ライフサイエンスコーパス: visceral

1 ic arterial wall (n = 7) and subcutaneous or visceral abdominal fat (n = 9) were most strongly implic

2 e individuals predict increases in total and visceral abdominal fat with treatment initiation.

3 The relationship between RSL and visceral abdominal fat, subcutaneous abdominal fat, tota

4 volvement of ZI in various functions such as visceral activities, arousal, attention, and locomotion,

5 lammatory response can be modulated, and the visceral adhesion formations and postsurgical complicati

6 a dysregulated inflammatory process causing visceral adhesions and postsurgical complications.

7 bioscaffold is very effective in inhibiting visceral adhesions formation and minimizing postsurgical

8 , leading to increased lipid accumulation in visceral adipocytes by an unknown mechanism.

9 We therefore compared the pattern of visceral adipose injury during acute pancreatitis and ac

10 We investigated whether visceral adipose NLRP3 impairs cognition by activating m

11 These results indicate that visceral adipose NLRP3 impairs memory via IL-1-mediated

12 Visceral adipose NLRP3 was required for deficits in long

13 iated with chronic low-grade inflammation of visceral adipose tissue (AT) characterized by an increas

14 ssion of E2F1 in adipocyte fraction of human visceral adipose tissue (hVAT) associates with a poor ca

15 e determined in biopsies of subcutaneous and visceral adipose tissue (SCAT and VAT, respectively) fro

16 between HIV-related microbiota signature and visceral adipose tissue (VAT) area (P for interaction =

17 Dual-energy x-ray absorptiometry (DXA) for visceral adipose tissue (VAT) assessment is used as an a

18 iver, subcutaneous adipose tissue (SAT), and visceral adipose tissue (VAT) in morbidly obese subjects

19 Visceral adipose tissue (VAT) is a primary site for stor

20 In particular, visceral adipose tissue (VAT) is critical for the regula

21 The degree of unsaturation was lower in visceral adipose tissue (VAT) phospholipids, indicating

22 he inflammasome protein cryopyrin (NLRP3) in visceral adipose tissue (VAT) promotes release of the pr

23 Visceral adipose tissue (VAT) was collected at d17.5 of

24 ide murine Treg cell clone isolated from the visceral adipose tissue (VAT), and identified surrogate

25 3 (DR3), a member of the TNF superfamily, on visceral adipose tissue (VAT)-derived murine and periphe

26 n both subcutaneous adipose tissue (SAT) and visceral adipose tissue (VAT).

27 pressed in Treg cells, particularly those in visceral adipose tissue (VAT).

28 inal subcutaneous fat [SAT], adipose tissue, visceral adipose tissue [VAT], and muscle) from patients

29 in adult locomotor activity, alterations in visceral adipose tissue and hepatic development, and per

30 Weight loss dose-dependently decreases visceral adipose tissue and liver fat content, and it im

31 Hence, excessive amounts of visceral adipose tissue and of ectopic fat largely defin

32 During obesity, macrophages infiltrate the visceral adipose tissue and promote inflammation that co

33 sponses and immunopathological phenotypes in visceral adipose tissue are drastically reduced in cavef

34 Body mass index, visceral adipose tissue area (cm(2)), lipids, leptin, fr

35 s, unlike diverticulitis, PNLIP leaking into visceral adipose tissue can cause excessive visceral adi

36 data set, for the subcutaneous, muscle, and visceral adipose tissue compartments, respectively.

37 Pancreatic lipase injected into mouse visceral adipose tissue hydrolyzed adipose triglyceride

38 measure skeletal muscle and subcutaneous and visceral adipose tissue indexes.

39 c measurements, greater skeletal muscle, and visceral adipose tissue indices were independently assoc

40 tion of adipose tissue into subcutaneous and visceral adipose tissue is required.

41 visceral adipose tissue can cause excessive visceral adipose tissue lipolysis independently of adipo

42 adipocyte-specific ATGL knockouts, had lower visceral adipose tissue lipolysis, milder inflammation,

43 d at overcoming glucocorticoid resistance in visceral adipose tissue may improve remodeling and help

44 lectively, these studies illuminate adaptive visceral adipose tissue plasticity in obese mice in resp

45 Visceral adipose tissue plays a critical role in numerou

46 ciation of the healthy plant-based diet with visceral adipose tissue remained statistically significa

47 sed activation of HIF-1alpha in ATM of obese visceral adipose tissue resulted in induction of IL-1bet

48 The omentum is a visceral adipose tissue rich in fat-associated lymphoid

49 ress type 2 cytokines, and share features of visceral adipose tissue Treg cells.

50 Given the unique association of visceral adipose tissue with obesity-related metabolic d

51 udies over the past 30 years have shown that visceral adipose tissue, accurately measured by CT or MR

52 in impaired accumulation of T(reg) cells in visceral adipose tissue, and is associated with enlarged

53 Lower liver fat content and visceral adipose tissue, greater insulin sensitivity and

54 t are at high risk if they have an excess of visceral adipose tissue-a condition often accompanied by

55 plant-based diets was associated with lower visceral adipose tissue.

56 city between human DM and non-diabetic (NDM) visceral adipose tissue.

57 plant-based diet index, percentage change in visceral adipose tissue: -4.9%, 95% CI: -8.6%, -2.0%).

58 riectomy and exhibited increased estrogen in visceral adipose tissues.

59 nt, notably peritoneal mesothelial cells and visceral adipose, secreted Wnt5a.

60 , body fat percentage (-2.6%; P < 0.001) and visceral adiposity (-0.2; P = 0.01) than unfit peers.

61 signaling may underlie correlations between visceral adiposity and cognitive impairment in humans.

62 Visceral Obesity summarises the evidence for visceral adiposity and ectopic fat as emerging risk fact

63 sma levels of this polyol are predictive for visceral adiposity gain and development of type 2 diabet

64 mong normal-weight patients, each SD greater visceral adiposity increased CVD risk by 70% (HR, 1.70 [

65 pants, MetScore, percentage of body fat, and visceral adiposity increased linearly across the BMI cat

66 A high visceral adiposity index was associated with adverse out

67 A high visceral adiposity index, either with or without dysglyc

68 Visceral afferents were back-labeled using retrograde tr

69 ody insulin sensitivity and also a marker of visceral and ectopic fat accumulation.

70 need for public health messaging to focus on visceral and ectopic fat in addition to excess bodyweigh

71 We discuss the measurement of visceral and ectopic fat, pathophysiology and contributi

72 ons from a public health programme targeting visceral and ectopic fat.

73 The visceral and hepatic adiposity associated with weight ga

74 itivity index and positively correlated with visceral and hepatic fat and SCD-1 activity in both grou

75 Excess visceral and intramuscular adiposity occurred across all

76 Visceral and intramuscular adiposity were associated wit

77 In the mediation analysis, visceral and liver fat jointly accounted for a statistic

78 Moreover, they showed increased visceral and liver fat, and a higher insulin resistance.

79 omplex events involved in the convergence of visceral and primitive streak-derived endoderm.

80 ities and differences in brain processing of visceral and somatic pain.

81 ticipants underwent MRI to assess volumes of visceral and subcutaneous abdominal adipose tissue and l

82 stically significant associations with lower visceral and subcutaneous abdominal adipose tissue volum

83 iations of the plant-based diet indices with visceral and subcutaneous abdominal fat volumes, LSI, an

84 ociated with accumulation and enlargement of visceral and subcutaneous adipose depots indicative of b

85 Paired visceral and subcutaneous adipose tissue samples were ob

86 distribution, such as total fat mass at DXA, visceral and subcutaneous adipose tissue, and liver and

87 tial infrastructure of the lipid networks in visceral and subcutaneous adipose tissues and suggests a

88 The visceral and subcutaneous fat areas were significantly r

89 ry artery disease, myocardial infarction and visceral and subcutaneous fat distribution; however, the

90 ract with gustatory, olfactory, homeostatic, visceral, and cognitive systems; and 4) discovering new

91 Total, visceral, and subcutaneous abdominal fat areas were meas

92 ed by vascular malformations (VMs) including visceral arteriovenous malformations and mucosal telangi

93 esity causes hyperinsulinemia and diminishes visceral AT (VAT) T reg number and function, but whether

94 ommon feature of macrophages residing in all visceral body cavities.

95 There was evidence that higher body fat and visceral body fat distribution caused elevated ACR, whil

96 ared adiposity of the trunk, intra-abdominal visceral cavity, and liver, adjusting for total fat mass

97 indicates a causal role for disgust-related visceral changes in disgust avoidance, supporting the hy

98 in subcutaneous depots are larger than their visceral counterparts (Pmeta = 9.8 x 10-7).

99 py as first-line therapy, except in cases of visceral crisis or rapidly progressive disease.

100 Leishmania (L.) infantum causes visceral, cutaneous, and mucosal leishmaniasis in humans

101 ubcutaneous fat tissues than in those in the visceral depot and that HSF1 is more abundant in differe

102 ock randomisation, stratified by presence of visceral disease and ECOG performance status.

103 0.59 versus 0.66 for local, nodal, bone, or visceral disease, respectively.

104 , and BMI-matched patients, all obese, whose visceral E2F1 protein expression was either high (E2F1(h

105 The insular cortex subserves visceral-emotional functions, including taste processing

106 lin in nutrient internalization by the early visceral endoderm and highlight its involvement in the f

107 The visceral endoderm is a polarized epithelial monolayer ne

108 We discover that Smad2 inactivation in the visceral endoderm results in increased numbers of PGCs d

109 I/HDL allows maternal nutrient uptake by the visceral endoderm.

110 ted them into the left and right side of the visceral falciform depot in six obese dogs.

111 Visceral fat (omentum) and abdominal subcutaneous fat of

112 Excessive visceral fat accumulation is a primary risk factor for m

113 eep white matter in obese subjects with high visceral fat accumulation, independent of common obesity

114 Visceral fat and age were strong individual predictors o

115 pecially in terms of increased risk of added visceral fat and increased risk of non-communicable dise

116 Lower visceral fat and SCD-1 activity may contribute to the pa

117 In multivariate models, visceral fat and SCD-1 were associated with total fastin

118 llowing abstinence, drug co-exposure reduced visceral fat and the amount of insulin required to clear

119 Notably, both visceral fat and the pro-inflammatory cytokine tumor nec

120 tissue specimens containing high amounts of visceral fat are challenging to analyze because of fat d

121 s aluminum oxide sample slide that minimizes visceral fat delocalization after thaw-mounting of tissu

122 ) mice exhibited accelerated weight gain and visceral fat deposition with age, when compared to wild

123 In a condition of dysfunctional visceral fat depots, as in the case of obesity, alterati

124 Higher visceral fat index, independent of fat mass index, was a

125 organ fat including subcutaneous fat index, visceral fat index, pericardial fat index, and liver fat

126 The visceral fat is highly susceptible to the availability o

127 it obese individuals had significantly lower visceral fat levels than unfit obese peers (-3.0; P = 0.

128 cible deletion of adipose OGT causes a rapid visceral fat loss by specifically promoting lipolysis in

129 Mechanistically, visceral fat maintains a high level of O-GlcNAcylation d

130 p experienced a ~73% reduction (~0.69 kg) in visceral fat mass (false discovery rate, FDR < 2.0 x 10(

131 icrobiota and diet have been shown to impact visceral fat mass (VFM), a major risk factor for cardiom

132 Patients with higher visceral fat mass are at a higher risk of developing sev

133 nsitivity improved (P < 0.001), and body and visceral fat mass decreased in all groups (P < 0.001).

134 A was: fat mass R2 = 0.88 male, 0.93 female; visceral fat mass R2 = 0.67 male, 0.75 female.

135 , most of the observed beneficial changes in visceral fat mass, and metabolomic and transcriptomic pr

136 aving three or more risk factors out of high visceral fat mass, high blood pressure, low high-density

137 expression itself was down-regulated in the visceral fat of two obese mouse models and obese patient

138 Acute lipolysis of visceral fat or circulating triglycerides may worsen acu

139 ester lipase (CEL), which may leak into the visceral fat or systemic circulation during pancreatitis

140 subcutaneous adipose tissue as the amount of visceral fat was independent of the level of chimerism.

141 whole-body insulin sensitivity, hepatic and visceral fat, and SCD-1 levels.

142 ipid components, fasting plasma glucose, and visceral fat, and there might be possible misclassificat

143 The obesity-asthma link is driven mainly by visceral fat, independent of total fat mass; therefore,

144 body composition, amount of subcutaneous and visceral fat, liver and heart ectopic fat, adipose tissu

145 ubcutaneous adipose tissue (SAT), but not in visceral fat.

146 ion in the liver and chronic inflammation in visceral fat.

147 loss by specifically promoting lipolysis in visceral fat.

148 lection and into 2019, we demonstrate that a visceral feeling of oneness (that is, psychological fusi

149 abnormal interoception (i.e., the sensing of visceral feelings), as observed in patients with cardiod

150 Not only is visceral founder cell specification not dependent on Poi

151 lop a multivariate classifier to distinguish visceral from somatic pain.

152 Underlying mechanisms include podocyte (visceral glomerular epithelial cell/GEC) injury.

153 9H;C* mice developed significant visceral glucosylceramide (GC) and glucosylsphingosine (

154 s right hemidiaphragm rupture with abdominal visceral herniation into the thoracic cavity several day

155 d in a rat model of mustard oil (MO)-induced visceral hyperalgesia whether the number and size of acu

156 Electroacupuncture (EA) relieves visceral hypersensitivity (VH) with underlying inflammat

157 Visceral hypersensitivity and pain result, at least in p

158 PGE2 by colonic mast cells appears to induce visceral hypersensitivity in patients with IBS-D.

159 ting peristaltic motion, fluid secretion and visceral hypersensitivity in the GI tract, and has been

160 Visceral hypersensitivity is common in patients with irr

161 her ingestion of FODMAPs can induce IBS-like visceral hypersensitivity mediated by fermentation produ

162 sies of patients with IBS-D failed to induce visceral hypersensitivity or increase the level of PGE2

163 on of mast cells in W/W(V) mice restored the visceral hypersensitivity response.

164 OX2, produced by mast cells, in mediation of visceral hypersensitivity using mice with the Y385F subs

165 in the gut, altered mucosal immune function, visceral hypersensitivity, and abnormal gastrointestinal

166 the brain, leading to motility disturbances, visceral hypersensitivity, and alterations in gastrointe

167 the brain, leading to motility disturbances, visceral hypersensitivity, and altered CNS processing.

168 d EP2) in the dorsal root ganglia to promote visceral hypersensitivity.

169 ts generated analgesic effects on MO-induced visceral hypersensitivity.

170 with older age, grade 1/grade 2 disease, no visceral involvement, higher comorbidity scores, and bei

171 Only 2 patients had visceral involvement.

172 ctate calcifications within it, and no other visceral involvement.

173 In 21 cutaneous and/or visceral Kaposi's sarcoma cases, occurring in patients l

174 ur patients with severe KSHV disease (2 with visceral KS, 1 with multicentric Castleman disease, and

175 unity and pathogen clearance in experimental visceral leishmaniasis (Leishmania donovani) but more se

176 ds to analyze longitudinal incidence data on visceral leishmaniasis (VL) and its sequela, post-kala-a

177 Visceral leishmaniasis (VL) is a parasitic infection tha

178 Control of visceral leishmaniasis (VL) on the Indian subcontinent r

179 No licensed vaccine exists against visceral leishmaniasis (VL), a disease caused by the Lei

180 In Tbilisi, Georgia, an endemic area for visceral leishmaniasis (VL), sand flies are abundant for

181 FN-gamma) are critical for the resolution of visceral leishmaniasis (VL).

182 Leishmania donovani is the dermal sequel of Visceral Leishmaniasis and importantly, is the proposed

183 d key roles in promoting inflammation during visceral leishmaniasis and malaria-two important parasit

184 nvenient, safe, and effective treatments for visceral leishmaniasis in Eastern African children are l

185 proteins of Lutzomyia longipalpis, vector of visceral leishmaniasis in the Americas.

186 m parasites, responsible for transmission of visceral leishmaniasis in the New World.

187 lebotomus argentipes, an important vector of visceral leishmaniasis in the Old World.

188 Visceral leishmaniasis is an infectious parasitic diseas

189 Visceral leishmaniasis is responsible for up to 30,000 d

190 The drugs currently used to treat visceral leishmaniasis suffer from toxicity and the emer

191 enylphosphonium bromide] in a mouse model of visceral leishmaniasis were established.

192 these events, their role remains elusive in visceral leishmaniasis, a disease associated with macrop

193 nducted a Phase II trial in 30 children with visceral leishmaniasis, aged 4-12 years, to test whether

194 phatic filariasis, trachoma, onchocerciasis, visceral leishmaniasis, and gambiense sleeping sickness)

195 Visceral leishmaniasis, caused by Leishmania infantum, i

196 ial vaccine candidates against cutaneous and visceral leishmaniasis, respectively.

197 al leishmaniasis (PKDL), clinical sequela of visceral leishmaniasis.

198 pproved oral drug for treating cutaneous and visceral leishmaniasis.

199 of this strategy to reduce the incidence of visceral leishmaniasis.

200 sized and tested against the murine model of visceral leishmaniasis.

201 iltefosine for Eastern African children with visceral leishmaniasis.

202 ni and L. infantum - cause the fatal disease visceral leishmaniasis.

203 nity are still ambiguous during experimental visceral leishmaniasis.

204 of new drug targets and their inhibitors for visceral leishmaniasis.

205 r systematic subunit vaccine testing against visceral leishmaniasis.

206 Nausea, the unpleasant sensation of visceral malaise, remains a mysterious process.

207 In Drosophila embryos, caudal visceral mesoderm (CVM) cells undergo bilateral migratio

208 undergo bilateral migration along the trunk visceral mesoderm (TVM) in order to form midgut muscles.

209 the predominant role of Ras signaling in the visceral mesoderm and that, accordingly, Ras signaling i

210 was stratified according to the presence of visceral metastases (yes vs no).

211 tch3 and MMP-3 compared with patient matched visceral metastases or osteolytic metastasis samples.

212 Bone and visceral metastases were detected in 21% (54) and 3.8% (

213 Bone and visceral metastases were detected in 40.2% of patients (

214 ave symptomatic bone metastases and no known visceral metastases.

215 tology, and bone-only disease; patients with visceral metastases; and patients aged up to 40 years.

216 used minimization factors: bone metastases; visceral metastases; investigational site; and prior abi

217 ions on bone scan, or presence of measurable visceral metastasis except lymph node metastasis).

218 gy Group performance status at baseline, and visceral metastasis.

219 s with more disseminated bone involvement or visceral metastasis.

220 that may influence visceromotor systems and visceral "mind-body" symptoms.

221 breaching the extracellular matrix (ECM) and visceral muscle.

222 pread along the actin-based muscle fibers of visceral muscles and accumulated on the surfaces of sali

223 fter acquisition start (daas), then moved to visceral muscles surrounding the midgut and to the hemoc

224 We demonstrate here that for the circular visceral muscles, despite superficial similarities, a si

225 a key determinant for the development of the visceral nervous system and branchiomotor nuclei in the

226 d to assess the impact of GABA signalling on visceral nociception, where test compounds were applied

227 citability of colonic afferents and suppress visceral nociception.

228 antinociception to both thermal and chemical visceral noxious stimuli.

229 ional risk (57%), were overweight (53%), had visceral obesity (62%), had a normal SMI (51%), had a lo

230 MUST and subcutaneous adiposity (P < 0.001), visceral obesity (P < 0.001), and low skeletal muscle in

231 Visceral obesity increases risks for all-cause mortality

232 Visceral obesity is associated with insulin resistance a

233 air on Cardiometabolic Risk Working Group on Visceral Obesity summarises the evidence for visceral ad

234 and help prevent metabolic complications of visceral obesity.

235 nce behaviors can be elicited or enhanced by visceral or cognitive threats that increase glucagon-lik

236 metastatic breast cancer (MBC) regardless of visceral or nonvisceral disease.

237 ients with anti-Scl70 Abs, at higher risk of visceral organ fibrosis, induced EndoMT and jeopardized

238 increasing primary tumor size, potentiating visceral organ metastasis, suppressing AR, and inducing

239 neous fistula, bleeding and perforation of a visceral organ) or death during 6 months of follow-up.

240 for the central integration of signals from visceral organs and contains preproglucagon (PPG) neuron

241 g in the accumulation of storage material in visceral organs and in some cases the brain of affected

242 overlapping functions.SIGNIFICANCE STATEMENT Visceral organs are innervated by sensory neurons whose

243 (PRC1) drives colonization of the bones and visceral organs in double-negative prostate cancer (DNPC

244 Neural control of the function of visceral organs is essential for homeostasis and health.

245 sa, local and distant lymph nodes, bones, or visceral organs was recorded.

246 Lesions in bone, nodes, prostate/bed, and visceral organs, as well as representative normal tissue

247 ans, which include laterality defects of the visceral organs, renal cysts as part of nephronophthisis

248 Visceral organs, such as the lungs, stomach and liver, a

249 Compared to other visceral organs, uterine spinal afferent endings display

250 cause heterotaxy, the incorrect placement of visceral organs.

251 te to the lymph nodes, peripheral blood, and visceral organs.

252 morrhagic fever that causes severe damage to visceral organs.

253 adiposity indices (abdominal [p = 0.02] and visceral [p = 0.03] fat volumes) may be higher in childr

254 Gastrointestinal (GI) pain - a form of visceral pain - is common in some disorders, such as irr

255 ypes, correlates with perceived intensity of visceral pain and discomfort, and shows specificity to p

256 al conditions may provide new treatments for visceral pain conditions.

257 , for >20% of the global population, chronic visceral pain is an unpleasant and often excruciating re

258 Accordingly, chronic visceral pain is debilitating, reduces the quality of li

259 d fear and anxiety (the affective aspects of visceral pain) are the domain of nodose afferents.

260 ExPANs mediate visceral pain, and myenteric neurons mediate colon motil

261 eased number and size along with severity of visceral pain, which may be associated with enhanced neu

262 r non-pain negative affect do not respond to visceral pain.

263 otor behavioural defects and ameliorated the visceral pathology.

264 r pain types reflect mixtures of somatic and visceral patterns.

265 how that adhesions derive primarily from the visceral peritoneum, consistent with our clinical experi

266 eliably distinguishes somatic (thermal) from visceral (rectal) stimulation in both cross-validation a

267 major fat depots located in subcutaneous and visceral regions may shed new light on adipose tissue-sp

268 w reduced interoceptive precision influences visceral regulation and interoceptively-guided decision-

269 . due to the variability of sensorimotor and visceral responses to physical exertion.

270 Major aortic branches of the arch and visceral segment add additional technical complexity to

271 apping innervation is proposed to facilitate visceral sensation and homeostasis, where sensation and

272 also the brain's primary sensory nucleus for visceral sensations relevant to symptoms in medical and

273 gical factors occurred, primarily in CNS and visceral sensitivity measures, providing new insights in

274 ns occurred between a combination of CNS and visceral sensitivity parameters, and GSRS-IBS total scor

275 of the sacral spinal cord horn that receives visceral sensory afferents from the bladder and distal c

276 G neurons, thereby diminishing the impact of visceral sensory information on GLP-1 receptor-expressin

277 Destroying visceral sensory nerves impacts pancreatic islet functio

278 hese findings suggest that InsCtx integrates visceral-sensory signals of current physiological state

279 chanisms subserving the integration of neuro-visceral signals.

280 The responses of these networks to visceral stimulation rather than their organisation at r

281 dentify immanent noise patterns during major visceral surgeries.

282 findings warrant active noise management for visceral surgery to reduce potential negative impacts of

283 hs for future research on noise reduction in visceral surgery.

284 els of 55 dBA are frequently exceeded during visceral surgical procedures.

285 tudy is to analyze noise patterns during 599 visceral surgical procedures.

286 ession was high in adipose tissue, higher in visceral than in subcutaneous tissue, increased in obesi

287 O2 in KSHV-infected KS tissue samples and KS visceral tissue microarrays.

288 In mice, Fam13a knockout (KO) have a lower visceral to subcutaneous fat (VAT/SAT) ratio after high-

289 on (L3-level attenuation), and fat (L1-level visceral-to-subcutaneous [V/S] ratio) measures were deri

290 utaneous fat but lower liver fat and a lower visceral-to-subcutaneous adipose tissue ratio.

291 human adipocytes from subcutaneous (SAT) and visceral (VAT) adipose tissues changes with age.

292 s of flavonoid subclasses and MRI-determined visceral (VAT) and subcutaneous (SAT) adipose tissue.

293 A disproportional accumulation of fat at visceral (VAT) compared to subcutaneous sites (SAT) has

294 pain types, and provide a new classifier for visceral versus somatic pain.

295 The visceral versus the somatic classifier reliably distingu

296 Preferential accumulation of visceral WAT is associated with increased risk for insul

297 ted whole-genome DNA methylation analysis of visceral WAT.

298 Visceral white adipose tissue (vWAT) expands and undergo

299 the heterogeneity of immune cells within the visceral white AT and their contributions to homeostasis

300 The expansion of visceral white AT promotes dysregulation of its immune c