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1 s also inhibit GS-1-B(4) from binding to the xenoantigen.
2 ly suppressed an evoked antibody response to xenoantigen.
3 es believed to be secreted in the absence of xenoantigens.
4 ting peripheral and intrathymic CD4 cells to xenoantigens.
5 onse to in vitro stimulation with guinea pig xenoantigens.
6 on both DTH and IgG antibodies to guinea pig xenoantigens.
7  genes that encode human Ab responses to pig xenoantigens.
8 rat monoclonal antibodies that recognize pig xenoantigens.
9 to react against the immunizing MHC class II xenoantigens.
10 eness to alloantigens and/or third-party pig xenoantigens.
11 e long-term immune response of recipients to xenoantigens.
12 and better control of the B-cell response to xenoantigens.
13              Long-term hyporesponsiveness to xenoantigen across both a concordant and discordant spec
14  are responsible for the production of major xenoantigens (aGal, Neu5Gc, Sda, and SLA-I) were sequent
15 ine cells devoid of three major carbohydrate xenoantigens, aGal, Neu5GC, and SDa (TKO) exhibit marked
16 identification of 2 further pig carbohydrate xenoantigens allowed the production of 'triple-knockout'
17 ral and elicited antibodies specific for pig xenoantigens, alpha-(1,3)-galactose (GAL) and N-glycolyl
18 ine cells devoid of three major carbohydrate xenoantigens, alphaGal, Neu5GC, and SDa (TKO) exhibit ma
19 le prepared from uninfected hamster tissues (xenoantigen and injection trauma control).
20 ymus donor and responded to nondonor porcine xenoantigens and alloantigens.
21 hat react with similar epitopes expressed on xenoantigens and bacteria may share structurally similar
22 aft recipients can be stimulated in vitro by xenoantigens and IL-2 to differentiate into highly react
23 be genetically engineered to eliminate three xenoantigens and to express nine human transgenes that e
24  for their capacity to respond to guinea pig xenoantigens and to lyse guinea pig target cells.
25 ve followed two major paths: deletion of pig xenoantigens, as well as insertion of "protective" human
26             Human CD4+ T cells can recognize xenoantigens by either a direct or indirect pathway.
27 ets are primarily CD4+ and recognize porcine xenoantigens by the indirect Ag pathway presentation.
28 lpha1,3-galactose (alpha1,3Gal) is the major xenoantigen causing hyperacute rejection in pig-to-human
29 a1,3Galbeta1,4GlcNAc-R), which are the major xenoantigens causing hyperacute rejection in pig-to-huma
30 nce to A/B-incompatible alloantigens and pig xenoantigens could be achieved in infant baboons.
31 he human cellular immune response to porcine xenoantigens, cytolytic T lymphocyte (CTL) cell lines we
32 t identified but is hypothesized to be a pig xenoantigen expressed on podocytes.
33 a sufficiently low level that any additional xenoantigens expressed on the cells can now be more easi
34       Presensitization of mice to guinea pig xenoantigens failed to increase the proportion of grafts
35                                  SLA-DQ is a xenoantigen for most patients.
36 al alpha(1-3)Gal epitope (xenograft antigen, xenoantigen) found on the cell surfaces of the donor org
37 pression of one of seven recently identified xenoantigens from the surface of pig aortic endothelial
38 on BHVs, particularly antibodies against the xenoantigens galactose-alpha1,3-galactose (alphaGal) and
39 re addition to the cultures, indicating that xenoantigens had to be processed in order to be recogniz
40  expression of all three of the known glycan xenoantigens has been deleted may be more challenging in
41               These results demonstrate that xenoantigen immunization can break tolerance to a self-A
42             To explore the potential role of xenoantigen immunization in cancer patients, we performe
43  alpha(1-3)-galactose (alpha-gal), the major xenoantigen in the pig to primate xenotransplant model.
44 0 pathways to inhibit the immune response to xenoantigen in the rat-to-mouse and pig-to-mouse models.
45 arked inhibition of the cellular response to xenoantigen in vivo and produced long-term acceptance of
46 tolerance to alloantigens, and in the future xenoantigens, in vivo is essential to progress in transp
47 ive PAECs recognized strain-specific porcine xenoantigens indirectly.
48 ptides that have been identified as allo- or xenoantigens is consistent with this hypothesis.
49                        T cell recognition of xenoantigens is likely to play a key role in rejection o
50                                 One of these xenoantigens is the swine major histocompatibility compl
51  cancer received two monthly vaccinations of xenoantigen-loaded dendritic cells with minimal treatmen
52  also suggest using IgGs lacking these major xenoantigens may improve safety and efficacy of ATG trea
53 s, before and after BAL exposure, recognized xenoantigens on PAECs with similar molecular weights, su
54 rum and Hu-PBL-SCID serum recognized similar xenoantigens on PI, indicating that Hu-PBL-SCID containe
55  presented here will add to our knowledge of xenoantigens on porcine red cells and be important for d
56 ns without the alphaGal epitope (nonalphaGal xenoantigens) on porcine erythrocytes using flow cytomet
57 lucose homeostasis in DKO pigs for two major xenoantigens paves the way to their use in (pre)clinical
58  in heterologous animal products can acquire xenoantigens, potentially limiting their utility.
59 ated that the clones are recognizing porcine xenoantigens presented by self-APCs.
60 ary-direct pathway for T cell activation via xenoantigen recognition/costimulation by endothelial-der
61 ession of Galalpha1,3-Gal epitope, the major xenoantigen recognized by human natural antibodies.
62 nce of self-APC, indicating that the primary xenoantigens recognized are peptides derived from SLA.
63 rmore, we demonstrate that the major porcine xenoantigens recognized are SLA class I molecules.
64                 Delineation of the major pig xenoantigens recognized by natural human xenoreactive an
65 alpha(1,3)-gal and N-glycolylneuraminic acid xenoantigens reduces human antibody binding to porcine p
66 s behind human T-cell recognition of porcine xenoantigens remain to be elucidated.
67 cNAc-R) has been identified as being a major xenoantigen responsible for hyperacute rejection, the re
68 tively adhered to PAEC and were activated by xenoantigen, resulting in highly efficient antigen prese
69 th alpha-galactosidase to cleave the primary xenoantigen-the alpha-Gal antigen.
70  phenotype and efficiently present allo- and xenoantigens to allogeneic T cells after co-culturing wi
71 tive immunogenicity of autoantigens, whereas xenoantigens, typically not presented during induction o
72  double knockout swine, lacking carbohydrate xenoantigens was already tested in nonhuman primates and
73 f pig aortic endothelial cell (PAEC) surface xenoantigens was analyzed by immunoprecipitation.
74                     Importantly, immunity to xenoantigens was only induced after xenotransplantation
75 ition receptors, and knock-down of the B4Gal xenoantigen were tested in various combinations.
76 on receptors, and knock-down of the beta4Gal xenoantigen were tested in various combinations.
77 ne responses of recipient mice to guinea pig xenoantigens were assessed.
78  directed at the gal carbohydrate or porcine xenoantigens were detected by enzyme-linked immunosorben
79  with GalT, but normal Ab responses to other xenoantigens were detected.
80 eras, but normal antibody responses to other xenoantigens were detected.
81                The identification of porcine xenoantigens whose recognition by human natural antibodi
82  IgM and IgG XNA from hu-PBL-SCID recognized xenoantigens with similar molecular mass as those recogn