1 l exploration without evidence of perforated viscus.

2 ne early death due to a perforated abdominal viscus.

3 asia or early malignant tumors of the hollow viscus.

4 0%) responded to stimulation of at least one viscus.

5 is (77.4%), followed by perforated abdominal viscus (11.5%), ileostomy closure (6.5%), trauma (2.7%),

6 r designed to simulate the lumen of a hollow viscus and were exposed to a rapid change in CO2 from 0%

7 cal and pathological roles of Pirt in hollow viscus are largely unknown.

8 oodstream infection and perforated abdominal viscus at Queen Elizabeth Central Hospital from 2008-201

9 mporary replacement of full-thickness hollow viscus defects, even in the face of heavy bacterial cont

10 w the current literature on pediatric hollow viscus injuries and emergency department disposition aft

11 graphy (CT) imaging and high rates of hollow viscus injury (HVI).

12 had no CT scan findings suspicious of hollow viscus injury were selected for clinical observation and

13 nce of CT scan findings suggestive of hollow viscus injury, the patients were observed with serial cl

14 tis as a result of a perforation of a hollow viscus or anastomotic insufficiency who had undergone OA

15 neurons responded to stimulation of a single viscus, the other five responded to two viscera.

16 to perforated appendicitis, other perforated viscus, traumatic injuries more than 4 hours old, or int