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1 ded unilateral or bilateral kidney agenesis, vesicoureteral junction obstruction, vesicoureteral refl
2  which confounds image interpretation at the vesicoureteral junction.
3 fection (UTI) must avoid inoculation-induced vesicoureteral reflex (VUR) yet still produce kidney and
4 , 2.47; 95% CI, 1.19-5.12), and grade 4 to 5 vesicoureteral reflux (0.60 per person-year; HR, 4.38; 9
5 ale (90%), 375 were white (78%), and 375 had vesicoureteral reflux (78%).
6 thral valves (four), urethral atresia (one), vesicoureteral reflux (one), and megacystis (one).
7                                      Primary vesicoureteral reflux (pVUR) is one of the most common c
8 he Randomized Intervention for Children with Vesicoureteral Reflux (RIVUR) Study and 295 controls, an
9 he Randomized Intervention for Children With Vesicoureteral Reflux (RIVUR) study, which will evaluate
10                                              Vesicoureteral reflux (VUR) (OMIM %193000), a common cau
11 INDINGS: The causal relationship between UTI-vesicoureteral reflux (VUR) and renal scarring has been
12  (UTI) and urinary tract abnormality such as vesicoureteral reflux (VUR) are given prophylactic antib
13 d an intermediate prevalence of GD-CNVs; and vesicoureteral reflux (VUR) had the fewest GD-CNVs but w
14 struction [ureteropelvic junction (UPJ)] and vesicoureteral reflux (VUR) have been identified and hav
15                                              Vesicoureteral reflux (VUR) is a common, familial genito
16                                              Vesicoureteral reflux (VUR) is a risk factor for acute p
17                                              Vesicoureteral reflux (VUR) is diverse in its natural hi
18                                      Primary vesicoureteral reflux (VUR) is one of the most common ur
19                                      Primary vesicoureteral reflux (VUR) is the most common congenita
20  in predicting acute pyelonephritis (APN) or vesicoureteral reflux (VUR) using the data of 288 patien
21 al-specific gene can therefore cause primary vesicoureteral reflux (VUR), a hereditary disease affect
22 d to ureteric bud (UB) induction defects and vesicoureteral reflux (VUR), although the mechanisms wer
23 the kidney and urinary tract (CAKUT) include vesicoureteral reflux (VUR).
24                          Among children with vesicoureteral reflux after urinary tract infection, ant
25 f pathology is lessened, however the risk of vesicoureteral reflux and consequent febrile urinary tra
26  to 5 months of age with grade III, IV, or V vesicoureteral reflux and no previous UTIs to receive co
27          In infants with grade III, IV, or V vesicoureteral reflux and no previous UTIs, continuous a
28  have greatly increased our understanding of vesicoureteral reflux and provide a promise of novel non
29 ients as bulking agents for the treatment of vesicoureteral reflux and urinary incontinence.
30 ing obstruction, may also be associated with vesicoureteral reflux and/or obstruction of the bladder
31 reteroceles, along with the recognition that vesicoureteral reflux associated with ureteroceles can b
32 nfants <= 2 years of age with a diagnosis of vesicoureteral reflux between 2009 and 2022 were recruit
33 el noninvasive molecular diagnostic tests of vesicoureteral reflux by proteomics methodology.
34 ch infants should be referred for endoscopic vesicoureteral reflux correction in their first visits.
35 le nature of bladder imaging in children for vesicoureteral reflux detection makes the search for non
36                    Traditional management of vesicoureteral reflux focuses on preventing renal compli
37 s in 298 individuals with confirmed UTIs and vesicoureteral reflux from the Randomized Intervention f
38 phylaxis decreased by 47% when adjusting for vesicoureteral reflux grade and bowel and bladder dysfun
39 trasonography imaging of the bladder to find vesicoureteral reflux has yet to be refined enough to be
40 ement of urinary tract infections (UTIs) and vesicoureteral reflux in children and examine new altern
41                             The treatment of vesicoureteral reflux in children has seen a shift from
42  an increasingly popular method for managing vesicoureteral reflux in children.
43 or detection of genetic mutations leading to vesicoureteral reflux in humans by studying differential
44 ion generates excessive apoptosis leading to vesicoureteral reflux in newborns, which underscores the
45 n may play an etiological role in congenital vesicoureteral reflux in otherwise normal children.
46                     The current diagnosis of vesicoureteral reflux involves voiding cystourethrograms
47                     The incidence of primary vesicoureteral reflux is about 1% to 2% of the general p
48 est that in some infants and older children, vesicoureteral reflux is associated with congenital stru
49 on (UTI) in infants with grade III, IV, or V vesicoureteral reflux is controversial.
50                            The management of vesicoureteral reflux is evolving, with advocacy ranging
51  tract infection (UTI) risk in children with vesicoureteral reflux is largely unknown.
52                                              Vesicoureteral reflux is neither necessary nor sufficien
53        It has been shown that the finding of vesicoureteral reflux is variable and that single studie
54 enged the traditional paradigm of aggressive vesicoureteral reflux management with surgery or antibio
55                                    Bilateral vesicoureteral reflux of infected urine was induced in 1
56 g and bladder dysfunction were predictors of vesicoureteral reflux outcomes when the infant was recei
57 y tract infections and/or renal scarring and vesicoureteral reflux persistence, respectively, in chil
58 y tract infections and/or renal scarring and vesicoureteral reflux persistence, the random forest was
59 ng UTIs but has not been investigated in the vesicoureteral reflux population.
60 sed to treat stress urinary incontinence and vesicoureteral reflux provide for similar rates of succe
61  voiding cystourethrogram to investigate for vesicoureteral reflux remains controversial.
62 eteral injection therapy, for patients whose vesicoureteral reflux requires correction, remains uncle
63 ications and seven (4.8%) patients developed vesicoureteral reflux requiring reoperation.
64                                Consequently, vesicoureteral reflux screening in siblings and offsprin
65 ve been tried as bulking agents to eliminate vesicoureteral reflux since the technique was introduced
66 he Randomized Intervention for Children With Vesicoureteral Reflux Study, a 2-year randomized clinica
67 he Randomized Intervention for Children with VesicoUreteral Reflux Study.
68 phritis and renal scarring may be related to vesicoureteral reflux that is missed by standard voiding
69 controlled trial involving 607 children with vesicoureteral reflux that was diagnosed after a first o
70 he Randomized Intervention for Children With Vesicoureteral Reflux trial and the Careful Urinary Trac
71 he Randomized Intervention for Children with Vesicoureteral Reflux trial or the Careful Urinary Tract
72                                              Vesicoureteral reflux was most common (15 patients).
73                                              Vesicoureteral reflux was simulated in four pigs, and 48
74       Although the presence of grade IV or V vesicoureteral reflux was the strongest predictor of ren
75 vel of greater than 40 mg/L, and presence of vesicoureteral reflux were all associated with the devel
76                         Sex and grade 1 to 3 vesicoureteral reflux were not associated with risk of r
77                           Early detection of vesicoureteral reflux will be valuable for prevention of
78  incontinence, decreasing surgical rates for vesicoureteral reflux, and decreasing recurrent urinary
79 phric blastema leads to renal hypodysplasia, vesicoureteral reflux, and ectopic ureters to name a few
80  of urinary leak, stricture, compression, or vesicoureteral reflux, and hospital costs were analyzed.
81 nary tract infections, indwelling catheters, vesicoureteral reflux, and immobilization hypercalcuria
82 ection (UTI) requiring antibiotics, grade of vesicoureteral reflux, and posttransplant bladder capaci
83 nary tract problems including recurrent UTI, vesicoureteral reflux, and renal scarring, the mechanism
84  circumcision debate, dysfunctional voiding, vesicoureteral reflux, and the diagnosis and follow-up o
85 bnormal screening renal sonograms often have vesicoureteral reflux, but a normal sonogram does not re
86  demonstrated moderate success in correcting vesicoureteral reflux, but little is known about its imp
87 y1 knockin mice also present megaureters and vesicoureteral reflux, caused by failure of ureters to s
88 chard Wahl reviews urinary tract infections, vesicoureteral reflux, dysfunctional voiding, and approp
89                   Both knockouts experienced vesicoureteral reflux, hydronephrosis, renal dysfunction
90 ecent studies have shown that in grades I-IV vesicoureteral reflux, open surgical intervention compar
91 inating COU from hydronephrosis secondary to vesicoureteral reflux, particularly when diagnostic imag
92 enesis, vesicoureteral junction obstruction, vesicoureteral reflux, posterior urethral valve, genital
93 e of MRU for the assessment of urolithiasis, vesicoureteral reflux, renal trauma, and fetal urinary t
94 ance of VCUG in a pediatric porcine model of vesicoureteral reflux, total radiation exposure can be r
95 f continuous antibiotic prophylaxis (CAP) in vesicoureteral reflux, train a model to predict the outc
96 porary data regarding the natural history of vesicoureteral reflux, urinary tract infection and renal
97 nts of irreversible bladder dysfunction with vesicoureteral reflux, urinary tract infection and resul
98 tion of focal liver lesions and diagnosis of vesicoureteral reflux.
99 o potentially milder manifestations, such as vesicoureteral reflux.
100 ebrile urinary tract infection commonly have vesicoureteral reflux.
101  kidney after bladder warming would indicate vesicoureteral reflux.
102 s, to potentially milder conditions, such as vesicoureteral reflux.
103 velopment of renal scarring in children with vesicoureteral reflux.
104 reased the risk of urologic complication and vesicoureteral reflux.
105 s are ureteropelvic junction obstruction and vesicoureteral reflux.
106 he two approaches in low-grade (grades I-II) vesicoureteral reflux.
107 tive value for ruling out high-grade (III-V) vesicoureteral reflux.
108 l need to reexamine treatment modalities for vesicoureteral reflux.
109 cystourethrography has been used to rule out vesicoureteral reflux.
110 place in the algorithm for the management of vesicoureteral reflux.
111 III genes result in the phenotype of primary vesicoureteral reflux.
112 hildren (112 of 117) had grade I, II, or III vesicoureteral reflux.
113 ant to prevent further damage, infection and vesicoureteral reflux.
114 ortical scarring, and long term follow-up of vesicoureteral reflux.
115 er of the 23 piglets with surgically created vesicoureteral reflux.
116 d 235 participants (80.5%) had grade IV or V vesicoureteral reflux.
117 nderwent cystourethrography (117 of 302) had vesicoureteral reflux; 96 percent of these children (112

 
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