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1 silent period was significantly prolonged in writer's cramp.
2 were converse and significantly different in writer's cramp.
3 anifest dystonias, including torticollis and writer's cramp.
4 during motor tasks that elicited dystonia or writer's cramp.
5 coherence in the patients with dystonia and writer's cramp.
6 dyskinesia (PD), cervical dystonia (CD) and writer's cramp.
7 ance has not been previously investigated in writer's cramp.
8 speech production tasks in 15 patients with writer's cramp and 15 matched healthy control subjects.
9 finger abduction movements in patients with writer's cramp and compared them with those of normal ag
11 as been implicated in the pathophysiology of writer's cramp and other primary dystonias, endogenous d
12 orted for DBS treatment of tardive dystonia, writer's cramp, cranial dystonia, myoclonus dystonia, an
16 e botulinum toxin is clinically effective in writer's cramp, it does not reverse the associated dysfu
18 Our voxel-based results also suggest that writer's cramp may be associated with reduced striatal d
22 ffected and unaffected hand in patients with writer's cramp showed significantly less reduction in 20
23 Twenty patients with torticollis, nine with writer's cramp, two with blepharospasm and 16 healthy co
24 umental practice, whereas many patients with writer's cramp (WC) have a history of average hand use.
26 fluence on the fine motor control network in writer's cramp, which could be artificially restored to
27 n healthy control subjects and patients with writer's cramp while they write a stereotyped word repet