ライフサイエンスコーパス: vitamin D receptor

1 netic loci, including the VDR gene (encoding vitamin D receptor).

2 cording to common genetic differences in the vitamin D receptor.

3 in D intake and genetic polymorphisms in the vitamin D receptor.

4 ced by common variations in the gene for the vitamin D receptor.

5 droxyvitamin-D3 (1,25D) was dependent on the vitamin D receptor.

6 optosis in intestinal epithelium lacking the vitamin D receptor.

7 P in tumor cells, likely through the nuclear vitamin D receptor.

8 ion of ambient vitamin D metabolites via the vitamin D receptor.

9 marily by lithocholic acid signaling via the vitamin D receptor.

10 cells revealed the presence of cell surface vitamin D receptors.

11 It seems likely that statins activate vitamin D receptors.

12 n genes encoding PD-L1 and PD-L2 through the vitamin D receptor, a ligand-regulated transcription fac

13 A recent article in Cell shows that vitamin D receptor activation reprograms reactive stroma

14 d the individual and combined effects of the vitamin D receptor activator paricalcitol (PARI) and die

15 Paricalcitol, a selective vitamin D receptor activator, decreased serum parathyroi

16 Furthermore, vitamin D receptor activators can restore Klotho express

17 storation of suppressed Klotho expression by vitamin D receptor activators conferred human aortic smo

18 y procalcific stressors could be restored by vitamin D receptor activators, in vitro and further conf

19 Administration of lithocholic acid or a vitamin D receptor agonist resulted in increased Slc30a1

20 The physiologic vitamin D receptor agonist, 1,25(OH)2D3 (calcitriol), is

21 Treatment with vitamin D receptor agonists (VDRAs) may have nephroprote

22 Vitamin D receptor agonists are known to suppress parath

23 evant synthetic lethal interactions and that vitamin D receptor agonists may show enhanced efficacy i

24 l induced a physical interaction between the vitamin D receptor and beta-catenin in podocytes, which

25 timicrobial effector pathway mediated by the vitamin D receptor and human beta defensin 2.

26 Increased binding of the vitamin D receptor and increased histone H4 acetylation

27 the miR-498 genome, which is occupied by the vitamin D receptor and its coactivators.

28 receptor DAF-12, a homolog of the mammalian vitamin D receptor and liver X receptor.

29 vation of TLR, that induce expression of the vitamin D receptor and localized synthesis of 1,25(OH)(2

30 aled new insights into the regulation of the vitamin D receptor and new targets for its action.

31 was markedly suppressed in mice lacking the vitamin D receptor and partially suppressed in vitamin D

32 ted with IL-23 plus IL-1beta upregulated the vitamin D receptor and responded to 1,25D with downregul

33 25-hydroxyvitamin D promoted binding of the vitamin D receptor and retinoid X receptor to the promot

34 Induction of the vitamin D receptor and the 1-alpha-hydroxylase genes was

35 n macrophages up-regulated expression of the vitamin D receptor and the vitamin D-1-hydroxylase genes

36 Whereas both the vitamin D receptor and vitamin D regulate iNKT cells, th

37 very that every tissue in the human body has vitamin D receptors and that vitamin D has pleiotropic e

38 Importantly, nuclear levels of CTSL, vitamin D receptor, and 53BP1 emerged as a novel triple

39 typed for TaqI and FokI polymorphisms of the vitamin D receptor, and interaction analyses were done t

40 o calcitriol in their ability to bind to the vitamin D receptor, and most of them exert significantly

41 educed concentrations of calcium-sensing and vitamin D receptors, and altered mRNA-binding protein ac

42 hese results indicate that vitamin D and the vitamin D receptor are required for the development of E

43 ion (SLC20A2, whose promoter has a predicted vitamin D receptor binding site, and XPR1), and one unas

44 o RUNX2, C/EBPbeta, retinoid X receptor, and vitamin D receptor binding sites, whereas adipocyte diff

45 ese lactation-induced increases, and reduced vitamin D receptor binding to promoter regions of Ca(2+)

46 hrough suppression of SOCS3 and induction of vitamin D receptor binding with the vitamin D-response e

47 key locus responsible for skin color, with a vitamin D receptor-binding interval.

48 oteinuric activity and podocytes express the vitamin D receptor, but whether vitamin D signaling in p

49 disrupting the interaction between SMAD3 and vitamin D receptor by altering SMAD3 ubiquitination.

50 ed by 1,25-dihydroxyvitamin D binding to the vitamin D receptor caused the decrease in hepcidin mRNA

51 1,25(OH)2D3 binds the vitamin D receptor complex present in many immune popula

52 but engaged distinct TGF-beta-dependent and vitamin D receptor-dependent (VDR-dependent) pathways, r

53 Vitamin D receptor-dependent changes in the expression o

54 IL-1beta in macrophages, and therefore-in a vitamin D receptor-dependent manner-inhibited the abilit

55 rast, activators of PPAR-alpha, RAR, RXR, or vitamin D receptor did not alter ABCA12 expression.

56 precipitation analysis demonstrated that the vitamin D receptor directly binds to the TRPC6 promoter.

57 1 alpha-hydroxylase, 24-hydroxylase, and the vitamin D receptor, each of which was regulated by 1,25(

58 Combined activation of beta-catenin and the vitamin D receptor enhanced differentiation of sebaceous

59 most, demonstrating the greatest increase in vitamin D receptor expression after cholecalciferol.

60 sis, as well as E(2)-mediated enhancement of vitamin D receptor expression in the inflamed CNS.

61 sed serum 25(OH)D levels four-fold, monocyte vitamin D receptor expression three-fold, and 24-hydroxy

62 In this study, we found that vitamin D receptor expression was induced in a CD4+ effe

63 However, antibodies to the vitamin D receptor failed to block 1,25D(3)-stimulated c

64 Vitamin D receptor FokI and BsmI genotypes and the (I/D)

65 n the MKP5 promoter that associated with the vitamin D receptor following 1,25D treatment.

66 sure, the principal source of vitamin D, and vitamin D receptor gene (VDR) polymorphisms (FokI, TaqI,

67 No single-nucleotide polymorphism at the vitamin D receptor gene met our corrected significance t

68 gging single-nucleotide polymorphisms in the vitamin D receptor gene, requiring P < .002 (0.05 divide

69 elopment and severity and that variations in vitamin D receptor genes are associated with asthma susc

70 ses were done to assess the influence of the vitamin D receptor genotype on response to vitamin D(3).

71 ed colorectal adenomas may vary according to vitamin D receptor genotype.

72 ral lines of evidence, including the role of vitamin D receptor genotypes, malnutrition's effects on

73 Vitamin D receptors have a broad tissue distribution tha

74 Vitamin D receptors have been found in all the major car

75 re associated with retinoic acid receptor or vitamin D receptor heterodimers by any of the agonists.

76 podocin promoter to target Flag-tagged human vitamin D receptor (hVDR) to podocytes in DBA/2J mice.

77 25(OH)(2)-vitamin D(3) (1,25D)/human nuclear vitamin D receptor (hVDR) transcription initiation compl

78 ar receptor superfamily, including the human vitamin D receptor (hVDR).

79 were (i) vitamin D-deficient, (ii) minus the vitamin D receptor, (iii) minus a vitamin D 25-hydroxyla

80 also describe the expression of a functional vitamin D receptor in IDEC.

81 Conversely, deletion of the vitamin D receptor in macrophages from diabetic patients

82 Conversely, deletion of the vitamin D receptor in macrophages from diabetic patients

83 on using conditional knockout of the myeloid vitamin D receptor in mice (KODMAC).

84 Conditional knockout of the vitamin D receptor in myeloid cells but not the endothel

85 ciated receptor gamma, liver X receptor, and vitamin D receptor in shaping the immune and metabolic f

86 Vitamin D receptor inactivation leads to hyperinflammato

87 Blocking the vitamin D receptor, inhibiting CYP27B1, or limiting 25D3

88 previously shown that the activation of the vitamin D receptor inhibits IgE production and that B ce

89 Using VDR affinity beads, the vitamin D receptor interacting protein (DRIP)/mediator c

90 plex from primary keratinocytes (KCs) as the vitamin D receptor-interacting protein complex.

91 se; MTHFR:C677T), and the vitamin D pathway (vitamin D receptor: Intron8G/T;).

92 In the intestine, the vitamin D receptor is activated by 1alpha, 25-dihydroxyv

93 The vitamin D receptor is highly expressed in the gastrointe

94 eptor and vitamin D regulate iNKT cells, the vitamin D receptor is required for both iNKT cell functi

95 study was designed to examine the effects of vitamin D receptor knockout (VDR(-/-)) and vitamin D def

96 xylase that converts vitamin D3 to an active vitamin D receptor ligand; P=1.4x10(-5)).

97 In addition to being a ligand for the vitamin D receptor, lithocholic acid is also a substrate

98 Egr-1, Egr-2, Vitamin D Receptor, MafB/c: Fos and PU.1:interferon regu

99 The vitamin D receptor-mediated increase in Smad3 expression

100 but not TLR4, stimulation markedly inhibited vitamin D receptor mRNA and protein expression, selectiv

101 Unlike the mineralization defect in Vitamin D receptor-null mice, the mineralization defect

102 d, genotypes in that pathway were important: vitamin D receptor (odds ratio [OR], 6.85 [95% confidenc

103 Vitamin D receptor, oestrogen receptor and mineralocorti

104 alcitriol, even though it binds to the human vitamin D receptor only about 1% as well as calcitriol.

105 Small interfering RNA silencing of the vitamin D receptor or SRC3 blocked the induction of cath

106 The vitamin D receptor participates in the control of IgE cl

107 ssing dentin matrix protein 1 (DMP1) via the vitamin D receptor pathway.

108 articipants with the tt genotype of the TaqI vitamin D receptor polymorphism.

109 Studies of vitamin D receptor polymorphisms have found that not all

110 In contrast, vitamin D receptor polymorphisms were not significantly

111 erleukin-6, tumor necrosis factor-alpha, and vitamin D receptor polymorphisms.

112 id activated proteins: farnesoid X receptor, vitamin D receptor, pregnane X receptor, and TGR5.

113 was 300 times more active in binding to the vitamin D receptor protein, 30 times more effective in c

114 The wide distribution of the vitamin D receptor provides a number of clinical targets

115 Expression of epidermal IL1f6, S100a8, vitamin D receptor, repetin, and major histocompatibilit

116 vity of VDR toward specific target genes.The vitamin D receptor/retinoid X receptor-alpha heterodimer

117 The vitamin D receptor/retinoid X receptor-alpha heterodimer

118 minor alleles for SNPs in genes encoding the vitamin D receptor (rs4334089, rs11568820) and 25-hydrox

119 These results identify reduced vitamin D receptor signaling as a potential mechanism un

120 In this article, we report that vitamin D receptor signaling attenuates TLR-mediated inf

121 crophage miR-106b-5p secretion from impaired vitamin D receptor signaling causes inflammation-induced

122 tective effect was associated with increased vitamin D receptor signaling.

123 Although both cell types have an intact vitamin D receptor-signaling axis, this study demonstrat

124 for the 25(OH)D(3)/DBP complex to stimulate vitamin D receptor targets and osteoblastogenesis.

125 , 1alpha,25-dihydroxyvitamin D, binds to the vitamin D receptor that regulates numerous genes involve

126 the development of synthetic ligands of the vitamin D receptor that target the TGF-beta-SMAD signali

127 , bone, and kidney and overexpression of the vitamin D receptor, thereby reproducing the human phenot

128 ith DAC and TSA increases the recruitment of vitamin D receptor to the CYP24A1 promoter.

129 These data may explain the recruitment of vitamin D receptor to the promoter region in MDEC but no

130 ulates B cells in vitro and mice without the vitamin D receptor (VDR knockout [KO]) have high serum I

131 or more >/=4 mm pockets were associated with vitamin D receptor (VDR) (rs2228570, P = 0.002, q = 0.04

132 P sequence analysis of binding sites for the vitamin D receptor (VDR) across the proximal intestine i

133 5D is competed by ZK159222, an antagonist of vitamin D receptor (VDR) action, and can occur in the pr

134 Vitamin D receptor (VDR) activation in HSCs inhibits liv

135 Here we demonstrate that vitamin D receptor (VDR) activation mitigates hepatic ER

136 ween observational studies that suggest that vitamin D receptor (VDR) activators provide a survival a

137 Here, we document that the vitamin D receptor (VDR) acts as a master transcriptiona

138 e of 1,25D in all cell types, while use of a vitamin D receptor (VDR) agonist (EB1089) and antagonist

139 o determined if corneas contain mRNA for the vitamin D receptor (VDR) and 1alpha-hydroxylase, the enz

140 e podocyte injury through down regulation of vitamin D receptor (VDR) and activation of renin angiote

141 ome analysis revealed an association between vitamin D receptor (VDR) and lipid metabolism in human t

142 The vitamin D receptor (VDR) and p65 formed a complex in tub

143 )D interacts with breast tumor expression of vitamin D receptor (VDR) and retinoid X receptor-alpha (

144 inhibitory interaction between the inactive vitamin D receptor (VDR) and Stat1, which was released u

145 ility to inhibit the interaction between the vitamin D receptor (VDR) and steroid receptor coactivato

146 Vitamin D receptor (VDR) and the megalin gene polymorphi

147 ived DNA precipitated with antibodies to the vitamin D receptor (VDR) and the retinoid X receptor (RX

148 cts by inhibiting cell growth and increasing vitamin D receptor (VDR) and VDR-mediated transcription.

149 Vitamin D receptor (VDR) antagonists prevent the VDR act

150 e idea that 1,25-dihydroxyvitamin D3 and the vitamin D receptor (VDR) are involved in regulating skin

151 Ligand-independent actions of the vitamin D receptor (VDR) are required for normal post-mo

152 is report, we explore the interaction of the vitamin D receptor (VDR) at regulatory sites within both

153 3-E1 cells, and assessed localization of the vitamin D receptor (VDR) at sites of action on a genome-

154 imed to investigate the relationship between vitamin D receptor (VDR) binding in lymphoblastoid cell

155 in vitro assays, vitamin D treatment led to vitamin D receptor (VDR) binding in the promoter region

156 Furthermore, we found vitamin D receptor (VDR) binding sites in the promoters

157 here was evidence of interaction between the vitamin D receptor (VDR) BsmI genotype and serum 25-hydr

158 Vitamin D receptor (VDR) deficiency (knockout [KO]) resu

159 Vitamin D receptor (VDR) deficiency in the intestine lea

160 Vitamin D and vitamin D receptor (VDR) deficiency results in severe sy

161 Global vitamin D receptor (VDR) deletion exaggerates colitis, b

162 lammatory bowel disease (IBD), whereas IL-10/vitamin D receptor (VDR) double KO mice developed fulmin

163 er, we discovered a marked downregulation of vitamin D receptor (VDR) during OIS, and a role for the

164 Recently, we found that vitamin D receptor (VDR) enhanced Claudin-2 expression i

165 dy was to identify determinants of placental vitamin D receptor (VDR) expression and placental calciu

166 Reduced vitamin D receptor (VDR) expression prompts skeletal mus

167 The effect of A. fumigatus on nuclear vitamin D receptor (VDR) expression was investigated usi

168 Suppression of endogenous vitamin D receptor (VDR) expression with siRNAs signific

169 ates breast cancer growth through regulating vitamin D receptor (VDR) expression.

170 The vitamin D receptor (VDR) FokI AA genotype and retinoid X

171 single nucleotide polymorphisms (SNP) in the vitamin D receptor (VDR) gene (Fok1, Bsm1, Cdx2) were as

172 The vitamin D receptor (VDR) gene has been involved in the m

173 Polymorphisms of the vitamin D receptor (VDR) gene have been implicated in su

174 Several polymorphisms in the vitamin D receptor (VDR) gene have been reported to be a

175 x technology we have selectively deleted the vitamin D receptor (VDR) gene in the cardiac myocyte in

176 on between 25-OHD level and variation at the vitamin D receptor (VDR) gene locus.

177 In addition, an association between vitamin D receptor (VDR) gene polymorphisms and diabetes

178 xamine sun exposure and its interaction with vitamin D receptor (VDR) gene variants on breast cancer

179 base substitution was found in exon 6 in the vitamin D receptor (VDR) gene.

180 der caused by mutations in hairless (HR) and vitamin D receptor (VDR) genes, respectively.

181 Vitamin D receptor (VDR) has been widely detected in the

182 olymorphisms (SNPs) in the gene encoding the vitamin D receptor (VDR) have been widely reported to as

183 We report that deletion of the vitamin D receptor (VDR) in hematopoietic cells did not

184 in the characterization of Vitamin D and the Vitamin D receptor (VDR) in immune function.

185 of the mediator complex as a coactivator for vitamin D receptor (VDR) in keratinocytes.

186 Emerging data suggest a role for the vitamin D receptor (VDR) in lipogenesis and adipocyte di

187 We demonstrate a role of the vitamin D receptor (VDR) in reducing cerebral soluble an

188 To address the role of the vitamin D receptor (VDR) in renal fibrogenesis, we subje

189 es, we analyzed the consequences of specific vitamin D receptor (Vdr) inactivation in the intestine a

190 The physiological ligand for the vitamin D receptor (VDR) is 1,25-dihydroxyvitamin D(3).

191 Vitamin D receptor (VDR) is a key genetic factor for sha

192 Vitamin D receptor (VDR) is a ligand (vitamin D(3))-depe

193 Vitamin D receptor (VDR) is a ligand-dependent transcrip

194 The vitamin D receptor (VDR) is a nuclear hormone receptor t

195 The vitamin D receptor (VDR) is a nuclear hormone receptor t

196 vitamins to the full-length rat recombinant vitamin D receptor (VDR) is either similar to or within

197 Here, we reveal that the vitamin D receptor (VDR) is expressed in stroma from hum

198 ster regulator of the hair follicle, and the vitamin D receptor (Vdr) is linked to coordinated contro

199 The data show that expression of the vitamin D receptor (VDR) is required for normal developm

200 The major physiological role of the vitamin D receptor (VDR) is the maintenance of mineral i

201 The vitamin D receptor (VDR) is the single known regulatory

202 Vitamin D receptor (VDR) knock-out (VDRKO) mice have def

203 Vitamin D receptor (VDR) knockdown partly abolished MART

204 The vitamin D receptor (VDR) ligand, 1,25 dihydroxyvitamin D

205 Vitamin D receptor (VDR) ligands are therapeutic agents

206 Here, we show that vitamin D receptor (VDR) ligands inhibit HSC activation

207 Vitamin D receptor (VDR) mutations in humans and mice ca

208 Little is known about the effects of the vitamin D receptor (VDR) on hepatic activity of human ch

209 nd thyroid hormone receptors (SMRT) from the vitamin D receptor (VDR) on those VDREs.

210 We show that transgenic expression of the vitamin D receptor (VDR) only in the distal intestine of

211 in D metabolite uptake and activation of the vitamin D receptor (VDR) pathway in colon cancer cells t

212 Vitamin D receptor (VDR) plays an essential role in gast

213 conducted to assess the association between vitamin D receptor (VDR) polymorphisms and genetic susce

214 onal 1,25-dihydroxyvitamin D (1,25D) and the vitamin D receptor (VDR) profoundly alter, through multi

215 1,25D3 alters vitamin D receptor (VDR) recruitment to the Cyp11a1 prom

216 Depletion of the vitamin D receptor (VDR) reduced these genoprotective ef

217 The vitamin D receptor (VDR) regulates a diverse set of gene

218 Tissue-specific modulation of vitamin D receptor (VDR) signaling had organ-restricted

219 Although the suppressive effect of vitamin D/vitamin D receptor (VDR) signaling has been shown in the

220 ihydroxyvitamin D3-bound [1,25(OH)2D3-bound] vitamin D receptor (VDR) specifically inhibits TGF-beta-

221 ting enzyme 1alpha-hydroxylase (CYP27B1) and vitamin D receptor (VDR) support anti-inflammatory respo

222 This is followed by discussion of the vitamin D receptor (VDR) that mediates the cellular acti

223 ydroxyvitamin D [25(OH)D] interacts with the vitamin D receptor (VDR) to decrease proliferation and i

224 D(3) (1,25(OH)(2)D(3)) hormone binds to the vitamin D receptor (VDR) to regulate gene expression.

225 The role of the vitamin D receptor (VDR) was also examined.

226 Vitamin D receptor (VDR) was significantly down-regulate

227 oxylase), CYP27B1 (1-alpha-hydroxylase), and vitamin D receptor (VDR) were downregulated in the liver

228 in cytochrome P-450 (CYP2R1)(rs10741657AG), vitamin D receptor (VDR)(rs2228570AG, rs1544410CT), olig

229 Treatment of mice with an active ligand of vitamin D receptor (VDR), 1,25-dihydroxyvitamin D(3) (1,

230 It is the ligand of the vitamin D receptor (VDR), a nuclear receptor with transa

231 The vitamin D receptor (VDR), an endocrine nuclear receptor

232 bly through genomic effects modulated by the vitamin D receptor (VDR), and autocrine/paracrine metabo

233 The enzyme is directly regulated by vitamin D receptor (VDR), and it is expressed mainly in

234 on of vitamin D, through its cognate nuclear vitamin D receptor (VDR), and its contribution to divers

235 erator-activated receptor gamma (PPARgamma), vitamin D receptor (VDR), and retinoic acid receptor alp

236 xpression of the calcium receptor (CaR), the vitamin D receptor (VDR), and the P450 cytochromes, CYP2

237 cytokines by 1,25(OH)(2)D(3) was mediated by vitamin D receptor (VDR), as 1,25(OH)(2)D(3) had no effe

238 l small-molecule compounds that activate the vitamin D receptor (VDR), but are devoid of hypercalcemi

239 Colonic mucosal expression concentrations of vitamin D receptor (VDR), E-cadherin, zonula occluden 1

240 ition led to the hyperphosphorylation of the vitamin D receptor (VDR), enabling an interaction betwee

241 [1alpha,25(OH)(2)D(3)] and its receptor, the vitamin D receptor (VDR), has resulted in significant co

242 sion induced by BXL0124 was blocked by siRNA vitamin D receptor (VDR), indicating that the regulation

243 Although mast cells express the vitamin D receptor (VDR), it is not clear to what extent

244 n-containing transcription factor 2 (Runx2), vitamin D receptor (Vdr), SRY (sex-determining region Y)

245 Vitamin D exerts its actions through the vitamin D receptor (VDR), the expression of which was re

246 dihydroxyvitamin D, are mediated through the vitamin D receptor (VDR), which heterodimerizes with ret

247 amin D, 1,25(OH)(2)D(3), are mediated by the vitamin D receptor (VDR), which heterodimerizes with ret

248 calcitriol are mediated at least in part by vitamin D receptor (VDR), which is expressed in many tis

249 amin D3 exerts its effects by binding to the vitamin D receptor (VDR), which regulates transcription

250 on in binding of a transcription factor, the vitamin D receptor (VDR), whose activating ligand vitami

251 vitamin D3 (1,25(OH)2D3) are mediated by the vitamin D receptor (VDR), whose expression in bone cells

252 1,20S(OH)2D3 interacts with the vitamin D receptor (VDR), with similar potency to its na

253 Vitamin D receptor (VDR)-dependent mechanisms regulate h

254 Vitamin D receptor (VDR)-knockout mice develop severe hy

255 Absence of vitamin D receptor (VDR)-mediated PPARgamma suppression

256 ever, about the role of SWI/SNF and PRMTs in vitamin D receptor (VDR)-mediated transcription.

257 Vitamin D receptor (VDR)-null mice develop polyuria, but

258 a high fat diet-resistant lean phenotype of vitamin D receptor (VDR)-null mutant mice mainly due to

259 ifferentiation was reduced, and beta-catenin/vitamin D receptor (VDR)-regulated gene expression was m

260 essed based on the gene expression levels of vitamin D receptor (VDR)-regulated genes osteocalcin and

261 Both VDREs bound the vitamin D receptor (VDR)-retinoid X receptor (RXR) compl

262 e in cancer development and acts through the vitamin D receptor (VDR).

263 transcriptional repression, mediated by the vitamin D receptor (VDR).

264 g protein (VDRE-BP) or 1,25(OH)(2)D(3)-bound vitamin D receptor (VDR).

265 t are different from genomic effects via the vitamin D receptor (VDR).

266 The effect of 20(OH)D3 was dependent on the vitamin D receptor (VDR).

267 alpha,25-Dihydroxyvitamin D3 signals via the vitamin D receptor (VDR).

268 )) is the biologically active ligand for the vitamin D receptor (VDR).

269 coactivator for nuclear receptors including vitamin D receptor (VDR).

270 ultiple tissue types by interacting with the vitamin D receptor (VDR).

271 EB) and a cluster termed C24-DS2 binding the vitamin D receptor (VDR).

272 s potent agonist of the transcription factor vitamin D receptor (VDR).

273 ous melanoma (CM) cells mediated through the vitamin D receptor (VDR).

274 dence that p63gamma specifically upregulates vitamin D Receptor (VDR).

275 l keratinocytes through interaction with the vitamin D receptor (VDR).

276 UV suppression of EAE in mice devoid of the vitamin D receptor (VDR).

277 erted via signaling mechanisms involving the vitamin D receptor (VDR).

278 egulatory role over mucosal immunity via the vitamin D receptor (VDR).

279 everal NRs in OPCs and OLGs, one of which is vitamin D receptor (VDR).

280 r-binding protein beta (C/EBPbeta), OSX, and vitamin D receptor (VDR).

281 ihydroxyvitamin D(3) (1,25(OH)(2)D(3)) via a vitamin D receptor (VDR)/retinoid X receptor (RXR) heter

282 at -312 (a DR4-type VDRE) could be bound by vitamin D receptor (VDR)/retinoid X receptor.

283 al keratinocytes, unliganded heterodimers of vitamin D receptor (VDR)/RXR-alpha and retinoic acid rec

284 lial cells signaling by the nuclear receptor Vitamin D Receptor (VDR, NR1I1) induces cell cycle arres

285 Calcitriol, acting through vitamin D receptors (VDR) in the parathyroid, suppresses

286 oma, including analysis of the expression of vitamin D receptors (VDR), the activating and inactivati

287 tic macrophages express the highest level of vitamin D receptors (VDRs) among nonparenchymal cells, w

288 Vitamin D receptors (VDRs) are found within multiple tis

289 nociceptors ("pain-sensing" nerves) express vitamin D receptors (VDRs), suggesting responsiveness to

290 d this might be due to genetic variations in vitamin D receptors (VDRs).

291 pression of vitamin D regulatory enzymes and vitamin D receptor was higher in Ksp-KL(-/-) mice than c

292 STAT3, Raf1, and PKCzeta, were increased and Vitamin D receptor was reduced in cancer cells.

293 STAT3, Raf1, and PKCzeta, were increased and vitamin D receptor was reduced in cancer cells.

294 When the vitamin D receptor was silenced with small interfering R

295 We also found that the vitamin D receptor was transiently expressed during MAC

296 ge in positioning of the 20R molecule in the vitamin D receptor when the 2-methylene group is present

297 tivity of VD3 is primarily attributed to the vitamin D receptor, whereas 5 affects Hh inhibition thro

298 transcription of PTH by associating with the vitamin D receptor, which heterodimerizes with retinoic

299 or hormonal receptors, namely PXR, AHR, and vitamin D receptor, which regulate major xenobiotic-meta

300 rmone transitorily down-regulates the kidney vitamin D receptor, which returns to normal levels with