79 he readily releasable pool (RRP) of synaptic vesicles, consistent with the isoproterenol-induced incr

80 Interestingly, these vesicles contain melanosome markers suggesting a melanom

81 h traditional exosomes and a novel subset of vesicles containing both beta1-integrin and Rab13.

82 scopy, which correlates with double-membrane vesicles containing vacuoles observed with electronic mi

83 phenotype and molecular composition of these vesicles could contribute to the therapeutic efficacy of

84 We propose that cargo recruitment into vesicles creates a crowded lumen that drives selectivity

85 chloride concentrations during the synaptic vesicle cycle to ensure normal synaptic transmission.

86 Tethered cytokine-containing vesicle (CytV)-endocytic organelle pairs are released se

87 lab, (3) a publicly available extracellular vesicles dataset, and (4) a publicly available phosphopr

88 nt dispersing factor (PDF) filled dense core vesicles (DCVs) to the terminals at the dorsal protocere

89 cles and lower vesicle density; by contrast, vesicle density in lobule X of CB (1) -KO remained uncha

90 O contained less synaptic vesicles and lower vesicle density; by contrast, vesicle density in lobule

91 ses the confined microenvironment of a giant vesicle, derived from a living cell.

92 Despite widely varying replication vesicle diameters, the resulting two rings of membrane i

93 11, and MbPex14) were visualized in numerous vesicles distinct from hydrogenosomes, the endoplasmic r

94 Although double-membrane vesicles (DMVs) appear to be a pan-CoV RO element, studi

95 ruits Ca(2+) channels and activates synaptic vesicle docking and priming via Munc13-1.

96 r step, both RIM and Munc13 mediate synaptic vesicle docking and priming.

97 t with the isoproterenol-induced increase in vesicle docking in cerebellar slices.

98 dent changes in global activity, in synaptic vesicle dynamics, in synapse size, and in synaptic mRNA

99 driven Cl(-) accumulation osmotically drives vesicle enlargement.

100 We performed extracellular vesicle (EV) analysis to identify markers of malignancy

101 Cells can communicate through extracellular vesicle (EV) secretion and uptake.

102 apy, (ii) immunotherapy, (iii) extracellular vesicle (EV) therapy, and (iv) machine learning-assisted

103 Extracellular vesicle (EV)-shuttled miRNAs have been proposed as an al

104 d exchange of various types of extracellular vesicle (EV).

105 Its presence in extracellular vesicles (EV) has been postulated to be important for th

106 easing evidence has shown that extracellular vesicles (EV) released by NK cells carry proteins and mi

107 Extracellular vesicles (EVs) are a heterogeneous group of natural part

108 Extracellular vesicles (EVs) are a universal feature of cellular funct

109 Extracellular vesicles (EVs) are cell-secreted nano-scale structures c

110 Extracellular vesicles (EVs) are important vectors of paracrine signal

111 Extracellular vesicles (EVs) are increasingly being recognized as impo

112 The mechanical properties of extracellular vesicles (EVs) are known to influence their biological f

113 Extracellular vesicles (EVs) are membrane vesicles secreted by cells a

114 Extracellular vesicles (EVs) are small, membrane-enclosed compartments

115 The biological significance of extracellular vesicles (EVs) as intercellular communication mediators

116 Extracellular vesicles (EVs) containing specific cargo molecules from

117 Extracellular vesicles (EVs) form an endogenous transport system for i

118 We found that extracellular vesicles (EVs) from young astrocyte were sufficient to c

119 Release of extracellular vesicles (EVs) is a common feature among eukaryotes, arc

120 cant increase in the amount of extracellular vesicles (EVs) isolated from DM mice and enhanced presen

121 vestigated the applications of extracellular vesicles (EVs) isolated from probiotic Lactobacillus pla

122 Extracellular vesicles (EVs) provide a critical means of bidirectional

123 Extracellular vesicles (EVs) represent an important method of intercel

124 a releases large quantities of extracellular vesicles (EVs) that likely facilitate communication betw

125 Cells release extracellular vesicles (EVs) to communicate over long distances, which

126 detect the formation of individual endocytic vesicles (EVs) with an unmatched temporal resolution of

127 e the diagnostic usefulness of extracellular vesicles (EVs), and their subpopulations (micro-vesicles

128 atica, are active secretors of extracellular vesicles (EVs), but research has not been undertaken inv

129 ine signaling 3 (SOCS3) within extracellular vesicles (EVs).

130 ical cytometry (VIEC) at an electrode as the vesicle exits the nanopore.

131 HIV-1 protein Nef secreted in extracellular vesicles (exNef) impairs neuronal functionality.

132 To capture synaptic vesicle exocytosis at cultured mouse hippocampal synapse

133 w intercellular cleft, keratinocyte synaptic vesicles expressing synaptophysin and synaptotagmin 1, a

134 also had a larger pool of readily-releasable vesicles, faster recovery following stimulation, and int

135 nger expressed on synaptic vesicles promotes vesicle filling with glutamate.

136 which the cell is partitioned into numerous vesicles for viral replication.

137 ronal endocytic protein that participates in vesicle formation by scission of invaginated membranes.

138 ggesting that CPSFL1 potentially facilitates vesicle formation by trafficking PA and/or PIP, known re

139 thout a priori hypotheses on the dynamics of vesicle formation.

140 Vesicles freed from synaptic clusters undergo ~1 um bout

141 Serum extracellular vesicles from 74 individuals presumed to have latent M.

142 tuberculosis peptides in serum extracellular vesicles from TB patients.

143 d basal insulin secretion, depleting insulin vesicles from the cytoplasm, and impairing GSIS.

144 ted transport, both kinesins localize to the vesicle front and can be engaged on the same vesicle.

145 Here, we use a reconstituted single-vesicle fusion assay under physiologically-relevant cond

146 affects dynamic control of COPI budding and vesicle fusion at the rims.

147 legans models and prevents alphaSyn-mediated vesicle fusion by altering the conformational properties

148 in is involved in the regulation of synaptic vesicle fusion, signifying the importance of alpha-syn-l

149 regulators of SNARE complex formation during vesicle fusion.

150 erent adaptor protein (AP) complexes promote vesicle generation at the TGN with different cargo speci

151 Our data support the vesicle germination and membrane invagination models of

152 some lamellar cristae may be organized by a vesicle germination process in the matrix, in addition t

153 [(11)C]UCB-J, a radioligand for the synaptic vesicle glycoprotein 2A (SV2A), were used to study hippo

154 h 1TC BP (ND) Conclusion: The novel synaptic vesicle glycoprotein 2A tracer, (18)F-SynVesT-1, display

155 Giant plasma membrane vesicles (GPMVs) are a widely used experimental platform

156 Here, giant plasma membrane vesicles (GPMVs) were employed to quantitatively show th

157 se (RP) measurements in a nanopore pipet and vesicle impact electrochemical cytometry (VIEC) at an el

158 Vesicle impact electrochemical cytometry (VIEC) was used

159 ens in Sjogren's syndrome, via extracellular vesicles in an apoptosis-independent mechanism.

160 n trafficking is mediated by LAMP-1-positive vesicles in association with cholesterol.

161 ffling of synaptic vesicle protein transport vesicles in axons.

162 There were as many vesicles in lobule V of CB (1) -KO as in CB (1) -WT, but

163 e central nervous system and associated with vesicles in neurons.

164 so found that neutrophils shed extracellular vesicles in the vascular lumen and that inhibition of ex

165 These results underscore the role of vesicles in thylakoid biogenesis and/or maintenance.

166 phenolic rings are nitrated, (6) cytoplasmic vesicles in vascular endothelial cells known to stain fo

167 is, as well as the replenishment of synaptic vesicles, in IHCs was not affected.

168 Extracellular vesicles, including exosomes/microvesicles (EMVs), have

169 infected cells revealed numerous membranous vesicles inside inclusions, as well as multivesicular bo

170 Uptake of vesicles into astrocytes follows a more classical pathwa

171 ally, transform originally large unilamellar vesicles into multilamellar vesicles with a collapsed in

172 -286) much more readily assembles with lipid vesicles into peptide/lipid coaggregates that resemble a

173 Exosomes are small extracellular vesicles involved in many physiological activities of ce

174 Plasmatic exosomes are extracellular vesicles involved in the intercellular communication sys

175 transport and transfer of tumor antigens in vesicles is a dominant pathway to load resident DCs for

176 Using fluorescence vesicle leakage assays, we demonstrate that TMD2 forms s

177 Circulating levels of large extracellular vesicles (lEVs), submicrometer-sized vesicles released f

178 tortion and the production of outer membrane vesicle-like features, while NPs bearing short cationic

179 toplasm with genomic RNA also in perinuclear vesicle-like structures near envelope glycoproteins or m

180 COPI vesicles likely become tethered while they bud, thereby

181 nstitute bo (3) oxidase in giant unilamellar vesicles made of PDMS-g-PEO and/or phosphatidylcholine (

182 Extracellular vesicle maturation was also defective in the vps27Delta

183 ts might benefit the understanding about the vesicles maturation, especially involving the "sorting b

184 is, and targeted inhibition of tumor-derived vesicles may be a promising therapeutic strategy to impr

185 f a discrete subset of mitochondrial-derived vesicles (MDVs) to facilitate delivery to lysosomes.

186 lipidation onto single-membrane perinuclear vesicles mediated by ATG16L1 via its WD40 domain, bypass

187 t protein receptor (SNARE) proteins from the vesicle membrane (R-SNAREs or vesicle-associated membran

188 (SEC4) positions the exocyst complex during vesicle membrane tethering, facilitating docking and fus

189 ers, we were able to quantify down to ~10(9) vesicles/mL, using as little as 60 muL of the sample.

190 mesenchymal stem cell-derived extracellular vesicles (MSC-EVs), which mediate cell-to-cell inflammat

191 Recently, extracellular membrane vesicle (MV) production has been proposed as a general s

192 of membrane interaction sites constrain the vesicle neck to a highly uniform shape.

193 while nCLCa-only mice had increased synaptic vesicle numbers, restoring normal neurotransmission.

194 Most cells form large vesicles of 350-600 nm in diameter at their apical side,

195 l and the bona fide CME population generates vesicles of a predominantly hexagonal-basket type; large

196 ng of the transporter, when reconstituted in vesicles of defined lipid composition.

197 release of ADAM10 on exosomes-extracellular vesicles of endosomal origin.

198 The outer membrane vesicles (OMVs) produced by P. gingivalis have been show

199 ve shed new light on how microbes secrete OM vesicles (OMVs) to influence inflammation, cell death, a

200 and selectively packaged into outer membrane vesicles (OMVs).

201 eased biogenesis of bacterial outer membrane vesicles (OMVs).

202 catecholamine molecules expelled from single vesicles onto an inner-wall carbon surface, where the du

203 axially close-by membranes, early endocytic vesicles, or tubular membrane structures.

204 tant roles in acidification of intracellular vesicles, organelles, and the extracellular milieu in eu

205 iposomes and penetration enhancer-containing vesicles (PEVs) modified with glucidex, a dextrin, and a

206 Mice with only nCLCb had a reduced synaptic vesicle pool and impaired neurotransmission compared to

207 activity-dependent augmentation of synaptic vesicle pool size relies exclusively on the action of Mu

208 f HuNoV virus-like particles (VLPs) to lipid vesicles produced from the individual HIE-lipid extracts

209 l acidification also increased extracellular vesicle production by mammary carcinoma cells.

210 a Na(+)/H(+) exchanger expressed on synaptic vesicles promotes vesicle filling with glutamate.

211 Neurally derived extracellular vesicle protein abnormalities also reveal a range of lat

212 we examined the distribution of the synaptic vesicle protein Synaptotagmin 2a (Syt2a) during developm

213 F1A known for its fast shuffling of synaptic vesicle protein transport vesicles in axons.

214 tion was analyzed with an ensemble of single vesicles providing detailed statistics.

215 In a mouse model, locally administered vesicles provoke significantly more potent and long-last

216 re assumed to act as platforms for supplying vesicles rapidly in the face of prolonged stimulation.

217 er, how neuronal activity regulates synaptic vesicle recycling is largely unknown.

218 The term 'extracellular vesicles' refers to a heterogeneous population of vesicu

219 Vesicle relaxation time scales suggest that the vesicula

220 The degree of vesicle release at the fewer, but larger, individual rem

221 r lumen and that inhibition of extracellular vesicle release blocks LTB4-mediated autocrine/paracrine

222 tic transmission and plasticity and synaptic vesicle release kinetics.

223 h is achieved by lowering the probability of vesicle release, promoting efficient vesicle replenishme

224 y neurons through SNARE-mediated (syntaxin1) vesicle release.

225 without altering the initial probability of vesicle release.

226 tamate loading as a function of the level of vesicle release.SIGNIFICANCE STATEMENT Auditory informat

227 of synapses in which neurotransmitter filled vesicles release their content independent of presynapti

228 ellular vesicles (lEVs), submicrometer-sized vesicles released from plasma membrane, from MetS patien

229 rapidly increasing the number of releasable vesicles reliably reproduce short-term plasticity and va

230 lity of vesicle release, promoting efficient vesicle replenishment, and increasing the readily releas

231 d release of neurotransmitters from synaptic vesicles requires mechanisms both to prevent un-initiate

232 brane flow and anterograde movement of lipid vesicles, resulting in cell polarization and locomotion.

233 When vesicles reverse direction, KIF13B relocates to the midd

234 a novel ultrasensitive digital extracellular vesicle screening technique (DEST), we measured putative

235 Extracellular vesicles (EVs) are membrane vesicles secreted by cells and can modulate biological a

236 ch is highly enriched in small extracellular vesicles secreted by many cell types-reduces the express

237 ained in hypoxia release small extracellular vesicles (sEVs) that activate mitochondrial dynamics, st

238 Small extracellular vesicles (sEVs), 50-150 nm in diameter, have been propos

239 Small extracellular vesicles (sEVs), including exosomes and small microvesic

240 atic tumor cells release small extracellular vesicles (sEVs, exosomes) that contain lipids and protei

241 ELVs are 100 nm diameter membrane vesicles shed into the urine by the cells lining the nep

242 experiments in POPC-based large unilamellar vesicles show that these main-group cations are highly s

243 release was quantified and correlated to the vesicle size all in the same nanotip.

244 demonstrate that ER leakage is influenced by vesicle size and cargo occupancy: overexpressing an iner

245 in the vps27Delta mutant, which had a larger vesicle size as measured by dynamic light scattering.

246 The vesicle size distribution testifies to a post-shock deco

247 e the dynamic behavior of the system; 3) the vesicle size does not affect the dynamics, but only the

248 of the system; and 4) heterogeneities in the vesicle size provoke stretching of the relaxation curves

249 xpressing an inert cargo protein or reducing vesicle size restores sorting stringency.

250 However, the vesicle size stays tunable by the glycyrrhizin content a

251 IAPP induced dramatic transformations of the vesicles, specifically disruption of the spherical shape

252 ned to probe the nature of the extracellular vesicle sRNAs from the parasitic nematode Heligmosomoide

253 ters are predominantly found on medium-sized vesicles, suggesting that they may be specific to microv

254 Vacuoles contained double-membrane vesicles suggestive of partially assembled virus.

255 Synaptic vesicle (SV) endocytosis is coupled to exocytosis to mai

256 ty.SIGNIFICANCE STATEMENT SV2A is a synaptic vesicle (SV) protein, the absence or dysfunction of whic

257 Vesicle tethering and fusion are thought to occur sequen

258 c fusion and protein sorting (HOPS)-mediated vesicle tethering, are required for the phosphorylation

259 y for the examination of exocyst function in vesicle tethering.

260 es (ERAS) can be visualized by labeling COPI vesicle tethers such as Tip20.

261 e that INVs are a generic class of transport vesicle that transfer cargo between these varied locatio

262 nomolar apparent affinity to PIP(2) in lipid vesicles that also contain background anionic lipids suc

263 Vesicles that are coated by coat protein complex II (COP

264 maintaining CD3zeta signalling, in endosomal vesicles that contain the insulin responsive aminopeptid

265 Small extracellular vesicles that efficiently package siRNA can significantl

266 ithin exosomes, which are small cell-derived vesicles that function in intercellular communication.

267 d trafficking of the newly formed autophagic vesicle to the recycling organelle, the lysosome.

268 ile microglia are known to use extracellular vesicles to communicate with neurons for maintaining hom

269 g, but rather exit the TGN into nonregulated vesicles to get incorporated to SGs at a later step.

270 s a dominant role, whereas KIF13B helps Rab6 vesicles to reach freshly polymerized microtubule ends,

271 w insights into how tethering factors bridge vesicles to target membranes, recruit multiple SNARE pro

272 b effector responsible for docking secretory vesicles to the plasma membrane before exocytosis.

273 lucose transporter type 4 (GLUT4)-containing vesicles to the plasma membrane in response to insulin s

274 t affected SNARE distribution and suppressed vesicle traffic similarly to the dominant-negative trunc

275 ion of Ca(2+) signaling, actin organization, vesicle trafficking and cell wall deposition, bearing co

276 larity protein biochemical interactions with vesicle trafficking to probe how various processes might

277 evealed a strong correlation between CD63(+) vesicle trafficking to the plasma membrane and focal adh

278 eval as well as functional clathrin-mediated vesicle trafficking were essential for the maintenance o

279 egulates various cellular functions, such as vesicle trafficking, activity of the Rab3 and Rab27 smal

280 e and polarity, cytokinesis, cell migration, vesicle trafficking, and receptor signaling.

281 l ATP synthesis, the chloroplast thylakoids, vesicle trafficking, and translation.

282 processes including cell division, endosomal vesicle trafficking, and viral budding.

283 34 Complex II, and thus had little impact on vesicle trafficking.

284 e in pattern-triggered immunity and the MIN7 vesicle-trafficking pathway, or a constitutively activat

285 We identified the vesicle-trafficking regulator GNOM and its suppressor, A

286 associated with calcium signaling and axonal vesicle transport (including the alpha4 nAChR subunit, t

287 calcium transfer, mitochondrial fission, and vesicle transport.

288 t yields fusion pore permeability divided by vesicle volume (g/V).

289 nd the content of the subsequently re-routed vesicles was determined by organelle proteomics.

290 4 levels and platelet-derived extracellular vesicles were increased in the SCS-groups.

291 Extracellular vesicles were prepared from hypothalamic tissues collect

292 s, zinc was detected in cortically-localized vesicles which underwent exocytosis upon activation.

293 that TPD54 defines a new class of transport vesicle, which we term intracellular nanovesicles (INVs)

294 with virus-induced cytosolic double-membrane vesicles, which may provide a tailored microenvironment

295 ction, KIF13B relocates to the middle of the vesicle, while KIF5B shifts to the back, suggesting that

296 e Ca(2+)-induced fusion of transmitter-laden vesicles whose coupling distance to Ca(2+) channels dete

297 arge unilamellar vesicles into multilamellar vesicles with a collapsed interbilayer spacing resulting

298 xpress the Escherichia coli gene mreB inside vesicles with bilayers made of lipid-polyethylene glycol

299 on of synaptotagmin-1-positive EYS secretory vesicles within the outer nuclear layer, and diminished

300 entration)()) showed spherical and irregular vesicles without perforations.