ライフサイエンスコーパス: visual stimulation

1 ht and left primary visual areas (full-field visual stimulation).

2 ravelling waves) similar to that elicited by visual stimulation.

3 ual cortex in the absence of any feedforward visual stimulation.

4 showed stronger, more reliable responses to visual stimulation.

5 as is the basis of their suppression during visual stimulation.

6 ived of V1 inputs showed robust responses to visual stimulation.

7 lamic excitation onto layer 4 neurons during visual stimulation.

8 resynaptic activation reduce AP responses to visual stimulation.

9 s of neocortex, are activated differently by visual stimulation.

10 iminative of data epochs before versus after visual stimulation.

11 ency measured during rest, in the absence of visual stimulation.

12 and glx changes in visual cortex induced by visual stimulation.

13 as potently as pyramidal cell spiking during visual stimulation.

14 te visual cortex in response to a structured visual stimulation.

15 supra-granular V1 neurons from responding to visual stimulation.

16 ynamic circuit mechanisms that are guided by visual stimulation.

17 spatially extensive, and was independent of visual stimulation.

18 greatly reduced by widespread and intensive visual stimulation.

19 tion or depression of targeted synapses with visual stimulation.

20 caque V4 cortical networks in the absence of visual stimulation.

21 ter a modulation block of prolonged (10 min) visual stimulation.

22 in macaque primary visual cortex (V1) during visual stimulation.

23 all-or-none' calcium transients modulated by visual stimulation.

24 ve the properties of a 'clock' signal during visual stimulation.

25 ntations that are activated by corresponding visual stimulation.

26 nditions, including spontaneous activity and visual stimulation.

27 in conflict with the reality of the physical visual stimulation.

28 e changing background synaptic activity with visual stimulation.

29 modulated by FEF stimulation, independent of visual stimulation.

30 spatially and temporally coincident auditory-visual stimulation.

31 persists for several minutes without further visual stimulation.

32 s and LGN neurons in male/female cats during visual stimulation.

33 via intense synaptic drive caused by natural visual stimulation.

34 EPSPs during spontaneous activity and during visual stimulation.

35 must still respond appropriately to relevant visual stimulation.

36 n neurons from animals after 4 h of constant visual stimulation.

37 thalamic input as a consequence of abnormal visual stimulation.

38 ncrease in gamma oscillations in response to visual stimulation.

39 normal, even exceeding the levels seen after visual stimulation.

40 at depends on search target identity but not visual stimulation.

41 tors beneath blood vessels are denied normal visual stimulation.

42 lels compensation of delays for time-varying visual stimulation.

43 Activation was compared with passive visual stimulation.

44 he thalamic nuclei was largely unaffected by visual stimulation.

45 ons in infected cortex responded normally to visual stimulation.

46 creased significantly (P <.05) during erotic visual stimulation.

47 ncreased baseline activity in the absence of visual stimulation.

48 activity in the retina-choroid complex after visual stimulation.

49 r 20 Hz (gamma band), that were activated by visual stimulation.

50 se of the regarded cell could be elicited by visual stimulation.

51 ned in working memory, in the absence of any visual stimulation.

52 any, saccade-related activity independent of visual stimulation.

53 r as a phenomenon that occurs independent of visual stimulation.

54 ained neurons that were highly responsive to visual stimulation.

55 elds was observed following brief periods of visual stimulation.

56 neuroscientists require accurate control of visual stimulation.

57 rrently memorized content, despite identical visual stimulation.

58 these pathways and how they interact during visual stimulation.

59 d-oxygen-level-dependent (BOLD) signal after visual stimulation.

60 esponses in the primary visual cortex during visual stimulation.

61 ntial dynamics regardless of the presence of visual stimulation.

62 and more consistent psychomotor responses to visual stimulation.

63 valence of plasticity after 4 hr of dark and visual stimulation.

64 intensity, with or without variable-contrast visual stimulation.

65 iChloC suppressed spiking activity evoked by visual stimulation.

66 homeostatic plasticity induced by patterned visual stimulation.

67 sleep and wakefulness, and after controlled visual stimulation.

68 of awake mice in the presence and absence of visual stimulation.

69 of visual alpha activity is possible through visual stimulation.

70 he normal rat retina associated with various visual stimulations.

71 nly a subset of neurons spike in response to visual stimulation, a far larger proportion of the circu

72 In response to visual stimulation, a subset of neurons in the striate a

73 Physostigmine also decreased activations to visual stimulation across all tasks within primary visua

74 isphere, and the lower-field and upper-field visual stimulations activate the superior and inferior p

75 Moreover, across conditions with identical visual stimulation, activation shifted the decision crit

76 Visual stimulation after repetitive TMS revealed long-te

77 However, how V4 neurons respond to visual stimulation after V1 injury remains unclear: Whil

78 ying a broadband input current mimicking the visual stimulation allowed us to estimate TRF between th

79 Interestingly, visual stimulation also resulted in an auditory WM-depen

80 Our data suggest that visual stimulation alters the interactions between rod-

81 st to the hemispheric symmetry observed with visual stimulation, an asymmetry emerged during VSTM wit

82 Targeted visual stimulation and computational inference demonstra

83 l areas is modulated by a combination of the visual stimulation and contextual factors, such as salie

84 t the interaction between exogenous rhythmic visual stimulation and endogenous brain rhythms can have

85 bilateral ongoing activity continues during visual stimulation and has a powerful additive impact on

86 s local sensitivity following strong, local, visual stimulation and has been shown to create a predic

87 -positive neurons in mouse V1 independent of visual stimulation and largely through nicotinic inputs

88 Visual stimulation and locomotion increased neuronal act

89 etal cortex contains neurons that respond to visual stimulation and motor behaviour.

90 ibitory neurons in awake mice during passive visual stimulation and performance of visual and auditor

91 e primary visual cortex of awake mice during visual stimulation and spontaneous activity.

92 tected in the anterior cingulate cortex upon visual stimulation and spread through the ventral retros

93 sts this activity is in response to both the visual stimulation and the abrupt appearance, or salienc

94 the relationship between the strength of the visual stimulation and the firing rate, we found that at

95 ented by blocking polyamine synthesis during visual stimulation and was rescued when Ca2+-permeable A

96 left primary visual cortex (right hemifield visual stimulation) and in both right and left primary v

97 result from decision formation as opposed to visual stimulation, and are specific to the oculomotor s

98 It is not related to foveal or peripheral visual stimulation, and it represents the position of th

99 Moreover, eEF2 phosphorylation is induced by visual stimulation, and NMDAR blockade before stimulatio

100 dently of evoked activity, persisted without visual stimulation, and predicted behavioral success in

101 assifies greater than 1700 neurons following visual stimulation; and stimulates individual neurons us

102 rally-presented crosshair while intermittent visual stimulation appeared in their top-right visual-fi

103 at during wakefulness, cortical responses to visual stimulation are dominated by synaptic inhibition,

104 and visual cortex blood flows in response to visual stimulation are poorly understood.

105 To determine whether OFF signals in visual stimulation are required for OFF RGC dendritic de

106 that the functional coupling observed during visual stimulation arises from coordinated or nearly syn

107 vivo two-photon imaging during hyperspectral visual stimulation as well as photolabeling of RGCs to p

108 deafened adults revealed robust responses to visual stimulation as well as receptive fields that coll

109 was largely complementary to that driven by visual stimulation, as well as the activity of other neu

110 whole-cell recordings, we show that pairing visual stimulation at a given retinal location with spik

111 The FF declined in response to visual stimulation at all tested locations, even in the

112 Eublepharis macularius) embryos to patterned visual stimulation beginning at either 1 week or 2 weeks

113 lain the changes of neural responsiveness to visual stimulation between states.

114 , both in the absence and in the presence of visual stimulation, biasing signals due to selective att

115 During periods with no visual stimulation, but while the subject was experienci

116 ecreased responses to low frequency periodic visual stimulation, but, while causing some increases in

117 During visual stimulation, BV responses to flickering light of

118 tate injection augmented the CBF response to visual stimulation by 38-53% in regions of the visual co

119 es to bolus lactate injection at rest and in visual stimulation by using positron-emission tomography

120 However, a brief period of visual stimulation can drive these neurons to start gene

121 lectrical stimulation of the NB, paired with visual stimulation, can induce significant potentiation

122 arvalbumin (PV)-positive interneurons during visual stimulation, challenging the disinhibition model.

123 Visual stimulation consisted of moving bars and full-fie

124 Visual stimulation, consisting of rotating hemicircles a

125 During visual stimulation, correlations increased when both cel

126 rapid loss of responsiveness to deprived-eye visual stimulation could be due to a decrease in intraco

127 Conversely, high-contrast visual stimulation could suppress the response to low-in

128 Embryos exposed to substantially augmented visual stimulation demonstrated a postnatal preference f

129 ically suppressing simple spikes only during visual stimulation demonstrated that simple spikes are r

130 et Ca(2+) elevations following physiological visual stimulation despite robust dilations of adjacent

131 In contrast, visual stimulation did not evoke gamma-band activity in

132 mbryos exposed to lesser amounts of prenatal visual stimulation did not show a preference for either

133 eloping brain circuits, specific patterns of visual stimulation drive functional plasticity of indivi

134 etal sulcus (IPS) and are revealed by direct visual stimulation during functional magnetic resonance

135 In this study, we show that visual stimulation during locomotion, which increases th

136 urons in mouse V1 increase their response to visual stimulation during locomotion.

137 It has recently been found that visual stimulation during visual WM maintenance reveals

138 The synaptic activity that is evoked by visual stimulation during wakefulness is unknown.

139 ttern formation that occurs for unstructured visual stimulation (e.g., empty-field flicker).

140 toward a spatial location in the absence of visual stimulation enhances future visual processing at

141 ic input within V1 at fixed delays following visual stimulation entrains neural responses that mimic

142 At eye opening, visual stimulation evokes robust patterns of modular cor

143 Over the initial 40 ms of visual stimulation, excitation from recurrent circuits i

144 hat at eye opening, the cortical response to visual stimulation exhibits several immaturities, includ

145 the modulated response amplitude to optimal visual stimulation (F1 values), significantly shortened

146 The influence of visual stimulation far from the receptive field center i

147 itored synaptic currents that were evoked by visual stimulation (flashing dark spots).

148 p10 mice were subjected to audio (70 db) and visual stimulation (flashing lights) for six hours per d

149 Rhythmic visual stimulation ("flicker") is primarily used to "tag

150 Visual stimulation for 4 h enhanced the stability of the

151 In this study, a fast periodic visual stimulation (FPVS) paradigm coupled with EEG was

152 fulness, it predicted them equally well, and visual stimulation further enhanced predictions of inhib

153 showed that inducing gamma oscillations with visual stimulation (gamma entrainment using sensory stim

154 nd oscillations (8-14 Hz) immediately before visual stimulation has been shown to predict perceptual

155 e substantially augmented amount of prenatal visual stimulation hatched significantly earlier than th

156 Neurons responsive to visual stimulation have now been described in the audito

157 ed LTP EEG paradigm that uses high-frequency visual stimulation (HFvS) to induce neural potentiation

158 id immune signaling response following 40 Hz visual stimulation, highlighting both the unique nature

159 gh overall movement enhanced V1 responses to visual stimulation, HOMs suppressed responses.

160 Visual stimulation, however, led to responses more consi

161 In the absence of visual stimulation, however, when single simple cells we

162 both amplitude and timing of the response to visual stimulation in advanced CAA.

163 f the retina-choroid complex associated with visual stimulation in anesthetized cats (n = 6).

164 ivity related to attention in the absence of visual stimulation in extrastriate cortex when subjects

165 o measure the spatial spread of responses to visual stimulation in human early visual cortex.

166 yramidal cells and PV(+) interneurons during visual stimulation in mouse primary visual cortex.

167 were made with healthy controls deprived of visual stimulation in one quadrant ["artificial scotoma"

168 found that combining touch on one hand with visual stimulation in the anatomically corresponding hem

169 bolites were measured at baseline and during visual stimulation in the occipital lobe using (31)P mag

170 Here we report a biphasic BOLD response to visual stimulation in the primary visual cortex of cats.

171 f the retinal and choroid vascular layers to visual stimulation in the retina.

172 rom the DRN may modulate c-Fos expression to visual stimulation in these subnuclei of the lateral gen

173 Pases in the structural plasticity driven by visual stimulation in vivo.

174 during treadmill running (in M1 neurons) and visual stimulation (in V1 neurons).

175 mic regulation in the retina when exposed to visual stimulation, in our case flicker.

176 h fluctuations are most prominent, prolonged visual stimulation increased the probability of the up s

177 e brain response that is phase locked to the visual stimulation increased with attention (as do stead

178 Although visual stimulation increases dendritic arbor growth rate

179 ing Xenopus tadpoles to 4-5 hr of persistent visual stimulation increases the intrinsic excitability

180 re specifically responsible for the enhanced visual stimulation-induced changes in neuronal responses

181 neural firing were only slightly modified by visual stimulation, irrespective of the sensory input.

182 h and the branch patterning, suggesting that visual stimulation is required for the acquisition of sp

183 However, it is not clear whether visual stimulation is required for the establishment of

184 ested that the shunting inhibition evoked by visual stimulation is responsible for the nonlinear comp

185 minate visually evoked responses, repetitive visual stimulation leads to long-term depression of GABA

186 nd three of them responded also to different visual stimulation (light-off, movement).

187 esponses after dark and increased them after visual stimulation, matching plasticity in excitatory ne

188 in the EOMs from P10 to P15 and suggest that visual stimulation may play a role in the signals that r

189 activity in visual cortex in the absence of visual stimulation may reflect a top-down bias of neural

190 sion in the occipital cortex with full-field visual stimulation (mean +/- standard error of the mean

191 l evoked potential (VEP) induced by repeated visual stimulation might reflect synaptic plasticity.

192 ed for normal development of V1 responses to visual stimulation, multiple forms of experience-depende

193 e, we demonstrate that adaptation with short visual stimulation of a direction-selective ganglion cel

194 Surprisingly, visual stimulation of different hotspots in the same cel

195 is believed to be triggered exclusively from visual stimulation of individual RF subregions.

196 activity with extracellular electrodes under visual stimulation of the center and surround.

197 ior optic tubercle (AOTu) of honey bees upon visual stimulation of the compound eye to analyze chroma

198 itory neurotransmitter GABA during monocular visual stimulation of the dominant and the non-dominant

199 h-aura patients and 6 normal controls during visual stimulation of the occipital cortex.

200 In this paradigm, visual stimulation of the receptive field and its near e

201 uit and compared these effects with those of visual stimulation of the same retinal ganglion cells.

202 iolet cones, and when transmitter release or visual stimulation of ultraviolet cones is perturbed.

203 The effect of visual stimulation on the climbing fiber activity was st

204 Our results show that the hemifield visual stimulation only activates LGN in the contralater

205 By contrast, visual stimulation only weakly modified co-activation pa

206 e activated in response to both auditory and visual stimulation, only the neural patterns recorded in

207 Four hours of visual stimulation or addition of intracellular spermine

208 Using either visual stimulation or current injection, we show that br

209 ference in the brain's response to a primary visual stimulation or in the physiology underlying BOLD

210 Visual stimulation or locomotion alone did not enhance r

211 exposed to the moderately augmented prenatal visual stimulation or not exposed to any prenatal visual

212 etinal ganglion cells (RGCs) is increased by visual stimulation or using chemogenetics, their axons r

213 athway in the context of arbitrarily complex visual stimulation, our understanding of visual system f

214 Visual stimulation outside the classical receptive field

215 enables simultaneous two-photon imaging and visual stimulation over a large range of wavelengths wit

216 l stimulation or not exposed to any prenatal visual stimulation (p < .01).

217 ncy (4, 8, and 16 Hz) reversing-checkerboard visual stimulation paradigm.

218 using brain functional magnetic imaging and visual stimulation paradigms.

219 With right hemifield visual stimulation, perfusion was significantly increase

220 Moreover, although visual stimulation plays a modulatory role, it is neithe

221 appear in over 95% of the brain for a simple visual stimulation plus attention control task.

222 Fish learned to swim in response to visual stimulation preceding tactile stimulation of the

223 strated in humans by showing that repetitive visual stimulation produces lasting enhancement of visua

224 hallucinogenic drugs, full-field flickering visual stimulation produces regular, geometric hallucina

225 The same visual stimulation protocol also induces a polyamine syn

226 is finding was not replicated in the case of visual stimulation, providing evidence for time-locked p

227 Parameters measured were reactivity to visual stimulation (quantified as blood oxygen level-dep

228 are the first, to our knowledge, to show how visual stimulation rapidly induces critical neuroimmune

229 We test whether 4 hr of increased visual stimulation regulates glutamatergic retino-tectal

230 We interpret these results as that visual stimulation regulates the maturation of RGC synap

231 ggest that attentional modulation of dynamic visual stimulation relies on two parallel cortical mecha

232 rated by sustained neurons during maintained visual stimulation remained sufficiently robust to allow

233 or moving ungrouped local elements while the visual stimulation remained the same.

234 s impulses, while Anonymous impulses (during visual stimulation) render the LGN slightly refractory f

235 right peripheral position in the absence of visual stimulation resulted in differential modulations

236 edly less reliable over time during rest and visual stimulation, resulting in unstable encoding of ba

237 measurements during spontaneous activity and visual stimulation reveal an intrinsic voltage-gated con

238 es in V1 and LGN, whereas in the presence of visual stimulation, saccades led to suppression of visua

239 erior cingulate cortex (CGp) is modulated by visual stimulation, saccades, and eye position, suggesti

240 echniques, we demonstrate that during normal visual stimulation scene information peaks in mid-layers

241 r (MCS) or parallel fiber (ZCS) input during visual stimulation; SCS cells fired complex spikes assoc

242 y assay, we first characterize how motor and visual stimulation sequences govern the selection of dis

243 processing colour, we developed a versatile visual stimulation setup to probe combined spatial, temp

244 nearby local populations driven by different visual stimulation showed different gamma frequencies.

245 nsitive to both visible and invisible cardio-visual stimulation, showing reduced activation for visua

246 Visual stimulation significantly modulated auditory acti

247 Furthermore, visual stimulation strongly modulates the bipolar cell p

248 e synthesis inhibitors blocked the effect of visual stimulation, suggesting that visual activity regu

249 In auditory cortices, visual stimulation suppressed activations, but amplified

250 neurons in anesthetized cats and found that visual stimulation suppressed low-frequency membrane pot

251 In response to visual stimulation, T1rho imaging revealed a significant

252 dings from an identified ganglion cell type, visual stimulation targeted at individual cone photorece

253 ed oscillations in membrane potential during visual stimulation that are largely absent during period

254 ing in conjunction with carefully calibrated visual stimulation that emulated either congruent or opp

255 D2 mice showed no response to visual stimulation that evoked robust optomotor response

256 duced fixation offset establishes the foveal visual stimulation that is required to restore the balan

257 a reduction in its response to deprived eye visual stimulation, the transgenic mouse V1 had already

258 functional coupling to be very common during visual stimulation: the simple cell's spikes tended to o

259 mmunication, bursts can also be triggered by visual stimulation, thereby transforming the retinal sig

260 Under visual stimulation, these areas in both hemispheres code

261 stimulus orientation and less responsive to visual stimulation through either eye.

262 cortical neurons unresponsive to subsequent visual stimulation through the deprived eye.

263 Using a new method for restricting visual stimulation to a selected retinal region, we exam

264 ell patch recordings from cat area 17 during visual stimulation to examine the generation and integra

265 y in healthy participants, we used ambiguous visual stimulation to probe the relationship between del

266 By presenting ultrasound and visual stimulation together, we found that ultrasonic st

267 Visual stimulation triggered regenerative local dendriti

268 Visual stimulation under anaesthesia with checkerboards

269 ase lock, or entrain, alpha through rhythmic visual stimulation under the assumption that this entrai

270 vert attention in the absence of significant visual stimulation using a threshold-contrast detection

271 With visual stimulation using two short pulses in the human b

272 n-stressful neutral, pleasant and unpleasant visual stimulation (VES) via emotionally laden slides.

273 i concerning comparable aspects of light and visual stimulation via collateralized axons.

274 s in the locust Schistocerca gregaria during visual stimulation via lateral LCD screens.

275 on may bias the neural processing of dynamic visual stimulation via two complementary neural mechanis

276 he functional deficit in retinal response to visual stimulation was also demonstrated in rAAV.sFLT-1-

277 WM maintenance, only the impulse response to visual stimulation was content-specific, suggesting that

278 The activation at three out of four rates of visual stimulation was greater for the patients with sch

279 resonance imaging (fMRI) signal response to visual stimulation was measured in retinotopic mapping-d

280 hermore, this modulation depended on whether visual stimulation was present or absent.

281 Visual stimulation was provided by the display of moving

282 Fine-grained visual stimulation was used to identify the location, ty

283 ecorded for 5 intensity tones with emotional visual stimulation was used, for the first time, to test

284 EEG data, recorded during bilateral rhythmic visual stimulation, we find the typical SSR gain effect

285 Despite robust subcortical responses to visual stimulation, we found little evidence for strengt

286 fMRI responses to visual stimulation were related to differences in RNFL t

287 cortex of cats, and the effects of prolonged visual stimulation were studied.

288 ed sub-additive responses to optogenetic and visual stimulation, which depended lawfully on stimulati

289 kes but enhanced voltage responses evoked by visual stimulation, which selectively boosted transmissi

290 t in the FEF, are most effectively driven by visual stimulation, while behavioral engagement is not s

291 during either full-field or right hemifield visual stimulation with a black and white reversing chec

292 man-comparable EEG in macaque monkeys during visual stimulation with colored dynamic random dot patte

293 Following prolonged visual stimulation with drifting gratings, we observed s

294 of orientation maps were induced by pairing visual stimulation with electrical activation of the mes

295 tex during sleep and wakefulness, and during visual stimulation with fixation.

296 the LFP oscillation became more entrained by visual stimulation with higher frequencies (>10 Hz).

297 ild-type littermate control) during rest and visual stimulation with moving full-field square-wave gr

298 tors in shaping the relay neuron response to visual stimulation with the AMPA component being importa

299 entrainment of ongoing alpha oscillations by visual stimulation, with concomitant consequences for pe

300 versal of the normal beta suppression during visual stimulation, with visual stimuli eliciting beta m