ライフサイエンスコーパス: vessel

1 duct in proximity to a perfused endothelial vessel.

2 es in the aqueous phase in a single reaction vessel.

3 observed following the formation of a mosaic vessel.

4 reased (18)F-fluoride uptake in at least one vessel.

5 vity (vessel grouping) with largely solitary vessels.

6 GF receptor beta-positive pericytes to blood vessels.

7 likely to be associated with transition zone vessels.

8 calcification within the walls of the great vessels.

9 -based procedure performed to treat bleeding vessels.

10 root to shoot via Pi export into root xylem vessels.

11 lood flow in the human fetal heart and major vessels.

12 uent angiogenic expansion of the reassembled vessels.

13 nd angles of surrounding parent and daughter vessels.

14 er metastasis occurs via blood and lymphatic vessels.

15 arine turtles were examined on-board fishing vessels.

16 chanical behavior similar to that of natural vessels.

17 gh an intricate network of penetrating small vessels.

18 achieve rapid hemostasis in acutely injured vessels.

19 A) was based on the area of absent capillary vessels.

20 nd angles of surrounding parent and daughter vessels.

21 C(20) phosphorylation was increased in these vessels.

22 oids also actively constricted and displaced vessels.

23 by ordering the development of intraretinal vessels.

24 o when astrocyte endfeet more fully ensheath vessels.

25 emonstrated that acellular tissue engineered vessels (A-TEVs) comprised of small intestinal submucosa

26 olism, has a strong impact on cerebral blood vessels, a phenomenon known as cerebrovascular reactivit

27 ed a highly back-scattering hyper-reflective vessel across the fovea with shadow effect and adhesions

28 ennia within the inorganic matrix of ceramic vessels, act as molecular fossils and provide manifold i

29 The formation of new blood vessels after myocardial infarction (MI) is essential fo

30 On per vessel analysis, there was positive correlation between

31 oeconomic status (SES) and measures of small vessel and cardiovascular disease on observed race diffe

32 Accounting for off-vessel and on-vessel pericytes, we observed no pericyte

33 le functional heterogeneity depending on the vessel and tissue in which they are located.

34 to 10 years in 1,800 patients with de novo 3-vessel and/or left main coronary artery disease randomiz

35 Focusing on type H vessels and exosomal miR-214-3p, this study examined the

36 n of new blood vessels from the pre-existing vessels and it has a vital role in the survival and grow

37 factors, resulting in the retention of blood vessels and macrophages.

38 ral amyloid angiopathy of the leptomeningeal vessels and may trigger secondary ischaemic injury in af

39 reviously achieved oximetry on major retinal vessels and measured the total retinal oxygen metabolic

40 A-Akt1WT/flx embryos had fewer visible blood vessels and more hemorrhages than their wild-type litter

41 ells in the brain parenchyma including blood vessels and neurons, and in particular NPY and POMC neur

42 -associated blood vessels differ from normal vessels and play key roles in tumor progression.

43 ells preferentially recruited to the retinal vessels and protect vessels from diabetic damage.

44 the shear stress imposed by navigating small vessels and sinusoids.

45 required for the normal growth of CNS blood vessels and that ablation of this gene results in microc

46 tion of "renovascular units" comprising both vessels and tubules with potential interaction.

47 uced hydraulic conductance, narrow metaxylem vessels), and improve penetrability of hard, dry soils (

48 within the alveolar septa, 14% around small vessels, and 7% around the airways.

49 pendent cellular radiosensitivity in culture vessels, and radiotherapeutic applications.

50 ing with minimal or no nasal displacement of vessels, and superior optic nerve thinning with inferior

51 PF lesions did not damage the phrenic nerve, vessels, and the esophagus.

52 ascular vitreous fissures overly the retinal vessels; and (3) cisterns are continuous with prevascula

53 rk was measured to be largely independent of vessel angle, hematocrit levels, and measurement signal-

54 a we show that when 50% or more tumour blood vessels are pericyte-FAK negative, melanoma patients are

55 It is likely that collecting lymphatic vessels are under axial strain in vivo and that the open

56 A concomitant small-vessel arteriopathy in addition to major pulmonary arter

57 to assess the presence and severity of small-vessel arteriopathy.

58 hin the meniscus appeared to have less blood vessels associated with them in the vascular region of t

59 e flow-generation capacity of the remodelled vessel at the expense of normal mitochondrial function a

60 better regressed corneal blood and lymphatic vessels at 1 week after the treatment compared to monoth

61 the evidence linking cerebral SVD with large vessel atheroma, atrial fibrillation, heart failure, and

62 h Bessel TPLSM, the fluorescence signal of a vessel becomes proportional to its size, which enables c

63 -1 ligands determines tip cell selection and vessel branching.

64 Blood vessels (BVs) are considered an integral component of ne

65 7.1% and increased the distensibility of the vessels by 14.6%.

66 maintains separation of blood and lymphatic vessels by limiting the plasticity of committed endothel

67 abetes with angiographic evidence of 2- or 3-vessel CAD who were treated with either PCI or isolated

68 Extended-zone retinal vessel caliber predicts moderate DR in adolescents with

69 ymphocytic vasculitis, intravascular fibrin, vessel caliber, extent of injury, C4d positivity, and in

70 Retinal vessel calibers were measured at an optic disc centered

71 els for the automated measurement of retinal-vessel calibre in retinal photographs, using diverse mul

72 hemorrhage, most often no actively bleeding vessel can be found during revision.

73 nondiseased organs (e.g., kidney) and blood vessels can express high levels of certain GSLs, not lea

74 Accurate radiocarbon dating of pottery vessels can reveal: (1) the period of use of pottery; (2

75 lipping, and complete or incomplete cortical vessel cautery.

76 callosum of a monkey brain reveal that blood vessels, cells, and vacuoles affect axonal diameter and

77 contractile frequency, force generation and vessel compliance, as well as decreasing flow-mediated c

78 Central nervous system (CNS) blood vessels contain a functional blood-brain barrier (BBB) t

79 phyte algae, but expanded dramatically among vessel-containing angiosperms.

80 shift in microglial migratory behavior along vessels corresponded to when astrocyte endfeet more full

81 ease states, such as low disease activity or vessel damage without active disease.

82 In contrast, flow areas (P = .003) and vessel densities (P < .000) in deep capillary plexus wer

83 Peripapillary superficial retinal vessel densities were significantly reduced in patients

84 LP_CD), macular superficial vascular complex vessel density (mSVC_VD), and foveal avascular zone (FAZ

85 Compared to ST and TG groups, optimal neural vessel density and branching index in SU and STG groups

86 specimens and xenografts exhibited decreased vessel density and increased hypoxia versus pre-resistan

87 e with increased tumour size, enhanced blood vessel density and metastasis.

88 y decrease than GCC thinning; faster macular vessel density decrease rate was associated significantl

89 two thirds of the eyes showed faster macular vessel density decrease than GCC thinning; faster macula

90 Both GCC thinning and macular vessel density decrease were detectable over time in all

91 the ONH GBC (AUROC = 0.91; P = .036) and the vessel density GBC (AUROC = 0.90; P = .010).

92 Retinal vessel density in the GA rim region decreased in SVC and

93 urrent study therefore evaluated the retinal vessel density in the peripapillary and macular region o

94 each projection, the GPD percentage and the vessel density percentage, the control metric, were comp

95 antly higher in DR eyes than in normal eyes; vessel density percentages were significantly lower in a

96 the test-retest repeatability of the GPD and vessel density percentages.

97 owever, perfused small-vessel density, small-vessel density, and percent perfused vessels were consis

98 ization by radial alveolar counts, pulmonary vessel density, and right ventricle hypertrophy (RVH).Me

99 ints included radial alveolar counts (RACs), vessel density, and right ventricular hypertrophy (RVH).

100 e a larger baseline lesion size, increase in vessel density, and vessel length density.

101 However, perfused small-vessel density, small-vessel density, and percent perfus

102 the temporal raphe of the deep retinal layer vessel density, using swept-source optical coherence tom

103 that TBX1 is required for systemic lymphatic vessel development in prenatal mice and it is critical f

104 uptake of differentiating xylem cells during vessel development.

105 late mechanobiologic stimuli known to affect vessel development.

106 A three-vessel diagnostic angiogram performed prior to MT led to

107 ta, including relative vascular volume, mean vessel diameter, and distance from vasculature to necrot

108 licker stimulation increased blood velocity, vessel diameter, and therefore flow in arterioles, capil

109 owed a predicted power-law dependence on the vessel diameter.

110 Tumor-associated blood vessels differ from normal vessels and play key roles in

111 ables convenient intensity-based analysis of vessel dilation and constriction dynamics in large volum

112 Umbilical vessel dimorphism is conserved in mammals, suggesting th

113 Associations between cerebral small vessel disease (SVD) and inflammation have been largely

114 loss of microvascular function, termed small vessel disease (SVD) underlying different potential clin

115 technique for assessing the degree of small-vessel disease and postoperative outcome after PEA in ch

116 idered for the prevention of ischaemic small vessel disease but the net benefit of such an approach w

117 t to assess the presence and degree of small-vessel disease in patients with chronic thromboembolic p

118 older adults with and without cerebral small vessel disease in vivo.

119 e. atherosclerosis) and/or of cerebral small vessel disease or worse multiple sclerosis pathology.

120 000 PY for no CAD to 34.7 per 1,000 PY for 3-vessel disease).

121 an impact in diseases such as cerebral small vessel disease, the leading cause of vascular dementia.

122 The small-vessel disorder cerebral autosomal dominant arteriopathy

123 We show that the remodelled lymphatic vessel displayed increasing intrinsic contractile freque

124 a previously uncharacterized gene, ENLARGED VESSEL ELEMENT (EVE), contributes to the dimensions of v

125 otyls, these enzymes had positive effects on vessel element expansion and fiber intrusive growth.

126 on of eudicots and the mechanisms underlying vessel element formation.

127 MENT (EVE), contributes to the dimensions of vessel elements in Populus, impacting hydraulic conducti

128 stem for the transport of water, composed of vessel elements.

129 Lymphatic vessel endothelial hyaluronan receptor 1 (LYVE-1) mediat

130 Lower extremities vessel enhancement and image noise were quantified, and

131 Pulmonary vascular abnormalities such as vessel enlargement and regional mosaic perfusion pattern

132 ions of GCA include aneurysm and stenosis of vessels, even in patients with apparently clinically ina

133 y end point was 12-month incidence of target vessel failure (defined as the composite of cardiac deat

134 antial and immediate occlusion of the tumour vessels followed by haemorrhage within the tissue and th

135 und (US), and only patients who had suitable vessels for AVF creation were enrolled (n = 162).

136 Two independent readers evaluated vessels for diameter, stenosis or occlusion, arterial di

137 gesting that HE4 may cause deregulated blood vessel formation and suppress proper T cell trafficking

138 While mosaic vessel formation was the most frequently observed intera

139 ease in microvessel density, and reduced new vessel formation.

140 tivity as a checkpoint to ensure appropriate vessel formation.

141 Combined cell therapy of vessel-forming cells and renal tubule-forming cells aime

142 renal injury, renal tubule-forming cells and vessel-forming cells infused into the renal artery did n

143 A total of 46 stents in 39 vessels from 25 patients were examined in this study, an

144 ction of the lesion and its associated blood vessels from a uCT scan.

145 n of dairy and carcass products in Neolithic vessels from Britain, Anatolia, central and western Euro

146 Using mesenteric lymphatic vessels from C57BL/6J, Ub-CreER(T2) ;Rasa1(fx/fx) , Foxc

147 Vessels from Cav-1(-/-) mice exhibited increased O(2)(-)

148 Vessels from Cav-1(-/-) mice were used to test proof of

149 recruited to the retinal vessels and protect vessels from diabetic damage.

150 Mesenteric lymphatic vessels from high-fructose diet-induced metabolic syndro

151 In vessels from mice with deletion of Kir6.1 (Kir6.1(-/-) )

152 nished lymphatic contractile activity in the vessels from the MetSyn animal is associated with the de

153 ular reconstruction is needed and additional vessels from the organ donor are not available.

154 gical process for the formation of new blood vessels from the pre-existing vessels and it has a vital

155 ambitious scenario of electrifying all U.S. vessel fuels results in up to 65% net reduction in air p

156 nally participate in altering ex vivo aortic vessel function.

157 information on the attraction of seabirds to vessels, giving access to crucial information for risk-a

158 ssociation of the number of injured vessels, vessel grade, and vessel type (internal carotid artery,

159 milar degrees of xylem network connectivity (vessel grouping) with largely solitary vessels.

160 dications require to unravel new pathways of vessel growth to identify potential drug targets.

161 nd pathology and treatments modulating these vessels having therapeutic potential across the spectrum

162 and connective tissue deposition, suggesting vessel healing and durable vessel occlusion.

163 contractile function in collecting lymphatic vessels; however, less is known about the role of axial

164 ts from the effects of adjacent mutant blood vessels (i.e., cell-non autonomous).

165 ) and HCl in a closed perfluoroalkoxy alkane vessel in 90 min.

166 ologous leukocytes to generate a same donor "vessel in a dish" bioassay.

167 odies injected intravenously in a peripheral vessel in the left arm.

168 usly with unlabeled antibody in a peripheral vessel in the right arm (10 mg/kg, providing therapeutic

169 Whole image and parafoveal deep macular vessels in glaucoma eyes (21.0%+/-8.7%, 24.4%+/-9.6%) we

170 Despite many reports about pulmonary blood vessels in lung fibrosis, the contribution of lymphatics

171 Lymphatic vessels in meninges drain interstitial fluid into the de

172 eads to misconnection of blood and lymphatic vessels in mice and humans.

173 rcts, reactive astrocytes, and damaged blood vessels in multi-infarct dementia when compared to AD.

174 fects the vascular response in small retinal vessels in order to maintain hemodynamic regulation in t

175 In contrast, CO(2) dilates vessels in other brain areas such as the amygdala.

176 Peripheral lymphatic vessels in patients with HFpEF exhibit structural and mo

177 e adipocytes controls the formation of blood vessels in the bone marrow, and also regulates the diffe

178 cal regulator for the remodeling of coronary vessels in the developing heart.

179 Lymphatic vessels in the donor lung exhibited active sprouting tow

180 construction of functional human-mouse blood vessels in the grafts that promoted cell survival in the

181 loss, is caused by damage to the small blood vessels in the retina.

182 Disorganized vessels in the tumor vasculature lead to impaired perfus

183 emerged controversial role of the lymphatic vessels in tumor dissemination and cancer immunotherapy.

184 ial cells and induced mispatterning of blood vessels in zebrafish.

185 CGRP-induced relaxations in mouse and human vessels, in vitro.

186 er patient (adjusted odds ratio, 1.6 per one-vessel increase [95% CI: 1.3, 2.2]; P < .001) were assoc

187 [5.2%]; P < .001), and increasing number of vessels injured per patient (adjusted odds ratio, 1.6 pe

188 cell-laden structures with 2 chambers and a vessel inlet and outlet.

189 and in the cytosol, supporting normal blood vessel integrity and development.

190 Recanalization of the occluded vessel is essential but not sufficient to guarantee brai

191 alization and experimental analysis of these vessels is challenging in mammals.

192 yloid (Abeta) deposits around cerebral blood vessels, is a major contributor of vascular dysfunction

193 (95% CI = 29-91%) increase in risk for small vessel ischemic stroke, a 197% increase (95% CI = 59-457

194 tions of morphology characterized by reduced vessel length and increased density of tip cells and per

195 Vessel length density (VLD), perfusion density (PD), and

196 lesion size, increase in vessel density, and vessel length density.

197 ered between the single- and triple-vascular-vessel lesion groups.

198 tion to enhance the radio-density within the vessel lumen.

199 WSS promoted monocyte interactions with the vessel lumen.

200 -induced apoptosis, indicating that low-flow vessels may be uniquely susceptible to YK-4-279-mediated

201 nd imply that therapeutic targeting of blood vessels may restore aged endocrine tissue function.

202 e.g. decreased flow or occlusion) in retinal vessels may serve as a useful diagnostic indicator of di

203 trasound mapping is routinely used to select vessels meeting minimal threshold diameters for surgical

204 Correlations between MCT and choroidal vessel metrics of CVV, CSV, CVI, and CSVR were studied.

205 time series is then mapped to a preoperative vessel model to determine the relative position of the c

206 brain angiogenesis by showing uncoupling of vessel morphogenesis and blood-brain barrier formation.

207 Abnormal control of blood vessel morphogenesis and maturation is linked to the pat

208 ular homeostasis that directly affects blood vessel morphogenesis, angiogenesis, and tissue permeabil

209 ally expressed genes (DEGs) related to blood vessel morphogenesis.

210 52 [95% CI, 1.23-5.18]; P=0.009), and target vessel myocardial infarction (8% versus 14%; hazard rati

211 ed as the composite of cardiac death, target vessel myocardial infarction, or target vessel revascula

212 significantly decreased in MetSyn lymphatic vessels, myosin light chain 20, MLC(20) phosphorylation

213 o a high prevalence of horizontally oriented vessels nasally and the horizontal optical defocus induc

214 Regions of metaxylem vessels near syncytia were found to have deviated from c

215 Here, we test the hypothesis that vessel noise level is a driver of disturbance, using hum

216 ium (o = 0.1 to 0.5 mm, P = 0.03), and large vessels (o >= 0.5 mm, P = 0.02).

217 e, the speed with which a patient with large vessel occlusion is transferred to a thrombectomy-capabl

218 r the 0.25-mg/kg dose in patients with large vessel occlusion ischemic stroke in whom endovascular th

219 Among patients with large vessel occlusion ischemic stroke, a dose of 0.40 mg/kg,

220 Conclusion Screening for large-vessel occlusion with CT angiography in patients with ac

221 nts with acute ischaemic stroke due to large vessel occlusion within a 12 h treatment window.

222 ition, suggesting vessel healing and durable vessel occlusion.

223 Spinal cord blood vessels of CMH-treated mice showed reduced expression of

224 of NOTCH3 protein in pathologically affected vessels of human CADASIL-affected brains.

225 trate the renal vascular network to generate vessels; only administering them into the kidney parench

226 ture in intact embryonic hearts and quantify vessel organization.

227 s fat was cultured in vitro to promote blood vessel outgrowth prior to implantation into immunocompro

228 sed displacement of the deep large choroidal vessels over the superior macular area even after tumor

229 iltration (P = .0044) and hyperplastic blood vessels (P = .0005).

230 dition to enabling identification of central vessel pathologic features, ferumoxytol-enhanced MR angi

231 Accounting for off-vessel and on-vessel pericytes, we observed no pericyte loss relative

232 The endothelial glycocalyx regulates blood vessel permeability and homeostasis.

233 in maintaining cell wall integrity of xylem vessels, physiological and molecular consequences due to

234 e C(16:0) and C(18:0) fatty acids in pottery vessels provide an internal quality control of the resul

235 Vessel reassembly was not limited by VEGFA neutralizatio

236 Neutralization of ANG2 did not prevent vessel reassembly, but did impair subsequent angiogenic

237 t was the composite of cardiac death, target vessel-related myocardial infarction, or target lesion r

238 Pericyte maturation was necessary to undergo vessel remodeling during angiogenesis.

239 es for the ANGPT2 N-terminal domain in blood vessel remodeling, tumor growth, metastasis, integrin bi

240 ss in the biofabrication of large- and small-vessel replacements, functional microvasculature has bee

241 A-treated targets showed significantly fewer vessel restrictions compared with IRF (P<=0.023).

242 have an inherent capacity to form new blood vessels, resulting in excessive lesion growth, and this

243 ses deficient cardiac remodeling of coronary vessels, resulting in improper coronary artery formation

244 rget vessel myocardial infarction, or target vessel revascularization).

245 utrophils were also found in atherosclerotic vessels, revealing that they arise across distinct conte

246 When a vessel's demersal catch exceeds owned and leased quota f

247 cerebrovascular flow resistance of all brain vessel segments, thereby controlling capillary flow whil

248 V), distal pulmonary venous blood volume for vessels smaller than 5 mm(2) in cross section (BV5), CT

249 ors were associated with lower risk of small vessel stroke (OR: 0.82, 95% CI = 0.75-0.89) and lower W

250 vels was associated with lower risk of small vessel stroke [odds ratio (OR) per standard deviation =

251 hemic stroke, large artery stroke, and small vessel stroke in all univariable MR analyses, but the as

252 not significantly different from those of WT vessels, suggesting that basal K(ATP) channel activity i

253 ) the use of a miniature vapor-equilibration vessel (the "saturator") to minimize the temperature gra

254 discuss the unique characteristics of blood vessels, the heart, and the kidney of giraffes and how t

255 ertrophy, dysfunction of blood and lymphatic vessels, the overall oestrogen dependence and the associ

256 vascular tufts while sparing healthy retinal vessels, thereby demonstrating the therapeutic potential

257 ng and entry of dendritic cells to lymphatic vessels through selective adhesion to its ligand hyaluro

258 thoracoabdominal NRP opening the aortic arch vessels to atmosphere allows collateral flow to be diver

259 equential lymph nodes and efferent lymphatic vessels to enter the bloodstream.

260 Streptococcus pyogenes also hijack lymphatic vessels to escape a local infection site, transiting thr

261 lly in human bladder cancer-associated blood vessels to find novel biomarkers and mechanisms involved

262 essels were >100-fold less sensitive than WT vessels to pinacidil.

263 nsisting of increased cup-disk ratio (3.8%), vessel tortuosity (2.5%), retinal pigment epithelium deg

264 haracterized over a 21 day cross-latitudinal vessel transit using a combination of molecular methods.

265 verse clinical outcomes, irrespective of the vessel treated.

266 relative position of the catheter within the vessel tree.

267 number of injured vessels, vessel grade, and vessel type (internal carotid artery, vertebral artery)

268 Cut-open vessels, uncertainties in maximum air volume estimations

269 horylation in vitro in aortic and mesenteric vessels using wire myography and membrane potential meas

270 the development and maintenance of lymphatic vessel valves.

271 disorders involving severe, systemic, small-vessel vasculitis and are characterized by the developme

272 valuate association of the number of injured vessels, vessel grade, and vessel type (internal carotid

273 cteristics of pulmonary AVMs, image quality, vessel visibility, and artifact grade.

274 ing the sprouting and remodeling of coronary vessels, visualized by a specific neural enhancer from t

275 termed lncRNA-MAP3K4 that is enriched in the vessel wall and regulates vascular inflammation.

276 a activation in cDCs is necessary to control vessel wall inflammation.

277 s blockade of leukocyte interaction with the vessel wall is being studied to reduce the inflammation

278 igher number and enhancement of intracranial vessel wall lesions at 7-T MRI in individuals evaluated

279 Contrast material-enhancing vessel wall lesions were associated only with increasing

280 was found between smoking and the number of vessel wall lesions.

281 fragmentation, and effacement of HA from the vessel wall of small pulmonary arteries.

282 The plaque-to-healthy vessel wall ratio of (68)Ga-FOL was significantly higher

283 ed by the accumulation of macrophages in the vessel wall.

284 s into the PCSK6 role in normal and diseased vessel wall.

285 ant source of the measured anisotropy in the vessel wall.

286 Abnormal HA accumulation within blood vessel walls is associated with tissue inflammation and

287 Mural cell coverage of the blood vessels was also reduced in betaA-Akt1WT/flx skin compar

288 Because cartilage does not have blood vessels, we studied ear AVMs to determine if overgrown c

289 However, these vessels were >100-fold less sensitive than WT vessels to

290 , small-vessel density, and percent perfused vessels were consistently higher and then stabilized 48

291 try, such as complex networks, buildings, or vessels, where the hypersensitive nature of multiple int

292 d formed a hierarchical network of remodeled vessels, whereas in betaA-Akt1WT/flx embryos, the capill

293 ntractile frequency and amplitude in control vessels, whereas, the reduced MetSyn lymphatic contracti

294 by changes in the diameter of intra-cerebral vessels, which control cerebrovascular resistance (CVR).

295 Leakage of retinal blood vessels, which is an essential element of diabetic retin

296 he draining mediastinal LN via the lymphatic vessels, which we term retrograde migration.

297 se with both higher CAC scores and number of vessels with obstructive disease (by CAC scores: 6.2 per

298 .3 per 1,000 PY for CAC >1,000; by number of vessels with obstructive disease: 6.1 per 1,000 PY for n

299 expression to the tunica adventitia of blood vessels within the tumor.

300 by tumor-associated or normal stromal blood vessels yet its significance may differ from the one fro