1 associated with increases in the activity of
voltage operated Ca(2+) channels (VOCCs).
2 These data suggest that
voltage operated Ca(2+) channels, mitochondrial Ca(2+) a
3 nal action potentials are mediated solely by
voltage-operated Ca(2)(+) channel activation, and slower
4 ght closes cyclic nucleotide-gated (CNG) and
voltage-operated Ca(2+) -permeable channels in mammalian
5 terization of the entire family of planarian
voltage-operated Ca(2+) channel alpha subunits (Ca(v)alp
6 ed by cytosolic calcium and inhibited by the
voltage-operated Ca(2+) channel blocker nifedipine.
7 Because L-type
voltage-operated Ca(2+) channel inhibitors substantially
8 To determine whether L-type
voltage-operated Ca(2+) channels (L-VOCCs) are required
9 It is becoming increasingly clear that
voltage-operated Ca(2+) channels (VOCCs) play a fundamen
10 Our results indicate that
voltage-operated Ca(2+) channels can modulate oligodendr
11 n, we used a conditional knock-out mouse for
voltage-operated Ca(2+) channels in oligodendrocyte prog
12 luxes via K(+) channels, Cl(-) channels, and
voltage-operated Ca(2+) channels were studied by compari
13 Nifedipine, an inhibitor of
voltage-operated Ca(2+) channels, inhibits progesterone-
14 gether, store depletion causes activation of
voltage-operated Ca(2+) entry and CCE.
15 rizontal cells without affecting presynaptic
voltage-operated Ca(2+) entry.
16 These kinases modulate
voltage-operated Ca(2+) uptake in OPCs and participate i
17 This influx of Ca2+ was not affected by the
voltage-operated Ca2+ channel (VOCC) antagonists nicardi
18 se in intracellular Ca2+ that was blocked by
voltage-operated Ca2+ channel antagonists.
19 The
voltage-operated Ca2+ channel blockers nifedipine (1 mum
20 lcineurin (protein phosphatase 2B (PP2B) and
voltage-operated Ca2+ channels (VOCCs) in NG108-15 cells
21 Voltage-operated Ca2+ channels and protein kinases are n
22 These studies establish that T-type
voltage-operated Ca2+ channels are required for cell cyc
23 The regulation of non-
voltage-operated Ca2+ channels in the plasma membrane re
24 reflect clusters of RyRs closely coupled to
voltage-operated Ca2+ channels in the sarcolemma.
25 lator (BAPTA) or Cd2+, a specific blocker of
voltage-operated Ca2+ channels, abolished the ability of
26 were mostly determined by the activation of
voltage-operated Ca2+ channels.
27 ppear to couple to dihydropyridine-sensitive
voltage-operated Ca2+ channels.
28 t to elicit a secondary activation of L-type
voltage-operated Ca2+ entry.
29 e examined the role of store-, receptor- and
voltage-operated Ca2+ influx pathways in rat intrapulmon
30 mutational modification strongly suppresses
voltage-operated calcium (Ca(V)1.2) channels while activ
31 induces Ca(2+)i mobilization via the L-type
voltage-operated calcium channel and the inositol 1,4,5-
32 erogeneity in the distribution of functional
voltage-operated calcium channel subtypes in the neostri
33 of extracellular calcium (Ca(2)(+)), L-type
voltage-operated calcium channels (L-VOCCs), Rho kinase
34 Finally, although Ca2+ influx through
voltage-operated calcium channels does not appear to con
35 he relative involvement of N-, P- and Q-type
voltage-operated calcium channels in the control of stri
36 This suggests a role for
voltage-operated calcium channels in the habituation pro
37 mbranes carry neurotransmitter receptors and
voltage-operated channels that are still functional, eve
38 We studied the properties of a
voltage-operated Na+ conductance in descending vasa rect
39 We conclude that a TTX-sensitive
voltage-operated Na+ conductance, with properties simila
40 Therefore, the
voltage-operated sparks phenotype is specific to the RyR
41 RyR1-transfected muscle lacked
voltage-operated sparks.