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1 igh rates of post-operative hearing loss and vestibular dysfunction.
2 mproves hearing sensitivity, and ameliorates vestibular dysfunction.
3 f hearing and vision, and varying degrees of vestibular dysfunction.
4 l defects and eventually to hearing loss and vestibular dysfunction.
5 ls only in the genetic background exhibiting vestibular dysfunction.
6 ilure of normal canal development results in vestibular dysfunction.
7 mic, hereditary hearing loss with associated vestibular dysfunction.
8        None of the strains displays signs of vestibular dysfunction.
9 ir death leads to permanent hearing loss and vestibular dysfunction.
10 that are useful models of human deafness and vestibular dysfunction.
11 ngenital deafness, retinitis pigmentosa, and vestibular dysfunction.
12 d for designing therapies to treat inner ear vestibular dysfunction.
13 of either central neurological or peripheral vestibular dysfunction.
14  target of mutations that cause deafness and vestibular dysfunction.
15 curring in DFNA9 leading to hearing loss and vestibular dysfunction.
16 t may cause or contribute to deafness and/or vestibular dysfunction.
17 itterbug (jbg), exhibit impaired hearing and vestibular dysfunction.
18 amed hurry-scurry (hscy) causes deafness and vestibular dysfunction.
19 tial for using antisense technology to treat vestibular dysfunction.
20 ) mouse carries a recessive mutation causing vestibular dysfunction.
21 re completely deaf and also display signs of vestibular dysfunction.
22 ated in ames waltzer and causes deafness and vestibular dysfunction.
23 causes hair cell degeneration, deafness, and vestibular dysfunction.
24 he inner ear that result in hearing loss and vestibular dysfunction.
25 het) is a recessive mutation in mice causing vestibular dysfunction.
26 ngenital deafness, retinitis pigmentosa, and vestibular dysfunction.
27 ion and navigational deficits observed after vestibular dysfunction.
28 ia was one indicating compensated peripheral vestibular dysfunction.
29  loss of vestibular hair cells and permanent vestibular dysfunction.
30 sociated with profound retinal, auditory and vestibular dysfunction.
31 tivity, Gpr156 mutant mice do not show overt vestibular dysfunction.
32 se likely contributes to the pathogenesis of vestibular dysfunction.
33 JLNS2), resulting in congenital deafness and vestibular dysfunction.
34 o treat synaptopathies linked to auditory or vestibular dysfunction.
35   Non-autonomic neurological causes included vestibular dysfunction (32 [5%]) and epilepsy (11 [1.7%]
36  low dose of DT caused profound SNHL without vestibular dysfunction and had no effect on wild-type (W
37 cargo whirlin are implicated in deafness and vestibular dysfunction and have been shown to localize a
38                  Autonomic manifestations of vestibular dysfunction and motion sickness are well esta
39  congenital sensorineural deafness, constant vestibular dysfunction and prepubertal onset of retiniti
40 th Bt2 significantly reduced the severity of vestibular dysfunction and prevented earlier mortality.
41 ice, which is characterized by hearing loss, vestibular dysfunction, and coat color dilution.
42  disorder manifested by congenital deafness, vestibular dysfunction, and progressive retinal degenera
43 d by progressive sensorineural hearing loss, vestibular dysfunction, and retinitis pigmentosa.
44                                 Deafness and vestibular dysfunction are extremely common sensory diso
45            In mammals, hair cell loss causes vestibular dysfunction as spontaneous regeneration is ne
46                                  Subclinical vestibular dysfunction, as identified by clinical tests,
47                       They are deaf and have vestibular dysfunction but do not develop photoreceptor
48 elial defects manifested by hearing loss and vestibular dysfunction but no retinal pathology.
49 t prove useful in ameliorating some forms of vestibular dysfunction by modifying ongoing primary vest
50 ty-related symptoms; and (3) the severity of vestibular dysfunction can predict whether hyperactivity
51 has been eliminated in supporting cells have vestibular dysfunction caused by failure of synapse form
52                                              Vestibular dysfunction, causing dizziness and imbalance,
53 al, Tmc1(Delta)Tmc2(Delta) mice had profound vestibular dysfunction, deafness, and structurally norma
54                                       Severe vestibular dysfunction developed in mice infected with B
55 een identified as causative for deafness and vestibular dysfunction (DFNB18B).
56 the severity rather than the age of onset of vestibular dysfunction differentiates whether hyperactiv
57 the waltzer (v) locus result in deafness and vestibular dysfunction due to degeneration of the neuroe
58 ouette (pi) locus result in hearing loss and vestibular dysfunction due to neuroepithelial defects in
59 inner (sr) locus results in hearing loss and vestibular dysfunction due to neuroepithelial defects in
60 ids have been used to treat hearing loss and vestibular dysfunction for many years.
61 etric effectively distinguishes animals with vestibular dysfunction from those with normal vestibular
62 ased gene therapy to ameliorate auditory and vestibular dysfunction has been proposed.
63 d overt behavioral defects characteristic of vestibular dysfunction have not been described.
64                                Patients with vestibular dysfunction have visual, perceptual, and post
65 nel-like 1 (TMC1) cause hearing loss without vestibular dysfunction in both mice and humans, we inves
66                        Hearing impairment or vestibular dysfunction in humans often results from a pe
67  target of mutations that cause deafness and vestibular dysfunction in mice and humans.
68 tal deafness in humans and both deafness and vestibular dysfunction in mice homozygous for the shaker
69 vision symptoms in patients, our findings of vestibular dysfunction in these Dfnb31 mutants raise the
70 zation on hair cells leading to deafness and vestibular dysfunction in waltzer mice.
71 dels are powerful for assessing auditory and vestibular dysfunction, in vitro human inner ear models
72 T conditions, while clarifying the impact of vestibular dysfunction, individual characteristics, vary
73                                        Human vestibular dysfunction is an increasing clinical problem
74                     The most common cause of vestibular dysfunction is arguably vestibular hair cell
75          A major cause of human deafness and vestibular dysfunction is permanent loss of the mechanos
76  possible modifiable risk factors, including vestibular dysfunction, is vital.
77  progressive, sensorineural hearing loss and vestibular dysfunction known as DFNA9.
78  here are a gene dose-sensitive cause of the vestibular dysfunction observed in EphB-Efnb2 signaling-
79 ar defect, but that may include mutants with vestibular dysfunction of the inner ear.
80 reflex (VOR) responses demonstrated that the vestibular dysfunction of the Zpld1 mutant mice is cause
81                               In the case of vestibular dysfunction or motion sickness, the unpleasan
82                     Behavioral recovery from vestibular dysfunction produced by unilateral labyrinthe
83  life cognitive-motor demands in people with vestibular dysfunction (PwVD), functional gait and falls
84 similar hair bundle defects may underlie the vestibular dysfunction reported in humans with mutations
85 used more severe arthritis, myocarditis, and vestibular dysfunction than serotype A.
86 ess associated with retinitis pigmentosa and vestibular dysfunction (USH1D) were previously mapped to
87                                              Vestibular dysfunction was also observed in mutant mice.
88                    However, a milder form of vestibular dysfunction was apparent from altered vestibu
89                                              Vestibular dysfunction was associated with space and mot
90 d without agoraphobia and to discern whether vestibular dysfunction was associated with specific symp
91  laboratory investigations, suggest that the vestibular dysfunction was immune mediated.
92 2a2 mutations cause hyperactivity; (2) it is vestibular dysfunction, which frequently co-occurs with
93 ke-wave seizures, cerebellar gait ataxia and vestibular dysfunction, which limit traditional approach
94 ain cause of balance disorders is peripheral vestibular dysfunction, which may occur as a result of h