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3 ring 20-minute frequency (0.25 Hz) and tidal volume-controlled breathing after intravenous injections
6 ation of 10 and 40 ppm to a test lung during volume-controlled (constant flow) and pressure-controlle
7 iated gas exchange with perflubron (n=10) or volume controlled continuous positive pressure breathing
9 al route improves survival time after severe volume-controlled hemorrhagic shock in rats without wors
11 rhagic shock models and partially realistic, volume-controlled hemorrhagic shock models to more reali
13 xpiratory pressure of 1 and 10 cm H2O, under volume-controlled mechanical ventilation in the settings
14 After injury, all animals were placed on volume-controlled mechanical ventilation to achieve PaO2
16 etric microfluidic DBS card were compared to volume-controlled pipetted DBS samples from the same fin
18 Only two (25%) of the eight animals in the volume controlled positive pressure breathing group were
19 ereas it remained unchanged over time in the volume controlled positive pressure breathing group.
21 IEPOX uptake by pure sulfate particles is a volume-controlled process, which results in particles wi
23 ntrolled ventilation (intervention group) or volume-controlled ventilation (control group) with ident
24 ressure-controlled ventilation compared with volume-controlled ventilation (nitric oxide concentratio
25 ciency of ventilation would be greatest with volume-controlled ventilation (VCV) compared with pressu
26 GI can be used either with pressure (PCV) or volume-controlled ventilation and continuously or only d
27 ndrome, E-cadherin expression was similar in volume-controlled ventilation and variable ventilation;
28 olled adaptive ventilation was compared with volume-controlled ventilation at the same levels of mean
29 layed was minimally altered by helium during volume-controlled ventilation but substantially decrease
30 rin expression in lung tissue was reduced in volume-controlled ventilation compared with nonventilate
31 ion and reduced lung elastance compared with volume-controlled ventilation in both acute respiratory
32 ry acute respiratory distress syndrome, only volume-controlled ventilation increased vascular cell ad
36 he time-controlled adaptive ventilation than volume-controlled ventilation with similar mean airway p
37 in time-controlled adaptive ventilation than volume-controlled ventilation with similar mean airway p
38 ion animals had bacteremia counts lower than volume-controlled ventilation with similar mean airway p
39 in time-controlled adaptive ventilation than volume-controlled ventilation with similar positive end-
40 ile time-controlled adaptive ventilation and volume-controlled ventilation with similar positive end-
41 ntilation)/positive end-expiratory pressure (volume-controlled ventilation) in a Pseudomonas aerugino
42 ncreased relative angiopoietin-1 expression (volume-controlled ventilation, 0.3 [0.2-0.5] vs variable
43 veolar damage (median [interquartile range]: volume-controlled ventilation, 12 [11-17] vs variable ve
44 1), and angiopoietin-2/angiopoietin-1 ratio (volume-controlled ventilation, 2.0 [1.3-2.1] vs variable
45 [8-10]; p < 0.01), interleukin-6 expression (volume-controlled ventilation, 21.5 [18.3-23.3] vs varia
46 dhesion molecule-1 messenger RNA expression (volume-controlled ventilation, 7.7 [5.7-18.6] vs nonvent
47 additional 14 animals were ventilated using volume-controlled ventilation, maintaining similar time-
48 e randomly assigned to receive conventional (volume-controlled ventilation, n = 6) or variable ventil
50 ntrolled adaptive ventilation, compared with volume-controlled ventilation, was associated with less
52 e-controlled ventilation: 14.5 to 130.5 ppm; volume-controlled ventilation: 21.6 to 104.7 ppm; nitric