ライフサイエンスコーパス: voltage-dependent channel

1 KCNE1 (MinK), producing a slowly activating voltage-dependent channel.

2 everely distorted measurements and recruited voltage-dependent channels.

3 micron) representing a high-density array of voltage-dependent channels.

4 through the opsin, rather than through other voltage-dependent channels.

5 E3) is essential for the specific control of voltage-dependent channel activation characteristics of

6 Corresponding to voltage-dependent channel activation, we observed a robu

7 ation and is not observed during ligand- and voltage-dependent channel activation.

8 hat reside within C99 significantly increase voltage-dependent channel activity beyond that induced b

9 trated sources of calcium are influx through voltage-dependent channels, and release of calcium from

10 odgkin-Katz (GHK) configuration that employs voltage-dependent channel binding functions for external

11 CNS physiology depend critically on powerful voltage-dependent channel block by external Mg(2+) (Mg(2

12 Voltage-dependent channel block by external Mg2+ (Mg2+(o

13 tification of their currents, which reflects voltage-dependent channel block by intracellular spermin

14 With the Woodhull model of voltage-dependent channel block, the values of Kd(0) wer

15 ty or at concentrations high enough to cause voltage-dependent channel block.

16 wn NMDAR antagonists are positively charged, voltage-dependent channel blockers.

17 specialized inactivation gate, as for other voltage-dependent channels, but by reclosure of the same

18 astrocytes occurs, at least in part, through voltage-dependent channels, but the exact mechanisms inv

19 Ca(2+) entry through voltage-dependent channels (Ca(V)3) enhances Ca(2+) rele

20 mitochondrial burden can be traced to L-type voltage-dependent channels (channels for which there are

21 452A nearly eliminated the effects of Rg3 on voltage-dependent channel gating but did not prevent the

22 ut not generally different from wild type in voltage-dependent channel gating.

23 of putative mechanisms underlying Ca2+- and voltage-dependent channel inactivation, and (b) close fi

24 currents by causing nearly complete loss of voltage-dependent channel inactivation.

25 alcium homeostasis modulator 1 (CALHM1) is a voltage-dependent channel involved in neuromodulation an

26 lving cytosolic calcium entry through L-type voltage-dependent channels is required for PACAP to evok

27 elated potassium channel (hERG, Kv11.1) is a voltage-dependent channel known for its role in repolari

28 Priming the stores with Ca2+ entry through voltage dependent channels modulated wave properties but

29 dependence on L-type Ca2+ channels and other voltage-dependent channels (Na+ and K+ channels) in the

30 ons remained negative to potentials at which voltage-dependent channels open.

31 ]i and tension are the entry of Ca2+ through voltage-dependent channels opened by depolarization or d

32 te how sensor coupling to the pore generates voltage-dependent channel opening, we solved the crystal

33 le impediments to trafficking and inhibiting voltage-dependent channel opening.

34 subunits on the cell surface and inhibiting voltage-dependent channel opening.

35 k calcium release from ER and influx through voltage-dependent channels prevented mitochondrial ROS a

36 In smooth muscle Ca(2)+ entry via voltage-dependent channels produces a uniform Ca(2)+ inc

37 as a result of multiple defects in time- and voltage-dependent channel properties.

38 t, and notch pathways, and ion-transport and voltage-dependent channel protein domains.

39 ubthreshold synaptic inputs are modulated by voltage-dependent channels, raising the possibility that

40 results establish the general principles of voltage-dependent channel structure in a biological memb

41 e common structural motif for members of the voltage-dependent channel superfamily.

42 The mechanism by which Ca(2+) entry through voltage-dependent channels supports both co-ordinated co

43 tion, part of which remained after all major voltage-dependent channels were blocked.

44 or changes in driving force occurred and no voltage-dependent channels were opened.

45 5.4 mmol/L K+; pipette, 140 mmol/L K+), two voltage-dependent channels were recorded with conductanc

46 nese hamster ovary cells produced Ca2+ - and voltage-dependent channels with macroscopic and unitary

47 wo structures provide atomic descriptions of voltage-dependent channels with unprecedented clarity.