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1 IS or TORC1 blocked both JH biosynthesis and vitellogenesis.
2 proteins for the provision of amino-acids in vitellogenesis.
3 he exponential tissue growth at the onset of vitellogenesis.
4 ibuted to protein catabolism associated with vitellogenesis.
5 and stored accumulation over the duration of vitellogenesis.
6 utophagy in the Aedes fat body at the end of vitellogenesis.
7 s of which are highly elevated at the end of vitellogenesis.
8 a melanogaster oocyte occurs actively during vitellogenesis.
9 oncentrated into subcortical clusters during vitellogenesis.
10 ILPs in controlling germline cyst growth and vitellogenesis.
11 a central event in egg maturation, is called vitellogenesis.
12 response to a protein-rich diet and to enter vitellogenesis.
13 d (2) stage 8 egg chambers that are entering vitellogenesis.
14 s, serves a similar function during mosquito vitellogenesis.
15 essed in the female mosquito fat body during vitellogenesis.
16 etabolism that accompany estrogen-stimulated vitellogenesis.
17 ed follicle growth with no obvious effect on vitellogenesis.
18 notably suppressed in animals with impaired vitellogenesis.
19 tissue in the ovotestis and enhanced ovarian vitellogenesis.
20 psin D mRNA were seen at around the onset of vitellogenesis, a time when the oocytes are starting to
21 ties in holometabolous insects, ranging from vitellogenesis and caste determination in adults to the
23 of egg chambers at stages 7-8, disruption of vitellogenesis and posterior follicle cell (PFC) hyperpl
26 to species require a blood meal to stimulate vitellogenesis and subsequent oviposition (anautogeny),
27 rthermore, certain amino acids essential for vitellogenesis are preferentially directed to the midgut
29 fection inhibits reproduction by suppressing vitellogenesis, causing testicular aberrations, and/or d
30 predominant during the termination phase of vitellogenesis, coinciding with the onset of lipogenesis
34 3 days before becoming competent to activate vitellogenesis in response to a blood-meal-initiated, el
36 acids (to restore TORC1 activity), restored vitellogenesis in the neck-ligated (IIS-, TORC1- and JH-
38 ock of JH biosynthesis or JH action arrested vitellogenesis, in part by inhibiting the expression of
43 have demonstrated that IIS and TORC1 promote vitellogenesis, mainly via inducing JH biosynthesis in t
44 nd in the fat body during the termination of vitellogenesis may serve as a transcriptional inhibitor.
45 ted to elucidate the molecular mechanisms of vitellogenesis regulation in the American cockroach, Per
46 Understanding the molecular mechanisms of vitellogenesis regulation will contribute significantly
48 ing neurons that gate both the initiation of vitellogenesis that occurs post-eclosion and its re-init
51 f the oocyte arrests temporarily just before vitellogenesis, the period when vitellogenins (yolk prot
53 ich stimulates immune induction and promotes vitellogenesis through the function of 20-hydroxyecdyson
54 eckpoint occurs at the onset of yolk uptake (vitellogenesis): under nutrient stress, egg chambers deg
55 sustained activity of SAG neurons shuts off vitellogenesis via continuous activation of the AstC neu