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1 ite (HA), and (3) binding to bacteria (e.g., viridans streptococci).
2 ifferentiating Streptococcus pneumoniae from viridans streptococci.
3 neumoniae DNA probe before identification as viridans streptococci.
4 e revealed the presence of this gene in most viridans streptococci.
5 ous recombination with DNA from PBP genes of viridans streptococci.
6 ccus pyogenes, Streptococcus agalactiae, and viridans streptococci.
7 s, whereas periodontal health yielded mainly viridans streptococci and Actinomyces species.
8 9 clinical isolates of S. pneumoniae and 101 viridans streptococci and were as follows: Pneumoslide,
9  homologs found in S. pneumoniae serotype 2, viridans streptococci, and Enterococcus faecalis.
10                                              Viridans streptococci are a heterogeneous group of gram-
11                  It is unclear, however, why viridans streptococci, bacteria that rarely produce absc
12 he presence of periapical abscesses and oral viridans streptococci DNA-positive thrombi was found (od
13  protein concentrations to adherence by oral viridans streptococci in microplates coated with whole s
14 ar coagulation, more than 60% of isolates of viridans streptococci induced platelet aggregation when
15 ichoic acid (LTA) purified from 2 strains of viridans streptococci induced the accumulation of tumor
16 ntal infection and oral bacteria, especially viridans streptococci, may be associated with the develo
17 lure [P =.01], bacterial etiology other than viridans streptococci [P<.001 except Staphylococcus aure
18                              Most human oral viridans streptococci participate in intrageneric coaggr
19 t interferon-gamma, LTA from both strains of viridans streptococci provoked the accumulation of induc
20                     Severe illness caused by viridans streptococci rarely occurs in immunocompetent h
21     PMNL ingested greater numbers of SMG and viridans streptococci than S. aureus but killed these or
22                                          The viridans streptococci (VS) are associated with the devel
23             Streptococcus gordonii and other viridans streptococci (VS) are primary etiologic agents
24 ypical for endodontic infection, mainly oral viridans streptococci, was measured in 78.2% of thrombi,
25 an did Staphylococcus aureus, in contrast to viridans streptococci, which caused greater chemotaxis t
26                                              Viridans streptococci, which include Streptococcus gordo